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1 ent species and in the SF3B1 K700E oncogenic mutant background.
2 genes either in the wild-type or in the rsp mutant background.
3 umulation of hydrogen peroxide in the dgs1-1 mutant background.
4 nes expressing pri-miR163 gene in the mir163 mutant background.
5 onto the LAT knock-in Bam32 knockout double-mutant background.
6 r body formation in the porB-1 porC-1 double mutant background.
7 hitecture, and motility inhibition in a sinR mutant background.
8 protein (IsdA) were most abundant in the agr mutant background.
9 from tissue-specific promoters in an Atsuc2 mutant background.
10 cent proteins of three colors in the quartet mutant background.
11 hs on the adaxial side of the leaf in an as1 mutant background.
12 a-4, rescued the rat phenotype in the impa-4 mutant background.
13 ible RNAi silencing of AtLCB2b in an Atlcb2a mutant background.
14 inhibit rhabdomeral degeneration in the tadr mutant background.
15 induce aneuploidy and tumorigenesis in a p53 mutant background.
16 ent, are largely lethal in a PINK1 or parkin mutant background.
17 ll-death defects in a genetically sensitized mutant background.
18 hanced defence that is suppressed in a myb30 mutant background.
19 ere required to accumulate NifB-co in a nifN mutant background.
20 positive regulator in the tga5-1 tga6-1 null mutant background.
21 cry1 subcellular localization in a cry1-null mutant background.
22 oteins of three different colors in the qrt1 mutant background.
23 oxyR plasmid was absent in a type I fimbrial mutant background.
24 1 during S phase, even in a sensitized ddb-1 mutant background.
25 strongly reduced in the jasmonate resistant1 mutant background.
26 per facial nerve formation even in the Hoxb1 mutant background.
27 ation during early development in the agb1-2 mutant background.
28 a signal that is not present in the spoIIIE mutant background.
29 enhance the late-flowering phenotype in a co mutant background.
30 nterferes with granule initiation in the ss4 mutant background.
31 and sensitivity to cytotoxic agents in a dam mutant background.
32 g of 35S-NPTII but not RD29A-LUC in the ros1 mutant background.
33 as observed in E7 mice on the Rb(DeltaLXCXE) mutant background.
34 s not able to rescue AR activity in the TIF2 mutant background.
35 stitutive activation of the response in this mutant background.
36 mal interaction differently depending on the mutant background.
37 lt in retinoblastoma formation even in a p53-mutant background.
38 ppresses Trp gene dysregulation in a cyp83B1 mutant background.
39 FRI to increase FLC mRNA levels in the abh1 mutant background.
40 that flk-1 signaling is up-regulated in the mutant background.
41 rly embryonic lethal defects in a Disp1-null mutant background.
42 screen was carried out in the bir1-1 pad4-1 mutant background.
43 escue of male viability in a roX1(-) roX2(-) mutant background.
44 rabidopsis carrying 35S::VP1 in an abi3 null mutant background.
45 ives totally glabrous plants only in the gl3 mutant background.
46 a2p, and Pea2p) are essential in a cla4delta mutant background.
47 anscription appears relatively robust in the mutant background.
48 onsistent with a cell division defect in the mutant background.
49 ene could be methylated de novo in the suvh4 mutant background.
50 Hoxd11 are present in the Hoxa11 homozygous mutant background.
51 ession of a COI1-YFP transgene in the coi1-1 mutant background.
52 d gain of L rescues ventral tissue loss in N mutant background.
53 n bud outgrowth in a strigolactone-deficient mutant background.
54 s further impaired in the C-terminal Hay/XPB mutant background.
55 domains of DEK1 using gene targeting in null mutant background.
56 channel-dependent behaviors in a slo-1-null mutant background.
57 n ABA signaling was alleviated in the ios1-1 mutant background.
58 localization appears near normal in a spe-29 mutant background.
59 ient to suppress its splicing defects in the mutant background.
60 ll caused cortex proliferation in the erecta mutant background.
61 ory action of CLE3 was abrogated in the clv1 mutant background.
62 ized and show increased activities in a upl3 mutant background.
63 e photorespiratory genes was observed in the mutant background.
64 steady-state photosynthesis and in the pgr5 mutant background.
65 se from 16 to 32 mug/ml in an embB codon 306 mutant background.
