ライフサイエンスコーパス: muscle pain

1 target for potential drug therapies to treat muscle pain.

2 y systemic symptoms such as fever, bone, and muscle pain.

3 els of peripheral neuropathy to evaluate for muscle pain.

4 nd transient, mild lower-extremity edema and muscle pain.

5 ot appear to modulate acute exercise-induced muscle pain.

6 nd was free of muscle weakness, and bone and muscle pain.

7 y accepted as a causal mechanism for chronic muscle pain.

8 d fever (temperature >38.5 degrees C), 7 had muscle pain, 6 had sinusitis, and 6 had biopsy evidence

9 (1832 [25.4%]), fatigue (1361 [18.9%]), and muscle pain (942 [13.1%]) were the most commonly reporte

10 al underlying reasons for the comorbidity of muscle pain and altered sympathetic reflexes in disease

11 usible that this finding partly explains the muscle pain and exercise intolerance that many patients

12 development of alternative therapeutics for muscle pain and may be especially relevant to pain cause

13 Muscle pain and weakness are the cardinal symptoms and o

14 Recovery occurred within 24 hrs, although muscle pain and weakness persisted for 2 months after st

15 wn statin-like lowering of LDL-C without the muscle pain and weakness responsible for discontinuation

16 the frequency of less-serious events such as muscle pain and weakness, which may affect 1% to 5% of p

17 derate levels of experimental sustained deep muscle pain, and a baseline, control state.

18 A prevalent side effect of statin therapy is muscle pain, and yet the basic mechanism behind it remai

19 Many clinicians believe that statins cause muscle pain, but this has not been observed in clinical

20 le of the cAMP pathway in a model of chronic muscle pain by assessing associated behavioral changes a

21 resolve within a few days, chronic joint and muscle pain can be long lasting.

22 was found at the ankle and on the arm during muscle pain compared to the control condition.

23 e TA muscle were found during saline-induced muscle pain compared to the infusion of isotonic saline.

24 ns in blood flow/oxygenation and a number of muscle pain disorders are based in problems of periphera

25 central command modulates the perception of muscle pain during exercise.

26 nemius muscle strongly enhanced acid-induced muscle pain in mice that was abolished by genetic inacti

27 bubble gum for 6 min) increases masticatory muscle pain in patients, but not in asymptomatic control

28 males and seven females, with no history of muscle pain in the masticatory system participated in th

29 ), headache (in 25.1% and 31.9%, vs. 16.9%), muscle pain (in 22.3% and 26.9%, vs. 13.3%), feverishnes

30 t study was to determine if exercise-induced muscle pain is modulated by central neural mechanisms (i

31 ults support the suggestion that neuropathic muscle pain may be a greater clinical problem than gener

32 ce for the involvement of Vi in craniofacial muscle pain mechanisms.

33 Evidence of muscle disease was indicated by muscle pain, muscle weakness and wasting, significant fa

34 -myo-neuropathy," characterized by joint and muscle pains, muscle fatigue, difficulty lifting, and ex

35 s, the placebo-corrected incidences of minor muscle pain, myopathy (with significant elevations in cr

36 The effect of experimental muscle pain on the amplitude and velocity sensitivity of

37 fects of the statin-are myopathy (defined as muscle pain or weakness combined with large increases in

38 Myopathy--muscle pain or weakness with blood creatine kinase level

39 py may cause symptomatic adverse events (eg, muscle pain or weakness) in up to about 50-100 patients

40 ay result in widespread deep hyperalgesia in muscle pain patients.

41 Ratings of muscle pain perception (MPP) and perceived exertion (RPE

42 with symptoms that include severe joint and muscle pain, rashes, and fever, as well as prolonged per

43 afferent function, leading to modulation of muscle pain-related behaviors and EPRs after BAO.

44 ll described in skeletal muscle, neuropathic muscle pain remains to be clarified.

45 Nonetheless, the role of SP in muscle pain remains unknown.

46 ponse pattern may be associated with chronic muscle pain states.

47 were neutropenia (38 [35%] of 110 patients), muscle pain (ten [9%]), rash (eight [7%]), cough, dyspno

48 The ability of muscle pain to generate somatosensory sensibility change

49 The inhibitory mechanism during muscle pain was shown to be effective for the deep tissu

50 Because chewing-induced masticatory muscle pain was significantly greater in female control

51 Mild cold exacerbation and significant muscle pain were equally common features in dominant and

52 hyperalgesia, and susceptibility to chronic muscle pain, which is unveiled by exposure to stress in

53 sh, fever episodes, arthralgia, and bone and muscle pain with cryopyrin-associated periodic syndromes

54 purposes, such as the fever, skin diseases, muscle pains, wounds and stomach problems.