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1 for the detection of white, black and brown mustard).
2 -activated prodrug of bromo-isophosphoramide mustard.
3 sicaceae species with the exception of white mustard.
4 ding fish, poultry, meats, whole grains, and mustard.
5 at M(2) D103N at high concentrations of the mustard.
6 emical weapon agents (CWAs) sarin and sulfur mustard.
7 mising tool for in-field detection of sulfur mustard.
8 h as acrolein in smoke or isothiocyanates in mustard.
9 era stricta (Brassicaceae), a perennial wild mustard.
10 , cauliflower, lettuce, celery, spinach, and mustard.
11 2) s(-1) and at 110-220 mumol m(-2) s(-1) in mustard.
12 loped to detect and quantify the presence of mustard.
13 otes displayed resistance to the most potent mustards.
14 in dehiscence-associated fruit structures in mustards.
15 itumor agents such as cisplatin and nitrogen mustards.
16 tional cross-linking agents such as nitrogen mustards.
17 all series are prodrugs of bis-iodo nitrogen mustards.
21 dition, an alkylating derivative, quinacrine mustard, affinity labeled a subset of the substituted cy
22 potent than the corresponding phosphoramide mustards against V-79 Chinese hamster lung fibroblasts i
23 f our study was to evaluate the potential of mustard AITC to induce thermogenesis (primary outcome) a
30 evented receptor alkylation by acetylcholine mustard, although modest alkylation still occurred at M(
31 blocked by atropine methylbromide and 4-DAMP mustard, an M(3) muscarinic receptor selective antagonis
35 ic receptors to identify where acetylcholine mustard and 4-[(2-bromoethyl)methyl-amino]-2-butynyl-N-(
36 to allow the detection of 1 ppm black/brown mustard and 50 ppm white mustard and celery in raw and b
37 hanism of action of nitrobenzylphosphoramide mustard and aziridinyl nitrobenzamide prodrugs, compound
38 f 1 ppm black/brown mustard and 50 ppm white mustard and celery in raw and brewed sausages with a pro
40 ausages containing defined concentrations of mustard and celery showed that the triplex assay is appl
42 xtremely toxic chemical warfare agent sulfur mustard and classified, respectively, into schedule 3.B.
43 content of total phenolics in nutmeg, white mustard and coriander seed oils extracted with chlorofor
44 te mustard, singleplex assay for black/brown mustard and duplex assay for the detection of white, bla
46 ergoes metabolic activation to phosphoramide mustard and nornitrogen mustard (NOR) which alkylate the
52 All the 6 surgical centers that performed Mustard and Senning operations in Sweden and Denmark ide
54 ment with bis-electrophiles such as nitrogen mustards and cisplatin is the N7 position of guanine, bu
56 lating agents, such as bifunctional nitrogen mustards and cisplatins, generate interstrand DNA cross-
58 s' distributions, Alliaria petiolata (garlic mustard) and Berberis thunbergii (Japanese barberry), ea
59 ica seeds (canola, Indian mustard, and white mustard) and in their hydraulically pressed seed meals,
60 hree mustard species (white, black and brown mustard) and three celery varieties (celery roots, celer
61 its native promoter, is expressed in Indian mustard, and transgenic pcs lines have been compared wit
62 ysis of Se in Brassica seeds (canola, Indian mustard, and white mustard) and in their hydraulically p
63 mitantly generated with the isophosphoramide mustard; and chloroacetaldehyde, a neurotoxic and nephro
64 tine, which contains a benzoic acid nitrogen mustard appended to the minor groove DNA-binding natural
66 ; 9 of 11 subjects received the uncapsulated mustard as the final intervention because this could not
67 administration of "super" doses of nitrogen mustard, autologous bone marrow was infused into 2 patie
69 tructures of three CEES-bound complexes, the mustard binds through the sulfur atom and lies along the
70 sis of the very toxic chemical warfare agent mustard (bis(2-chloroethyl)sulfide) in the environment a
72 nvironmental changes in the clade of Buckler mustard (Biscutelleae): a mesopolyploidy event from the
77 in food: white mustard (Sinapis alba), black mustard (Brassica nigra) and brown mustard (Brassica jun
78 he simultaneous detection of traces of black mustard (Brassica nigra) and brown mustard (Brassica jun
79 rbivory), on glucosinolate concentrations of mustard (Brassica nigra) and collard (B. oleracea var. a
81 3,5-difluorophenol and -aniline (4)-nitrogen mustards by the enzyme carboxypeptidase G2 (CPG2) in gen