66 e Dictyostelium protein and expressed in the mutant background.
67 modulation of the RRP is blocked in the rim mutant background.
68 tomatal opening mediated by PHOT1 in a phot2 mutant background.
69 lerate the depletion of MvaT in an DeltamvaU mutant background.
70 1 expression and nodule formation in an ern1 mutant background.
71 ctional 35S::NRT2.1 transgene in an atnrt2.1 mutant background.
72 e the wake-promoting effects of DA in a DopR mutant background.
73 be used to image Pax2 expressing cells in a mutant background.
74 l adhesin ApiA, was not affected in the same mutant background.
75 during heterocyst differentiation in a sigE mutant background.
76 length SMN are also stabilized in the degron mutant background.
77 ficient to mediate the Hth function in the L mutant background.
78 io protein is greatly diminished in the seh1 mutant background.
79 d localization of MRP observed in an ECA3265 mutant background.
80 stablishment in a series of dot1 and histone mutant backgrounds.
81 expression and reduced proliferation in both mutant backgrounds.
82 effect on genome stability in wild-type and mutant backgrounds.
83 o promote epidermal differentiation in these mutant backgrounds.
84 ed maintenance of RPS methylation in various mutant backgrounds.
85 examined their expression in each of the QS mutant backgrounds.
86 he ROS1 down-regulation that occurs in these mutant backgrounds.
87 found in mice with different Cited2 and Wt1 mutant backgrounds.
88 r complex, becomes essential in mcs6 or pmh1 mutant backgrounds.
89 OSM-9 motility is disrupted in certain IFT mutant backgrounds.
90 ct formation in leafy, apetala1 and apetala2 mutant backgrounds.
91 ly flowering in wild-type and late-flowering-mutant backgrounds.
92 mice were crossed onto Ink4a/Arf and/or p53 mutant backgrounds.
93 by introducing a loss of her7 function into mutant backgrounds.
94 f PHYB in photoreceptor and clock-associated mutant backgrounds.
95 gregation in both the wild-type and the pilA mutant backgrounds.
96 cistronic transcripts in enhanced RNAi (Eri) mutant backgrounds.
97 both chd1Delta and, as has been shown, K123A mutant backgrounds.
98 in phytochrome (phy) and cryptochrome (cry) mutant backgrounds.
99 n both large T (63 mutants) and middle T (51 mutants) backgrounds.
101 stil elongation and stigma exsertion in this mutant background, a process that requires SRK catalytic
102 -Activated Protein Kinase 6 (MPK6) in a mpk3 mutant background all have abscission-defective phenotyp
104 ns of gene expression in different zebrafish mutant backgrounds allow further quantitative evaluation
106 g wve-1 levels in either unc-34/ena or wsp-1 mutant backgrounds also leads to a significant enhanceme
107 (upstream activating sequence)-ben in a ben mutant background and identified a well defined critical
108 introduced the HG transgene onto a Gbx2-null mutant background and recreated a new Otx2/Gbx2 border i
110 porter-gene activity increased in a u-shaped mutant background and that forced expression of SerpentN
111 Despite of this striking effect in the spi mutant background and the expression of EcR signaling co
112 alize the anatomy of specific neurons in Lhx mutant backgrounds and find that the development of the
113 expression in a subset of autonomous-pathway-mutant backgrounds and functions both to promote activat
114 ly different activities in the sinI and expR mutant backgrounds and in response to added SinI AHLs.
116 lle and Twist were introduced into different mutant backgrounds and the patterning activities were vi
117 s were expressed in both secA2 wild-type and mutant backgrounds and were tested for their ability to
118 lly to hth, (ii) Hth is upregulated in the L mutant background, and (iii) MH domain of Hth is require
119 ted with chromosomal translocations in a p53 mutant background, and heterozygosity for the mutant all
120 enced the candidate gene, NCU02713.3, in the mutant background, and phenocopied the mutation by gene
121 thermore, STR2 is haplo-insufficient in str1 mutant background, and str1(-/-)/str2(+/-) embryos were
122 acnA mutations were introduced into an rpiRc mutant background, and the effects on RNAIII were determ
124 ed in bacteria, but primarily in specialized mutant backgrounds, and the factors responsible for DNA
125 that heat shock-induced puffs in JIL-1 null mutant backgrounds are strongly labeled by antibody to t
127 Etv loss-of-function alleles into the Rfwd2 mutant background attenuated the branching phenotype, su
128 ented by any of the lspA(Mx) genes in an lpp mutant background, but not in an E. coli lpp(+) backgrou
129 icacy of genotoxic cancer therapies in a p53 mutant background by eliminating one of the checkpoints
130 d, after elimination of CESA redundancy in a mutant background, by coimmunoprecipitation and mass spe
131 pression of hDAT-L368Q in the Drosophila DAT mutant background caused developmental lethality, indica
132 show that disruption of PDAT1 in the tgd1-1 mutant background causes serious growth retardation, gam
136 of Baboon in the patched domain in a patched mutant background completely rescues the head defects.
