ライフサイエンスコーパス: myeloid cell

1 levels of CYP27B1 compared with homeostatic myeloid cells.

2 the host response to cytosolic viral RNA in myeloid cells.

3 g and proinflammatory cytokine production in myeloid cells.

4 direct interaction of activated T cells and myeloid cells.

5 mors, but is undermined by immunosuppressive myeloid cells.

6 t liver macrophages and infiltrating Gr-1(+) myeloid cells.

7 expression patterns resembling inflammatory myeloid cells.

8 ails broad pro-inflammatory gene programs in myeloid cells.

9 ability, and increased brain infiltration by myeloid cells.

10 ch, in turn, regulated these key outcomes in myeloid cells.

11 ses have been characterized, particularly in myeloid cells.

12 ng this technology, we deleted IRF5 in human myeloid cells.

13 igh degree of heterogeneity among human lung myeloid cells.

14 jury of newly recruited peripherally-derived myeloid cells.

15 aining the phagocytic ability of homeostatic myeloid cells.

16 yte immunoglobulin-like receptors (LILRs) on myeloid cells.

17 R1 and its roles influencing the function of myeloid cells.

18 ressions of alpha7nAChR in differential lung myeloid cells.

19 expression systems and Traf3ip3(-/-) primary myeloid cells.

20 ognition receptor (PRR)-induced cytokines in myeloid cells.

21 mia by invading and replicating in mammalian myeloid cells.

22 self-renewal, and proliferation of immature myeloid cells.

23 ppressive function of bone marrow Ly6C(high) myeloid cells.

24 ion of VDR in intestinal epithelial cells or myeloid cells.

25 amine the role of IL-33 and ST2 signaling in myeloid cells.

26 ccompanied by an infiltration of T cells and myeloid cells.

27 , including triggering receptor expressed on myeloid cells 1 (TREM-1) signalling.

28 n amplifier triggering receptor expressed on myeloid cells-1 (TREM-1) and its putative ligand the neu

29 ils through triggering receptor expressed on myeloid cells-1 (TREM-1) and the ICAM-1(+) neutrophils i

30 and soluble triggering receptor expressed on myeloid cells 2 (sTREM2), as well as a marker of neurona

31 variants of triggering receptor expressed on myeloid cells 2 (TREM2) are linked with an enhanced risk

32 a-expressed triggering receptor expressed on myeloid cells 2 (TREM2) gene increase AD risk 2- to 4-fo

33 Triggering receptor expressed in myeloid cells 2 (TREM2) genetic variants are risk factor

34 Mutation of Triggering receptor expressed on myeloid cells 2 (TREM2) impairs the response of microgli

35 ia receptor triggering receptor expressed on myeloid cells 2 (TREM2) increase AD risk, and activation

36 iant of the Triggering-Receptor-Expressed on Myeloid cells 2 (TREM2) increases the risk of Alzheimer'

37 ed stimulation of the triggering receptor of myeloid cells 2 (TREM2) is on the forefront of these can

38 Microglial triggering receptor expressed on myeloid cells 2 (TREM2) signaling plays pivotal roles in

39 Triggering receptor expressed on myeloid cells 2 (TREM2), a receptor exclusively expresse

40 roglia gene triggering receptor expressed on myeloid cells 2 (TREM2).

41 sing TREM2 (triggering receptor expressed on myeloid cells 2) and displaying a fibrosis-promoting phe

42 A member 1, triggering receptor expressed on myeloid cells 2, CD163; P < 0.05-0.0001), endosomal proc

43 we showed that circulating pro-inflammatory myeloid cells accumulated in atherosclerotic plaques and

44 x found upregulated gene pathways related to myeloid cell activation, myeloid cell migration, and pha

45 We discovered that myeloid cells actively engulf invading living Th17 lymph

46 Arterial myeloid cell adhesion was quantified by intravital micro

47 racterized the phenotypes of CP-infiltrating myeloid cells after transient middle cerebral artery occ

48 We speculate that vascular and myeloid cell ageing synergize, via IL-6 signalling, to a

49 re we show in mice that loss of C9orf72 from myeloid cells alone is sufficient to recapitulate the ag

50 ulate unifying inflammatory features in both myeloid cells and adipocytes and hint at an underappreci

51 alveolar epithelium to metabolically process myeloid cells and advance antibacterial inflammation.