82 No effective antidote exists, and sulfur mustard can be fairly easily produced in large quantity.
85 s for the monofunctional binding of nitrogen mustard class of anticancer drugs to purine bases of DNA
89 larger mustard group of the dinitrobenzamide mustard compound SN 23862 forces the prodrug to bind at
91 nd turmeric can be ignored because in common mustard containing foodstuffs these biological species a
100 bility of two commercial mustard ELISA kits (Mustard ELISA Kit-specific and Mustard ELISA Kit-total)
101 d ELISA kits (Mustard ELISA Kit-specific and Mustard ELISA Kit-total) and three in-house developed re
102 compares the applicability of two commercial mustard ELISA kits (Mustard ELISA Kit-specific and Musta
103 ress responses of Brassica nigra (wild black mustard) exposed consecutively to O3 and the specialist
104 We show that toxicity resulting from topical mustard exposure is mediated in part by initiating exagg
105 dent origins of rosette flowering within the mustard family and attempted to evaluate the extent to w
107 Eight of these were benzoic acid L-glutamate mustards for evaluation as prodrugs and the other eight
108 eterogeneous catalyst for the oxidation of a mustard gas analogue, 2-chloroethyl ethyl sulfide (CEES)
117 Sixteen novel polyfluorinated benzoic acid mustards have been synthesized for use in gene-directed
118 imethyl methylphosphonate (DMMP)) as well as mustard (HD) in both liquid and gas phases at ambient te
119 er than 95% decomposition of adsorbed sulfur mustard (HD), sarin, and VX was achieved at ambient temp
120 n of the chemical warfare agent (CWA) sulfur mustard (HD, bis(2-chlororethyl) sulfide) and a range (c
123 nthesized a panel of model unhooked nitrogen mustard ICLs to systematically investigate how the state
124 es, with a 75% to 83% lower dose of nitrogen mustard in addition to omission of procarbazine and melp
125 owed the detection of white, black and brown mustard in brewed model sausages down to a concentration
130 el sausages containing white and black/brown mustard in the range from 1 to 50 ppm indicate that both
131 ystander effects of nitrobenzylphosphoramide mustards in NTR-expressing cells suggest that they could
132 uffs differing in their labelling concerning mustard, in one sample mustard was detected with both EL
133 omly selected for the placebo and capsulated mustard intervention; 9 of 11 subjects received the unca
140 oxic alkylating agent bromo-isophosphoramide mustard, is preferentially activated in hypoxic conditio
141 tes from low-dose melphalan (L-phenylalanine mustard (L-PAM))-treated MOPC-315 tumor bearers led to I
143 cessing of a substrate containing a nitrogen mustard-like ICL two nucleotides in the duplex region be
144 -37-7), consisting of a DNA-damaging aniline mustard linked to an androgen receptor (AR) ligand, is k
146 ntional crosslinking agents such as nitrogen mustard, melphalan or cisplatin which bind in the DNA ma
148 ecies suitable for in-the-field application: mustard, miscanthus, and 16 willow species and cultivars
149 quencies, we found that cisplatin-, nitrogen mustard-, mitomycin-, and carmustine-induced DNA adducts
150 nterstrand 5'-GNC-3' cross-links by nitrogen mustards, modify the electrostatics of the major groove
151 This genetic locus, which we have named mustard (mtd), contains a LysM domain, often involved in
154 ion to phosphoramide mustard and nornitrogen mustard (NOR) which alkylate the N-7 position of guanine
155 on of capsaicin (1 microm; 8 of 9 LF cells), mustard oil (100 microm; 10 of 12 LF cells), and low pH
156 rents were sensitized by capsaicin (3 of 9), mustard oil (2 of 7), or low pH (1 of 6) application.
