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1 ic blasts obtained from a patient with acute myeloblastic leukemia.
2 Abl oncogene, which is causative for chronic myeloblastic leukemia.
3 enic CD40L to protect against the CD40- WEHI myeloblastic leukemia.
4 d highly in some karyotypically normal acute myeloblastic leukemias.
5 n 40 patients (7 Hodgkin's Disease, 13 Acute Myeloblastic Leukemia, 5 Acute Lymphoblastic Leukemia, 8
6 iocytomas) and six other malignancies (acute myeloblastic leukemia, acute lymphoblastic leukemia, men
8 riteria for complete remission (CR) in acute myeloblastic leukemia (AML) require the absence of perip
9 overall survival (OS) and evolution to acute myeloblastic leukemia (AML) were FAB and WHO CMML subtyp
10 e critical steps for classification of acute myeloblastic leukemia (AML) which influences the selecti
12 lastic leukemia (ALL) and 418 cases of acute myeloblastic leukemia (AML)) with 30,000 contemporaneous
13 ood acute lymphoblastic leukemia (ALL)/acute myeloblastic leukemia (AML), for further postnatal expos
18 ]/Abelson [ABL] kinase inhibitors in chronic myeloblastic leukemia and ATRA in acute promyelocytic le
23 ephosphorylation was determined in the human myeloblastic leukemia cell line HL-60 using subtype-sele
28 tivated protein kinase (MAPK) of HL-60 human myeloblastic leukemia cells before causing myeloid diffe
29 protein synthesis following induction of M1 myeloblastic leukemia cells for terminal differentiation
30 eloid and monocytic differentiation of HL-60 myeloblastic leukemia cells in response to retinoic acid
31 zinc finger transcription factor Egr-1 in M1 myeloblastic leukemia cells promotes terminal differenti
32 identified based on increased expression in myeloblastic leukemia cells undergoing differentiation.
37 le arrest and differentiation of HL-60 human myeloblastic leukemia cells, motivating the present anal
38 radiation to induce DNA damage in human ML-1 myeloblastic leukemia cells, the promoter and intronic r
39 nduced terminal differentiation of murine M1 myeloblastic leukemia cells, where the cells growth arre
42 primarily a 100-kD protein and a CD34+ acute myeloblastic leukemia expressing mainly 130-kD and 145-k
43 cdc25A during terminal differentiation using myeloblastic leukemia M1 cells, that can be induced to u
44 developed myelodysplastic syndrome and acute myeloblastic leukemia (MDS/AML), which raises the questi
46 present study, we found that the exposure of myeloblastic leukemia ML-1 cells to UV light (UVC) cause
48 that an early event in the cell membrane of myeloblastic leukemia (ML-1) cells was the vigorous acti
49 transformation of these disorders into acute myeloblastic leukemia probably relate to the underlying
50 secondary myelodysplasia and secondary acute myeloblastic leukemia, resulting in 15 patient deaths.
51 dence rate of myelodysplastic syndrome/acute myeloblastic leukemia was 0.50% versus 0.07% in (90)Y-ib
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