52 rotein and TREM2-regulated function in human myeloid cells and are the first to show a role for MEK1/

53 erized by the excessive production of mature myeloid cells and arise from the acquisition of somatic

54 rgans in vivo and implicate tumor-associated myeloid cells and associated signals as potential therap

55 s is associated with inflammatory changes of myeloid cells and atherosclerosis, but the underlying me

56 Cre induction was highest in myeloid cells and B cells and substantially lower in T c

57 ers of brain populating peripherally-derived myeloid cells and endogenous microglia.

58 Communication between myeloid cells and epithelium plays critical role in main

59 (SLE), these antibodies bind Fc receptors on myeloid cells and induce proinflammatory cytokine produc

60 zed and delivered to the cytoplasm of target myeloid cells and leukemic cells.

61 ore efficient bacterial killing by Ly6B.2(+) myeloid cells and macrophages compared to WT neonates.

62 rive the generation of Tregs and tolerogenic myeloid cells and PD-1 up-regulation in CD8(+) T cells.

63 ed bacterial-induced cytokines in intestinal myeloid cells and PRR-enhanced antimicrobial pathways in

64 ditional ablation of EP2 receptors in immune myeloid cells and show that systemic EP2 antagonism bloc

65 in the tumor microenvironment (TME) in which myeloid cells and T cells were the most abundant stromal

66 nctions by dampening the interaction between myeloid cells and T cells, orthogonal to PD-1 and other

67 th cis-eQTLs have response eQTLs (reQTLs) in myeloid cells and T cells, respectively.

68 ly expressed in circulating and intratumoral myeloid cells and that high IL8 expression is associated

69 onse matrix that can infer representation of myeloid cells and the cytokine environment they encounte

70 x interactions between resident lymphoid and myeloid cells and their communications with the surround

71 eloid cells to cocultures of LACC1-deficient myeloid cells and wild-type CD4(+) T cells restored T-ce

72 tinal microbiota, diminished infiltration by myeloid cells, and an accumulation of intraluminal polya

73 osomal pathway is diminished in C9orf72(-/-) myeloid cells, and blocking STING suppresses hyperactive

74 i-tumor immunity through effects on T cells, myeloid cells, and other cell types within the tumor mic

75 ly augment T cell responses in parallel with myeloid cell- and microbiome-targeted approaches that ma

76 Myeloid cells are a major culprit of IBMIR.

77 Myeloid cells are a vital component of innate immunity a

78 Myeloid cells are critical to the development of fibrosi

79 These myeloid cells are known to secrete several proangiogenic

80 LILRB1 gene expression in lymphoid and myeloid cells arises from two distinct promoters that ar

81 osis and warrants further investigation into myeloid cells as drivers of fibrotic disease.

82 identifies Hyal2-expressing tumor-associated myeloid cells as key players in the accumulation of LMW-

83 ideography to reveal the dynamic behavior of myeloid cells as they interact, extravasate and survey t

84 n significantly decreased brain-infiltrating myeloid cells as well as attenuated cachexia during PDAC

85 of HA-degrading activity of Hyal2-expressing myeloid cells, as the engagement of CD44 receptor with s

86 Unexpectedly, mice without Asxl2 only in myeloid cells (Asxl2DeltaLysM) were completely resistant

87 ology toolbox, enabling the study of complex myeloid cell behaviour dynamically.