158 ted capsaicin (a specific TRPV1 agonist) and mustard oil (a specific TRPA1 agonist) behavioral respon
160 in lamina II neurons that also responded to mustard oil (allyl isothiocyanate), indicating a presyna
161 s in outward rectification of single channel mustard oil (I(MO)) current-voltage relationships (I-V)
162 ects of ACEA on the TRPA1-selective agonist, mustard oil (MO), for calcitonin gene-related peptide (C
163 cular injection of an inflammatory irritant, mustard oil (MO), induces significant edema formation in
164 ion, NGF evoked a time-dependent increase of mustard oil (MO)-evoked TRPA1 activation in trigeminal g
167 rring electrophilic plant compounds, such as mustard oil and cinnamaldehyde, are TRPA1 agonists, it i
168 show that derivatives of two such compounds, mustard oil and cinnamaldehyde, covalently bind mouse TR
170 his channel is the sole target through which mustard oil and garlic activate primary afferent nocicep
171 fants receiving topical cord applications of mustard oil and other potentially unclean substances, re
173 spinalised, pentobarbitone-sedated animals, mustard oil applied to any site on the ipsilateral hind
175 that reduces plant predation, the so-called "mustard oil bomb," in which vacuole breakage in cells ha
178 zation of TRPV1 after TRPA1 stimulation with mustard oil in a calcium and cAMP/protein kinase A (PKA)
179 al desensitization of TRPA1 by capsaicin and mustard oil is not influenced by activation of protein p
180 Saturating activation by cinnamaldehyde or mustard oil occluded potentiation but did not interfere
184 ere we show that AITC (allyl isothiocyanate; mustard oil) and menthol represent two distinct types of
185 pounds that cause a burning sensation (e.g., mustard oil) and, indirectly, by components of the infla
186 ted cells that respond to capsaicin (but not mustard oil) as well as large-diameter myelinated neuron
189 by the TRPA1 agonists allyl-isothiocyanate (mustard oil), carvacrol, and polyunsaturated fatty acids
193 in cinnamon oil, wintergreen oil, clove oil, mustard oil, and ginger all activate TRPA1 (ANKTM1).
195 osensor, we establish that capsaicin, unlike mustard oil, consistently activates phospholipase C in s
196 lowing stimulation of peripheral nerves with mustard oil, demonstrating that NMB contributes to neuro
197 so exhibited by ART-OE mice to capsaicin and mustard oil, measured using a two-choice drinking test.
198 racolonic application of either capsaicin or mustard oil, stimuli known to evoke sustained nociceptor
199 e display behavioral deficits in response to mustard oil, to cold ( approximately 0 degrees C), and t
201 of trigeminal neurons that express TRPA1, a mustard oil- and cinnamaldehyde-sensitive channel, and t
203 n is regulated by TRPV1, and it appears that mustard oil-induced TRPA1 internalization is prevented b
204 are innervated by menthol-, capsaicin-, and mustard oil-responsive sensory neurons and are required
212 ow that TRPA1 is desensitized by homologous (mustard oil; a TRPA1 agonist) and heterologous (capsaici
213 cosinolate-myrosinase system, the so-called "mustard-oil bomb." Tissue damage caused by insect feedin
215 is study, the erucic acid content in several mustard oils and prepared mustard samples from Germany a
216 d molecular target for the pungent action of mustard oils and support an emerging role for TRP channe
217 e, the pungent principle of wasabi and other mustard oils, produces pain by activating TRPA1, an exci
218 f 10 g of capsulated mustard or uncapsulated mustard or a capsulated placebo mixture, measurements of
220 s have often had an atrial switch procedure (Mustard or Senning operation) performed, which leaves th
226 anates and sulfur compounds with bitterness, mustard, peppery, warming and initial heat mouthfeel tra
228 Silencing of eIF2Bbeta in a TuMV-susceptible mustard plant line and expression of eIF2Bbeta from a Tu
229 a TuMV-susceptible line in a TuMV-resistant mustard plant line confirmed the new resistance mechanis
230 , the pungent ingredient in wasabi and other mustard plants that induces pain and inflammation by act
232 gs that liberate the cytotoxic phosphoramide mustards (PM, IPM, and tetrakis-PM) via beta-elimination
233 roimidazole mustard TH-302, dinitrobenzamide mustard PR-104A, and benzotriazine N-oxide SN30000.
