ライフサイエンスコーパス: mutant strain

1 sistant to oxidants than a catalase-deletion mutant strain.

2 type M59 GAS, we constructed an isogenic mga mutant strain.

3 failed to support growth of an E. coli acpP mutant strain.

4 x II failed to support growth of the E. coli mutant strain.

5 gitis more frequently than did the DeltasfbA mutant strain.

6 sue infected with a wild-type or rim101Delta mutant strain.

7 e higher or detectable only in the RNase III mutant strain.

8 spectrometry, and the use of a p13 deletion mutant strain.

9 onization numbers were 158-fold less for the mutant strain.

10 ) to insert MifM and to complement a SpoIIIJ mutant strain.

11 this as determined by phenotypes of the mbfA mutant strain.

12 he increased resistance of the corresponding mutant strain.

13 e domain, is sufficient to complement a rocA mutant strain.

14 uted to an altered membrane potential in the mutant strain.

15 the DeltawcaM DeltacsgA DeltayihO DeltabcsE mutant strain.

16 ated iron acquisition is impaired in an sbnI mutant strain.

17 gnificantly upregulated in the fabT deletion mutant strain.

18 h a newly characterized curved-rod Delta1228 mutant strain.

19 lated RNA ends in ribonuclease wild-type and mutant strains.

20 and cellular phenotypes of the wild-type and mutant strains.

21 in both the E. coli parent and aceE deletion mutant strains.

22 d A isolated from the corresponding deletion mutant strains.

23 Hsp70 biology has emerged from studying ssa mutant strains.

24 eron, by developing knock-out and functional mutant strains.

25 antially accelerated PR degeneration in both mutant strains.

26 ntly different between the wild type and the mutant strains.

27 ehavior of the system under the injection of mutant strains.

28 ls between the wild-type and rex-inactivated mutant strains.

29 f Deltainv, DeltayadA, and DeltainvDeltayadA mutant strains.

30 present in the wild type but lacking in syp mutant strains.

31 les to F1 pups to yield an allelic series of mutant strains.

32 n to the fitness and evolutionary success of mutant strains.

33 eral recombinant inbred lines and cerebellar mutant strains.

34 these processes can be uncoupled in specific mutant strains.

35 duction was increased in planktonic cells of mutant strains.

36 alize several ciliogenesis phenotypes of IFT mutant strains.

37 ced immunogenicity phenotype of the original mutant strains.

38 a dose-dependent stimulation of response for mutant strains.

39 in and determined the virulence phenotype of mutant strains.

40 xposed to H pylori, including cagPAI and cgt mutant strains.

41 sequences in both wild type and degradosome mutant strains.

42 ainst the wild type virus and drug-resistant mutant strains.

43 did not lead to the development of resistant mutant strains.

44 genetic interactions in single and multiple mutant strains, (2) can identify drug targets, (3) detec

45 Using an E. coli mutant (strain 536DeltafyuA) unable to acquire yersiniab

46 Both WT and mutant strains acquired Fe from Fh, although Fe acquisit

47 A DeltaeseJ mutant strain adheres to epithelioma papillosum of carp

48 Chemical analyses of generated mutant strains allowed for the identification of a trike

49 Comparative metabolomics of different mutant strains allowed to propose a pathway for HAS bios

50 terized straight-rod Deltapgp1 and Deltapgp2 mutant strains, along with a newly characterized curved-

51 The mutant strains also produced very reduced amounts of eth

52 A rhd_2599-tusA-dsrE2-deficient mutant strain, although not viable in liquid culture, wa

53 not observed in response to a cdtB isogenic mutant strain and (2) present in cells expressing CdtB.

54 t (Saccharomyces cerevisiae) SEIPIN deletion mutant strain and a plant (Nicotiana benthamiana) transi

55 ry aspergillosis since a sulfate transporter mutant strain and a sulfite reductase mutant strain are

56 onal asRNAs were identified in the RNase III mutant strain and are encoded primarily opposite to the

57 We then constructed these novel mutant strains and compared their observed phenotypes to

58 rofile across multiple HCV genotypes and key mutant strains and for its good in vitro ADME profiles a

59 cantly increased Agr expression in both hemB mutant strains and S. aureus grown with HQNO and signifi

60 e are increased in both fast and slow Pol II mutant strains and the magnitude of half-life changes co

61 nfections in Drosophila melanogaster involve mutant strains and their reference strains that act as e

62 different inoculation techniques, bacterial mutant strains, and assays for the hypersensitive respon

63 ences were examined in the single and double mutant strains, and the virulence of select strains was

64 troso-guanidine mutagenesis and selection, a mutant strain Apmu4 was derived, in which the rate of lo

65 Akinetes in the mutant strain appeared normal, but these cultures were l

66 porter mutant strain and a sulfite reductase mutant strain are fully virulent.