88 cation; GAd with the pan-EC-targeting ligand myeloid cell-binding peptide (MBP) incorporated in the c

89 ovides a significant advance in the study of myeloid cell biology, thus enabling pathway discovery an

90 cells (CNS-myeloids) and bone-marrow-derived myeloid cells (BMDMs) cooperatively regulate brain immun

91 follows: (1) CH was consistently present in myeloid cells but varied in penetrance in B and T cells;

92 r target of CD11b-driven Cre was circulating myeloid cells but, unexpectedly, not microglia.

93 isoform is activated by Gi-coupled GPCRs in myeloid cells, but the extent to which the two endogenou

94 equences, viral replication was inhibited in myeloid cells by harnessing the RNA interference pathway

95 with zirconium-89 and fluorophores to detect myeloid cells by in vivo positron emission tomography im

96 While myeloid cells can traffic into the brain via multiple ro

97 that deficiency of angiogenin (Ang) in mouse myeloid cells caused impairment of epithelial barrier in

98 id cells leading to the expansion of certain myeloid cell clones and induces changes in myeloid cell

99 Central nervous system-native myeloid cells (CNS-myeloids) and bone-marrow-derived mye

100 Myeloid cells comprise a major component of the tumor mi

101 monocyte/macrophages, neutrophils, and fetal myeloid cells contained viral RNA and infectious virus,

102 was required for airway inflammation, ST2(+) myeloid cells contributed to exacerbation of airway infl

103 ite adipose tissue inflammation, in part via myeloid cell contribution, is central to obesity pathoge

104 he downregulation of key pathways regulating myeloid cell cycle, maturation and regenerative function

105 This generated a myeloid cell-cytokine-specific response matrix that can

106 l or smooth muscle cells or cardiomyocyte or myeloid cell deficiency of IDO and challenged with acute

107 endent on monocyte recruitment, as mice with myeloid cells deficient in the adhesion molecule integri

108 Knockdown of Wnt5A specifically in the myeloid cells demonstrated a clear decrease in Wnt5A exp

109 Importantly, mice with IRF5 deleted from myeloid cells demonstrated T cell outcomes similar to th

110 role of the histone deacetylase Hdac3 within myeloid cells demonstrated that Hdac3 promotes M2 activa

111 Myeloid cells derived from bone marrow contribute to the

112 Consistent with mouse models, myeloid cells derived from human peripheral blood monocy

113 Mechanistically, myeloid cell-derived angiogenin promoted IEC survival an

114 Meanwhile, myeloid cell-derived IL-33 was required for airway infla

115 In this study, we demonstrate that myeloid cell-derived TGF-beta1 signaling is increased in

116 RNA) that plays a pivotal role in regulating myeloid cell development via targeting HOXA1 gene expres

117 capture three distinct biological processes: myeloid cell differentiation, protein phosphorylation an

118 hial epithelial repair, and implicate ST2 in myeloid cell differentiation.

119 Mice lacking AnxA1 in all cells or only in myeloid cells displayed a defect in this reparative proc

120 While the magnitude and the phenotypes of myeloid cells diverged between tMCAO and TLR2 stimulatio

121 These results reveal mechanisms whereby myeloid cells drive human cancer progression by thwartin

122 Deletion of RIPK3 in myeloid cells, DRP1 or MMP12 suppression in ALI-inflicte

123 ith myocardial infarction, we observed rapid myeloid cell egress from the spleen and bone marrow by i

124 mobilizes immune-suppressive and angiogenic myeloid cells, emerging studies reveal that IL-17 can di

125 tion or growth inhibition and a reduction in myeloid cells endogenously expressing high levels of PD-

126 aNKs reduced levels of macrophages and other myeloid cells endogenously expressing high PD-L1 in peri

127 rrow responds to stress signals by expanding myeloid cells endowed with immunosuppressive functions,

128 f m(6)A methyltransferase subunit METTL14 in myeloid cells exacerbates macrophage responses to acute

