1 acquired thermotolerance were identified by
mutant analysis.
2 ry cell wall synthesis, were investigated by
mutant analysis.
3 sequence motif identified here by a deletion
mutant analysis.
4 ther PLL2 or PLL3 based on single and double
mutant analysis.
5 ification, and differentiation has come from
mutant analysis.
6 meristem determinacy was revealed by double
mutant analysis.
7 cassettes for subsequent transformation and
mutant analysis.
8 ose synthase (SS) by immunoprecipitation and
mutant analysis.
9 protection, oligonucleotide competition, and
mutant analysis.
10 een shown to be critical for its function by
mutant analysis.
11 ription were identified through an extensive
mutant analysis.
12 ctivity by LEAFY, as was deduced from double
mutant analysis.
13 at amino acid position 291, as indicated by
mutant analysis.
14 gen (HBsAg), HBeAg levels, HBV genotype, and
mutant analysis.
15 ng, is becoming a powerful tool for en-masse
mutant analysis.
16 racing, electrophysiology, pharmacology, and
mutant analysis.
17 wer than half have roles established through
mutant analysis.
18 in the germination process were confirmed by
mutant analysis.
19 saul1 phenotypes, as demonstrated by double
mutant analysis.
20 We used designer arrays for defined
mutant analysis,
a high-throughput subtractive competiti
21 The
mutant analysis also confirms the proposed role of Frh i
22 Mutant analysis also revealed interactions of ABA and LR
23 A loss-of-function
mutant analysis also revealed that single mutants of bHL
24 s an approximately 85% reduction in omega-7s
Mutant analysis also showed that FATTY ACID ELONGASE1 is
25 Crystal structures followed by
mutant analysis and affinity pull-downs have revealed th
26 Mutant analysis and asymmetric reconstructions show that
27 Using a combination of double
mutant analysis and biochemistry, we found that in maize
28 Double
mutant analysis and comparison of AP-3(-) and BLOC-1(-)
29 Molecular epistasis tests, double
mutant analysis and dosage-sensitive interactions demons
30 We tested a small set of these genes through
mutant analysis and found that one significantly increas
31 Reciprocal grafting, double
mutant analysis and gene cloning suggest that all MAX ge
32 ng a combination of in vitro explant assays,
mutant analysis and gene delivery into mouse embryos cul
33 Both the
mutant analysis and gene regulation studies suggest that
34 Using gene expression studies, genetic
mutant analysis and genetic mosaics, we show that egl-38
35 e CER2 homologs CER2-LIKE1 and CER2-LIKE2 by
mutant analysis and heterologous expression in yeast.
36 Quadruple
mutant analysis and in situ localization of A, B, C and
37 Using a combination of
mutant analysis and in vitro explant assays, we demonstr
38 studies, in vitro migration assays, genetic
mutant analysis and in vivo fate mapping in mice, we fou
39 Mutant analysis and live cell imaging indicate that PDS5
40 Mutant analysis and optogenetic studies reveal that GABA
41 igands at the midline, and we show by double
mutant analysis and physical interaction tests that WRK-
42 A combination of loss-of-function
mutant analysis and protein interaction data indicates t
43 al gene 20 product (gp20) were determined by
mutant analysis and sequence localization within the str
44 By means of deletion
mutant analysis and site-directed mutagenesis, we have i
45 teraction as previously determined by escape
mutant analysis and site-directed mutation, is located i
46 Mutant analysis and subsequent molecular studies have re
47 To confirm the results of the
mutant analysis and to determine the relative contributi
48 Using global expression studies, double
mutant analysis,
and protein interaction assays, we find
49 nt systems have been addressed by a targeted
mutant analysis approach and almost all are shown to be
50 pylori stress resistance was evaluated by a
mutant analysis approach.
51 gene targets in CGNs using inhibitor and Nfi
mutant analysis as well as chromatin immunoprecipitation
52 A dimerization motif, an alanine scan double
mutant analysis at the helix-helix interface was carried
53 Based on double
mutant analysis,
AXL1 in a MATalpha strain acted genetic
54 Double-
mutant analysis between abi3-3 and wri1-1 suggested that
55 Double-
mutant analysis between mnp-1(RNAi), ina-1, and vab-1 mu
56 Double-
mutant analysis between scraps, a mutation in anillin th
57 ion observed in mig-10 mutants, while double
mutant analysis between unc-53 and mig-10 showed no incr
58 , we previously used the multistep method of
mutant analysis by PCR and enzyme cleavage (MAPREC).
