ライフサイエンスコーパス: muscle damage

1 ytokines, including IL-6, to repair skeletal muscle damage.

2 in early events in the myogenic response to muscle damage.

3 of the vasoconstrictor response and eventual muscle damage.

4 exercise that causes an increase in skeletal muscle damage.

5 retained at 85% of the synaptic sites after muscle damage.

6 ells enter muscles in response to repetitive muscle damage.

7 iotoxin (CTX)-induced tibialis anterior (TA) muscle damage.

8 invasive method of detecting and quantifying muscle damage.

9 direct link between anti-FHL1 responses and muscle damage.

10 e removal of those with potential iatrogenic muscle damage.

11 ng that RARgamma agonist may oppose skeletal muscle damage.

12 iltration, indicating exacerbated dystrophic muscle damage.

13 howing that Tregs can directly contribute to muscle damage.

14 on partners) in myofibril assembly and after muscle damage.

15 e immunoreactivity for Xin was indicative of muscle damage.

16 -SRP and anti-HMGCR Abs could be involved in muscle damage.

17 al integrity and increased susceptibility to muscle damage.

18 lopment of muscle atrophy, in statin-induced muscle damage.

19 educed HGF levels and c-met activation after muscle damage.

20 ays responsible for atrogin-1 expression and muscle damage.

21 onnects the immune and nonimmune pathways of muscle damage.

22 te in Pompe myofibers and may cause profound muscle damage.

23 ty infiltration of the liver and evidence of muscle damage.

24 prolonged endplate currents, and consequent muscle damage.

25 been found to be upregulated by exercise or muscle damage.

26 or satellite cell activation following local muscle damage.

27 Our findings indicate that muscle damage affects the lineage choices of muscle SP c

28 controls, Akita mice demonstrated increased muscle damage after eccentric exercise along with a decl

29 y, dramatically prevented lovastatin-induced muscle damage and abrogated atrogin-1 induction both in

30 estoring muscle ERRgamma pathway ameliorated muscle damage and also prevented DMD hallmarks of postex

31 oprene units to protein targets cause statin muscle damage and atrogin-1 induction in cultured cells

32 ), occurs as a result of contraction-induced muscle damage and deficient muscular repair.

33 nonimmune mechanism responsible for skeletal muscle damage and dysfunction in autoimmune myositis.

34 D 2B and 2C, daily prednisone dosing reduced muscle damage and fibroinflammatory infiltration.

35 deficiency reduced quadriceps and diaphragm muscle damage and fibrosis at 14 wk but not at 6 mo, and

36 890F)/mdx showed a significant resistance to muscle damage and force loss following repeated eccentri

37 amma signaling in muscular dystrophy reduces muscle damage and improves motor performance by promotin

38 marked and significant reduction in cardiac muscle damage and infarct size.

39 hed protein biomarkers associated with heart muscle damage and point-of-care monitoring of both these

40 romosome that result in skeletal and cardiac muscle damage and premature death.

41 agic buildup may be responsible for skeletal muscle damage and prevent efficient trafficking of repla

42 can to the cytoplasm is sufficient to induce muscle damage and provides a new model of muscular dystr

43 ion-reambulation model was used for inducing muscle damage and recovery in the lower hindlimbs in mic

44 that can be used for imaging and quantifying muscle damage and recovery in vivo.

45 All morphant fish showed muscle damage and vascular abnormalities at day 1 post-f

46 The strong correlation between the degree of muscle damage and Xin immunoreactivity suggests that Xin

47 their varied etiologies, are associated with muscle damage and, often, other organ system involvement

48 channels leads to defective Ca2+ signaling, muscle damage, and impaired exercise capacity.

49 line diet develop hepatosteatosis, liver and muscle damage, and lymphocyte apoptosis.

50 indicating that MTR may be more sensitive to muscle damaged by denervation than conventional MRI.

51 es and transiently increases in regenerating muscles damaged by bupivacaine.

52 ral treatment with UDCA prevents gallbladder muscle damage caused by BDL, whereas oral treatment with

53 ndlimb was found to have a greater amount of muscle damage compared to that in the contralateral noni

54 ceptibility to eccentric contraction-induced muscle damage compared with mdx controls.

55 ted the elevation of inflammatory markers of muscle damage (creatine kinase activity, C-reactive prot

56 ewing the ECT complication, it appeared that muscle damage due to catatonic immobility led to acute h

57 Two cohorts of young adults-the Eccentric Muscle Damage (EMD; n = 156) cohort and the Functional S

58 e-induced microtrauma. kg(-1) . d(-1)) after muscle-damaging exercise (300 eccentric contractions).