129 expressing lower levels of Mcl and Mincle on myeloid cells exhibited a drastic reduction in EAE incid

130 While peripherally-derived myeloid cells exhibited increased process movement in th

131 SCs), a heterogeneous population of immature myeloid cells, expand during chronic viral (HCV, HIV) in

132 s, such as extracellular vesicle release and myeloid cell expansion, in the establishment of pre-meta

133 h was downregulated in both human and murine myeloid cells exposed to LPS as well as other TLR ligand

134 Proinflammatory myeloid cells expressed high levels of CYP27B1 compared

135 ex exhibits increased number of inflammatory myeloid cells expressing cell-surface P2X7Rs.

136 n(+) mesenchymal cells expressing CXCL12 and myeloid cells expressing CXCL12 receptor CXCR4.

137 ffects of this molecule on the regulation of myeloid cells expressing RM CD200R, mimicking interactio

138 Overall, myeloid cells from anti-41BB-treated tumors had an impro

139 ippocampus with macrophages and inflammatory myeloid cells from the periphery, along with elevated fr

140 more often associated with innate immune and myeloid cell function pathways, whereas AA-dominant path

141 patient, we also evaluated the alteration of myeloid cell functional activity.RESULTSWe provide evide

142 ssue and fibroblasts suggests involvement in myeloid cell functions during periodontal inflammation.

143 n myeloid cell clones and induces changes in myeloid cell functions that promote atherosclerosis via

144 Mice with LACC1-deficient myeloid cells had an increased burden of bacteria and al

145 , whereas mutation of REVERBalpha in club or myeloid cells had no effect on the bleomycin phenotype.

146 hat soluble triggering receptor expressed on myeloid cells had the best prognostic accuracy for 30-da

147 production of calcitriol in proinflammatory myeloid cells has the potential to reduce the risk for a

148 Trained immunity, a functional state of myeloid cells, has been proposed as a compelling immune-

149 While myeloid cells have been shown to initiate and maintain r

150 Trx1 system is dispensable for steady-state myeloid-cell hematopoiesis due to their capacity to tap

151 n lymphoid organ formation, in COPD.Methods: Myeloid cell heterogeneity and phenotype were studied in

152 Genetic deletion of the HIF-2alpha gene in myeloid cells (HIF-2alpha(mye/-) ) markedly exacerbated

153 t time to our knowledge, a novel mediator of myeloid cell-IEC crosstalk in maintaining epithelial bar

154 of heterogeneous cells derived from immature myeloid cells (IMCs).

155 Plexin-B2 deletion in myeloid cells impairs corralling, leading to diffuse tis

156 ivery of immunomodulatory compounds to tumor myeloid cells in a variety of cancers.

157 d in myeloid-specific enhancers, implicating myeloid cells in AD etiology.

158 Although radiotherapy remodels myeloid cells in both models, only transplant tumors are

159 immunotherapies targeting immunosuppressive myeloid cells in CCA.

160 ation, we can delineate biological roles for myeloid cells in different cytokine environments during

161 ells, which has implications for the role of myeloid cells in disease and the utilization of these ce

162 The critical role of suppressive myeloid cells in immune regulation has come to the foref

163 ant advancement in understanding the role of myeloid cells in lung cancer.

164 LACC1 expression by myeloid cells in mice is required to clear bacteria and

165 Depleting CD4 T cells led to increased myeloid cells in peripheral blood, spleen, and bone marr

166 However, the true requirement for myeloid cells in PH development has not been demonstrate

167 To study the role of myeloid cells in the central nervous system (CNS) in the

168 Microglia are the resident myeloid cells in the central nervous system (CNS).

169 Myeloid cells in the fetus were not depleted in this exp

170 IDO1 induction in myeloid cells in the inducible bronchus-associated lymph

171 ty affects the expressions of alpha7nAChR in myeloid cells in the lung.