67 eins as novel antimicrobial targets, and LTA mutant strains as improved probiotics are highlighted.

68 han-normal accumulation of AIP-III in a codY mutant strain, as determined using ultrahigh-performance

69 rocessing of 16S rRNA is also delayed in the mutant strain, as indicated by increased levels of precu

70 Even fecal-recovery numbers for both mutant strains at several time points prior to the anima

71 onsistent with these observations, the H273D mutant strain attenuated GAS virulence, whereas the H273

72 at the espACD regulatory region of the phoP mutant strain because of PhoP-EspR protein-protein inter

73 s study, we determined that an rpoE deletion mutant strain BHM2578 compared to the wild type (WT) was

74 expression decreased in the CN3718 codY-null mutant strain but significantly increased in the SM101 c

75 ucts of cysteine rescue the growth of a thiI mutant strain by a mechanism that requires the transport

76 tes for the limited DDK activity in a cdc7-1 mutant strain by allowing increased, premature phosphory

77 We also analyzed rescue of the mreB mutant strain by overproduction of YvcK in which the pho

78 re infected intravenously with the Deltairs4 mutant, strain CAI-12 (1 x 10(5) CFU), or a mixture of t

79 V. cholerae mutant strains carrying inactivated AI synthase genes we

80 ed to the wild-type strain, a H201R isogenic mutant strain caused significantly larger skin lesions i

81 cytogenes infection, a reduced number of the mutant strain compared to the parental strain was observ

82 on by the epithelium in response to the sapA mutant strain compared to the parental strain.

83 th phase-dependent hierarchies of polymerase mutant strain competitiveness.

84 All madC mutant strains contain loss-of-function point mutations

85 gas was detected in a hydrogenase quadruple-mutant strain containing deletions in the hya, hyb, hyd,

86 vma mutant strains containing rsp5 or rim8 mutations maintai

87 Motility and attachment of a gcbA mutant strain could be restored to wild-type levels via

88 It was previously reported that a thiI mutant strain could grow independent of exogenous thiami

89 ity level, we asked whether matrix-defective mutant strains could be coaxed to produce functional mat

90 Here we generate a new deletion mutant strain, CVS-N2c(DeltaG), and examine its neuronal

91 ingae pv tomato DC3000 and the nonpathogenic mutant strain DC3000hrpA- allowed us to establish causal

92 Mutant strains defective in insulin signaling (daf-2) al

93 riptional profiling of roots inoculated with mutant strains defective in the synthesis of Nod Factor

94 Mutant strains, defective in sRNAs or in ARGONAUTE3 (a k

95 In contrast, both wild-type and mutant strains deficient for peroxiredoxins and glutathi

96 erexpression of COY1 inhibited the growth of mutant strains deficient in fusion activity at the Golgi

97 We found that mutant strains, deficient in the pilus proteins (Deltagb

98 A MakatG1 deletion mutant strain (DeltaMakatG1) showed decreased catalase a

99 riptional profiles were compared between the mutant strain DeltarelA [a (p)ppGpp(0) strain under gluc

100 rium Synechocystis sp. PCC 6803, a series of mutant strains, Deltasll1981, Deltaslr0370, Deltaslr1022

101 Yeast mutant strains DeltaTIM11 and DeltaATP20 (lacking the e

102 gnificantly increased in the SM101 codY-null mutant strain, demonstrating CodY-dependent regulation d

103 nvasion of the WT but not the sfbA-deficient mutant strain, demonstrating the importance of an SfbA-f

104 er, we found that a slowly constricting fzlA mutant strain develops 'pointy' poles, suggesting that F

105 Despite high expression of ACT, the rseA mutant strain did not infect the murine airway as effici

106 This bacterial mutant strain did not stimulate colitis in dnKO mice.

107 addition of a symbiotic partner exudate, the mutant strains differentiated hormogonium-like filaments

108 However, the same mutant strain displayed no survival defect in murine mod

109 Mice immunized with these mutant strains displayed >10-fold protective effects aga

110 the general fitness of the amoebae with the mutant strain displaying a substantial growth defect.

