ライフサイエンスコーパス: myelin-associated glycoprotein

1 igodendrocyte-myelin glycoprotein), and MAG (myelin-associated glycoprotein).

2 eled fatty acid and immunostaining of P0 and myelin associated glycoprotein.

3 in the growth cone induced by the repellent myelin-associated glycoprotein.

4 drocyte apoptosis and a preferential loss of myelin-associated glycoprotein.

5 vation and neurite inhibition in response to myelin-associated glycoprotein.

6 wth and overcame growth inhibition caused by myelin-associated glycoprotein.

7 PP1 mediate growth cone repulsion induced by myelin-associated glycoprotein.

8 In line with these data, myelin-associated glycoprotein addition to cerebellar gr

9 go, oligodendrocyte-myelin glycoprotein, and myelin-associated glycoprotein, all bind to an axonal No

10 glecs were known, namely sialoadhesin, CD22, myelin-associated glycoprotein and CD33.

11 ditions, but also promotes neurite growth on myelin-associated glycoprotein and chondroitin sulfate p

12 ing of neuronal growth cones to netrin-1 and myelin-associated glycoprotein and for netrin-1/DCC-depe

13 Immunofluorescence labeling for myelin-associated glycoprotein and for the protein Caspr

14 However, myelin-associated glycoprotein and semaphorin 3A regulat

15 inding of the myelin inhibitors Nogo-A, MAG (myelin-associated glycoprotein), and OMgp (oligodendrocy

16 Three myelin proteins, Nogo, MAG (myelin-associated glycoprotein), and OMgp (oligodendrocy

17 (sialoadhesin), siglec-3 (CD33), siglec-4a (myelin-associated glycoprotein), and siglec-5 to alpha2-

18 ntified constituents of this activity (Nogo, myelin-associated glycoprotein, and oligodendrocyte myel

19 iated, structurally distinct proteins, Nogo, myelin-associated glycoprotein, and oligodendrocyte myel

20 nervous system (CNS) myelin proteins, Nogo, myelin-associated glycoprotein, and oligodendrocyte myel

21 The 7E11 monoclonal antibody blocks Nogo, myelin-associated glycoprotein, and oligodendrocyte myel

22 t or second Ig domains of the I-type lectins myelin-associated glycoprotein, and sialoadhesin did not

23 her known Siglec family members (CD22, CD33, myelin-associated glycoprotein, and sialoadhesin) show t

24 valuated Ig gene usage in patients with anti-myelin-associated glycoprotein (anti-MAG) neuropathy, an

25 usion body myositis and paraproteinemic anti-myelin-associated glycoprotein antibody demyelinating po

26 pain substrates [myelin basic protein (MBP); myelin-associated glycoprotein] are degraded in this dis

27 S-100 antigen as a pan-Schwann cell marker, myelin-associated glycoprotein as a marker for myelinati

28 Myelin-associated glycoprotein binds to LRP1 with high a

29 rin-1, brain-derived neurotrophic factor and myelin-associated glycoprotein, but not to semaphorin 3A

30 action and stabilization of oligodendrocytic myelin-associated glycoprotein by neuronal gangliosides

31 at cleaves one class of axonal receptors for myelin-associated glycoprotein, enhances spinal axon out

32 ets for autoantibodies include gangliosides, myelin associated glycoprotein, Hu antigen and extractab

33 Moreover, the GPR12-mediated rescue from myelin-associated glycoprotein inhibition was attributab

34 elin basic protein and protein zero, whereas myelin-associated glycoprotein is largely unaffected.

