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1 asciitis precedes and frequently accompanies muscle necrosis.
2 acrophages changes between the stage of peak muscle necrosis (4 weeks of age) and muscle regeneration
3 ic area) and severe (>5% mean necrotic area) muscle necrosis, an area under the receiver operating ch
4 ated macrophages to promote inflammation and muscle necrosis and in skeletal muscle fibers to limit r
5 ologically, there was evidence of widespread muscle necrosis and regeneration, fiber splitting, and r
6 tendinous strain injury that help to prevent muscle necrosis and retain the function of necessary mus
7 arrying marker genes that are unable to halt muscle necrosis and the difficulty of stable transfer of
8 razolium staining to determine the amount of muscle necrosis and the location of muscle protection.
9 (MD) is a disease characterized by skeletal muscle necrosis and the progressive accumulation of fibr
10 absence of dystrophin; calpastatin prevents muscle necrosis; and nitric oxide synthase prevents infl
12 (calpain) activity in dystrophic muscle and muscle necrosis, but have not tested whether calpain act
13 ing myocarditis, myocardial and red skeletal muscle necrosis, correlate with the intensity of the inf
14 muscle regenerative capacity and preventing muscle necrosis could be an effective treatment for the
16 tly was not due to differences in degrees of muscle necrosis, hemolysis, acute renal heme loading, or
17 ve membrane resealing in skeletal muscle and muscle necrosis; however, the function of dysferlin in t
18 in and the biochemical mechanisms leading to muscle necrosis in Becker muscular dystrophy are still u
19 m oxidative damage, and its absence leads to muscle necrosis in response to injury in Stra13-deficien
21 py for DMD may hold promise for ameliorating muscle necrosis, inflammation, and fibrosis by inhibitin
25 nicity was greater in patients who developed muscle necrosis (n = 15) than in those who did not (8.2%
26 istopathologic findings demonstrated limited muscle necrosis, reduced microvascular thrombosis, and e
28 ll muscular dystrophies are characterized by muscle necrosis that overtakes the regenerative capacity
29 odels for muscular dystrophy, showed ongoing muscle necrosis with age, a hallmark of the human diseas
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