59 fibers, implicating mild direct or indirect muscle damage from the WNV infection.

60 Muscle damage has been shown to enhance the contribution

61 roles of ER stress and autophagy in skeletal muscle damage have been explored in multiple muscle dise

62 lemmal nNOS leads to functional ischemia and muscle damage; however, the mechanism of nNOS subcellula

63 also prevented DMD hallmarks of postexercise muscle damage, hypoxia, and fatigue in mdx mice.

64 ced biochemical and histological evidence of muscle damage, improved muscle function and increased ex

65 vities diminished or enhanced (respectively) muscle damage in a dystrophy model.

66 markably, loss of Fnip1 profoundly mitigated muscle damage in a murine model of Duchenne muscular dys

67 lovastatin-induced atrogin-1 expression and muscle damage in cultured mouse myotubes and zebrafish c

68 ata demonstrate Xin as a useful biomarker of muscle damage in healthy individuals and in patients wit

69 muscle regeneration give rise that ischemic muscle damage in limb transplantation might be reversibl

70 st muscle susceptible to contraction-induced muscle damage in mdx mice.

71 AC4 in muscle fibers is sufficient to induce muscle damage in mice.

72 plasma in response to acute notexin-induced muscle damage in rats.

73 are robust and promising biomarkers of acute muscle damage in rats.

74 evated myoplasmic[Ca(2+)](rest), and present muscle damage in soleus with a strong sex bias.

75 on of IL-10 expression in mdx mice increased muscle damage in vivo and reduced mouse strength.

76 In contrast, ablation of IFN-gamma reduced muscle damage in vivo during the regenerative stage of t

77 ve of severe inflammation, coagulopathy, and muscle damage including less bacterial clearance, hypogl

78 in some Dysf(-/-) mice, indicating possible muscle damage induced by hypertrophy.

79 atrogin-1 may be a critical mediator of the muscle damage induced by statins.

80 Skeletal muscle damage is an often-occurring event.

81 Muscle damage is currently assessed through methods such

82 y of inflammatory cells and thereby increase muscle damage is unknown.

83 Significant muscle damage is very rare when statin therapy is used i

84 In skeletal muscle, DAMAGE is at the postsynaptic membrane and is as

85 Conversely, muscle damage leading to regeneration may promote some i

86 Two hours after cardiotoxin-induced muscle damage, local activin A protein expression increa

87 ggest that myalgias and relatively low-level muscle damage may occur in a substantial number of patie

88 Other muscle damage measures also showed a correlation with se

89 otassium homeostasis mechanisms may minimize muscle damage of myopathies due to certain RyR1 mutation

90 Muscle damage or disease leads to progressive weakness a

91 exercise lead to protection against striated muscle damage, oxidative stress and injury.

92 to muscle size) and histological markers of muscle damage (percentage of regenerating fibers and fib

93 Macrophages recruited at the site of sterile muscle damage play an essential role in the regeneration

94 low-choline diet, some people presented with muscle damage rather than liver damage; several effect a

95 h Pompe disease are associated with skeletal muscle damage, rather than acute myocardial injury.

96 = 0.6175, P = <0.0001) with the severity of muscle damage, regardless of myopathy type.

97 Although the mechanism of muscle damage remains to be fully elucidated, a number o

98 that cachexia was associated with a type of muscle damage resulting in activation of both satellite

99 Muscle damage results in rapid recruitment of eosinophil

100 GTP-binding proteins lead to statin-induced muscle damage since these molecules require modification

101 t of HSPs may provide protection against the muscle damage that occurs by a pathological increase in

102 e kinase activity, an established measure of muscle damage, to near-normal levels.

103 CK), CK-myocardial band levels, and skeletal muscle damage was assessed.

104 Renal and organ muscle damage was enhanced in PC(+/-) mice, as shown by

105 Confirmation of muscle damage was obtained using in vivo indocyanine gre

106 Using notexin-induced muscle damage, we have shown that regeneration is attenu

107 e diagnostic importance of CK in determining muscle damage, we tested the association of the variant

108 muscle force/endurance as well as increased muscle damage when compared to regular mdx mice.

109 ostmenopausal women developed fatty liver or muscle damage, whereas only 44% of premenopausal women d

110 d signs of organ dysfunction (fatty liver or muscle damage), while less than half of premenopausal wo

111 glycogen accumulation in skeletal muscle and muscle damage with exertion.

112 FER animals displayed progressive muscle damage with myofiber necrosis, internalized nucle