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1 he paranode and ultimately the physiology of myelinated nerve.
2 in the spinal cord and peripherally in large myelinated nerves.
3 ly placed, nonmyelinated axon segments along myelinated nerves.
4 l terminals and the juxtaparanodal region of myelinated nerves.
5 response protein 2) transcription factors in myelinated nerves.
6 lial junctions flank the nodes of Ranvier in myelinated nerves.
7 ed to regions before the transition zones of myelinated nerves.
8 Nerve terminal arborizations, arising from myelinated nerves and exhibiting variable morphology, we
10 tor in vivo during development and repair of myelinated nerves explaining the deficiency of myelin ob
11 tional ordering of membrane compounds in the myelinated nerve fiber by means of polarized Raman micro
12 nerve fibers and synthetic gel fibers, a non-myelinated nerve fiber carrying an impulse was treated a
13 sults further demonstrate the important role myelinated nerve fiber degeneration plays in the pathoge
14 ermal nerve fiber density, as well as dermal myelinated nerve fiber density, which persisted througho
22 erest because the global effects of aging on myelinated nerve fibers are more complex and profound th
24 that in aging there is a ubiquitous loss of myelinated nerve fibers from the brain and that fiber lo
25 d the middle-aged monkeys the mean number of myelinated nerve fibers in the anterior commissure is 2.
26 correlated with a reduction in the number of myelinated nerve fibers in the anterior commissure, an i
27 ed the morphology and area number density of myelinated nerve fibers in the cingulate bundle and genu
30 g results in a 20% decrease in the number of myelinated nerve fibers per unit area, while remaining n
31 which may be related to the vulnerability of myelinated nerve fibers to the normal process of aging.
33 sequential nodal and axonal injury of intact myelinated nerve fibers, recapitulating pathologic featu
34 ction, it had no effect on measures of large myelinated nerve fibers, specifically sural or sciatic n
40 14 cutaneous nerve biopsies revealed loss of myelinated nerve fibres (86%), increased regenerative cl
41 phology and molecular architecture of dermal myelinated nerve fibres were examined using immunohistoc
44 gated sodium channels at nodes of Ranvier in myelinated nerves: here, we investigate its role in AIS
51 n depends on specialized membrane domains in myelinated nerve, the node of Ranvier, the paranode, and
53 e alterations in ion channel localization in myelinated nerves; this provides a rationale for the aud
54 etic screens for mutants with disruptions in myelinated nerves, we identified mutations in erbb3 and
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