59 g of oral poliovirus vaccine with the use of
mutant analysis by PCR and restriction enzyme cleavage (
60 Mutant analysis by PCR and restriction enzyme cleavage (
61 Direct RNA extraction and
mutant analysis by polymerase chain reaction and restric
62 Inr consensus sequence based on an extensive
mutant analysis carried out in HeLa cell extracts.
63 Mutant analysis confirms that affinity proteomics is a v
64 Mutant analysis confirms that Asp-97 and Glu-108 are pro
65 However,
mutant analysis correlated specific HMW2 domains with co
66 The
mutant analysis,
coupled with physiological data, indica
67 ular localization in response to Shh, double
mutant analysis demonstrates that Rab23 does not work th
68 Mutant analysis demonstrates that Vac8p functions separa
69 Deletion
mutant analysis determined that the N- and C-termini are
70 Deletion
mutant analysis determines that there is a difference in
71 -mapping studies using neutralization escape
mutant analysis,
deuterium exchange mass spectrometry, a
72 ed subtly elevated levels of lesions, double
mutant analysis disagreed with a simple epistatic model
73 Mutant analysis established residues that were involved
74 tro findings to in vivo biological function,
mutants analysis establishes the role of Shr in GAS grow
75 The
mutant analysis further allows us to propose that a subs
76 Mutant analysis has defined two parallel genetic pathway
77 Mutant analysis has identified mycoplasma proteins assoc
78 Although deletion
mutant analysis has suggested that the region of the gen
79 The
mutant analysis highlights some important motifs for sub
80 enzymes in H. pylori have been studied using
mutant analysis;
however, the gene encoding adenosine de
81 r downstream of ETR1 and ERS based on double
mutant analysis;
however, the signaling mechanisms leadi
82 ading to the epidermis in L. japonicus While
mutant analysis identified redundancy in several biosynt
83 ter activity in lymphoid cells, and deletion
mutant analysis identified three distinct domains of TEL
84 Mutant analysis in Arabidopsis has indicated that absenc
85 By combining physical perturbations and
mutant analysis in both species, we show that difference
86 Mutant analysis in Caenorhabditis elegans reveals that c
87 Recent
mutant analysis in zebrafish points to an important role
88 Results from deletion and
mutant analysis indicate that Pak1 regulates cyclin D1 t
89 Double-
mutant analysis indicated that blocking the salicylic ac
90 Double
mutant analysis indicated that ERS2, similar to ETR1, ET
91 In addition, double-
mutant analysis indicated that gamma-tubulin-dependent n
92 ATR
mutant analysis indicated that it is required for checkp
93 A
mutant analysis indicated that the Ino2p/Ino4p/Opi1p reg
94 Double
mutant analysis indicated that the SOS genes function in
95 Double
mutant analysis indicated that the thk1 mutant is epista
96 Double
mutant analysis indicated that the two auxin transport s
97 Mutant analysis indicates a progressive loss in the amph
98 Mutant analysis indicates a role for PIAL1 and 2 in salt
99 Double
mutant analysis indicates an interaction energy between
100 Double-
mutant analysis indicates that a related helix-loop-heli
101 Double-
mutant analysis indicates that BK and Erg K(+) channels
102 Double-
mutant analysis indicates that both sag-1 and eat-16 act
103 termediate columns of the neuroectoderm, and
mutant analysis indicates that Dichaete regulates cell f
104 Double-
mutant analysis indicates that early flowering is depend
105 Double
mutant analysis indicates that ETR2 acts upstream of CTR
106 Double
mutant analysis indicates that Gli3 repressor activity i
107 Essential for viability, plx
mutant analysis indicates that larval death is attributa
108 Double
mutant analysis indicates that phyB is epistatic to hrb1
109 uces increases in the abundance of PIF3, and
mutant analysis indicates that PIF3 acts, in conjunction
110 r the S/M or G2/M checkpoint, however double
mutant analysis indicates that rad31 acts in a process w
111 Genetically, sos2sos3 double
mutant analysis indicates that SOS2 and SOS3 function in
112 Mutant analysis indicates that the cytokinin receptors A
113 Deletion
mutant analysis indicates that the juxtamembrane region
114 Double
mutant analysis indicates that the primary target of Rab
115 Genetic double-
mutant analysis is consistent with ptp-3A acting with th
116 Double
mutant analysis is consistent with these results, showin
117 Using
mutant analysis,
laser ablation, optogenetics, and Ca2+
118 depressor, we used YFP:actin to monitor, and
mutant analysis,
laser-ablation and transgenic feminizat