111 The feoB mutant strain does not have an intrinsically faster grow

112 Importantly, the DeltaeseJ mutant strain does not replicate efficiently in EPC cell

113 etween M. tuberculosis H37Rv and a DeltanarL mutant strain during exponential growth in broth culture

114 g. 1-phenyl-2-thiourea, PTU) or pigmentation mutant strains (e.g. casper mutant).

115 Individual DeltatrxA and DeltatrxC deletion mutant strains each show a greater abundance of lipid pe

116 he hypothesis that vaccination with the batA mutant strain elicits protective immunity against subseq

117 ed defects in Mia40 oxidation, only one erv1 mutant strain (erv1-1) had significantly decreased activ

118 The mutant strains exhibit increased levels of phosphorylate

119 Mice infected with this mutant strain exhibited a 4-log-unit reduction in bacter

120 Further, this mutant strain exhibited significantly enhanced survival

121 t H273 and H327 relieved inhibition, and the mutant strains exhibited a wild-type phenotype.

122 We observe that a ste50Delta mutant strain exhibits highly heterogeneous response to

123 n original strategy that combined the use of mutant strains expressing catalytically inactive variant

124 strain expressing both HMW1 and HMW2 and the mutant strains expressing either HMW1 or HMW2 were able

125 monitoring the relative fitness of isogenic mutant strains expressing only one alternative polymeras

126 Three E. coli K-12 gcvB mutant strains failed to demonstrate a change in DCS sen

127 A screen with S. pombe transcription factor mutant strains for growth sensitivity to the AEO fractio

128 We consistently found that mutant strains for RNAi (dcr1Delta, ago1Delta, rdp1Delta

129 ld-type strain and an isogenic fabT deletion mutant strain found that between 3.7 and 28.5% of the S.

130 1;3 and OsPIP2;6 were displayed in yeast HD9 mutant strain (fps1acr3ycf1) as a result of increased B

131 An exogenous EL mimic protected mutant strains from hNP-1 and hBD-2 but not RP-1, indica

132 on techniques can be used to distinguish FKS mutant strains from wild type, but testing C. glabrata w

133 We have sequenced 394 mutant strains, generated in a chemical mutagenesis scre

134 The mutant strain grew normally under iron-limiting conditio

135 for all phenotypes mentioned above, but all mutant strains grew slower than UA159 in medium suppleme

136 orescent reporter proteins revealed that the mutant strain had greatly enlarged peroxisomes up to 10

137 Animals infected with the cagA mutant strain had low levels of gastric inflammation and

138 virulence characteristics, and the quadruple-mutant strain had the same (greatly attenuated) phenotyp

139 The genes disrupted in these mutant strains had disparate predicted functions.

140 Mutant strains harbouring transposon insertions in two s

141 release, and an ato5Delta ATO1(G53D) double mutant strain has additive alkalinization and ammonia re

142 cytotoxin-associated gene A (CagA) isogenic mutant strain HP238(CagAm) failed to induce IFN-gamma re

143 V. cholerae and a locked low-cell-density QS-mutant strain identified 7,240 transcriptional start sit

144 In all mutant strains, impaired maturation of white matter astr

145 The virulence of the mutant strain in a murine model of melioidosis demonstra

146 w that Cu strongly inhibits growth of a copA mutant strain in acidic cultures.

147 experiments with a PEP carboxylase-deficient mutant strain in blood and cerebrospinal fluid.

148 CMV infection resolution and emergence of a mutant strain in high-risk recipients of kidney transpla

149 se4 was stabilized in a Psh1 phosphodepleted mutant strain in which the major phosphorylation sites w

150 t mortality between wild-type and DSB repair mutant strains in any model of infection.

151 re limited data on disease manifestations of mutant strains in children.

152 ded EC50 values <1.0 nM against HIV-1 WT and mutant strains in MT-4 cells.

153 roteome changes of wild type UA159 and lrgAB mutant strains in response to these same stresses.

154 vironments, to characterization of different mutant strains, including those harbouring synthetic cir

155 ested by the hypersensitivity of a Deltasod1 mutant strain, increased resistance afforded by the supe

156 We show that this mutant strain is able to metabolise xylose to acetate on

157 in mouse lung 24 h post infection while the mutant strain is cleared by host defense mechanisms.

