1              Mice lacking Kcnc3 also exhibit muscle twitches.

2  argue has formal similarity to the skeletal muscle twitch, albeit manifest over a much longer time s

3   One sciatic nerve was stimulated to induce muscle twitch, and the other nerve remained unstimulated

4                                  Heartbeats, muscle twitches, and lightning-fast thoughts are all man

5 so nontoxic in vivo, had no effect on evoked muscle twitch, but was able to cleave syntaxin.

6  have been of the motor cortex where a focal muscle twitch can be produced, called the motor-evoked p

7 ABA/glycine release, resulted in exaggerated muscle twitches during REM sleep that were similar to th

8 tonia, taking the form of exaggerated phasic muscle twitches, during REM sleep.

9 y triggers hundreds of thousands of skeletal muscle twitches each day; sensory feedback from the resu

10 4AP (1-2 mg/kg) increased nerve-evoked whole muscle twitch force and electromyograms (EMG) to a great

11 toxic to mice, had no effect on nerve-evoked muscle twitch in vitro, and was not able to cleave synta

12 enting with 2 months of diffuse, involuntary muscle twitching in the absence of myasthenic symptoms,

13            TMS of motor cortex can produce a muscle twitch or block movement; TMS of occipital cortex

14 subtle convulsive movements such as rhythmic muscle twitches or tonic eye deviation).

15 roduced early defects in the rate of cardiac muscle twitch relaxation and ventricular torsion.

16                    Furthermore, nerve-evoked muscle twitch tension was reduced.

17 tic transmission and an increase in skeletal muscle twitch tension.

18                            In addition, slow muscle twitch, tetanus tension, and susceptibility to in

19 eduction of ACh release that compromised the muscle twitch triggered by the nerve stimulation.

20 uration curves for electrical stimulation of muscle twitches were measured for each group and chronax