172 ocytes, suggest that the sustained influx of myeloid cells in the lungs of juvenile mice during acute

173 these data suggest that monocyte/macrophage myeloid cells in the placenta play a significant role in

174 s associated with their proximity to CD73(+) myeloid cells in tumor tissue.

175 s reveal a new role for TGFbeta signaling on myeloid cells in tumorigenesis.

176 ith target immune cells and to accumulate in myeloid cells in tumours and systemic compartments.

177 ate immune cells, including ILC2s and ST2(+) myeloid cells, in RSV infection-triggered pathophysiolog

178 Myeloid cells included 2 populations of proinflammatory

179 iglec-15 was more broadly expressed on human myeloid cells, including human osteoclasts.

180 mice inoculated with control ZIKV.IMPORTANCE Myeloid cells, including monocytes, play a crucial role

181 In addition, ACE overexpression in myeloid cells increases their immune function.

182 These results suggest that increased myeloid cell infiltration contributes to autoreactive CD

183 The investigation of myeloid cell infiltration in HCC, NET and intrahepatic C

184 CCL7 blockade in mice reduced myeloid cell infiltration into the kidney and ameliorate

185 ST2 deficiency in mice leads to reduced lung myeloid cell infiltration, abnormal alternatively activa

186 bust proinflammatory cytokine expression and myeloid cell infiltration.

187 Thus, IRF5 acts as a regulator of myeloid cell inflammatory cytokine production during IAV

188 Myeloid cells interact with intestinal epithelial cells

189 or the roles of PML-RARalpha in transforming myeloid cells into leukemia cells, but further uncover a

190 tivates STAT3 and the infiltration of CD11b+ myeloid cells into the lung.

191 These data establish that myeloid cell-intrinsic IRF5 regulates multiple distinct

192 However, how myeloid cell-intrinsic IRF5 regulates the multiple disti

193 recent study by Strauss et al. describes how myeloid cell-intrinsic PD1 signaling limits myelopoiesis

194 systems regulate development and function of myeloid cells is barely understood.

195 In addition, IL-33 signaling in myeloid cells is crucial for airway inflammation.

196 e MS lesions and that activation of STAT3 in myeloid cells is essential for leukocyte infiltration, n

197 this costimulatory axis in tumor-associated myeloid cells is poorly understood.

198 iscuss how ageing affects the development of myeloid cells leading to the expansion of certain myeloi

199 Myeloid cell leukemia 1 (Mcl-1) has emerged as an attrac

200 Incubation with the myeloid cell leukemia 1 (MCL1) inhibitor S63845, a proap

201 How myeloid cell lineage affects activation states in respon

202 correlated with increased expression of the myeloid cell lineage-specific transcription factor IRF8

203 in clone-specific expansion within different myeloid cell lineages.

204 Mice deficient for LKB1 in myeloid cells (LysM-cre x Stk11fl/fl ) or neutrophils (M

205 human TB, and overlap between fibroblast and myeloid cell markers in tissues.

206 ing in supporting remyelination by promoting myeloid cell-mediated inflammatory responses via TNF-alp

207 pathways related to myeloid cell activation, myeloid cell migration, and phagocytosis.

208 in-3 and key adhesome members PXN/LPXN limit myeloid cell motility and phagocytosis, thereby providin

209 ly, we focus on pathways that raise systemic myeloid cell numbers and modulate immune cell phenotypes

210 ysregulated immune responses involving T and myeloid cells occur in COVID-19 patients with severe cli

211 a lack of type I IFNs, reduced HLA-DR in the myeloid cells of patients with severe COVID-19, and tran

212 atory response mediated by plaque-associated myeloid cells of the brain.

213 itulated by IL-10Ralpha deletion in CD11c(+) myeloid cells or local IL-10Ralpha blockade.

