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1 ing pathway and miRNAs regulate the skeletal muscle development.
2 scriptome are known to play central roles in muscle development.
3 scribed player during cardiac and pharyngeal muscle development.
4 wn about the regulatory mechanisms of tongue muscle development.
5 l JAM family paralogues may also function in muscle development.
6 nes in the muscle precursors during forelimb muscle development.
7 s required for homeostasis during Drosophila muscle development.
8 s in decreased Wnt signaling and lung smooth-muscle development.
9 ulatory factor Myf5 plays important roles in muscle development.
10 s play an instructive role in guiding tongue muscle development.
11 llite cell activation and therefore skeletal muscle development.
12 transcriptional responses and impaired early muscle development.
13 targets of EBF activity with known roles in muscle development.
14 known function of EBF proteins in vertebrate muscle development.
15 f the earliest aspects of lung airway smooth muscle development.
16 e the earliest aspects of lung airway smooth muscle development.
17 tors that is important in heart and skeletal muscle development.
18 ue-tissue interaction during tongue skeletal muscle development.
19 factor availability during craniofacial and muscle development.
20 ell-cell interactions in regulating skeletal muscle development.
21 ethylase and promote adipogenesis and smooth muscle development.
22 tributing factor in early postnatal skeletal muscle development.
23 plasticity of titin splicing during skeletal muscle development.
24 nuclear co-repressor that regulates bone and muscle development.
25 strong deficits in notochord but not somitic muscle development.
26 rimarily in regulation of cardiac and smooth muscle development.
27 me, without abnormalities of vascular smooth muscle development.
28 e critical step in myofiber formation during muscle development.
29 pel and Even-skipped expression required for muscle development.
30 nd bomb2 (mib2) gene is a novel regulator of muscle development.
31 tically important for fast and slow skeletal muscle development.
32 l pathways that control cardiac and skeletal muscle development.
33 II activity resulted in inefficient skeletal muscle development.
34 s MyoD and myogenin are crucial for skeletal muscle development.
35 n factors are well-established regulators of muscle development.
36 tional regulation combine to direct skeletal muscle development.
37 ranscriptional processes during normal fetal muscle development.
38 urther suggesting that Maml1 is required for muscle development.
39 se to delete Myog before and after embryonic muscle development.
40 ulmonary septal defects, and abnormal smooth muscle development.
41 embryogenesis in the developing mesoderm for muscle development.
42 nscripts and proteins consistent with smooth muscle development.
43 into mice caused repression of gastrocnemius muscle development.
44 o be a major target of Pcs regulation during muscle development.
45 inent roles in regulating smooth and cardiac muscle development.
46 d-gestation period is important for skeletal muscle development.
47 cally in the developing mesoderm, throughout muscle development.
48 2C transcription and is essential for proper muscle development.
49 ecluding analysis of its effects on skeletal muscle development.
50 regulatory network involved in Drosophila's muscle development.
51 promoter during the late stages of embryonic muscle development.
52 ly growing rats may be requisite for optimal muscle development.
53 tion of the abdomen and defects in embryonic muscle development.
54 ssential roles in transcriptional control of muscle development.
55 ll behavior is likely specified during early muscle development.
56 eletal muscle repair but not during skeletal muscle development.
57 ct transcriptional complexes during skeletal muscle development.
58 yocardin is a critical determinant of smooth muscle development.
59 tiple muscle genes during cardiac and smooth muscle development.
60 ess also plays an important role in skeletal muscle development.
61 ications for the physiological regulation of muscle development.
62 myogenin at T87 is dispensable for skeletal muscle development.
63 at mice lacking CDO display delayed skeletal muscle development.
64 hanisms that regulate MEF2 expression during muscle development.
65 16 of the mouse embryo, a critical stage of muscle development.
66 mains to suggest possible interaction during muscle development.
67 nd fast twitch muscle fibers are crucial for muscle development.
68 nteracting genes in heart, blood vessel, and muscle development.
69 r insights into the merits and mechanisms of muscle development.
