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1 yncytial formation is a hallmark of skeletal muscle differentiation.
2 low fiber type specification during skeletal muscle differentiation.
3 ors and histone-modifying enzymes that shape muscle differentiation.
4 gulator Myocardin (Myocd) and induces smooth muscle differentiation.
5 ctor is important for all types of embryonic muscle differentiation.
6 erentiation with subsequent increased smooth muscle differentiation.
7 hich arrested during the process of skeletal muscle differentiation.
8 ulates myogenic transcription factors during muscle differentiation.
9 indicating that CTCF regulates MRF-mediated muscle differentiation.
10 ciated genes in ARMS cells, thereby blocking muscle differentiation.
11 s agents that mediate cytostasis and promote muscle differentiation.
12 ing M2c macrophage activation and modulating muscle differentiation.
13 overcome the inhibitory effects of MBNL3 on muscle differentiation.
14 ox transcription factor that is critical for muscle differentiation.
15 through induction of microRNAs during smooth muscle differentiation.
16 ying an important role in attenuating smooth muscle differentiation.
17 and two KLF3 isoforms are upregulated during muscle differentiation.
18 ription factor that plays essential roles in muscle differentiation.
19 to Igf2 promoters is also an early event in muscle differentiation.
20 erating stem cells and later promotes smooth muscle differentiation.
21 ity to study the systems-level properties of muscle differentiation.
22 fferentiation, and Hand2 potentiates cardiac muscle differentiation.
23 is, we discovered one compound that controls muscle differentiation.
24 s may serve as markers of striated or smooth muscle differentiation.
25 activation of the Myog promoter to initiate muscle differentiation.
26 grin subunit, is essential for morphological muscle differentiation.
27 ng FAK regulation of myogenin expression and muscle differentiation.
28 t role for Pbx proteins in promoting cardiac muscle differentiation.
29 ortant signaling pathways linked to skeletal muscle differentiation.
30 keleton is a critical early step in skeletal muscle differentiation.
31 hich contains PRDM16, of which loss promotes muscle differentiation.
32 n at random, we identify 49 involved in late muscle differentiation.
33 t beta-catenin, not TCF/LEF, is required for muscle differentiation.
34 ss of brown fat characteristics and promotes muscle differentiation.
35 fied direct Stat3 target genes important for muscle differentiation.
36 xpression throughout the program of skeletal muscle differentiation.
37 APC, have been suggested to govern terminal muscle differentiation.
38 ence that Akt1 and not Akt2 is essential for muscle differentiation.
39 teins, Skp2 and Myf5, for proteolysis during muscle differentiation.
40 of primitive mesenchyme exhibiting skeletal muscle differentiation.
41 t regulates myogenin expression and skeletal muscle differentiation.
42 ct of the Msx1 gene is a potent inhibitor of muscle differentiation.
43 PK pathway directly during processes such as muscle differentiation.
44 functions in myoblasts to modulate skeletal muscle differentiation.
45 Myoblast fusion is an essential step during muscle differentiation.
46 uning, clearance of neuronal debris, and for muscle differentiation.
47 wn of Cdh1 by siRNA significantly attenuates muscle differentiation.
48 ctions as a negative regulator of late-stage muscle differentiation.
49 es associated with proliferation or terminal muscle differentiation.
50 transcription of target genes essential for muscle differentiation.
51 ential for sustaining the earliest events in muscle differentiation.
52 Ig/fibronectin superfamily members linked to muscle differentiation.
53 ssion of the late genes that induce terminal muscle differentiation.
54 protein, may play a critical role in cardiac muscle differentiation.
55 ack loop in the transcriptional circuitry of muscle differentiation.
56 that repress gene expression during skeletal muscle differentiation.
57 on, and the Ski and Hes6 genes have roles in muscle differentiation.
58 on of genes involved in cardiac and skeletal muscle differentiation.
59 although one that is not required for smooth muscle differentiation.
60 own about how these proteins regulate smooth muscle differentiation.
61 yoblasts by RNA interference blocks skeletal muscle differentiation.
62 P-2 expression by RNA interference inhibited muscle differentiation.
63 rams of gene expression linked to growth and muscle differentiation.
64 or of cardiomyocyte hypertrophy and skeletal muscle differentiation.
65 nascent buds coincided with patterned smooth muscle differentiation.
66 t to promote notochord formation and prevent muscle differentiation.
67 ed for myogenic gene expression and skeletal muscle differentiation.
68 meostasis which is downregulated in skeletal muscle differentiation.
69 p, both of which are induced during skeletal muscle differentiation.
70 nvestigated the function of AUF1 in skeletal muscle differentiation.
71 3 ubiquitin ligase to repress human skeletal muscle differentiation.
72 sion between progenitor cell maintenance and muscle differentiation.
