ライフサイエンスコーパス: muscle relaxation

1 on and the consequent MLCP activation during muscle relaxation.

2 -activated K+ channels and results in smooth muscle relaxation.

3 s, suggesting decreased cGMP-mediated smooth muscle relaxation.

4 tes chloride channels on the muscle to cause muscle relaxation.

5 oxicity and Ca(2+) extrusion during skeletal muscle relaxation.

6 omyotonia, an exercise-induced impairment of muscle relaxation.

7 n relief from bronchospasm via airway smooth muscle relaxation.

8 ) (PGE(2)) production and bronchiolar smooth muscle relaxation.

9 dothelium is integral to coordinating smooth muscle relaxation.

10 tching might limit the rate of fast skeletal muscle relaxation.

11 unknown mechanism, possibly involving smooth muscle relaxation.

12 helix relieves SERCA inhibition, initiating muscle relaxation.

13 es not always accompany cGMP-mediated smooth muscle relaxation.

14 yed colonic emptying, and decreased circular muscle relaxation.

15 pnotics and opioids, promotes intraoperative muscle relaxation.

16 e decrease in MLC phosphorylation and smooth muscle relaxation.

17 en of the sarcoplasmic reticulum, initiating muscle relaxation.

18 ork together to modulate the rate of cardiac muscle relaxation.

19 vity and induces MLC20 dephosphorylation and muscle relaxation.

20 pparent Ca(2+) affinity and thereby enabling muscle relaxation.

21 osin phosphatase, leading to vascular smooth muscle relaxation.

22 the sensitivity to 8-Br-cGMP-mediated smooth muscle relaxation.

23 ha-Tm) have been shown to cause slow cardiac muscle relaxation.

24 cogen metabolism, cell-cycle progression and muscle relaxation.

25 rotein that regulates PP1 function in smooth muscle relaxation.

26 gested to be required for NO-mediated smooth muscle relaxation.

27 myosin light chain (MLC) and vascular smooth muscle relaxation.

28 fectively controls the development of smooth muscle relaxation.

29 band thereby preventing misfolding events on muscle relaxation.

30 tein kinase I suggested a pathway for smooth muscle relaxation.

31 ing soluble guanylyl cyclase to cause smooth muscle relaxation.

32 ormance in an animal model of slowed cardiac muscle relaxation.

33 ng cascade that ultimately transduces smooth muscle relaxation, a significant "spare receptor" pool h

34 1 gene (CLCN1), is characterized by delayed muscle relaxation after contraction.

35 the endothelium coordinates vascular smooth muscle relaxation along resistance arteries during blood

36 hosphatase is the primary effector of smooth muscle relaxation and a target of signaling pathways tha

37 olinoleate [LNO2]) that can stimulate smooth muscle relaxation and block platelet activation by eithe

38 EDTA recovered its ability to contribute to muscle relaxation and Ca2+ sequestration at its Ca2+ off

39 Like PA, EDTA's contribution to muscle relaxation and Ca2+ sequestration was more clearl

40 ns are a general mechanism both for inducing muscle relaxation and for switching off myosin II-based

41 omyopathy, characterized by impaired cardiac muscle relaxation and force development.

42 ems such as neurotransmitter release, smooth muscle relaxation and frequency tuning of auditory hair

43 f signaling through GAR-3 inhibit pharyngeal muscle relaxation and impair feeding--but do not block m

44 Rapamycin, an mTORC1 inhibitor, improved muscle relaxation and increased muscle force in HSALR mi

45 Muscle relaxation and inhibitory neurotransmission are r

46 -ATPase of skeletal muscle, is essential for muscle relaxation and maintenance of low resting Ca(2+)

47 n cyclic nucleotide-mediated vascular smooth muscle relaxation and may play a role in the increased p

48 lates such physiological processes as smooth muscle relaxation and neuronal survival.

49 in animals controls processes such as smooth muscle relaxation and neurotransmission by activation of

50 ells, may be associated with vascular smooth muscle relaxation and peripheral blood flow increase in

51 ascular homeostasis via regulation of smooth muscle relaxation and platelet aggregation.

52 oponin-I, and C-protein, causing accelerated muscle relaxation and reduced myofilament sensitivity to

53 Reactivity to endothelial-independent smooth muscle relaxation and subsequent vessel dilation was sim

54 however, in the regulation of uterine smooth muscle relaxation and that of other smooth muscles and c

55 tor for limiting NO-mediated vascular smooth muscle relaxation and tissue survival following ischemic

56 fter binding nitric oxide, leading to smooth muscle relaxation and vasodilation.

57 enhances cardiac contractility, accelerates muscle relaxation, and amplifies the inotropic and lusit

58 en limiting side effects including sedation, muscle relaxation, and ataxia.

59 t, PAR(2)-mediated PGE(2) production, smooth muscle relaxation, and decreased baseline airway resista

60 d, but did not eliminate, NO release, smooth muscle relaxation, and hyperpolarization.

61 roendosecretion, visual transduction, smooth muscle relaxation, and microbial killing.

62 ts of non-linear contractile responses, slow muscle relaxation, and neuromodulation.