137 PR gene expression, is abolished in a sid2-1 mutant background, consistent with a requirement for sal
138 this purpose, ETR1 levels were quantified in mutant backgrounds containing receptor loss-of-function
139 levels are reduced in the dcr-1(ok247) null mutant background; conversely, a reduction of DCR-1 prot
140 55A and M377V, made in the active FAR8-S363P mutant background converted its specificity from 16:0-Co
141 specific restoration of Tbx1 expression in a mutant background corrected most of those defects in the
143 ss on rop6(DN) plants, which was retained in mutant backgrounds defective in SA biosynthesis or signa
144 iated response in rop6(DN) was suppressed in mutant backgrounds defective in SA signaling (nonexpress
145 losis was lost in DO11.10 mice bred in a rag mutant background, demonstrating that the immune respons
146 on also occurs in the rps2, ndr1, and Atrar1 mutant backgrounds, demonstrating that this activity can
150 heat shock mRNAs, SSA4 and HSP104, in these mutant backgrounds; each transcript concentrates in a si
151 red when MET1 is re-introduced into the met1 mutant background, either via genetic cross or DNA trans
155 converted into carpelloid organs in certain mutant backgrounds even in the absence of AG activity in
156 nic males carrying this construct in a mab-9 mutant background exhibit tail abnormalities including m
157 m is preserved across Drosophila species and mutant backgrounds exhibiting a range of eggshell phenot
158 rammed DNA double-strand breaks in a spo11-1 mutant background failed to rescue the DNA fragmentation
159 d VE-cadherin(+) vascular cells in the flt-1 mutant background first occurs between day 5 and day 6.
160 Our findings question the prudence of using mutant backgrounds for genetic screens and underscore a
162 dition, disruption of ihfA in an hns or toxT mutant background had no effect on tcpA expression.
165 sed epistasis experiments in an atonal (ato) mutant background, identified 188 genes induced by ey.
166 had shown that removal of Pax6 from the Gsh2 mutant background improves the molecular identity of the
167 inbred ddm1 (decrease in DNA methylation 1) mutant background in which various genetic and epigeneti
169 DNA looping in a variety of operator and CI mutant backgrounds in vivo, and our methodology can be a
170 eased the frequency of rootless seedlings in mutant backgrounds in which auxin regulation of basal po
171 n (Psyr_1620-1616, paoABCDE), only in a pdhQ mutant background, in addition to pyruvate, are herein a
172 roxylase 1/beta-hydroxylase 2 (b1 b2) double-mutant background, in which both Arabidopsis beta-hydrox
173 in meiotic entry, as is exhibited in certain mutant backgrounds, inappropriately juxtaposes undiffere
174 that the taa mutants in several known auxin mutant backgrounds, including pid and npy1, mimic all of
175 phyA' in the DNA methyl transferase I (met1) mutant background increased PHYA expression and restored
176 ind that Rim20-GFP foci accumulate in a vps4 mutant background independently of external pH, Rim101 p
177 protein-protein interactions in a variety of mutant backgrounds indicate that substrates are probably
178 ESL and dnaKJ operons transformed into these mutant backgrounds indicated that sig1 is required for t
179 ffects of ethylene and auxin in a variety of mutant backgrounds indicates that auxin biosynthesis, tr
180 to restore Syt1 function in a syt1(-/-) null mutant background, indicating both C2A and C2B are speci
181 ational repression were impaired in the AGO3 mutant background, indicating that AGO3 can mediate both
182 b-1 mutant lethality was enhanced in a pyr-1 mutant background, indicating that HSPG synthesis is ver
183 background, whereas it persisted in the sspA mutant background, indicating that SspA/Lpa mediate the
184 anced and blue shifted in the Cys154 --> Gly mutant background, indicating that the latter mutation c
185 binding, confers complete rescue in a dli-1 mutant background, indicating this is not an essential d
188 of S motility in the mglA-8 masK-815 double mutant background is different than that resulting from
190 eed produced from VP1 expression in the abi3 mutant background is nearly indistinguishable from wild
191 n that the upregulation of FLC in FRI- or AP-mutant backgrounds is correlated to an increase in histo
192 oxylase observed in both the daf-2 and unc-2 mutant backgrounds is suppressible either genetically by
195 is abrogated in the mrt-2, hus-1, and clk-2 mutant backgrounds, lack of the apoptotic branch of the
196 s observed only for attractant addition in a mutant background lacking the coupling proteins, CheW an
197 ts and, importantly, deleting Yap in the Nf2 mutant background largely restores hippocampal developme
198 th and ethylene responsiveness in the double-mutant background, leading to the conclusion that canoni
199 in PHB development, loss of egl-5 in a ham-1 mutant background leads to PHB differentiation defects.
