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1 eded by long-duration waves of airway smooth muscle contraction.
2 on of the muscle action potential crucial to muscle contraction.
3 , reduces the local vasodilatory response to muscle contraction.
4 the fundamental force-generating elements of muscle contraction.
5 ular Ca(2+) release via RyR1 during skeletal muscle contraction.
6 sin light chain, which induces airway smooth muscle contraction.
7 functions such as synaptic transmission and muscle contraction.
8 olvement of ZIPK in the regulation of smooth muscle contraction.
9 ated the receptor, inhibiting nerve-mediated muscle contraction.
10 uencies (50-100 Hz) that would induce strong muscle contraction.
11 ominant-negative effect of mutant protein on muscle contraction.
12 tion of Mb (SmbO2) and PmbO2 kinetics during muscle contraction.
13 lentivirus-mediated shRNA attenuated smooth muscle contraction.
14 ty of only a few motor units active during a muscle contraction.
15 sarcoplasmic reticulum to initiate skeletal muscle contraction.
16 capable of detecting and modifying body wall muscle contraction.
17 molecule in the regulation of human cardiac muscle contraction.
18 function as a regulator of blood flow during muscle contraction.
19 rated exercise pressor reflex in response to muscle contraction.
20 ular signaling, which is required for proper muscle contraction.
21 ose transporter 4 in response to insulin and muscle contraction.
22 nderstanding cMyBP-C's modulation of cardiac muscle contraction.
23 gn receptor activity to circadian rhythm for muscle contraction.
24 ctivating RhoA and suppressing airway smooth muscle contraction.
25 AVD vary with the intensity and duration of muscle contraction.
26 protein that has a role in modulating smooth muscle contraction.
27 uscle fibers, and is critical for control of muscle contraction.
28 ble for reaction with DAF-FM during bouts of muscle contraction.
29 e thick filaments and a modulator of cardiac muscle contraction.
30 s system and are responsible for controlling muscle contraction.
31 oxia and oxidative stress, inflammation, and muscle contraction.
32 l alterations of epithelial cells and smooth muscle contraction.
33 f Ca(2+) from the sarcoplasmic reticulum and muscle contraction.
34 oint for thin filaments as they slide during muscle contraction.
35 ts that appear to be primarily influenced by muscle contraction.
36 linked to the detoxification process and to muscle contraction.
37 ulatory protein troponin switches on and off muscle contraction.
38 ivation of motor neurons controlling enteric muscle contraction.
39 a critical role in the regulation of smooth muscle contraction.
40 myocytes and determines the force of cardiac muscle contraction.
41 minating Ca(2+)-induced signaling such as in muscle contraction.
42 e of Ca(2+) from internal stores to initiate muscle contraction.
43 s actin and myosin interactions to influence muscle contraction.
44 albumin-mediated guinea pig bronchial smooth muscle contraction.
45 cTnC), plays an important role in regulating muscle contraction.
46 -HT1A receptors inhibit firing and, thereby, muscle contraction.
47 tores upon nerve impulse to trigger skeletal muscle contraction.
48 ion of thick filament length, such as smooth muscle contraction.
49 echanical stress-mediated decrease in smooth muscle contraction.
50 modelling of the artery, or increased smooth muscle contraction.
51 ate (CAS) have been shown to regulate smooth muscle contraction.
52 mally exceeds the minimum necessary to cause muscle contraction.
53 We also investigated the effects of evoked muscle contraction.
54 ium mobilization and efficient and effective muscle contraction.
55 with the intensity and duration of skeletal muscle contraction.
56 re dynamics, with important consequences for muscle contraction.
57 It is essential for controlling muscle contraction.
58 ysophosphatidic acid-induced vascular smooth muscle contraction.
59 s that reflect the intensity and duration of muscle contraction.
60 eracts with troponin I and initiates cardiac muscle contraction.
61 as cell motility, embryonic development, and muscle contraction.
62 a(2+)) release channel required for skeletal muscle contraction.
63 uring a myosin transgene known to facilitate muscle contraction.
64 lved in blood pressure regulation and smooth muscle contraction.
65 three times greater than those possible via muscle contraction.
66 s system and are responsible for controlling muscle contraction.
67 eraction with actin, a process that leads to muscle contraction.