158 Congruous with ex vivo findings, a nfu mutant strain is more susceptible to oxidative killing b

159 munity afforded by vaccination with the batA mutant strain is predominantly mediated by IgG antibodie

160 The csfV mutant strain is severely attenuated compared to wild-ty

161 Two Helicobacter mutant strains (katA(H56A) and katA(Y339A)) containing c

162 a mechanism different from that by which the mutant strains kill mice.

163 The mutant strain lacking both SPN and SLO production is sev

164 read changes in transcriptional profile in a mutant strain lacking CRP(Mt) during exponential growth,

165 to colonize more frequently than the double mutant strain lacking HMW1 and HMW2.

166 tion model and that HA levels increased in a mutant strain lacking hyaluronidase (HysA).

167 A mutant strain lacking mapA was constructed and found to

168 by Pseudomonas aeruginosa wild-type versus a mutant strain lacking multiple porins.

169 e wild type were compared with patterns in a mutant strain lacking polynucleotide phosphorylase (PNPa

170 ia and soft tissue infection models, and the mutant strain lacking production of both toxins is furth

171 An S. Typhimurium mutant strain lacking these two effectors exhibits marke

172 Analysis of two mutant strains lacking distinct actin cross-linkers (mhc

173 iffusion in wild-type V. cholerae to that in mutant strains lacking either toxR or the toxT promoter,

174 s lysozyme resistance of L. monocytogenes as mutant strains lacking gpsB showed an increased lysozyme

175 Experiments with mutant strains lacking mitochondrial superoxide dismutas

176 Mutant strains lacking mneP are Mn(II) sensitive and acc

177 Similarly to a carR mutant, strains lacking almE, almF, and almG exhibited e

178 e wild-type phenotype of an Escherichia coli mutant-strain lacking sirohydrochlorin-ferrochelatase ac

179 Mongolian gerbils with an H. pylori pgdA(-) mutant strain led to significantly decreased levels of i

180 on of LipA from Xac306 and verified that the mutant strain lost most of its lipase and esterase activ

181 y the successful isolation of a M. smegmatis mutant strain mc(2)155, whose efficient plasmid transfor

182 ot essential for this organism; however, the mutant strain (MIG1) showed reduced motility and increas

183 The OhrR mutant strain (MSDeltaohrR) showed severalfold upregulat

184 quire a live, stable, lipid-rich E. gracilis mutant strain, named B1ZFeL, with 40% more lipid content

185 clude the generation of large collections of mutant strains, now available for forward- and reverse-g

186 An ibeA deletion mutant strain (NRG857cDeltaibeA) was constructed, and th

187 The DeltarpoE DeltanepR mutant strain of B. quintana established that RpoE-media

188 compared to a DeltarelADeltaspoT (ppGpp(0)) mutant strain of E. coli, deficient in the stringent res

189 sed together in a temperature-sensitive ftsH mutant strain of Escherichia coli were found to be nonfu

190 A mutant strain of L. monocytogenes expressing the cystein

191 Compared to wild-type, a hip1 mutant strain of M. tuberculosis induced enhanced levels

192 easure the mechanical properties of T4P of a mutant strain of Pseudomonas aeruginosa PAO1 unable to r

193 1) from S. atroolivacues SB3033, a DeltalnmE mutant strain of S. atroolivaceus S-140.

194 because killed bacteria and a protease-null mutant strain of S. aureus were unable to penetrate.

195 c-psbA4 gene from Cyanothece 51142 in a 4E-3 mutant strain of the model non-nitrogen-fixing cyanobact

196 We use a dynactin mutant strain of the multinucleate fungus Ashbya gossypi

197 Bs2 gene since no effect was observed when a mutant strain of Xcc with a disruption in avrBs2 gene wa

198 much faster "colony surfing" still occurs in mutant strains of Bacillus subtilis lacking flagella.

199 -producing and a noninvasive hypha-producing mutant strains of C. albicans.

200 xperimental data for different wild type and mutant strains of C. botulinum Group I type A1.

201 rates as well as their ability to detect fks mutant strains of Candida albicans (11 mutants), Candida

202 of photoautotrophically grown wild-type and mutant strains of Chlamydomonas reinhardtii to determine

203 ease progression using Caenorhaditis elegans mutant strains of dnj-14, the worm orthologue of DNAJC5.