200 into a 'sensitised' zygotic headless (tcf3) mutant background leads to severe forebrain and eye defe
201 n by RNAi of ats1-1 RNA levels in the ats1-1 mutant background led to a more severe growth phenotype
202 t of these cells (the leaf margin) in a dwf4 mutant background led to both restoration of differentia
203 Phenotypic analysis of the transgenes in Trl mutant background led us to the conclusion that GAGA-519
206 ion of this RGS1 protein variant in the rgs1 mutant background makes plants hypersensitive to a subse
208 oss of KANADI activity in a Class III HD-Zip mutant background mitigates the defects in bilateral sym
209 l function in ovule development: in the MPK6 mutant background, MPK3 is haplo-insufficient, giving fe
211 nor ARE2 transcription is altered in an are mutant background, nor does overexpression of either ARE
212 which can be reversed upon transfer into the mutant backgrounds of decrease in DNA methylation 1 (ddm
213 auer arrest: it forms dauers in the deletion mutant backgrounds of ncr-1 or daf-28/insulin; as a sing
216 l types using the GAL4/UAS system in an amon mutant background partially rescues larval molting and g
217 f a single mutant allele of Nodal in the Tet mutant background partially restored patterning, suggest
218 he pathway was reduced by RNAi in the ats1-1 mutant background, phosphatidylglycerol amounts decrease
221 application and the ssi2 mutation in various mutant backgrounds produce similar effects and that rest
222 CeTRAP240/let-19(RNAi) in a CeTRAP230/dpy-22 mutant background produced a strong synthetic lethal phe
226 ic ftz cis-regulatory element (CRE) in a ftz mutant background rescued for segmentation defects.
228 ion of AtBI1 transgene in the two homozygous mutant backgrounds rescued the accelerated cell death ph
229 c restoration of Gata3 function in the Gata3 mutant background rescues the expression phenotypes of t
231 pression of Shaw(OX) and NKCC(RNAi) in a qsm mutant background restored LL-induced behavioral arrhyth
233 Overexpression of this gene in the rcp1 mutant background restores carotenoid production and, un
234 Deletion of sloR or sdhB in the DeltaserR mutant background restores growth to wild-type levels, s
236 ld-type, an equivalent loss in the fshr-1(0) mutant background resulted in a highly penetrant germlin
237 Mutation of this gene in the triple-FDH mutant background resulted in a methanol-negative phenot
239 Deletion of flhA in a wild-type or luxS mutant background resulted in identical loss of motility
240 C1, using artificial microRNA in the exo70C2 mutant background, resulted in a complete pollen-specifi
241 gene, Bmpr1b, in the retina-specific Bmpr1a mutant background, results in abnormal retinal dorsovent
243 e (Pol) II distribution in single and double mutant backgrounds revealed that Swi6 and Chp2 proteins
244 alysis between the tumors in the various E2F-mutant backgrounds revealed that there was extensive com
245 mal promoter was placed in a scarecrow (scr) mutant background, revealing a possible role for SCR in
246 ity and female-specific lethality in a piwi2 mutant background, revealing the zygotic function of piw
247 ngly, ruvC mutants, in both recA(+) and recA mutant backgrounds, scored as hyperrecombinational.