63 intubation and procedures requiring profound muscle relaxation, and to minimize the amounts of anesth

64 Furthermore, the prolongation of muscle relaxation appeared to correlate with the levels

65 lays important roles in both vascular smooth muscle relaxation as well as prevention of blood coagula

66 R), plays important roles in vascular smooth muscle relaxation as well as the prevention of platelet

67 In rats, ocinaplon produces significant muscle relaxation, ataxia, and sedation only at doses >2

68 suggesting that the effects of NO on smooth muscle relaxation, blood pressure regulation and inhibit

69 sed for controlling the depth of anesthesia, muscle relaxation, blood pressure, intravascular volume,

70 locking agents (NMBAs) induce dose-dependent muscle relaxation, but their effects vary widely between

71 Smooth muscle relaxation by acetylcholine and a number of other

72 ated by cAMP (Epac), induces vascular smooth muscle relaxation by increasing the activity of ryanodin

73 asmic reticulum Ca(2+)-ATPase SERCA promotes muscle relaxation by pumping calcium ions from the cytop

74 hibit SERCA, the membrane pump that controls muscle relaxation by regulating Ca(2+) uptake into the s

75 Smooth muscle relaxation can also be elicited by inhibitors of

76 We report further that oviduct muscle relaxation can be induced by activating OA neuron

77 Patients experience delayed muscle relaxation causing functionally limiting stiffnes

78 rates of LC(20) dephosphorylation and smooth muscle relaxation compared with LC(20) phosphorylated ex

79 reinforce and temporally fine-tune striated muscle relaxation-contraction cycles.

80 cellular Ca(2+) concentration that generates muscle relaxation/contraction cycles.

81 hat selected measures of muscle strength and muscle relaxation correlate with changes in clinical sta

82 and the sensitivity to cGMP-mediated smooth muscle relaxation correlates with the relative expressio

83 ears that the mutant behavior is caused by a muscle relaxation defect due to the impairment of Ca2+ r

84 Although it is impossible to improve muscle relaxation (defined as absence of electrical acti

85 s lead to Brody disease, an exercise-induced muscle relaxation disorder, zebrafish accordion mutants

86 Nitric oxide (NO) acts as a smooth muscle relaxation factor and plays a crucial role in mai

87 Optimal muscle relaxation for ambulatory surgery results from a

88 The rate-limiting step of cardiac muscle relaxation has been proposed to reside in the myo

89 he principle stimulator of cavernosal smooth muscle relaxation, however, the inhibition of vasoconstr

90 appears to be a suitable agent for providing muscle relaxation in critically ill patients.

91 this series mediate very long-lasting smooth muscle relaxation in guinea pig tracheal strips.

92 nthase in regulating the NO-dependent smooth muscle relaxation in human penile corpus cavernosum tiss

93 inhibitory junctional potentials (IJPs) and muscle relaxation in mammalian gastrointestinal (GI) mus

94 tly, 6-PBC 24 also reversed diazepam-induced muscle relaxation in mice.

95 PKG subsequently causes smooth muscle relaxation in part via activation of myosin light

96 Atracurium is sometimes used for muscle relaxation in patients undergoing mechanical vent

97 in vascular homeostasis by mediating smooth muscle relaxation in response to nitric oxide, but littl

98 Smooth muscle relaxation in response to NO signaling is due, in

99 gnificant enhancement of NO-dependent smooth muscle relaxation in this tissue.

100 revents strong binding to actin and promotes muscle relaxation in vitro and in vivo.

101 , these cyclodextrin derivatives reverse the muscle relaxation induced by rocuronium in vitro and in

102 ability to bind Ca2+ and facilitate skeletal muscle relaxation is limited by its Mg2+ off-rate.

103 Muscle relaxation is triggered by the dephosphorylation

104 The aim of this study was to investigate the muscle relaxation of human gallbladders with cholesterol

105 The mechanism underlying smooth muscle relaxations of cerebral arteries in response to n

106 The effects of smooth muscle relaxation on arterial wall mechanics are controv

107 Peptides that cause muscle relaxation or contraction or that modulate electr

108 ontributing to feedback inhibition of smooth muscle relaxation or other processes.

109 Central to the muscle relaxation phase is a dynamic membrane protein co

110 Current models postulate that during muscle relaxation, phosphatases other than MLCP dephosph

111 Defective cardiac muscle relaxation plays a causal role in heart failure.

112 ractions, coupled with nitric oxide-mediated muscle relaxation, promote intestinal transit and parasi

113 in differentiated C2C12 slows the timing of muscle relaxation, promotes nuclear localization of calc

114 nberger technique was used, with respiratory muscle relaxation provided by brief manual ventilation.

115 ion may influence a proportion of the smooth muscle relaxation that occurs in asthma.

116 c oxide (NO) synthesis and subsequent smooth muscle relaxation, the signaling pathways downstream of

117 a myosin-blocking state, aberrantly favoring muscle relaxation, thus mimicking the low-Ca(2+) effect

118 Muscle relaxation time was much more prolonged than was

119 f cytosolic calcium and consequent prolonged muscle relaxation times.

120 Vascular smooth-muscle relaxation to diethylamine was enhanced in endoth

121 me-in-bed restrictions; n = 25), progressive muscle relaxation training (RT; n = 25), or a quasi-dese

122 target protein, HSP20, which mediates smooth muscle relaxation via actin depolymerization.

123 Muscle relaxation was commonly used, with 40% of the cen

124 Smooth muscle relaxation was complete in both groups.

125 Anesthesia and muscle relaxation were maintained continuously throughou

126 lcium to the sarcoplasmic reticulum to allow muscle relaxation, whereas PLB inhibits cardiac SERCA un

127 thelium-independent coronary vascular smooth muscle relaxation, whereas the abnormal response to cold

128 augments beta-agonist-mediated airway smooth muscle relaxation, while augmenting beta-agonist-stimula

129 der baseline conditions and following smooth muscle relaxation with nitroglycerin (NTG).

130 Smooth muscle relaxation with NTG increases isobaric compliance

131 of HSP20 on Ser16 may have a role in smooth muscle relaxation without MLC dephosphorylation.

132 crease in [cGMP], can induce arterial smooth muscle relaxation without proportional reduction in myos