119 at integrates multiple approaches, including
mutant analysis,
lineage tracing, cell purification, gen
120 A refined
mutant analysis localized this element to nucleotides -8
121 Based on deletion
mutant analysis,
MdmX inhibition of Smad transactivation
122 stion and have shown by microarray analysis,
mutant analysis,
metabolite measurements, and (13)C-labe
123 res, we propose how the information from the
mutant analysis might facilitate the use of a simplified
124 This work provides the first
mutant analysis of a GYF-domain protein in either C. ele
125 tructure of the AdpA-DBD-DNA complex and the
mutant analysis of AdpA-DBD revealed its unique manner o
126 Double
mutant analysis of Atrar1 in combination with the R sign
127 Genetic evidence from double-
mutant analysis of cow1-1 and other loci involved in roo
128 Double
mutant analysis of dlf1 and indeterminate1 (id1), anothe
129 Double
mutant analysis of hos5-1 and the ABA-deficient aba1-1 a
130 Mutant analysis of induced plant defense pathways showed
131 Double-
mutant analysis of lls1 with two maize mutants oil-yello
132 Mutant analysis of plants deficient in any of three in-f
133 Double-
mutant analysis of polar residues in the distal beta-hai
134 Surprisingly, a detailed substitution
mutant analysis of the amino-terminal domain revealed a
135 Here, we report a detailed
mutant analysis of the D' element which suggests that an
136 Deletion
mutant analysis of the LTR of a PERV-NIH isolate identif
137 Finally, double
mutant analysis of unc-71 with other axon guidance signa
138 Deltahns, DeltarpoS, and Deltahns DeltarpoS
mutants, analysis of RpoS reporter fusions, quantitative
139 otein 70 (Hsp70) family, in conjunction with
mutant analysis,
permitted the characterization of a mot
140 Mutant analysis,
pharmacology and patch-clamp recording
141 Through
mutant analysis,
protein depletion and rescue experiment
142 In this issue, Oda et al. use
mutant analysis,
protein tagging, and cryoelectron tomog
143 Transplantation experiments and
mutant analysis reveal that cephalic mesoderm is the sou
144 Deletion and site-specific
mutant analysis revealed a critical role of a potential
145 Mutant analysis revealed a new relationship between the
146 In addition, double
mutant analysis revealed epistasis between EDM2 and the
147 Mutant analysis revealed extensive ligand redundancy in
148 A comprehensive
mutant analysis revealed only one element upstream of th
149 Mutant analysis revealed that a variable residue within
150 Double
mutant analysis revealed that all edr1-associated phenot
151 Deletion
mutant analysis revealed that both the kinase domain and
152 Mutant analysis revealed that complement-protective CD55
153 Double-
mutant analysis revealed that edr2-mediated resistance i
154 Double
mutant analysis revealed that espA was epistatic to pktA
155 Mutant analysis revealed that GndA and FDH4 are crucial
156 Double
mutant analysis revealed that SmD3b is also involved in
157 The stf lfl double
mutant analysis revealed that STF and LFL act mainly ind
158 Mutant analysis revealed that the C-terminal region of C
159 Double
mutant analysis revealed that the coi1 mutation (causing
160 Deletion
mutant analysis revealed that the complement control pro
161 Double
mutant analysis revealed that the nlp7-1 phenotype depen
162 Mutant analysis revealed that the protective function of
163 Mutant analysis revealed that the single chemotaxis syst
164 Double
mutant analysis revealed that these drought-induced phen
165 Mutant analysis revealed that yKu70 and Sir1 act collect
166 induce transcription of a reporter, deletion
mutant analysis revealed the presence of a strong activa
167 This
mutant analysis revealed the presence of a third glycosy
168 Furthermore, MFSD2A
mutant analysis reveals an important function of the C t
169 Double
mutant analysis reveals an unexpected, redundant negativ
170 spatially stereotyped in wild-type animals,
mutant analysis reveals that each cell has the potential
171 wun wun2 double
mutant analysis reveals that the two genes, hereafter co
172 Deletion
mutant analysis reveals that WASP residues 101-151 are n
173 Tryptic phosphopeptide and
mutant analysis reveals that, as in mitosis, stress-indu
174 Our
mutant analysis reveals the contribution of mechanical e
175 AHF-Cre genetic tracing experiments and Tbx1
mutant analysis show that nonsomitic neck muscles share
176 Deletion and point
mutant analysis show that the activity of TNRC4 on tau E
177 Mutant analysis showed that AAD2 also contributes to ome
178 formation depends on ZCF membrane curvature:
mutant analysis showed that Cdc42p localization is negat
179 Explant essays and
mutant analysis showed that cellular guidance involved r
180 Mutant analysis showed that crdA cells were delayed in d
181 Double
mutant analysis showed that dopA interacts genetically w
182 genes involved in glycogen homeostasis, and
mutant analysis showed that Gcn4p suppresses glycogen le
183 Mutant analysis showed that HDA19/HD1 mediated deacetyla
184 Double
mutant analysis showed that only re rer1 and rer5 rer6 e
185 Mutant analysis showed that the glutamate residue corres
186 Mutant analysis showed that the RsbT kinase, which is re
187 involved in this response were identified by
mutant analysis,
showing that the EARLY FLOWERING 4 gene
188 Phospho-
mutant analysis shows that Aurora contributes to the mic
189 Our
mutant analysis shows that eliminating UNC-34 function r
190 Mutant analysis shows that RAD51B is essential for the m
191 Double
mutant analysis shows that trio interacts with Rac in a
192 In addition, our homeotic
mutant analysis shows that wing transformation in T1 ori
193 Double
mutant analysis shows that zig-3 and zig-4 act together
194 Our double-
mutant analysis shows that, in contrast to Zw3, Nkd acts
195 Precursor metabolite incorporation and
mutant analysis studies support the mode-of-action, bloc
196 Mutant analysis substantiates this idea.
197 heterozygous genetic interactions and double
mutant analysis suggest that 18W affects the Rho-GTPase-
198 Deletion
mutant analysis suggested a requirement of RopGEF activi
199 Binding studies in vitro and
mutant analysis suggested that GFP-PH bound PtdIns(4,5)P
200 not been demonstrated in vitro, but previous
mutant analysis suggested that Rhizobium etli gene wreQ
201 Mutant analysis suggested the role of the cysteine prote
202 Double
mutant analysis suggests a possible inaccessibility of e
203 In addition,
mutant analysis suggests a possible novel role for phyC
204 Double
mutant analysis suggests that ETTIN interacts with TSL,
205 Double
mutant analysis suggests that HST acts in parallel to SQ
206 Double
mutant analysis suggests that pgp-2(+) functions in para
207 Double
mutant analysis suggests that sur-6 PP2A-B acts downstre
208 Mutant analysis suggests that VE accumulation in these f
209 Mutant analysis supports the assignment of the primary i
210 Here, we show through systematic
mutant analysis that GA20ox1, -2, and -3 are the dominan
211 We present double
mutant analysis that indicates that daf-28(sa191) acts a
212 he aid of genetic engineering and subsequent
mutant analysis,
the functional role of conserved cystei
213 we have developed an approach termed triple-
mutant analysis (
TMA).
214 We used double
mutant analysis to determine the relative positions of t
215 FRUITFULL, which we show through comparative
mutant analysis to modulate fruit shape during post-fert
216 We have used double
mutant analysis to position mei-217 in the meiotic recom
217 Through single- and double-
mutant analysis to study the mitotic cohesion proteins S
218 We used mouse in utero electroporation and
mutant analysis to test whether cortical signaling sourc
219 tinin (Pta), a surface-associated cytotoxin,
mutant analysis was used in conjunction with a mouse mod
220 Mutant analysis was used to identify Moraxella catarrhal
221 Mutant analysis was used to prove that the HupA protein
222 to be composed of PFs by cross-sectional and
mutant analysis,
was found to extend along the entire le
223 Using fate mapping and
mutant analysis,
we find that PAA progenitors are derive
224 Using antibody labeling and
mutant analysis,
we have localized 12 of 13 core APC/C c
225 Based on our
mutant analysis,
we revised the rec27 open reading frame
226 Through phenotypic and double
mutant analysis,
we show that AGO1 regulates stem cell f
227 By conditional Shh
mutant analysis,
we show that Shh signaling regulates hC
228 We also report an escape
mutant analysis,
which allows the mapping of heterotypic
229 ure focused physiological studies, including
mutant analysis,
which will provide further details into
230 Double-
mutant analysis with anther ear1 and tassel seed2 reveal
231 Double-
mutant analysis with atr1D, an overexpression allele of
232 Double
mutant analysis with mutations in other genes affecting
233 Mutant analysis with potential upstream genes was used t
234 Double-
mutant analysis with suppressors and enhancers of lin-12
235 We performed
mutant analysis with the null alleles of ABP1, abp1-c1 a
236 Double-
mutant analysis with the peroxisomal ATP-binding cassett
237 Double
mutant analysis with this Hoxb1(3'RARE) allele and other