73 gesting a role for this protein during early muscle differentiation.
74 ction of myocardin, a key mediator of smooth muscle differentiation.
75 role in the regulation of the final stage of muscle differentiation.
76 d cells, whereas an increase is required for muscle differentiation.
77 ity and function as an inhibitor of skeletal muscle differentiation.
78 us Wnt signalling inhibitor for normal heart muscle differentiation.
79 ent, including mesenchymal growth and smooth muscle differentiation.
80 channels is a pivotal event during skeletal muscle differentiation.
81 of Map4k4 is essential for its inhibition of muscle differentiation.
82 ressive pediatric cancer exhibiting skeletal-muscle differentiation.
83 f DEFB103, a human defensin that can inhibit muscle differentiation.
84 oth muscle precursors is required for smooth muscle differentiation.
85 activity and consequently inhibited skeletal muscle differentiation.
86 key molecular link between O(2) and skeletal muscle differentiation.
87 s involved in Sharp-1-mediated inhibition of muscle differentiation.
88 as a negative regulator of mTOR activity and muscle differentiation.
89 atively regulates mTOR activity and skeletal muscle differentiation.
90 s slow fiber gene expression during skeletal muscle differentiation.
91 res in which H19 depletion causes precocious muscle differentiation, a phenotype recapitulated by let
92 minant-negative Set7 mutant impairs skeletal muscle differentiation, accompanied by a decrease in lev
93 hat Map4k4 is a novel suppressor of skeletal muscle differentiation, acting through a Myf5-dependent
94 do not display overt abnormalities in smooth muscle differentiation, although they show a significant
95 e expression during myofibroblast and smooth muscle differentiation, an event that is important for w
98 exit sites takes place early during skeletal muscle differentiation and completely remodels the secre
99 oblasts have the ability to undergo skeletal muscle differentiation and cross the blood vessel wall r
101 oop-helix transcription factor essential for muscle differentiation and enhances its binding to E box
102 ve formation, vascular development, skeletal muscle differentiation and fiber-type switching, and car
104 functions are reflected in defects in smooth muscle differentiation and function in mice with mutatio
107 ction between MyoD and Id1 in the process of muscle differentiation and have implications for the inv
108 K(v)7.4 plays a permissive role in skeletal muscle differentiation and highlight REST as a crucial t
110 roRNAs that are dynamically regulated during muscle differentiation and hypertrophy identified microR
111 T14 and Y15 in cell cycle quiescence during muscle differentiation and implicate two muscle differen
112 of cytoskeletal remodelling processes during muscle differentiation and in adult cardiomyocytes.
114 y regions of genes expressed during skeletal muscle differentiation and initiates chromatin remodelin
116 on factor, is a potent repressor of skeletal muscle differentiation and is dysregulated in muscle pat
119 etween transient and permanent repression of muscle differentiation and lineage commitment genes and
120 protein degradation and protein synthesis in muscle differentiation and metabolism under abnormal and
121 The spatial and temporal control of head muscle differentiation and morphogenesis are very site s
122 ther signal transduction pathways including: muscle differentiation and myopathies (MEF2C), tumor sup
124 SRF is an essential regulator of skeletal muscle differentiation and numerous components of the mu
125 tor (SRF) activates genes involved in smooth muscle differentiation and proliferation by recruiting m
126 lated genes were mainly involved in skeletal muscle differentiation and proliferation, including IGF2
127 nts define a biological function for Set7 in muscle differentiation and provide a molecular mechanism
128 fine a novel biological function for 4.1R in muscle differentiation and provide a molecular mechanism
130 itical trans-regulatory function in skeletal muscle differentiation and regeneration that is mediated
133 A expression and results in deficient smooth muscle differentiation and resultant aortic arch artery
134 ged1 in neural crest impairs vascular smooth muscle differentiation and results in aortic arch artery
135 w that FGF9 represses peribronchiolar smooth muscle differentiation and stimulates vascular developme
136 f SMN protein in the intrinsic regulation of muscle differentiation and suggest that abnormal muscle
137 icated TNFalpha-dependent miRNA circuitry in muscle differentiation and survival pathways in cancer.
140 ays as diverse as B-cell development, smooth muscle differentiation, and hepatic gluconeogenesis.
141 trophin (Utrn) is suppressed during skeletal muscle differentiation, and it is replaced at the sarcol
142 serum response factor, a master regulator of muscle differentiation, and negatively regulates its lev
143 ome-wide analysis of epigenetic marks during muscle differentiation, and strikingly, we observed a ne
144 cognised role in the earliest steps of heart muscle differentiation, and that partial complementation
146 modulate chromatin structure during skeletal muscle differentiation are still poorly understood.