68 d a particularly active cell associated with muscle contractions.
69 o electrically-induced intermittent hindlimb muscle contractions.
70 iffening of titin stretched during eccentric muscle contractions.
71 tumor tissue and the unintended induction of muscle contractions.
72 ary twisting postures arising from sustained muscle contractions.
73 with enhanced synaptic release and stronger muscle contractions.
74 y of spontaneous colonic longitudinal smooth muscle contractions.
75 ogether generate appropriate intensities for muscle contractions.
76 normal pacemaker function, but lack enteric muscle contractions.
77 aberrant patterning of peristaltic waves of muscle contractions.
78 of motoneuron recruitment to the strength of muscle contractions.
79 atment planning and the induction of intense muscle contractions.
80 ic (e.g., heel pad) mechanisms, or by active muscle contractions.
81 d generate situationally appropriate, graded muscle contractions.
82 n regulates the sensory feedback controlling muscle contractions.
83 rtical stimulations in motor cortex elicited muscle contractions.
84 arges reliably evoke contra-lateral hindlimb muscle contractions.
86 is characterized by a weakness in voluntary muscle contraction, a direct consequence of greatly redu
87 unified understanding of swimming, caused by muscle contractions ("active" swimming) or by forces imp
89 rall volume as it elongates, suggesting that muscle contraction against the tongue's fixed volume (i.
90 TMEM16A control neuronal signalling, smooth muscle contraction, airway and exocrine gland secretion,
91 leather or parchment, stresses comparable to muscle contraction already occur at small osmotic pressu
93 interneurons disrupted bilaterally symmetric muscle contraction amplitude, without affecting the timi
94 s, this study is focused on the dynamics and muscle contraction analysis to assess loads on bones and
95 precise control of neurotransmitter release, muscle contraction and cell excitability, among many oth
96 findings, authors suggest that both ciliary muscle contraction and ciliary body edema may play role
98 essure, eventually exacerbated by expiratory muscle contraction and dynamic hyperinflation, all incre
100 ificantly associated with G-protein-mediated muscle contraction and extracellular matrix remodeling.
101 es with potential regulatory roles in smooth muscle contraction and extracellular matrix-receptor int
102 r responsible for biological motions such as muscle contraction and intracellular cargo transport, fo
104 sarcoplasmic reticulum to initiate skeletal muscle contraction and is associated with muscle disease
105 nitiates functional hyperaemia upon skeletal muscle contraction and is attenuated during ageing via a
108 enes, activation of pathways associated with muscle contraction and physiology, and downregulation of
110 d to variable cytoplasmic strain produced by muscle contraction and relaxation, but their morphology
114 erms of estimation, the proteins involved in muscle contraction and structure were preferentially enz
115 how Tyr(1065) phosphorylation affects smooth muscle contraction and the conformation and cellular fun
116 l-wide release of Ca2+, which in turn causes muscle contraction and the generation of the mechanical
117 e Z-band will help us understand its role in muscle contraction and the role of these proteins in the
118 primary motor cortex (M1) directly regulates muscle contraction and thereby controls specific movemen
119 l mechanism for the control of airway smooth muscle contraction and thus are a critical factor in air
120 l mechanism for the control of airway smooth muscle contraction and thus are a critical factor in air
121 tion may identify agents that inhibit smooth muscle contraction and/or restrain or reverse obstructiv
124 creases the interval between systemic smooth muscle contractions and increases the rate of morphogene
125 r characterized by sustained or intermittent muscle contractions and its pathophysiological mechanism
126 ect to the spinal cord to control particular muscle contractions and movements have been found to dis
128 The H-FIRE protocol substantially reduces muscle contractions and the therapy can be delivered wit
130 responses, including dilatation to skeletal muscle contraction) and in relation to the spread of vas
131 al role in regulating actin dynamics, smooth muscle contraction, and airway hyperresponsiveness in as
133 e proliferator-activated receptor signaling, muscle contraction, and ion transport were already prese
134 es a highly conserved protein that regulates muscle contraction, and is a tumour-suppressor gene.
135 sin, the molecular motor that powers cardiac muscle contraction, and its accessory protein, cardiac m
136 ptor signaling, circadian exercise, striated muscle contraction, and lipid and carbohydrate oxidative
141 rocesses, such as rapid organelle transport, muscle contraction, and tension-sensitive anchoring.
