ライフサイエンスコーパス: muscle stiffness

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1 scle action potentials (myotonia), producing muscle stiffness.

2 ormal cardiac output by sustaining force and muscle stiffness.

3 scle action potentials (myotonia), producing muscle stiffness.

4 accompanied by elevated titin-based passive muscle stiffness.

5 The rate of relief from muscle stiffness after 12 weeks was almost twice as high

6 nd thin filaments, and gives rise to passive muscle stiffness; an important determinant of diastolic

7 element and its deficiency leads to loss of muscle stiffness and aberrant mechanotransduction in ske

8 in cable number is accompanied by increased muscle stiffness and an increase in the number of collag

9 per motor neuron syndrome and often leads to muscle stiffness and disability.

10 ff person syndrome (SPS) is characterized by muscle stiffness and gait difficulties.

11 As the activation progressed, the muscle stiffness and phase angle steadily increased; the

12 rgic) neurotransmission is believed to cause muscle stiffness and spasms.

13 sient adverse events such as increased pain, muscle stiffness, and headache were reported 50% to 67%

14 reduced body size, evoked tremor, seizures, muscle stiffness, and morbidity by postnatal day 21.

15 both the enhanced excitability of myotonia (muscle stiffness due to repetitive discharges) and the i

16 ce, F, approximates the isometric force, the muscle stiffness, E, is large, and the muscle hysteresiv

17 e (CRS) measuring patient reported change in muscle stiffness from baseline.

18 tion of EDL and SOL indicated an increase in muscle stiffness in KO animals.

19 ority of CE over placebo in the treatment of muscle stiffness in MS.

20 Muscle stiffness in stiff-person syndrome (SPS) is produ

21 e does not occur, although during shortening muscle stiffness is reduced compared to the isometric st

22 displays three important characteristics: 1) muscle stiffness leads force during force development; 2

23 In patients, muscle stiffness lessens with exercise, a change known a

24 e contraction, the frequency response of the muscle stiffness of single beta-escin permeabilized smoo

25 e tense LR seems inconsistent with intrinsic muscle stiffness or diffuse orbital fat pressure but sug

26 identified in families with either myotonia (muscle stiffness) or periodic paralysis, or both.

27 l with eight components addressing symptoms (muscle stiffness, pain and discomfort and muscle spasms,

28 t hypercontractile phenotype with congenital muscle stiffness, rather than weakness, and respiratory

29 Except for global muscle stiffness, results of neurologic examination are

30 associated with chronic symptoms, including muscle stiffness, spasms, pain and insomnia.

31 Multiple sclerosis is associated with muscle stiffness, spasms, pain, and tremor.

32 putative roles of orbital fat and intrinsic muscle stiffness suggested to be alternatives to connect

33 e producing myotonic discharges (observed as muscle stiffness) that are worsened by elevated K+ level

34 amplitude required to cause active force or muscle stiffness to fall by half, or hysteresivity to do

35 amplitude required to cause active force or muscle stiffness to fall by half, or hysteresivity to do

36 Muscle stiffness was estimated during pCa 6.0 activation

37 However, titin-based muscle stiffness was reduced in the mice that expressed

38 rom these hearts (n=8) revealed that passive muscle stiffness was significantly reduced in patients w

39 Muscle stiffness was the most prominent symptom overall,

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