ライフサイエンスコーパス: muscle fatigue

1 tion in conditions of weakness and premature muscle fatigue.

2 response to 50 Hz stimulation and increased muscle fatigue.

3 experimental studies into the mechanisms of muscle fatigue.

4 lactic acid accumulation are major causes of muscle fatigue.

5 D) is limited by both breathlessness and leg muscle fatigue.

6 equent prey movement by inducing involuntary muscle fatigue.

7 k of breathing-related changes in quadriceps muscle fatigue.

8 nce, membrane potentials, contractility, and muscle fatigue.

9 the thermal reaction norm of limpet adductor muscle fatigue.

10 ts when attempting to understand and predict muscle fatigue.

11 tressors, from dystrophy to heart failure to muscle fatigue.

12 nd can lead to muscle weakness and premature muscle fatigue.

13 ractions can be sustained after the onset of muscle fatigue.

14 the physiological impairments that can cause muscle fatigue.

15 there is no global mechanism responsible for muscle fatigue.

16 it task failure rather than those that cause muscle fatigue.

17 cidosis is being re-evaluated as a factor in muscle fatigue.

18 cal processes is causal to the use-dependent muscle fatigue.

19 n limb blood flow also accompany inspiratory muscle fatigue.

20 along with oxidative phosphorylation without muscle fatigue.

21 ometric contraction would be expected as the muscle fatigued.

22 Fast-timescale disturbances occur when muscles fatigue.

23 r exercise on: (1) exercise performance, (2) muscle fatigue, (3) capillarity, and (4) mitochondrial b

24 condition of fishlike body odor and chronic muscle fatigue, accompanied by elevated levels of the mu

25 of muscle has recently been shown to prevent muscle fatigue after exercise.

26 Muscle fatigue after ligation was related to the extent

27 be a major neural mechanism contributing to muscle fatigue and associated performance impairment.

28 CaMKII) and Ca(2+) store refilling to reduce muscle fatigue and atrophy.

29 accumulation are two of the major causes of muscle fatigue and exhaustion.

30 en with acute severe asthma with respiratory muscle fatigue and failure of medical treatment.

31 Shorter-term treatment protected against muscle fatigue and increased mdx hindlimb muscle force b

32 dvance our understanding of exercise-induced muscle fatigue and its role in muscle disease.

33 prey muscle preparations result in profound muscle fatigue and loss of contractile force.

34 By describing the effects of muscle fatigue and recovery in terms of two phenomenolog

35 reased central motor output and end-exercise muscle fatigue and reduced endurance performance.

36 Muscle fatigue and subsequent recruitment of poorly effi

37 the pivotal role of amino acid catabolism in muscle fatigue and type 2 diabetes pathogenesis.

38 ury, understanding the mechanisms underlying muscle fatigue and weakness has been the focus of much i

39 Ss) are increasingly recognized as causes of muscle fatigue and weakness.

40 O2 debt following exercise, a major cause of muscle fatigue, and a key factor in acidosis-induced tis

41 lactate] comparable to those observed during muscle fatigue, and accounted for this paradoxical conse

42 reliance on carbohydrate fuels, exaggerated muscle fatigue, and impaired endurance exercise capacity

43 f sarcoplasmic reticulum Ca(2+) stores, slow muscle fatigue, and increase running endurance without n

44 ase in cell pH) was thought to contribute to muscle fatigue by direct inhibition of the cross-bridge

45 work was to re-evaluate the role of H(+) in muscle fatigue by studying the effect of low pH (6.2) on

46 Muscle fatigue can be defined as the transient decrease

47 t or obese, populations in which respiratory muscle fatigue can be limiting.

48 a visuomotor rotation, or internal, such as muscle fatigue, can create a difference between the moto

49 with a known level of pre-existing locomotor muscle fatigue (DeltaQ(tw,pot) -20%) (PFT(67%)-TT).

50 with a known level of pre-existing locomotor muscle fatigue (DeltaQ(tw,pot) -36%) (PFT(83%)-TT).

51 arkedly enhances endurance and resistance to muscle fatigue, despite reducing muscle force.