35 This shortened induction was also found in myelin-associated glycoprotein knock-out mice, but not i

36 ys revealed that C1q interacts directly with myelin associated glycoprotein (MAG) in myelin, resultin

37 binds the myelin inhibitors NogoA, OMgp, and myelin-associated glycoprotein (MAG) and has been propos

38 In addition to myelin-associated glycoprotein (MAG) and Nogo-A, availab

39 The neural sialoadhesins, myelin-associated glycoprotein (MAG) and Schwann cell my

40 We have analyzed mice that lack both the myelin-associated glycoprotein (MAG) and the myelin gala

41 Myelin-associated glycoprotein (MAG) binds to the nerve

42 brain-derived neurotrophic factor (BDNF), or myelin-associated glycoprotein (MAG) but not by a gradie

43 hmidt-Lantermann (S-L) incisures contain the myelin-associated glycoprotein (MAG) but not P(0) or pro

44 nel of myelin antibodies, we discovered that myelin-associated glycoprotein (MAG) expression is drama

45 trates that mice with a null mutation of the myelin-associated glycoprotein (MAG) gene have a chronic

46 Sox10 synergy, including a novel site in the Myelin-associated glycoprotein (Mag) gene, is also reduc

47 Myelin-associated glycoprotein (MAG) has been implicated

48 I(3,5)P2 deficiency leads to accumulation of myelin-associated glycoprotein (MAG) in LAMP1(+)perinucl

49 Immunocytochemical detection of myelin-associated glycoprotein (MAG) indicated that Schw

50 The myelin-associated glycoprotein (MAG) is a member of the

51 Myelin-associated glycoprotein (MAG) is a potent inhibit

52 Myelin-associated glycoprotein (MAG) is a sialic acid-bi

53 Myelin-associated glycoprotein (MAG) is a transmembrane

54 Myelin-associated glycoprotein (MAG) is an abundant myel

55 Myelin-associated glycoprotein (MAG) is expressed in per

56 Myelin-associated glycoprotein (MAG) is susceptible to p

57 In the injured nervous system, myelin-associated glycoprotein (MAG) on residual myelin

58 n was enhanced, and the repulsion induced by myelin-associated glycoprotein (MAG) or myelin membrane

59 godendrocyte myelin glycoprotein (OMgp), and myelin-associated glycoprotein (MAG) to mediate neurite

60 ic protein (MBP), proteolipid protein (PLP), myelin-associated glycoprotein (MAG), "Rip," and the neu

61 crease in calpain content and degradation of myelin-associated glycoprotein (MAG), a calpain substrat

62 Myelin-associated glycoprotein (MAG), a membrane-bound p

63 e reported in mice with a disrupted gene for myelin-associated glycoprotein (MAG), a myelin receptor

64 Myelin-associated glycoprotein (MAG), a nervous system c

65 Myelin-associated glycoprotein (MAG), a sialic acid bind

66 We now demonstrate that myelin-associated glycoprotein (MAG), a well known inhib

67 Myelin-associated glycoprotein (MAG), an inhibitor of ax

68 Antibodies to P0, myelin-associated glycoprotein (MAG), and neural cell ad

69 und along axons: myelin basic protein (MBP), myelin-associated glycoprotein (MAG), and neuron-glia ce

70 the myelin inhibitory proteins Nogo/Nogo-66, myelin-associated glycoprotein (MAG), and oligodendrocyt

71 '-cyclic nucleotide phosphodiesterase (CNP), myelin-associated glycoprotein (MAG), and P0.

72 mutants, is defective for the expression of myelin-associated glycoprotein (MAG), and the misregulat

73 One inhibitor, myelin-associated glycoprotein (MAG), binds to sialoglyc

74 growth cone turning induced by a gradient of myelin-associated glycoprotein (MAG), cAMP acts by modul

75 igodendrocyte myelin glycoprotein (OMgp) and myelin-associated glycoprotein (MAG), exert their effect

76 For example, the endogenous lectin, myelin-associated glycoprotein (MAG), is reported to bin

77 One glial molecule, the myelin-associated glycoprotein (MAG), located in the ada

78 part to myelin-associated inhibitors such as myelin-associated glycoprotein (MAG), Nogo-A, and oligod