248 cessive ppd mutants on a spring-flowering hr mutant background show early, photoperiod-insensitive fl
249 scopy of the P(nifHD)-gfp reporter in a sigE mutant background showed delayed development and undetec
250 -of-function 14-3-3 lambda mutant in a phot1 mutant background showed that the 14-3-3 lambda protein
251 ression of regulatory genes in wild-type and mutant backgrounds showed that RspR controls transcripti
252 on of the flgM promoter to lacZ in different mutant backgrounds showed that the flgM promoter was not
253 Mating these mutant mice to various gene mutant backgrounds showed that the mouse disease phenoty
254 Assay of the FUS1-lacZ reporter (in a hog1 mutant background) showed that sho1 and msb2 mutations b
255 on of FHY1 are abolished in a PHYA-deficient mutant background, showing that FHY1 requires a signal f
256 n a feedback-insensitive threonine deaminase mutant background, shows that these two enzymes have ove
257 of one allele of Pax6 on the homozygous Tlx mutant background significantly worsens the phenotype.
259 dev promoter activity in wild-type and devS mutant backgrounds strongly suggest that upstream and do
260 ts is abolished in the ssi2 act1 coi1 triple-mutant background, suggesting that both JA- and act1-gen
261 phenotype of dao is suppressed in a seizure mutant background, suggesting that Dao acts by an effect
262 ependent and rates are not altered in a CUL1 mutant background, suggesting that this ARF is targeted
263 ffects were lost or altered in patS and hetN mutant backgrounds, supporting a role of SepJ in the int
265 r screen in a gastrulation-sensitized double-mutant background, targeting genes likely to be expresse
267 sis and are hyperactive PTI suppressors in a mutant background that lacks the cell death response.
268 erwise isogenic background, as well as a nod mutant background that primarily undergoes loss of chrom
269 e expressed from a separate cistron in a VSV mutant background that readily establishes persistent in
271 al of 72 tested, we identify 6 miRNA allelic mutant backgrounds that modulate the survival response t
277 d overexpression in a desiccation-intolerant mutant background to play an important role in seed DT.
278 nts were generated in either of the permease mutant backgrounds to identify the ATPase(s) interacting
279 s9 activity in heterozygous loss-of-function mutant backgrounds, to rapidly evaluate the phenotypic i
280 nd phyC deficiency had no effect in the phyA mutant background under FRc, suggesting that phyC does n
281 evels of HFR1 protein accumulate in the spa1 mutant background under various light conditions, includ
282 d score a double interval in a wild-type and mutant background using this protocol will take 22-27 we
283 reen for synthetic lethals in an unc-34 null mutant background utilizing an RNAi feeding approach.
284 h L, while its homeodomain is not, (iv) in L mutant background ventral eye suppression function of Ht
285 olecular and lineage analysis in this double mutant background we demonstrate that presumptive rhombo
286 uction of SPCH is compromised in a GATA gene mutant background, we hypothesize that PIF- and light-re
288 holino-mediated knockdown of cdx1a in a cdx4 mutant background, we show that a deficiency in both cdx
289 crossing a Bmp4 mutation into the RBPJ/Axin2 mutant background, we show that Wnt and Bmp4 signaling a
290 HO-1:GFP) in wild-type and kinesin (unc-104) mutant backgrounds, we establish in the living nematode
291 nic lines expressing wild-type CG7780 in the mutant background were generated and subsequently shown
293 sations of ABCB19 and PIN1 in the reciprocal mutant backgrounds were like those in wild type, PIN1 pl
294 tations did not affect virulence in the msbB mutant background when administered orally to BALB/c mic
295 ion and trichome placement occur normally in mutant backgrounds where asymmetry of polarity protein d
297 uorescent kinesin-5, KLP61F-GFP, in a klp61f mutant background, where it rescues mitosis and viabilit
298 n decreased approximately fivefold in a gidA mutant background, with a concurrent decrease in the exp
299 ion, which was broken in the pmr4 disruption mutant background, with callose deposits at the site of
300 nal fusion was placed in 11 of the 12 Tda(-) mutant backgrounds, with cysI being the sole exception.
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