52 ents limits CMD but also minimizes locomotor muscle fatigue development by stimulating adequate venti

53 nfluence on the rate of peripheral locomotor muscle fatigue development.

54 y," characterized by joint and muscle pains, muscle fatigue, difficulty lifting, and extremity parest

55 the working muscle in order that peripheral muscle fatigue does not exceed a critical threshold.

56 ns into the mechanisms underlying peripheral muscle fatigue due to energetic supply/demand mismatch a

57 action analysis to assess loads on bones and muscle fatigue during simulation of surgical interventio

58 r the central effects of fatiguing locomotor muscle fatigue exert an inhibitory influence on central

59 However, the magnitude of locomotor muscle fatigue following various TTs was not different (

60 unit fatigue as a tractable means to predict muscle fatigue for a variety of tasks and to illustrate

61 Increases in pre-existing locomotor muscle fatigue from control TT to PFT(83%)-TT resulted i

62 These data demonstrate in humans, that muscle fatigue, generated in the initial minutes of exer

63 Taken together, in the absence of locomotor muscle fatigue, group III/IV-mediated leg muscle afferen

64 he fatigue task, suggesting that significant muscle fatigue had occurred.

65 Muscle fatigue has been known to differentially affect t

66 own to impair contractile function and cause muscle fatigue in dystrophic (mdx) mice.

67 Despite greater muscle fatigue in individuals with spinal cord injury (S

68 between healthy controls and FM patients in muscle fatigue in response to exercise.

69 ring ISC, despite adjustment for the greater muscle fatigue in this condition (P < 0.001).

70 reat mild to moderate anxiety, insomnia, and muscle fatigue in Western countries, leading to its emer

71 Additionally, JTV519 improved skeletal muscle fatigue in WT and calstabin-2-/- mice with HF by

72 The purpose of this study was to examine muscle fatigue-induced resting-state interhemispheric mo

73 How muscle fatigue influences these two types of cell popula

74 Muscle fatigue is a temporary decline in the force and p

75 utput, so that the development of peripheral muscle fatigue is confined to a certain level.

76 sible relevance of these effects to skeletal muscle fatigue is considered.

77 paired by the absence of GLUT4, and onset of muscle fatigue is hastened.

78 The development of muscle fatigue is typically quantified as a decline in t

79 ry in the field, that lactic acidosis causes muscle fatigue, is unlikely to tell the whole story.

80 se due to dyspnea (n = 16) (as compared with muscle fatigue, n = 11) displayed weaker respiratory mus

81 gnal changes of the brain and muscles during muscle fatigue processes induced by maximal voluntary co

82 IRL) exacerbated exercise-induced quadriceps muscle fatigue (Q(tw) = -12 +/- 8% IRL-CTRL versus-20 +/

83 Surprisingly, however, while muscle fatigue reached a plateau, oxygen uptake continue

84 ce ( approximately 50%) and enhanced in situ muscle fatigue resistance ( approximately 30%) were obse

85 ification of training responses, and altered muscle fatigue resistance.

86 Diaphragm and forearm muscle fatigue showed very similar time-dependent effect

87 The model provides a possible mechanism for muscle fatigue, suggesting that at low but nonzero glyco

88 be the primary contributor to the premature muscle fatigue that is a hallmark of PVD.

89 ease the voltage delivered to prey, inducing muscle fatigue that turns challenging prey items into ea

90 wer for both the EEG and EMG activities with muscle fatigue, the fatigue weakens strength of brain-mu

91 With muscle fatigue, the locus of flow control resides in dis

92 Unexpectedly, muscle fatigue was unaffected by nNOS depletion, reveali

93 microcirculation and contribute to enhanced muscle fatigue, whereas formation of oxygen free radical

94 in brain and muscle signals during voluntary muscle fatigue, which may suggest weakening of functiona

95 ndgrip contractions that induced significant muscle fatigue, with resting state fMRI data collected b

96 We tested the hypothesis that muscle fatigue would attenuate vasodilatory responsivene

97 d oxidative ATP synthesis, and (2) ischaemic muscle fatigue would be related to the accumulation of i

98 We hypothesized that muscle fatigue would create a temporary "disrupted state