214 e immunity, induced via modulation of mature myeloid cells or their bone marrow progenitors, mediates

215 We previously reported that myeloid cells, particularly dendritic cells, from the th

216 Brain-resident microglia and myeloid cells (perivascular macrophages) are important H

217 f evidence suggests that bone marrow-derived myeloid cells play a critical role in the pathophysiolog

218 Myeloid cells play critical and diverse roles in mammali

219 monstrated, and a specific disease-promoting myeloid cell population has not been identified.

220 T cell engager leads to changes in the host myeloid cell population, both of which contribute to tre

221 on, specifically reducing cytokine-producing myeloid cell populations in Irf5 (-/-) mice but not impa

222 To better characterize these different myeloid cell populations, we used long-term in vivo 2-ph

223 nd pairings shaped the distribution of blood myeloid cell populations.

224 hors identify resident microglia as the only myeloid cells present at beta-amyloid deposits.

225 Both CD4(+) T cells and myeloid cells produced pathogenic levels of VEGF-A withi

226 ld be targeted in LSCs to normalize leukemic myeloid cell production.

227 alpha-dependent factor(s) which regulate the myeloid cells proliferation.

228 These findings demonstrate that myeloid cells provide a major source of Wnt5A to facilit

229 ely, these data reveal that tumor-associated myeloid cells provide signals critical for T-ALL growth

230 Instead, myeloid cells provide signals that directly support T-AL

231 overed that deletion of TGFbeta signaling on myeloid cells (PyMT/TGFbetaRII(LysM)) affects extracellu

232 ine axis, which may orchestrate inflammatory myeloid cell recruitment and expression of damage mediat

233 We report that CX3CR1-CCR2-mediated myeloid cell recruitment contributes to stroke injury.

234 oxinemia, thus suggesting that NETs regulate myeloid cell recruitment.

235 ther, we show that microglia, not peripheral myeloid cells, release IL-1alpha ex vivo.

236 recruitment to sites of injury or pathology, myeloid cells represent therapeutic targets for a broad

237 Hence, by dissecting how different myeloid cells respond to cytokine activation, we can del

238 ic approaches aimed at resolving detrimental myeloid cell responses in tissues, including those occur

239 AREG enhanced myeloid cell responses via inducing chemokine and colony

240 nt mice decreases the number of inflammatory myeloid cells, restores cortical dendritic spine dynamic

241 with this possibility, in vivo depletion of myeloid cells results in a significant reduction in leuk

242 ence in mice that lack Beclin 1 or FIP200 in myeloid cells results in spontaneous immune activation a

243 However, it is unknown whether innate myeloid cells retain memory of prior antigenic stimulati

244 ompounds with known pharmacology using human myeloid cells, searching for those that enhance TREM2 pr

245 Here, we describe a myeloid cell-selective NF-kappaB inhibitor using an miR-

246 ic plasticity and convergence among distinct myeloid cells so that they may act as a functional unit

247 Myeloid cell-specific deletion of the neutrophil cytosol

248 one marrow transplantation, and in mice with myeloid cell-specific EGFR deficiency.

249 Our results demonstrate that myeloid cells, specifically cells of the nonclassical mo

250 fies a previously unrecognized requisite for myeloid cell STAT3 in the activation of myelin-reactive

251 lncRNA LUCAT1 which is upregulated in human myeloid cells stimulated with lipopolysaccharide and oth

252 ne (soluble triggering receptor expressed on myeloid cells [sTREM-1], interleukin-6, interleukin-8, c

253 Little is known about the roles of myeloid cell subsets in kidney injury and in the limited

254 Two myeloid cells subsets.

255 receptor CD200R which is highly expressed on myeloid cells such as macrophages and neutrophils.