147 control splicing patterns during neuron and muscle differentiation are the polypyrimidine tract-bind
148 ermed the Bruno body, and after the onset of muscle differentiation, Aret disperses in the nucleus.
150 mors that displayed an inability to complete muscle differentiation as determined by histological app
151 y in MyoD transcription delayed the onset of muscle differentiation, as assayed by expression of the
152 trates that TBP2 does not replace TBP during muscle differentiation, as previously proposed, with lim
153 ISPR-Cas9 genome editing and in vitro smooth muscle differentiation assay, we demonstrate that CD146
154 irk exerts its anti-apoptotic effects during muscle differentiation at least in part through effects
155 ltransferase Prmt5 was required for skeletal muscle differentiation at the early stages of myogenesis
156 that sfrp1 is not only able to promote heart muscle differentiation but is also required for the form
157 pression of c-Ski/SnoN also induces skeletal muscle differentiation, but how c-Ski/SnoN function in m
158 ng promoted proliferation and induced smooth muscle differentiation, but inhibited myocardial differe
159 in expression are induced as early events in muscle differentiation, but the responsible molecular me
160 f2 family of transcription factors regulates muscle differentiation, but the specific gene programs c
161 implicated in regulating vertebrate skeletal muscle differentiation, but their precise role(s) in viv
162 in differentiated muscle cells and promotes muscle differentiation by activating serum response fact
163 -regulatory modules (CRMs) in human skeletal muscle differentiation by combining myogenic TF binding
164 te MEF2 transcriptional function in skeletal muscle differentiation by depleting individual MEF2 prot
165 These data suggest that MBNL3 antagonizes muscle differentiation by disrupting Mef2 beta-exon spli
166 we demonstrated myocardin coordinate smooth muscle differentiation by inducing transcription of micr
167 sed in ureteral mesenchyme, regulates smooth muscle differentiation by maintaining Shh1 responsivenes
168 ed changes in the chromatin landscape during muscle differentiation by mapping the genome-wide locati
171 blast are utilized in the somites to promote muscle differentiation by serving as a source of Noggin.
173 enhancer that confers promoter regulation by muscle differentiation, changes in intracellular calcium
174 chanisms in mediating the complex program of muscle differentiation determined by the E2A proteins.
176 factors that play pivotal roles in striated muscle differentiation, development, and metabolism, in
177 l induction that propagates a wave of smooth muscle differentiation during aortic arch artery develop
178 ranscription factors play essential roles in muscle differentiation during embryogenesis, but their p
179 myoblasts, demonstrating that the process of muscle differentiation enhances the loss of normal ISCU
182 neurin plays an important regulatory role in muscle differentiation, fiber-type determination, hypert
183 are employed at different times of skeletal muscle differentiation for the purpose of facilitating A
185 18, which are important regulators of smooth muscle differentiation from the mesothelium and related
186 Here, we demonstrate that expression of the muscle differentiation gene Myocyte enhancer factor-2 (M
187 TGF-beta1 promotes the expression of smooth muscle differentiation genes through the inhibition of N
189 ve lipid whose signaling activities regulate muscle differentiation, homeostasis, and satellite cell
190 regulation during the initiation of skeletal muscle differentiation; however, there is less informati
191 lts in embryos that lack detectable bodywall muscle differentiation, identifying this trio as a set t
192 yme (micromass) and that Follistatin rescues muscle differentiation in a concentration-dependent mann
193 he absence of MyoD, was sufficient to induce muscle differentiation in a manner entirely dependent on
194 Moreover, Msx1 and H1b cooperate to inhibit muscle differentiation in cell culture and in Xenopus an
196 the GSK3beta inhibitor BIO induced skeletal muscle differentiation in human induced pluripotent stem
198 nstrate further that Hh ligand drives smooth muscle differentiation in primary intestinal mesenchyme
199 1 kinases to identify those that may repress muscle differentiation in proliferating myoblasts in the
200 rmal cardiovascular morphogenesis and smooth muscle differentiation in the aorta and pulmonary artery
201 Notch ligand Jagged1, a key driver of smooth muscle differentiation in the aortic arch arteries.
202 omponent Ezh2 is required to restrict smooth muscle differentiation in the developing lung mesotheliu
204 As NAM-NAD(+) biosynthesis also impacts muscle differentiation in vertebrates, we propose that s
205 muscle fibers, and knockdown of A20 impairs muscle differentiation in vitro, which suggests that A20
206 oss of brown fat characteristics and induces muscle differentiation in vivo through demethylation of
209 OBEC2 blocks TGFbeta signaling, and promotes muscle differentiation, in a mammalian myoblastic cell l
210 e decreased levels of proteins important for muscle differentiation including pRB, MyoD, desmin, and
212 ing muscle differentiation and implicate two muscle differentiation-induced microRNAs in the process.