70 atin from executing its inhibitory effect on muscle development.
71 pression and altered function during cardiac muscle development.
72 is transiently expressed in vascular smooth muscle development.
73 these proteins, as well as genes involved in muscle development.
74 rchestrates the early phases of adult flight muscle development.
75 tion in mammals and plays important roles in muscle development.
76 showed that this gene is necessary for early muscle development.
77 in several stages of prenatal and postnatal muscle development.
78 DNA damage and apoptosis, impairing skeletal muscle development.
79 le maturation and maintenance than for early muscle development.
80 loping zebrafish to determine the effects on muscle development.
81 m centrosomes to the nuclear envelope during muscle development.
82 genes and plays a critical role in skeletal muscle development.
83 (G12D) expression at different stages during muscle development.
84 genous nhp2l1 in zebrafish disrupts skeletal muscle development.
85 r Myod to regulate fast-muscle, but not slow-muscle, development.
86 s, which, due to its unique mode of hypaxial muscle development, allows us to examine myoblast develo
87 al muscle would be valuable in understanding muscle development and a variety of muscle diseases that
88 nin plays a dual role as both a regulator of muscle development and an inducer of neurogenic atrophy.
89 ELF) family contribute to heart and skeletal muscle development and are implicated in myotonic dystro
90 genes that fall into heart, vasculature and muscle development and body growth categories, which pro
93 le precursor cells is a key step during limb muscle development and depends on the activity of PAX3 a
94 OB mothers, which may play a role in altered muscle development and development of insulin resistance
97 platform enables novel studies into skeletal muscle development and disease and chronic drug testing
98 function of MTMR14 and its specific role in muscle development and disease is much less well underst
99 r signaling axis that is critical for smooth muscle development and disease progression in the lung.
100 Given the role of Zasp proteins in mammalian muscle development and disease, our results have relevan
102 tubes such as is necessary for both skeletal muscle development and during regeneration, by remodelin
103 down-regulates a key splicing factor during muscle development and establishes a role for microRNAs
105 din die during the earliest stages of smooth muscle development and fail to express multiple smooth m
106 ynein-interacting proteins during Drosophila muscle development and found that several factors, inclu
109 ibution of the NAM-NAD(+) salvage pathway to muscle development and function using Caenorhabditis ele
110 ological roles for SOCE and ECCE in skeletal muscle development and function, as well as other curren
111 on as a novel mechanism for regulating heart muscle development and function, in particular the assem
112 he role of alpha7 integrin and dystrophin in muscle development and function, we generated integrin a
113 determinant of cardiac and skeletal striated muscle development and function, with misexpression freq
122 ggest unanticipated biological insights into muscle development and highlight new directions for furt
123 osphatidylinositol 3-phosphate metabolism in muscle development and homeostasis remain poorly underst
124 s of WNT/beta-catenin signaling molecules in muscle development and homeostasis, we used in vitro cul
125 We have applied this technique to skeletal muscle development and identified new genes with interes
126 own to regulate the formation of extraocular muscle development and in this report we show that its e
127 ypes define pathways essential for nerve and muscle development and interactions between these two ce
128 x3 plays an essential role in early skeletal muscle development and is a key component in alveolar rh
130 h that MYMK activity is necessary for normal muscle development and maintenance in humans, and expand
131 rst evidence that FLNc has a crucial role in muscle development and maintenance of muscle structural
133 on and lactation exhibited impaired skeletal muscle development and metabolic disorders normally asso
134 athways through which GHR regulates skeletal muscle development and modulates nutrient metabolism.
