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1 Contactin is downregulated along the entire myelinated nerve fiber.
2 adhesion molecules, such as MAG, present in myelinated nerve fibers.
3 e fornix, but there is a significant loss of myelinated nerve fibers.
4 rs, associated with a dramatic loss of large myelinated nerve fibers.
5 to distinct subcellular domains in mammalian myelinated nerve fibers.
6 sent a new method to quantify differences in myelinated nerve fibers.
12 erest because the global effects of aging on myelinated nerve fibers are more complex and profound th
13 tional ordering of membrane compounds in the myelinated nerve fiber by means of polarized Raman micro
14 nerve fibers and synthetic gel fibers, a non-myelinated nerve fiber carrying an impulse was treated a
15 sults further demonstrate the important role myelinated nerve fiber degeneration plays in the pathoge
16 ermal nerve fiber density, as well as dermal myelinated nerve fiber density, which persisted througho
18 that in aging there is a ubiquitous loss of myelinated nerve fibers from the brain and that fiber lo
19 d the middle-aged monkeys the mean number of myelinated nerve fibers in the anterior commissure is 2.
20 correlated with a reduction in the number of myelinated nerve fibers in the anterior commissure, an i
21 ed the morphology and area number density of myelinated nerve fibers in the cingulate bundle and genu
25 g results in a 20% decrease in the number of myelinated nerve fibers per unit area, while remaining n
26 sequential nodal and axonal injury of intact myelinated nerve fibers, recapitulating pathologic featu
27 ction, it had no effect on measures of large myelinated nerve fibers, specifically sural or sciatic n
28 which may be related to the vulnerability of myelinated nerve fibers to the normal process of aging.
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