214 Moreover, follistatin's ability to enhance muscle differentiation is at least partially due to its
221 demonstrate that neural crest-derived smooth muscle differentiation is specifically required for norm
222 a, a malignancy showing features of skeletal muscle differentiation, is the most common soft tissue s
223 vestigate the role of dysferlin during early muscle differentiation, its localization was studied at
224 ckdown of LncMyoD strongly inhibits terminal muscle differentiation, largely due to a failure to exit
225 LMOD3 was expressed from early stages of muscle differentiation; localized to actin thin filament
226 (Epha4, Pmp22, Nrp1, Gap43, Ndn) and smooth muscle differentiation (Madh3, Nrp1, Tagln Calponin, Cal
228 iption of a wide range of cardiac and smooth muscle differentiation markers in non-muscle cell types.
230 lly, revealed by the premature expression of muscle differentiation markers, and, especially, by a re
231 ng SMN inhibited the premature expression of muscle differentiation markers, corrected the cytoskelet
234 research groups have used them for skeletal muscle differentiation, most were based on gene over-exp
236 /protein expression of phenotypic markers of muscle differentiation, namely paired box 7 (satellite c
237 nown transcriptional repressors for terminal muscle differentiation, namely ZBTB38, Bhlhe41, and Id1.
238 espite the role played by Msx1 in inhibiting muscle differentiation, nothing is known of the mechanis
242 Bmp4 expression, which then enhanced smooth muscle differentiation of neural crest cells (NCCs) in t
245 d Hippo pathways converge to regulate smooth muscle differentiation of the neural crest, which is cru
246 f Mef2 in adult myoblasts leads to premature muscle differentiation, our results explain how and why
247 al differentiation genes and to override the muscle differentiation pathways, and they suggest that t
248 , coinciding with a reduction in both smooth muscle differentiation potential and TGFbeta1 responsive
249 isrupting this stereotyped pattern of smooth muscle differentiation prevents terminal bifurcation.
250 e a reduction in quantity of ureteral smooth muscle, differentiation proceeded without Smad4, produci
252 ed role for Myocd in repressing the skeletal muscle differentiation program and suggest that this tra
253 n, our results explain how and why the adult muscle differentiation program is attenuated prior to pu
255 e for migration and initiation of the smooth muscle differentiation program, however, it is essential
259 se factor (SRF) is a key regulator of smooth muscle differentiation, proliferation, and migration.
260 genes are direct transcriptional targets of muscle differentiation regulators including serum respon
268 ion of beta1 integrin at the onset of smooth muscle differentiation resulted in interrupted aortic ar
270 , bioartificial muscle engineering, skeletal muscle differentiation studies and for better understand
271 n myogenic differentiation using an in vitro muscle differentiation system based on C2C12 cells.
273 en associated with roles in immune response, muscle differentiation, testes development and DNA damag
274 devastating cancer with specific features of muscle differentiation that can result from mutational a
275 this system provides a novel model of smooth muscle differentiation that reliably recapitulates the p
279 egulator myocyte enhancer factor 2 (MEF2) in muscle differentiation, the interaction of MEF2 with cof
280 important ubiquitin E3 ligase that modulates muscle differentiation through coordinating cell cycle p
281 Pbx proteins are also necessary for cardiac muscle differentiation through interacting with Hand2.
282 tin E3 ligase UBR5 as an activator of smooth muscle differentiation through its ability to stabilize
283 ar axis, which functionally acts in skeletal muscle differentiation through the modulation of TrxR1 b
284 tive splicing regulator nPTB during skeletal muscle differentiation to control a potential network of
285 us Wnt ligand required for controlling heart muscle differentiation via canonical Wnt/beta-catenin si
286 e repair enzyme TDG as a repressor of smooth muscle differentiation via competing with SRF for bindin
288 role of GATA-6 in regulating vascular smooth muscle differentiation, we directly examined its ability
289 em with reporters of early and late skeletal muscle differentiation, we examined the influence of 2,4
290 criptional targets of MyoD prior to skeletal muscle differentiation, we have undertaken a transcripto
291 Due to their identified roles for terminal muscle differentiation, we hypothesize that the accumula
292 roRNAs (miRNAs) that participate in skeletal muscle differentiation were among the most differentiall
294 ent of MyoD, a central regulator of skeletal muscle differentiation, where they induce repressed chro
295 s markedly diminished from the very onset of muscle differentiation, whereas MyoD abundance was reduc
296 y member ponsin in nascent costameres during muscle differentiation, which is mediated by an interact
297 ional repressor of Myog, inhibiting skeletal muscle differentiation while activating SMC-specific gen
299 bility of Notch to promote or inhibit smooth muscle differentiation, while the physiological role for
300 sible and transcription factors that control muscle differentiation, yet we still have only rudimenta
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