204 Anesthetic-resistant mutant strains of Drosophila exhibit a different pattern

205 tive stress resistance of wild-type and putA mutant strains of Escherichia coli.

206 eered capsulated and unencapsulated isogenic mutant strains of groups A, B, C, W, and Y meningococci

207 oughput sequencing (Hi-C) with wild-type and mutant strains of Neurospora crassa to gain insight into

208 Here we show that mutant strains of S. praecaptivus that lack genetic comp

209 Furthermore, mutant strains of T. forsythia, devoid of either mirolys

210 mpared with whole-cell vaccines, circulating mutant strains of the bacterium, and parents refusing va

211 to a toxic concentration of toluene, a tolR mutant strain or a strain overexpressing a diguanylate c

212 did not promote hemolysin expression in hemB mutant strains or S. aureus grown with HQNO.

213 In contrast, in rabbits exposed to the mutant strains or the LPS preparations, the microbial lo

214 iated C4b deposition when IgM binding to the mutant strain pairs was normalized.

215 Using a pde2 mutant strain, pApA was detected for the first time in S

216 On plants, the Deltanmo2 mutant strain penetrated host cuticles like wild type, b

217 Analyses indicate that a flgK-flgL double mutant strain phenocopies deletion of flgN and that over

218 Overexpression of mch in the mtdB mutant strain, predicted to reduce methenyl-dH(4)MPT acc

219 Detailed metabolic profiling of mutant strains produced by systematic inactivation of PK

220 Analysis of the reporter in a p53 mutant strain putatively indicates that the PCDs are a r

221 during the growth of the wild-type and sigL mutant strains reduced expression of the toxin genes, in

222 s upregulated expression of FliC in the fliC mutant strain reduces its virulence.

223 that the majority of cells in a Deltasll1130 mutant strain remained unicellular and viable after prol

224 produce an mif(-/-) strain of L. major This mutant strain replicated normally in vitro but had a 2-f

225 SMG-free, quadruple vic mutant strains representing both allelic backgrounds of

226 n situ reconstitution of fnm in the deletion mutant strain restored adherence.

227 Expression of PutA in the putA mutant strain restored oxidative stress resistance, conf

228 Our analyses of wild-type and mutant strains reveal key elements of chromosome archite

229 ic Cu(+) Analysis of DeltacopR and DeltacueR mutant strains revealed a CopR regulon composed of genes

230 scriptome analysis of the wild-type and dcl3 mutant strains revealed a further difference from higher

231 letion of GCN2 from the eEF1A actin bundling mutant strains revealed a second defect in translation.

232 Analysis of several mutant strains revealed that different thresholds of myo

233 DeltadevR, DeltadevSDeltadosT, and DeltanarL mutant strains revealed that in response to nitrite prod

234 nal carbon; the core oscillator in the prd-1 mutant strain runs with a long period under glucose-suff

235 itch variability is heritable, and comparing mutant strains selectively bred to high and low penetran

236 ated MRSA parental and their respective sarA mutant strain sets.

237 Unexpectedly, the mutant strain showed increased serum resistance.

238 tion in the wild type and an IsoMO-disrupted mutant strain showed that epoxyisoprene, or a subsequent

239 for correct fungal development as the AaGPx3 mutant strains showed a severe reduction in conidiation.

240 EM of the compensatory mutant strains showed complete virus particles, but thes

241 g N. gonorrhoeae 1291 wild type and isogenic mutant strains showed that cytoplasmic LdhA (NAD(+)-depe

242 omycetemcomitans and DeltaihfA and DeltaihfB mutant strains showed that IHF differentially regulates

243 However, in agnA-/agnB- double mutant strains strongly reduced accumulation of extrachr

244 transcripts that were more abundant in dcl3 mutant strains than in wild-type cells were not due to s

245 we describe the genome engineering of a RF1 mutant strain that enhances suppression efficiency durin

246 Specifically, we generated a mutant strain that lacks all 13 PTS transporters, and fr

247 ding increasing concentrations of sulfate to mutant strains that are unable to incorporate H2S effici

248 combine phenotypic and genotypic analyses of mutant strains that suggest discrepancies in the literat

249 In the two mutant strains, the flexural rigidity is not significant

250 . pylori, whereas 5 of 12 mice contained the mutant strain; the mean colonization numbers were 158-fo

251 ully restored the wild-type phenotype of the mutant strain; this indicates that P55 plays no importan

252 still substantially impact the phenotypes of mutant strains through epistasis.