256 ate and adaptive immune responses that prime myeloid cells, such as macrophages, protect against path

257 Increased cellular ATP underpins increased myeloid cell superoxide production and phagocytosis asso

258 Our results show the potential of using myeloid cell-targeted miR-146a mimics for the treatment

259 SCs, and its deletion transformed MDSCs into myeloid cells that activated CD8(+) T cell-mediated immu

260 ) are a heterogeneous population of immature myeloid cells that proliferate in the setting of cancer

261 Monocytes are among the major myeloid cells that respond to Toxoplasma, a ubiquitous f

262 ct populations of nerve-resident homeostatic myeloid cells that transcriptionally differed from centr

263 plications in the view of the growing use of myeloid cell therapies to treat brain disease in humans.

264 C formation, involving a direct contact with myeloid cells through CD40L-CD40 interaction and IFN-gam

265 Tumour-infiltrating myeloid cells (TIMCs) are critical regulators of cancer

266 Complementation of cytokines produced by myeloid cells to cocultures of LACC1-deficient myeloid c

267 These findings demonstrate the ability of myeloid cells to directly react to pathogenic T cell inf

268 10 from effector T cells signals to CD11c(+) myeloid cells to suppress an atypical and pathogenic IFN

269 Macropinocytosis is essential for myeloid cells to survey their environment and for growth

270 TLR2 agonist rapidly recruited myeloid cells to the CP, increased leukocytosis in the C

271 sure through recruitment of PD-L1-expressing myeloid cells to the wound site.

272 itch of IL6 signaling from a canonical mode (myeloid cells) to a noncanonical trans-signaling mode (a

273 oro-crown ether payload ((19)F-HDL) to allow myeloid cell tracking by (19)F magnetic resonance imagin

274 To understand the mechanisms of myeloid cell trafficking via the CP and their influence

275 Analysis of myeloid cell transcriptomics revealed that ADAMTS5 is en

276 poral and spatial effects of MI on different myeloid cell types.

277 supporting immunoregulatory polarization of myeloid cells upon infiltration into tumors remain large

278 tibodies or conditional depletion of Insr in myeloid cells using the Cre-loxP system protects mice fr

279 costimulatory signals from tumor-associated myeloid cells,v leading to enhanced TIL expansion and fu

280 The proliferation of myeloid cells was further promoted by osteocytes lacking

281 The fragmentation of HA by myeloid cells was mediated by the membrane-bound enzyme

282 y, selective abrogation of EGFR signaling in myeloid cells was sufficient to protect against nephriti

283 The suppressive capacity of myeloid cells was tested in cocultures with autologous l

284 ion to IL-10's classic inhibitory effects on myeloid cells, we also describe the nonclassic roles att

285 B, T, and myeloid cells were analyzed before anti-CD20 administrat

286 Gene networks enriched for myeloid cells were anticorrelated with IL-15 treatment a

287 Increased numbers of Hyal2(+)CD11b(+) myeloid cells were detected in the tumor tissue as well

288 We found myeloid cells were elevated in the placenta of pregnant

289 Overall, not T cells, but myeloid cells were most strongly enriched in AD, and we

290 Myeloid cells were normal in number and function.

291 of chronic autoimmune neuritis, homeostatic myeloid cells were outnumbered by infiltrating lymphocyt

292 It is also well-expressed by myeloid cells, where its role is unknown.

293 AD risk genes are specifically expressed in myeloid cells, whereas others are ubiquitously expressed

294 roglia as well as newly recruited peripheral myeloid cells, which has implications for the role of my

295 taplegic homolog 7 (SMAD7) in CD34(+)PRLR(+) myeloid cells, which reduced the production of transform

296 L-1beta, and IL-6)-conditioning cytokines by myeloid cells, which then cross-regulated Th2 and regula

297 pply of bone marrow-derived brain-engrafting myeloid cells with donor wild-type CSF1R to repopulate t

298 evealed a wide heterogeneity of infiltrating myeloid cells with increased infiltration of PD-L1(+) TA

299 ng the migratory patterns of CP-infiltrating myeloid cells with intact and disrupted CX3CR1-CCR2 sign

300 tal cells, which are analogous to vertebrate myeloid cells, yet molecular underpinnings of the lymph