79 nhibitors of regeneration in myelin, such as myelin-associated glycoprotein (MAG), play an important

80 olecules associated with CNS myelin, such as myelin-associated glycoprotein (MAG), represent major ob

81 obulin-like family member lectins (siglecs), myelin-associated glycoprotein (MAG), Schwann cell myeli

82 general and by a specific myelin component, myelin-associated glycoprotein (MAG), than in the same t

83 lated expression of splice variants encoding myelin-associated glycoprotein (MAG), which generates tw

84 eurofascin and NrCAM at sites independent of myelin-associated glycoprotein (MAG)-staining Schwann ce

85 igodendrocytes and oligodendrocyte- derived, myelin-associated glycoprotein (MAG).

86 part, via the cell-surface adhesion protein, myelin-associated glycoprotein (MAG).

87 ed by inhibitory proteins in myelin, such as myelin-associated glycoprotein (MAG).

88 n receptors during chemorepulsion induced by myelin-associated glycoprotein (MAG).

89 d responsiveness to neurotrophin 3 (NT3) and myelin-associated glycoprotein (MAG).

90 ssociated inhibitors of regeneration such as myelin-associated glycoprotein (MAG).

91 owth cone chemotaxis, including netrin-1 and myelin-associated glycoprotein (MAG).

92 limited by two proteins in myelin, Nogo and myelin-associated glycoprotein (MAG).

93 g junctional adhesion molecule-C (JAM-C) and myelin-associated glycoprotein (MAG).

94 Myelin-associated glycoprotein (MAG, Siglec-4) is one of

95 nhibitory cytokines (TNFalpha, IL-1beta) and myelin-associated glycoproteins (MAG, Nogo).

96 Anti-MAG (myelin-associated glycoprotein) neuropathy is a disablin

97 ARIs include myelin-associated glycoprotein, Nogo, oligodendrocyte-my

98 We show that myelin-associated glycoprotein or CNS myelin, in general

99 g molecules--such as myelin-derived Nogo and myelin-associated glycoprotein--or reactive astrocyte-pr

100 ite matter and preferentially became Olig2+, Myelin Associated Glycoprotein-positive, Myelin Basic Pr

101 Others showed that siglec-4a (myelin-associated glycoprotein) prefers Neu5Ac over Neu5

102 Instead, myelin-associated glycoprotein recruited p75 neurotrophi

103 erized siglecs, sialoadhesin, CD22, CD33 and myelin-associated glycoprotein, several new ones, siglec

104 Most remarkable was the finding that myelin-associated glycoprotein (Siglec-4) binds with 500

105 ctions of hypoxia-inducible factors, and the myelin associated glycoprotein to proteolipid protein 1

106 amined the relationship between the ratio of myelin-associated glycoprotein to proteolipid protein 1

107 We previously showed the ratio of myelin-associated glycoprotein to proteolipid protein 1

108 th factor in the context of reduced ratio of myelin-associated glycoprotein to proteolipid protein 1

109 ignificant correlations between the ratio of myelin-associated glycoprotein to proteolipid protein 1

110 matter perfusion, indicated by the ratio of myelin-associated glycoprotein to proteolipid protein 1,

111 Collapsin-1/Semaphorin III/D (Sema III) and myelin-associated glycoprotein trigger repulsive turning

112 Expression of myelin-associated glycoprotein was also decreased by abo

113 The myelin-associated glycoprotein was appropriately sorted

114 in, myelin oligodendrocyte glycoprotein, and myelin-associated glycoprotein, was delayed in the spina

115 by 206.5% while the levels of 68 kDa NFP and myelin-associated glycoprotein were decreased by 42.9 an

116 n overcame neurite outgrowth inhibition from myelin-associated glycoprotein, which was mirrored by ac

117 Indeed, there is an early loss of myelin-associated glycoprotein within the lesion, despit

118 ing of beta1-integrin adhesions triggered by myelin-associated glycoprotein, yet permitted integrin c