ライフサイエンスコーパス: myelinolysis

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1 wo of three liver transplanted patients with myelinolysis.

2 rognostic factors in central or extrapontine myelinolysis.

3 oencephalopathy (12%, 7/60), central pontine myelinolysis (8%, 5/60), and malignancy (3%, 2/60) were

4 ly ill patients with central or extrapontine myelinolysis and according to available evidence, the pr

5 aemia results in brain lesions demonstrating myelinolysis and cellular necrosis; (b) normalization of

6 gh unproven, a potential for central pontine myelinolysis and rebound intracranial hypertension exist

7 pathology: perivascular astrocyte depletion, myelinolysis, and complement and Ig deposition.

8 EAE suggest a major role for this enzyme in myelinolysis associated with autoimmune demyelinating di

9 ated the clinical and pathologic features of myelinolysis by rapidly reversing hyponatremia in animal

10 Extrapontine myelinolysis can be found isolated or associated with CP

11 admission, and occurrence of central pontine myelinolysis (CPM).

12 Central pontine and extrapontine myelinolysis (CPM/EPM) are severe neurologic complicatio

13 The motor and localizing signs of myelinolysis differ from the generalized encephalopathy

14 untreated hyponatremia against the risk for myelinolysis due to correction of hyponatremia.

15 The exact pathogenesis of myelinolysis has not been determined.

16 this proteinase may play a critical role in myelinolysis in autoimmune demyelinating diseases such a

17 The separation of patients with myelinolysis in central pontine and extrapontine locatio

18 alitis, leucodystrophies and central pontine myelinolysis), infections [malaria, acquired immunodefic

19 Myelinolysis is a neurologic disorder that can occur aft

20 ly ill patients with central or extrapontine myelinolysis is better than thus far thought despite ini

21 Myelinolysis is more likely to occur after the treatment

22 In hypernatraemic rats there was myelinolysis of brain white matter, with karyorrhexis an

23 Central pontine and extrapontine myelinolysis patients experienced higher intraoperative

24 ransplantation (43%, 26/60); central pontine myelinolysis, subdural hematoma, acute infarcts, and Asp

25 Initially named "central pontine myelinolysis," this disease is now known to also affect

26 Manifestations of myelinolysis usually evolve several days after correctio

27 Patients at risk of central myelinolysis who are subjected to aggressive osmolar cor

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