1 scriptionally repressed at the transition to myelocytes.

2  immature crystalloid granules in eosinophil myelocytes.

3 osegmented neutrophils, consisting mainly of myelocytes and metamyelocytes, was noted.

4 ll population, which includes promyelocytes, myelocytes and metamyelocytes; mean (+/- SD) cell recove

5 ranscription was restricted to promyelocyte, myelocyte, and very early metamyelocyte stages of the gr

6  gangliosides characteristic of neutrophils, myelocytes, and other blood cells.

7 zed by increased bone marrow myeloblasts and myelocytes, as well as extramedullary myelopoiesis.

8 ripheral blood eosinophils and an eosinophil myelocyte cell line (AML14.3D10).

9 actors in nuclear extracts of the eosinophil myelocyte cell line, AML14.3D10.

10 s, SHP-2 and PD-1-SHP-2 signaling restrained myelocyte differentiation resulting in a myeloid landsca

11                           Although roles for myelocytes have been suggested in the pathophysiology of

12 turation from myeloblasts with peak level in myelocytes (MC)/metamyelocytes (MM), when the cells stop

13 pmental stages, including promyelocyte (PM), myelocyte & metamyelocyte (MC & MM), and band & segmente

14 myeloid precursors such as promyelocytes and myelocytes, only mature monocytes and granulocytes were

15 crease in NOX-2S and NOX-2 expression in the myelocyte rather than promyelocyte stages of differentia

16 ll shear stress significantly enhances HL-60 myelocyte rolling on P- and E-selectin at site densities

17 hologies by using both global (AEG-1 KO) and myelocyte-specific knockout (AEG-1DeltaMAC) transgenic m

18 ension yielded minimal maturation beyond the myelocyte stage, after 72 hours of exposure to ATRA on l

19 e granulocytic lineage expressed MEFV at the myelocyte stage, concurrently with lineage commitment.

20 ng with an increase in immature eosinophilic myelocytes that showed abnormal basophilic granulation.

21 rement for C/EBPepsilon for the promyelocyte-myelocyte transition in myeloid differentiation.

22 T signaling, and confining B lymphocytes and myelocytes within their dedicated functional environment