234 genic DNA crosslinking chemotherapy nitrogen mustard predicted to elicit HRR, selected against cells
235 04A, an experimental DNA alkylating nitrogen mustard prodrug currently under investigation for the tr
237 PR-104, a phosphate ester of the nitrogen mustard prodrug PR-104A, has shown evidence of efficacy
238 igned and synthesized a new type of nitrogen mustard prodrug that can be activated by high level of r
240 oper activation of the leaving group, sulfur mustards react with Grignard reagents with neighboring g
242 uch as methylators and crosslinking nitrogen mustards, represent a major risk factor for acute myeloi
243 and 65% activity in yellow, black and brown mustard, respectively, whereas the corresponding activit
249 content in several mustard oils and prepared mustard samples from Germany and Australia was determine
251 amine and glucose were added to the degummed mustard seed oil (20.16mumol/g oil) to prepare blank oil
261 tients after intra-atrial baffle procedures (Mustard, Senning, or total cavopulmonary connection).
262 d-transposition of the great arteries after Mustard/Senning (n=2), tetralogy of Fallot (n=2), aortic
265 (DECP) and selective oxidation of the sulfur mustard simulant, 2-chloroethyl ethyl sulfide (CEES).
267 selective and sensitive detection of sulfur mustard simulants in water that uses a metal-ion indicat
269 on of traces of potentially allergenic white mustard (Sinapis alba) and celery roots (Apium graveolen
270 calized Chlamydomonas CRR1 to the nucleus in mustard (Sinapis alba) seedlings, a location consistent
271 mustard species commonly used in food: white mustard (Sinapis alba), black mustard (Brassica nigra) a
273 -time PCR assays (singleplex assay for white mustard, singleplex assay for black/brown mustard and du
274 oration of the chemical warfare agent sulfur mustard (SM) produces a covalent adduct with human serum
275 allowing the simultaneous detection of three mustard species (white, black and brown mustard) and thr
276 us detection of three potentially allergenic mustard species commonly used in food: white mustard (Si
279 eed, the endogenous activities from Japanese mustard spinach, lemon, and spinach have the same substr
280 terized, and examined for binding the sulfur mustard surrogate, 2-chloroethyl ethyl sulfide (CEES).
281 ncluding the clinical stage 2-nitroimidazole mustard TH-302, dinitrobenzamide mustard PR-104A, and be
282 strip for rapid detection of gaseous sulfur mustard that is based on its degradation by the enzyme h
283 a series of ROS-activated aromatic nitrogen mustards that selectively kill chronic lymphocytic leuke
284 ly diluted DNA extracts from black and brown mustard, the DNA of both mustard species could be detect
285 atin synthase improves the ability of Indian mustard to tolerate higher levels of the heavy metal Cd
286 Therefore, selectively targeting nitrogen mustards to cancer cell mitochondria based on Deltapsimt
287 ific ICLs mimicking those formed by nitrogen mustards to facilitate the studies of cellular responses
288 tirapazamine analogue 18a bearing a nitrogen mustard unit at the 6-position, it was found that remova
289 a substantial increase in reactivity of the mustard unit, as measured by hydrolysis rates and DNA-al
293 nylates of the form 1 and a related nitrogen mustard variant have been constructed using a novel vari
294 labelling concerning mustard, in one sample mustard was detected with both ELISAs and the three real
295 and the three real-time PCR assays although mustard was not indicated on the food ingredient list.
296 e 3 conditions.The highest tolerable dose of mustard we were able to use did not elicit a relevant th
297 uctase, a series of nitrobenzylphosphoramide mustards were designed and synthesized incorporating a s
299 mpounds spontaneously liberate phosphoramide mustards with half-lives in the range of 0.08-15.2 h und
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