253 or screening mutants and establishing stable mutant strains through genetic crosses.

254 The ability of the mutant strain to acidify the defined medium during growt

255 s did not alter the ability of the Deltapgp2 mutant strain to survive within human epithelial cells o

256 rface lipoprotein-deficient lgt pneumococcal mutant strain to test the hypothesis that lipoproteins a

257 of Chlamydomonas reinhardtii and the use of mutant strains to analyze photosynthesis was conducted i

258 notation of new genes, and the generation of mutant strains to define the role of genes in complex en

259 Here, we utilize P. gingivalis mutant strains to show that pathogen-differentiated mDCs

260 profiles from DeltavttR(A) and DeltavttR(B) mutant strains to the isogenic parent strain confirmed t

261 ssociated with acm allelic replacement (cat) mutant strain TX6051 used in that study.

262 satisfies fluxomic data for wild-type and 25 mutant strains under different substrates and growth con

263 ease in the linear electron transport in the mutant strain versus the wild type, while the cyclic ele

264 The capsule mutant strain was also impaired for survival in guinea p

265 The mutant strain was approximately 10-fold more sensitive t

266 -induced gene in A. pleuropneumoniae, an hfq mutant strain was constructed.

267 beta-galactosidase gene lacZ (WBWlacZ), the mutant strain was defective in colonization compared to

268 Finally, a conditional Bmi1 mutant strain was generated and used to determine the co

269 The B. abortus bab1_1517 mutant strain was significantly attenuated in macrophage

270 Initial stress assays revealed that the putA mutant strain was significantly more sensitive to oxidat

271 hat the immune response to the F. tularensis mutant strains was significantly different from that obs

272 xpression by epithelial cells exposed to psm mutant strains was significantly increased compared to t

273 Using isogenic mutant strains, we demonstrate that deleting one or more

274 Using isoallelic mutant strains, we found that 3 polymorphisms in this to

275 Using FGDelta-mutant strains, we showed that specific combinations of

276 S(0) globules from a Chlorobaculum tepidum mutant strain were purified and used to show that the wi

277 id stress conditions when wild-type and ylxM mutant strains were cultured together.

278 dsRNA from Escherichia coli RNase III WT and mutant strains were deep-sequenced.

279 The mutant strains were examined for CPS quantity, size, and

280 Two mutant strains were generated: one where expression of t

281 Notably, deletion mutant strains were more resistant to ciprofloxacin trea

282 Whereas DeltaagrB mutant strains were not able to produce biofilms, a Delt

283 ptation, exocytosis, and endocytosis in sec3 mutant strains were similarly alleviated by mutation of

284 d Pseudomonas putida G7 Y1, a nonchemotactic mutant strain, were simultaneously introduced into the s

285 egulated by RpoE, and surprisingly, the rseA mutant strain where RpoE activity was elevated expressed

286 In this study, we generated imKO mice, a mutant strain whose LRP4 gene can be deleted in muscles

287 Using a mutant strain with a high differentiation rate and fluor

288 rbidity and mortality, particularly if a CMV mutant strain with antiviral resistance emerges.

289 we constructed a Yersinia pseudotuberculosis mutant strain with arabinose-dependent regulated and del

290 Mutation of pagPBPa resulted in a mutant strain with increased sensitivity to antimicrobia

291 nserted into the C. besciigenome, creating a mutant strain with its S-layer extensively decorated wit

292 mplementation studies of a M. smegmatis glgE mutant strain with these GlgE derivatives, together with

293 generate and phenotypically characterize 29 mutant strains with deletions of individual transporter

294 al phase contrast and confocal microscopy of mutant strains with fluorescently labeled actin and myos

295 type (WT) strain of Escherichia coli K12 and mutant strains with lesions affecting ammonium-assimilat

296 Mutant strains with reduced function in the insulin/IGF-

297 hpA The expression of uhpT was absent in the mutant strains with uhpT deletion and was not inducible

298 ing molecular mass in both the wild-type and mutant strains with various subsets of phenylalanine-gly

299 One (+/0) mutant strain, with multiple mutations at the mating loc

300 -infection of mice with wild type and a feoB mutant strain yielded a different outcome: FeoB is clear