142 sforming growth factor-beta, vascular smooth muscle contraction, and the hedgehog and Wnt signaling p
143 orphogenesis, the frequency of airway smooth muscle contraction, and the rate of developmental matura
144 hways, induce inhibition of the ileal smooth muscle contractions, and affect distinct physiological v
145 results suggest that the Mb dynamics during muscle contraction are contributing factors to faster VO
148 blood pressure responses to static hindlimb muscle contractions are greater in rats with femoral art
153 ment to understanding the connection between muscle contraction at the molecular and macro scale.
154 near nature of musculotendon systems, cyclic muscle contractions at the passive natural frequency of
155 he fact that single nonmuscle cell and whole-muscle contraction both follow a Hill-like force-velocit
156 osin cross-bridges is the molecular basis of muscle contraction, but generally accepted ideas about c
157 al circuits that direct precise sequences of muscle contraction, but the identity and organizational
158 cooperativity are likely coordinated during muscle contraction, but the relative contribution from e
159 several methods to measure cardiomyocyte and muscle contraction, but these require customized hardwar
160 ing involuntary cardiac muscle, Tm regulates muscle contraction by coupling Ca(2+) binding to Tn with
161 onstrate an anti-bone-resorptive activity of muscle contraction by ES that develops rapidly and is in
162 findings suggest that Plk1 regulates smooth muscle contraction by modulating vimentin phosphorylatio
163 e alpha2 subunit plays a key role in cardiac muscle contraction by regulating intracellular Ca(2+), w
165 activation of the motor neurons that induce muscle contraction can facilitate quantitative studies o
166 motor manifestations, primarily involuntary muscle contractions causing twisting movements and abnor
167 ical phenomena, such as synaptic plasticity, muscle contraction, cell cycle, and circadian rhythms.
168 are key players in cytoskeletal remodeling, muscle contraction, cell migration and differentiation p
169 more than 2,000 J/kg of specific work during muscle contraction, compared with just 40 J/kg for natur
170 ation of SER-2 facilitates ventral body wall muscle contraction, contributing to the tight ventral tu
171 nectin-dependent component of the integrated muscle contraction-dependent arteriolar vasodilatation i
173 n phosphorylation (RLC) necessary for smooth muscle contraction depends on the respective activities
176 nisms to release energy more rapidly than by muscle contraction directly, thus amplifying power outpu
181 ls generate electrical currents that control muscle contraction, encode neuronal information, and tri
182 at plays an important role in regulating the muscle contraction, especially in Ca(2+) activation.
185 of 1947 mRNAs encoding proteins involved in muscle contraction, focal adhesion, integrin, JAK/STAT,
186 eurologic disorder characterized by abnormal muscle contractions for which standard medical therapy i
187 lar system development and function, such as muscle contraction genes TNNI2, TNNT1, and CKM; contract
190 ) regulate many physiological functions like muscle contraction, hormone secretion, gene expression,
191 uxley's integrative approach to the study of muscle contraction; how he persevered throughout his car
194 M3-mAChR phosphorylation in bronchial smooth muscle contraction in health and in a disease state with
196 custs [7, 14-20]; serotonin, which regulates muscle contraction in locust oviducts [21]; and the FMRF
197 t data to show that Rac1 is activated during muscle contraction in murine and human skeletal muscle a
200 d in muscles and directly mediates sphincter muscle contraction in the defecation cycle in hermaphrod
203 rosophila heart muscle [22] and may regulate muscle contractions in the reproductive tract, if it is
205 showed a concentration-dependent decrease in muscle contraction, increase in heart rate, and accelera
207 equence decreased the dilatation to skeletal muscle contraction, indicating that there is a physiolog
208 cle weakness and increased susceptibility to muscle contraction-induced injury have not been extensiv
209 dentified distinct pathways linked to smooth muscle contraction, inflammatory cytokines, immune media
212 n of force generation during vascular smooth muscle contraction involves a rise in intracellular calc
216 ay hyper-responsiveness, and the strength of muscle contraction is determined by the frequency of osc
217 ay hyper-responsiveness, and the strength of muscle contraction is determined by the frequency of osc
219 Together, these findings suggest that smooth muscle contraction is mediated by the recruitment of bet
222 Myocardial depolarization leading to cardiac muscle contraction is reflected by the amplitude and dur
229 The actin-myosin system, responsible for muscle contraction, is also the force-generating element
230 Increases in plasma insulin and exercise/muscle contraction lead to recruitment of additional MVU
231 mplex and involves the mechanical effects of muscle contraction; local metabolic, red blood cell and
233 the muscular hydrostat, and, thus, intrinsic muscle contraction may simultaneously increase the lengt
234 The molecular motor myosin teams up to drive muscle contraction, membrane traffic, and cell division
235 oth oxidant cascades have complex effects on muscle contraction, metabolic function and tissue perfus
236 However, whether the change in PmbO2 during muscle contraction modulates mVO2 and whether the O2 rel
237 To support the high energy requirements of muscle contraction, myogenesis entails an increase in mi
239 nsport, olfaction, phototransduction, smooth muscle contraction, nociception, cell proliferation and
241 cus secretion, neuronal excitability, smooth muscle contraction, olfactory signal transduction, and c
242 the effects of in vivo exercise and ex vivo muscle contractions on Rac1 signaling and its regulatory
243 ix (ECM) deformation, which can be caused by muscle contraction or changes in hydrostatic pressure.
