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1 to how myosin both generates and responds to muscle force.
2 terminant of physiological levels of passive muscle force.
3 n myosin structure, myosin biochemistry, and muscle force.
4 age-related decreases in DHPR number and in muscle force.
5 sistance to muscle fatigue, despite reducing muscle force.
6 s impairs regeneration, leading to decreased muscle force.
7 biochemistry, and significant enhancement of muscle force.
8 he dystrophin-associated protein complex and muscle force.
9 advances to explain how they could influence muscle force.
10 tor nerve activity served as a surrogate for muscle force.
11 enge, as well as greater decrement in biceps muscle force.
12 ce and those that assess evoked (stimulated) muscle force.
13 accounts for Ca(2+) sensitization of smooth muscle force.
14 eeded to reduce muscle pathology and improve muscle force.
15 by a dynamic balance of surface, tissue, and muscle forces.
16 ment of new IOLs designed to harness ciliary muscle forces.
17 uscles determines the magnitude of joint and muscle forces.
18 ATP hydrolysis (ATPase) reaction coupled to muscle force?.
19 macrophage density and a faster recovery in muscle force (20%), combined with an increase in muscle
20 otor deficit characterized by a reduction in muscle force, abnormal muscle contractile characteristic
24 muscle generated significantly less absolute muscle force and became more susceptible to contraction-
26 Arimoclomol significantly improved hindlimb muscle force and contractile characteristics, rescued mo
27 w diet, and it determines the maintenance of muscle force and exercise performance upon a HFD regimen
30 keletal-troponin complex to calcium improves muscle force and grip strength immediately after adminis
31 survived at least 18 months, and had normal muscle force and intracellular organization of muscle fi
32 tic stage of disease, resulting in increased muscle force and motor unit survival and a significant i
33 st cancer, caused remarkable improvements of muscle force and of diaphragm and cardiac structure in t
34 els are expressed on ASM and regulate smooth muscle force and offer a novel target for therapeutic re
35 n (SA), a mechanical property that increases muscle force and oscillatory power generation, is not kn
36 low-level dystrophin expression, we compared muscle force and pathology in mdx3cv and mdx4cv mice.
37 ponin activator that may be used to increase muscle force and power in conditions of muscle weakness.
38 osed with CK-2066260 show increased hindlimb muscle force and power in response to submaximal rates o
39 atment ameliorated histopathology, increased muscle force and protected against eccentric contraction
42 the mean number of attached bridges, depress muscle force and stiffness, and increase force-length hy
43 thetized, tracheotomized rat in which tongue muscle force and the neural drive to the protrudor and r
44 el with the user's calf muscles, off-loading muscle force and thereby reducing the metabolic energy c
46 Ross et al. is not only due to variations in muscle forces and cranial morphology, but also due to va
47 ed to simulate MU firing rates and isometric muscle forces and, to that model, we added fatigue-relat
48 and organ masses, muscle histology, in vitro muscle force, and creatine kinase levels were measured.
49 amping properties that may aid or oppose the muscle force; and the environment produces reaction forc
50 and grip is a common task during which high muscle forces are sustained, especially at the proximal
54 gested to cause a progressive decline in the muscle force at which motor units are recruited during r
56 del of the skull that can be used to predict muscle forces, bite forces, and joint reaction forces wo
57 d utrophin levels, may help maintain minimal muscle force but not arrest muscle degeneration or necro
58 cycle and contributed almost elastically to muscle force, but the rapidly cycling cross-bridge distr
59 st muscle fatigue and increased mdx hindlimb muscle force by 40%, a value comparable to current dystr
60 n an isometric task, where joint torques and muscle forces can be straightforwardly computed from lim
66 with confidence when model input parameters (muscle forces, detailed material properties) and/or outp
69 and rate of demembranated (skinned) cardiac muscle force development by exchanging native cardiac tr
70 S; an adapter protein associated with smooth muscle force development) from cytoskeletal vimentin.
79 study tested the hypothesis that masticatory muscle forces exerted during static biting are consisten
80 timal length), the steady-state value of the muscle force, F, approximates the isometric force, the m
82 coupling deficits, we compared the papillary muscle force generated by electrically stimulated versus
83 es a myopathy characterized by reductions in muscle force-generating capacity, atrophy (loss of muscl
84 -I, there were significant increases both in muscle force generation and cross-sectional area at all
86 ance were independent of central command and muscle force generation, were not activated in anticipat
92 les, taking into consideration surrogates of muscle force [ie, muscle cross-sectional area (MCSA) and
93 or, improved muscle relaxation and increased muscle force in HSALR mice without affecting splicing.
95 that CK-2017357 increases the production of muscle force in situ at sub-maximal nerve stimulation ra
102 erefore, quantitative assessment of skeletal muscle force is important for diagnosis of intensive car
105 alis; in particular, motoneuronal inputs and muscle force levels are chosen to approximately achieve
108 tor activity, histopathology, and individual muscle force measurements of mdx and mdx((5)cv) mice.
110 x (M1): relatively low-level parameters like muscle force, or more abstract parameters like handpath?
111 tates of a key behavior produce asymmetry in muscle forces, passive joint forces can be coadapted to
112 mpt)) leading to a hypermuscular yet reduced muscle-force phenotype was compared to that in wild-type
113 orylation plays a prominent role in skeletal muscle force potentiation of fast-twitch type IIb but no
114 tal issue in locomotion is to understand how muscle forcing produces apparently complex deformation k
118 icle-shortening velocity at the time of peak muscle force production increased with walking speed, im
120 zed expression of NMJ-related genes, in situ muscle force production, and clearance of glycogen in co
121 e model regarding the possibility of maximal muscle force production, and suggest that only 97% of th
124 cted tissue and modify AChR mRNA expression, muscle force production, motor endplate area, and innerv
127 taking into account the state dependence of muscle-force production and multijoint mechanics, I show
128 stress we predict working to balancing side muscle force ratios, peak bite forces, and joint reactio
129 assive muscle fire in direct relationship to muscle force-related variables, rather than length-relat
131 stimating or measuring the joint torques and muscle forces that underlie movements made by biological
132 factors regulate and/or maintain extraocular muscle force through a rapid mechanism that appears to i
134 rom contraction-induced injury and corrected muscle force to the same level as that observed in contr
136 dynamic parameters of jaw function including muscle force, torque, effective mechanical advantage, ja
139 wisdom, is based in part on studies in which muscle force was shown to decline more rapidly when stim
140 week after injection, muscle morphology and muscle force were compared between the IGF-treated and c
141 n, systolic blood pressure, and LV papillary muscle force while LV end-diastolic and systolic volume
142 ing, and calculations of how quickly passive muscle force would slow limb movement as limb size varie
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