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1 e role of E2F3 in vivo, we generated an E2f3 mutant mouse strain.
2 ele of Dscam to use alongside existing Dscam mutant mouse strains.
3 ating glucose metabolism and fat mass in two mutant mouse strains.
4 e scientific community for the generation of mutant mouse strains.
5 ossed Myo15(sh2/sh2) mice to the three other mutant mouse strains.
6 nous imprinted genes in inbred wild-type and mutant mouse strains.
7 n of (59)Fe absorption in both wild-type and mutant mouse strains.
8 ked compositional differences between WT and mutant mouse strains.
9 els were quantitatively similar in the three mutant mouse strains and normal mice.
10  complementation system (LCS) makes use of a mutant mouse strain, aphakia (ak), homozygotes of which
11           Here we show that odors of two MHC mutant mouse strains (bm1 and bm3) can be distinguished,
12  in individual hippocampi from the mast cell mutant mouse strain, C57BL/6 Kit(W-sh/W-sh).
13                               We generated a mutant mouse strain carrying an F832A mutation in Rb1 th
14 ed a forward genetics approach to identify a mutant mouse strain characterized by the absence of CNS
15                                              Mutant mouse strains deficient in RGS7 or -11 were chara
16         Compared with wild-type mice, 2 FHM1 mutant mouse strains developed earlier onset of anoxic d
17  which are defective in the pallid and muted mutant mouse strains, encode small, coiled-coil-forming
18                                              Mutant mouse strains expressing either p31 or p41 Ii cha
19 y important yet simple approach to establish mutant mouse strains for functional study at defined sta
20                   The recent construction of mutant mouse strains for several of these diseases shoul
21                     Here we describe a novel mutant mouse strain, Goldenticket (Gt), that fails to pr
22                                  At least 15 mutant mouse strains have been classified as models of H
23                                   Studies of mutant mouse strains have revealed that each family memb
24                              We identified a mutant mouse strain, HcB-19/Dem (HcB-19), that shares fe
25                                              Mutant mouse strains in 60 different GPCRs were generate
26                     The fast accumulation of mutant mouse strains in recent years has provided an inv
27                                A conditional mutant mouse strain, in which Pitx2 function is inactiva
28 we allografted carotid artery loops from six mutant mouse strains into immunocompetent CBA/CaJ recipi
29                                              Mutant mouse strains lacking Ii chain expression have be
30          The class II molecules expressed by mutant mouse strains lacking Ii chain or DM activities d
31                              The spontaneous mutant mouse strain, plt/plt, lacks the secondary lympho
32                                              Mutant mouse strains producing only p31 or p41 under con
33                                         This mutant mouse strain provides a unique model to further e
34                        We show that a CRD-BP mutant mouse strain retains expression of the shorter tr
35 er (EOC) in vivo We used the Pten/Kras(G12D)-mutant mouse strain that develops serous EOC with 100% p
36      Surprisingly, our studies of a knock-in mutant mouse strain that expresses a stabilized and tran
37 ological conditions was investigated using a mutant mouse strain that expresses a truncated Cabin1 la
38                               We generated a mutant mouse strain that lacks exons 13-20 of L3mbtl1.
39  strains, ACAID cannot be induced in several mutant mouse strains that are coincidentally deficient i
40 small intestinal phenotype found in most Apc-mutant mouse strains, this strain has been designated th
41 tic defects of GATA-2-/- cells, we interbred mutant mouse strains to assess the effects of p53 loss o
42                                  Here we use mutant mouse strains to investigate E2F3's role in vivo.
43                                            A mutant mouse strain was generated by replacing the IL-2
44 he lungs of infected mice revealed that both mutant mouse strains were only transiently impaired in t
45                                          The mutant mouse strains were subjected to lethal doses of b
46                                              Mutant mouse strains with "autistic-like" phenotypes (Fm
47 e-driven breast tumorigenesis in a series of mutant mouse strains with specific DDR deficiencies to r

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