247 l activity can be transformed into sustained muscle contractions, particularly in systems with slow m
252 w a reversible reduction in the amplitude of muscle contractions, representing a surrogate marker for
253 s involved in metabolism, cardiac disorders, muscle contraction, reproduction, behavior, and signalin
255 Ca(2+) release channel required for skeletal muscle contraction; RyR1) from aged MCat mice was less o
258 ed during behavior in live moving worms, and muscle contraction strength as a result of this acidific
259 ograms that require multiple precisely timed muscle contractions, such as, for example, playing the p
260 mble motor neuron activity that induces fast muscle contraction, suggesting that eel high-voltage vol
262 eal a simple circuit of neurons controls the muscle contractions that allow fruit fly larvae to crawl
263 r characterized by involuntary and sustained muscle contractions that can lead to paralysis and abnor
264 rd injury (SCI) are debilitating involuntary muscle contractions that have been associated with incre
265 s set the origin and frequency of the smooth muscle contractions that propel wastes from the kidney t
266 is involved in regulation of vascular smooth muscle contraction through direct phosphorylation of LC2
267 basement membrane and transmit the force of muscle contraction through integrin adhesion complexes.
268 r about every 50 s, trigger rhythmic enteric muscle contractions through downstream GABAergic neurons
269 used a recently discovered drug to eliminate muscle contraction, thus allowing prolonged intracellula
272 niches, regulating processes that range from muscle contraction to acidification of intracellular org
274 specifically the M-wave, was analysed during muscle contraction to assess the ability of the tibialis
276 nging from endocrine disruption and impaired muscle contraction to effects on aquatic ecosystems.
277 PK is also a prerequisite for the ability of muscle contraction to increase insulin sensitivity.
280 problem, had impact far beyond the field of muscle contraction to the benefit of multiple fields of
281 Flexibility in the bilateral coordination of muscle contraction underpins variable locomotor movement
283 optic nerve ultimately drive iris-sphincter-muscle contraction via excitatory cholinergic parasympat
287 light chain-20, a key regulator of lymphatic muscle contraction, was observed in insulin-resistant LM
288 ur experiments demonstrated that coordinated muscle contraction waves are associated with asymmetric
289 ons as a second messenger in tracheal smooth muscle contraction, we used the criteria set out by Suth
290 dings in humans maintaining steady isometric muscle contractions, we found evidence that the cortex s
292 ning period, deoxygenated Mb kinetics during muscle contraction were measured using near-infrared spe
294 ntify a novel mechanism acting downstream of muscle contraction, where YAP activates JAG2 expression
296 light chain (RLC) phosphorylation for smooth muscle contraction with subsequent dephosphorylation for
297 ics were estimated by combining the model of muscle contraction with validated models of lower limb m
298 PCA sensory-motor neurons, to coordinate sex muscle contractions with initiation and continuation of
299 production, storage of sperm, and changes in muscle contraction within the reproductive tract (RT).
300 ) attenuated actin polymerization and smooth muscle contraction without affecting myosin light chain
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