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1  control of cholesterol biosynthesis for CNS myelinogenesis.
2 rescues MBP expression from the beginning of myelinogenesis.
3  reduced in the qkV/qkV mutant during normal myelinogenesis.
4  indicating the essential function of QKI in myelinogenesis.
5 ecifically in myelinating glia during active myelinogenesis.
6 rapid accumulation of MBP mRNA during active myelinogenesis.
7 tegrin receptor signaling may be involved in myelinogenesis.
8 l patterns of oligodendrocyte generation and myelinogenesis.
9 roliferation and (2) delay maturation and/or myelinogenesis.
10 a central role in brain VLCFA metabolism and myelinogenesis.
11 regulator of myelin membrane trafficking and myelinogenesis.
12 vity and proteolipid protein function during myelinogenesis.
13 fic miRNA-regulated checkpoints that control myelinogenesis and a therapeutic role for miR-219 in CNS
14 tes with Sox10 to regulate the initiation of myelinogenesis and acts as a molecular nexus of regulato
15 ts crucial for axon-glial recognition during myelinogenesis and axon-myelin stability.
16 ogression, its molecular targets involved in myelinogenesis are mostly unidentified.
17 ecular mechanisms underlying oligodendrocyte myelinogenesis are poorly defined.
18 ights the significance of PLP1 dosage in CNS myelinogenesis as well as the importance of accurate det
19 n pathways, active during specific phases of myelinogenesis for regulating myelin formation, remain p
20 itical for Schwann cell (SC) development and myelinogenesis in a manner dependent on transmembrane ty
21  promoting accelerated MBP expression during myelinogenesis in a MBP isoform-preferential manner, and
22 g1 is a central regulator of oligodendrocyte myelinogenesis in brain and that axonal recognition and
23 nscription factor, Olig1, in oligodendrocyte myelinogenesis in brain development.
24 liferation of purified OLP in culture and on myelinogenesis in cerebellar slices.
25  which to modulate their differentiation and myelinogenesis in vivo following transplantation.
26 rapping of that membrane around axons during myelinogenesis, or to stabilize the myelin spiral that f
27  attenuated during the most active period of myelinogenesis (P13 and P20).
28  molecular mechanisms for Fyn to control CNS myelinogenesis remain elusive.
29 d axons suggests that NT-3- and BDNF-induced myelinogenesis resulted, at least in part, from expansio
30  pivotal roles to advance OL development and myelinogenesis through Akt/mTOR activation.
31    This peptide may normally function during myelinogenesis to detect conformational changes originat
32 gests a novel mechanism for accelerating CNS myelinogenesis via regulating mRNA metabolism.
33               To determine whether augmented myelinogenesis was associated with increased proliferati
34 f apoptotic cells during the later stages of myelinogenesis were observed.
35            Oligodendrocytes (OLs) engaged in myelinogenesis were selectively vulnerable, in contrast

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