1 ich is apparent from reporter expression and
mutant analyses.
2 interspecies rescue experiments and further
mutant analyses.
3 med by single-mutant and compensatory double-
mutant analyses.
4 onsistent with previous predictions based on
mutant analyses.
5 Double
mutant analyses also demonstrate that ETT functions inde
6 Double
mutant analyses also reveal that IDN2 and three uncharac
7 Double-
mutant analyses also reveal that SIN2 shares functional
8 Mutant analyses and (13)C NMR studies also confirmed tha
9 Finally, the combination of
mutant analyses and characterization of P-body movement
10 with 3-hydroxypropionate, as demonstrated by
mutant analyses and enzyme activity measurements; (ii) t
11 Double
mutant analyses and expression studies show that althoug
12 Through transcriptome profiling,
mutant analyses and transgenic experiments, we aim to es
13 A combination of promoter deletion
mutant analyses,
and the response of promoter mutants to
14 Double
mutant analyses between Ya and gnu suggest that YA plays
15 Pharmacological and
mutant analyses confirmed the functional involvement of
16 Double-
mutant analyses demonstrate that Bif1 exhibits an epista
17 Double-
mutant analyses demonstrated that pif1phyA, pif1phyB, pi
18 Through a combination of loss-of-function
mutant analyses,
genetic mapping, and transgenic rescue
19 Mutant analyses have identified more than 10 positive co
20 lation of plant cell development, and double-
mutant analyses have revealed a complex genetic network
21 New
mutant analyses have revealed separable roles for Dap160
22 We report tissue-specific mouse
mutant analyses identifying the bone morphogenetic prote
23 Mutant analyses implicate ARP2 as an important subunit f
24 Point
mutant analyses in the two-hybrid system and in vitro sh
25 Interestingly, double, triple, and quadruple
mutant analyses indicate that individual SCAR genes vary
26 Double
mutant analyses indicate that tcl-2 can act synergistica
27 Mutant analyses indicate that the cardia bifida locus na
28 Double-
mutant analyses indicate that these pleiotropic cop/det/
29 Double
mutant analyses indicate that VosA and VelB play an inte
30 Mutant analyses indicated a correlation between dsRNA-bi
31 Additional
mutant analyses indicated that acs regulation was accomp
32 Mutant analyses indicated that endogenous ABA levels rep
33 ogenesis provides new insights for strategic
mutant analyses necessary to integrate the roles of tran
34 this study, comparative transcriptional and
mutant analyses of Desulfovibrio alaskensis strain G20 a
35 Double-
mutant analyses of era1 with the ABA-insensitive mutants
36 We present double
mutant analyses of eta2 axr1 plants indicating that libe
37 Double-
mutant analyses of the ABA-hypersensitive signaling muta
38 ve binding ELISA and by alanine substitution
mutant analyses of the hIFNAR1-IgG.
39 Double-
mutant analyses of tsh4 and several highly branched muta
40 Sox4
mutant analyses reveal a requirement for Sox4 in IPC spe
41 Double-
mutant analyses reveal that vem-1 genetically interacts
42 The 64
mutant analyses revealed all the genetic determinants re
43 Mutant analyses revealed that apical Baz accumulations c
44 Tissue-specific knockdowns and genetic
mutant analyses revealed that both Tachykinin and Tachyk
45 Double
mutant analyses revealed that I9H functioned redundantly
46 In the present study, double
mutant analyses revealed that only rhd1-4 hypersusceptib
47 Systematic double-
mutant analyses revealed the genetic network context in
48 in the conserved Asp51 phosphorylation site,
mutant analyses show that cell-cycle-controlled CtrA pro
49 Our
mutant analyses show that Lis1/dynactin have at least tw
50 Double
mutant analyses show that the hypersensitive phenotype o
51 Deletion
mutant analyses show that the most proximal 852 kb of th
52 P factors in regulating these promoters, and
mutant analyses showed that ABI4 and these bZIPs share s
53 Double
mutant analyses showed that PHYTOALEXIN DEFICIENT4 (PAD4
54 Instead, double
mutant analyses showed that the phyB mutation suppressed
55 Mutant analyses showed the inhibitory function of DC-HIL
56 ogether with results from abh1/abi1-1 double-
mutant analyses suggest that abh1 shows enhanced sensiti
57 Furthermore, results of double-
mutant analyses suggest that Ark1p and Prk1p function in
58 Target gene expression and double-
mutant analyses suggest that C2cd3 is an essential regul
59 Lastly, double
mutant analyses suggest that ETT control of floral organ
60 Gene-expression profiles and
mutant analyses suggest that methanol is the dominant ca
61 In addition, double-
mutant analyses suggest that the gene products of claret
62 Mutant analyses suggest that the primary motors for mito
63 Genetic double
mutant analyses suggest that the SUR-7-mediated effect i
64 Double
mutant analyses suggest that tube-size control by septat
65 Mutant analyses suggested a requirement of the p-coumaro
66 Double-
mutant analyses suggested that both wild-type Smo and Sm
67 In zebrafish, previous lineage-tracing and
mutant analyses suggested that SHF ventricular and OFT p
68 Gene expression and
mutant analyses support a model in which DnaA and Rok co
69 Mutant analyses supported this proposed model at a signi
70 This review summarizes the
mutant analyses that give insight into these patterning
71 As result of the
mutant analyses,
the antagonistic cytokinin and auxin si
72 e been implicated in xylan synthesis through
mutant analyses,
the biochemical mechanisms responsible
73 oinformatics-driven hypothesis building with
mutant analyses to identify potential epigenetic mechani
74 equired for cell-to-cell trafficking through
mutant analyses using a zucchini yellow mosaic virus exp
75 suppressor gene functions that eluded single
mutant analyses,
using a Caenorhabditis elegans genome-w
76 single site-directed mutagenesis and double
mutant analyses,
we conduct a detailed analysis on the r
77 n the activity of the recombinant enzyme and
mutant analyses,
we demonstrate its prominent role in th
78 in situ hybridization and double and triple
mutant analyses,
we showed that this enhanced defect was
79 Mutant analyses were consistent with predictions that Cy
80 Thus,
mutant analyses will require multi-generational studies.
81 Double
mutant analyses with dcl2-1, dcl3-1, and dcl4-2 alleles
82 ethylation (H3K9me2) levels at AtMu1c Double
mutant analyses with epigenetic silencing mutants sugges
83 Combining
mutant analyses with gain- and loss-of-function approach
84 By combining
mutant analyses with in vivo electrophysiological record
85 Double
mutant analyses with M. truncatula single leaflet1 (sgl1
86 Double-
mutant analyses with mutations in MUCILAGE MODIFIED2 and