136 tently resulted in grossly abnormal skeletal muscle development and myofibrillar disorganization at t
137 ific phenotypes, such as vascular and smooth muscle development and neonatal epithelial and mast cell
140 results indicate that BIN1 and DNM2 regulate muscle development and organization, function through a
141 MRTFs in sarcomere formation during skeletal muscle development and point to the potential involvemen
143 any protein regulatory factors essential for muscle development and regeneration are degraded via the
145 otein abundance of MyoD, a master switch for muscle development and regeneration, but not that of its
154 s sperm motility (Fer-1), mammalian skeletal muscle development and repair (MYOF and dysferlin), and
157 play a synergistic effect on zebrafish early muscle development and some effects of GRK2 knockdown ca
158 myogenin is absolutely required for skeletal muscle development and survival until birth, but it is d
159 transcriptional network during ductal smooth muscle development and that disruption of this pathway m
160 omplexity in even this well-studied model of muscle development and that it can improve transcriptome
161 d that the receptors were present throughout muscle development and that, in adult muscle fibres, the
162 lude that Ci-MRF is required for larval tail muscle development and thus that an MRF-dependent myogen
163 s distinct from those occurring in embryonic muscle development and uncover an unsuspected non-cell a
164 essential for cardiac, skeletal, and smooth muscle development and uses its N-terminal TEA domain (T
165 strongly suggesting its importance in smooth muscle development and/or function, which could explain
166 a severe neonatal disorder in which skeletal muscle development and/or regeneration is impaired.
167 levels of genes implicated in cell adhesion, muscle development, and contraction, in structural const
168 Minion as a novel microprotein required for muscle development, and define a two-component programme
169 to categories related to the nervous system, muscle development, and especially to metabolic diseases
171 linking Fat1 to cancer, abnormal kidney and muscle development, and neuropsychiatric disease, this F
172 ndrogens drive sex differentiation, bone and muscle development, and promote growth of hormone-depend
174 orm a regulatory loop with Notch1 to repress muscle development, and this result expands our understa
175 ragm's muscle connective tissue and regulate muscle development, and we show that the striking migrat
178 beta1 integrins, suggesting that defects in muscle development are not primarily caused by defects i
180 s, which have crucial functions in embryonic muscle development, are assumed to have similar roles in
181 cise control of Mef2 levels is essential for muscle development as different Mef2 protein levels acti
182 the 6664 CNVs are enriched for immunity and muscle development, as well as head and limb morphology.
184 n signaling are capable of restricting heart muscle development at these relatively late stages of de
185 a new maturation step in vertebrate skeletal muscle development at which thick filament gene expressi
186 S deficiency contributes to misregulation of muscle development, blood flow, fatigue, inflammation an
188 e found that genes and gene sets involved in muscle development, bone synthesis, immunity, reproducti
189 data support a model in which Hhip regulates muscle development both by sequestering Hedgehog and by
190 ses differentiation of myoblasts in skeletal muscle development by attenuating the function of miR-30
191 ) has emerged as a key regulator of skeletal muscle development by governing distinct stages of myoge
192 le-specific miRNAs that are regulated during muscle development by the SRF transcription factor.
194 le differentiation and suggest that abnormal muscle development contributes to the manifestation of S
197 nscription factor, an essential regulator of muscle development, directly activates transcription of
199 HLH) transcription factors to drive skeletal muscle development during embryogenesis, but little is k
201 t ex-myomiR levels are elevated in perinatal muscle development, during the regenerative phase that f
203 e into myotubes and is required for skeletal muscle development, expression of muscle contractile pro
204 ased expression and reduced induction of key muscle development factors, such as MyoD and myogenin, w
206 n which Myog was deleted following embryonic muscle development had normal skeletal muscle, except fo
210 (G12D) expression at different stages during muscle development has profound effects on the ability o
212 Developmental mechanisms underlying somatic muscle development have mostly been studied in vertebrat
214 nsive genetic and genomic analyses to follow muscle development in a mouse model of BWS to dissect th
215 s been shown to be important for appropriate muscle development in chick limb buds; however, Fgfr4 nu
217 ant embryos were strikingly normal; however, muscle development in double morphants was severely disr
218 on factor Mef2 has well established roles in muscle development in Drosophila and in the differentiat
219 ption factor that is necessary for embryonic muscle development in Drosophila and vertebrates; howeve
221 s, signalling through this receptor promotes muscle development in growing embryos and angiogenesis i
222 skeletal muscle and supports normal skeletal muscle development in laminin-alpha2-deficient muscle bu
224 s also upregulated during perinatal skeletal muscle development in mice in vivo and that both miR-1 a
226 mises activation of WNT signaling and proper muscle development in the soft palate through tissue-tis
227 tiotemporal profiling of the early stages of muscle development in the zebrafish embryo, we identifie
231 he involvement of Trpc1 channels in skeletal muscle development in vitro and in vivo, and identify a
235 analysis showed that hnrpdl is important for muscle development in zebrafish, causing a myopathic phe
237 examined the roles of Brg1 and Brm in smooth muscle development, in vivo, through generation and anal
239 signals are essential for several aspects of muscle development, including myofibrillogenesis-the ter
240 is necessary for aspects of Xenopus skeletal muscle development, including somite organization, migra
245 the defects in lung development, esophageal muscle development is disrupted in Foxp2(-/-);Foxp1(+/-)
247 Although the genetic control of craniofacial muscle development is known to involve pathways distinct
248 ate muscle development, whereas invertebrate muscle development is largely independent of MRF functio
251 that a primary function of lbx1 in hypaxial muscle development is to repress myoD, allowing myoblast
253 verall, cofilin-2, although not critical for muscle development, is essential for muscle maintenance.
254 d that pomk function is necessary for normal muscle development, leading to locomotor dysfuction in t
256 that Hsp90alpha1 plays an important role in muscle development, likely through facilitating myosin f
257 like growth factors (IGFs) play key roles in muscle development, maintenance, and repair, but their m
259 osed to regulate various aspects of skeletal muscle development, metabolism, and mitochondrial functi
260 the alpha2/delta1 subunit may be crucial for muscle development, muscle repair and at times in which
262 dinated changes in H3K27ac and RNA implicate muscle development, neuronal regulation, and sensory res
265 r, genetic studies have shown that body wall muscle development occurs in the absence of HLH-1 activi
266 e indicates the total dependence of skeletal muscle development on the presence of RLC-f during embry
268 does not play a critical role in regulating muscle development or energy expenditure, but it is an i
271 fusion is an indispensable step for skeletal muscle development, postnatal growth, and regeneration.
273 ression pattern changes of genes involved in muscle development, protein degradation and biosynthesis
274 nd a positive relationship in expression for muscle development related genes, and a negative relatio
277 relative to CHC17 in membrane traffic during muscle development, repair, and at neuromuscular and myo
282 alpha7B is tightly regulated during skeletal muscle development suggests different and distinct roles
283 or knockout of myostatin, a key repressor of muscle development that can regulate Smad3 and mTOR sign
284 , possess a primary defect in bladder smooth muscle development that is apparent by embryonic day 15.
287 disease), and Rolling pebble (a regulator of muscle development); these findings are expected to faci
288 clude that although BAG3 is not required for muscle development, this co-chaperone appears to be crit
289 extend the role of Fn14 in wound repair and muscle development to involvement in the etiology of cac
290 Here we investigated the role of SMN in muscle development using muscle cell lines and primary m
291 initiate gene transcription during heart and muscle development, was reported to bind histones H3 and
293 ntial role of COUP-TFII in limb and skeletal muscle development, we bypassed the early embryonic leth
294 investigate the functions of SRF in skeletal muscle development, we conditionally deleted the Srf gen
295 es of Dicer and miRNAs in mammalian skeletal muscle development, we eliminated Dicer activity specifi
296 ression and the function of FGF4 in limb and muscle development, we generated mutant mice in which th
297 tter assess the role of kindlin-2 in cardiac muscle development, we used morpholinos to knockdown the
298 that talin 1 and 2 are crucial for skeletal muscle development, where they regulate myoblast fusion,
299 ctor (MRF) genes is essential for vertebrate muscle development, whereas invertebrate muscle developm
300 as neurogenesis, neuron differentiation and muscle development, which are increasingly disturbed in
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