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1 multiplex, 4 myopathy, 3 motor neuropathy, 2 myelopathy).
2 opical spastic paraparesis/HTLV-I-associated myelopathy.
3 sc herniation can result in acute or chronic myelopathy.
4 e, of whom none had hearing loss, ataxia, or myelopathy.
5 + T cells in patients with HTLV-I-associated myelopathy.
6 adult T-cell leukemia and HTLV-1-associated myelopathy.
7 ine, after development of objective signs of myelopathy.
8 opical spastic paraparesis/HTLV-I-associated myelopathy.
9 copper deficiency usually co-occurring with myelopathy.
10 here were no localizing signs or evidence of myelopathy.
11 adiological, and neurophysiological signs of myelopathy.
12 uggests that spondylosis is the cause of the myelopathy.
13 r NMO-IgG may lead to an alternate cause for myelopathy.
14 nal damage in a reversible phase of cervical myelopathy.
15 hic lateral sclerosis, and radiation-induced myelopathy.
16 ll leukemia and tropical spastic paraparesis/myelopathy.
17 opical spastic paraparesis/HTLV-1-associated myelopathy.
18 T lymphocytes that may lead to leukemia and myelopathy.
19 tion; some had acute monophasic or relapsing myelopathy.
20 myelitis, and unspecified neurodegenerative myelopathy.
21 riteria for BD; (2) clinically suggestive of myelopathy; (3) simultaneous spinal cord and brain magne
22 han other causes of longitudinally extensive myelopathy (50/156 (32%) vs 0/66 (0%); p</=0.001) but di
25 ical spine stenosis and symptomatic cervical myelopathy after decompressive surgery using (18)F-FDG P
26 eurodegenerative disorder, HTLV-I associated myelopathy (also known as tropical spastic paraparesis),
27 T-cell leukemia (ATL) and HTLV-1 associated myelopathy, also called tropical spastic paraparesis (HA
28 number of inflammatory conditions, including myelopathy, although the majority of individuals who are
30 tropical spastic paraparesis-HTLV-associated myelopathy and ATLL or healthy carriers may be relevant
32 apsin response-mediator protein-5 autoimmune myelopathy and occult neoplasia are important considerat
36 ly diagnosed with neoplastic or inflammatory myelopathies, and decompressive surgery was delayed by a
38 (MRIs) within 1 month of acute worsening of myelopathy; and (4) follow-up duration >/= 1 year after
44 ed with an increased risk of HTLV-associated myelopathy, but little is known about variation in HTLV-
46 cortex of patients with cervical spondylotic myelopathy (CSM) by using proton magnetic resonance (MR)
48 measure of severity of cervical spondylotic myelopathy (CSM), which will be of use in determining th
50 l, older patients with degenerative cervical myelopathy (DCM) are felt to have lower recovery potenti
51 ventions for patients with acute and chronic myelopathies depend on preclinical animal models of inju
52 s, number of steps taken over this distance, myelopathy disability index (MDI), and Nurick scores.
59 erized by recurrent optic neuritis, cervical myelopathy from syringomyelia, paraparesis, amenorrhea-g
61 us type 1 (HTLV-I) causing HTLV-I-associated myelopathy (HAM)/tropical spastic paraparesis (TSP)] and
65 causes T-cell leukemia and HTLV-I-associated myelopathy in humans, has been determined by NMR methods
67 spinal cord thinning in chronic progressive myelopathies, including human T-lymphotropic virus 1 (HT
68 sociated neuroinflammatory diseases, notably myelopathy induced by retrovirus human T leukemia virus-
69 ell lymphotropic virus-I (HTLV-I)-associated myelopathy is a slowly progressive neurologic disease ch
72 ephalopathy, chorea, brain stem dysfunction, myelopathy, mononeuritis multiplex, Guillain-Barre-like
73 long myelopathy (n = 9), both long and short myelopathy (n = 2) and short myelopathy (n = 2) was obse
76 esis (HAM/TSP) is a progressive inflammatory myelopathy occurring in a subset of HTLV-1-infected indi
77 patients with: (1) longitudinally extensive myelopathy of other cause (n=66) and (2) myelitis in the
78 patients in whom serological evaluation for myelopathy of uncertain cause demonstrated collapsin res
79 ntrol patients had multiple sclerosis, other myelopathies, optic neuropathies, and miscellaneous diso
81 Felty's syndrome, cervical spine fusion for myelopathy, or total knee arthroplasty at hospitals in C
82 been observed in other forms of longitudinal myelopathy outside of BD, including neuromyelitis optica
83 opical spastic paraparesis-HTLV-1-associated myelopathy patients contain a Lys at position 88 in some
84 s, myoclonus, or both), cerebellar syndrome, myelopathy, peripheral neuropathy, cranial neuropathy, m
85 ation: multilevel decompression, preexisting myelopathy, pulmonary disease, cardiovascular disease, h
86 sing frequency): neuropathy, encephalopathy, myelopathy, stiff-man phenomena, and cerebellar syndrome
87 mphotropic virus type 1 (HTLV-1) -associated myelopathy/tropic spastic paraparesis is a demyelinating
90 neuroinflammatory disease HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) and ad
91 luding multiple sclerosis, HTLV-I associated myelopathy/tropical spastic paraparesis (HAM/TSP) and ch
92 n T-lymphotropic virus 1 (HTLV-1)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) and mu
93 ymphotropic virus type 1 (HTLV-1)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) and mu
94 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) are kn
95 lymphotropic virus (HTLV) type I-associated myelopathy/tropical spastic paraparesis (HAM/TSP) based
96 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) exhibi
97 virus type I-associated (HTLV-I-associated) myelopathy/tropical spastic paraparesis (HAM/TSP) has be
98 ll leukemia virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is a c
99 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is a n
100 ymphotropic virus type 1 (HTLV-1)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is a p
101 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is an
102 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is an
103 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is an
104 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) is ass
105 eukemia/lymphoma (ATL) and HTLV-I associated myelopathy/tropical spastic paraparesis (HAM/TSP) is poo
106 ymphotropic virus type I (HTLV-I)-associated myelopathy/tropical spastic paraparesis (HAM/TSP) prolif
107 (+) Tregs in patients with HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP) result
108 sult in the development of HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP), a chr
109 ymphotropic virus type 1 (HTLV-1)-associated myelopathy/tropical spastic paraparesis (HAM/TSP), a dis
110 us type I (HTLV-I) develop HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP), an im
111 four of five patients with HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP), mono-
112 1 (HTLV-1) is the agent of HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP), obser
127 tory neurological disease (HTLV-I-associated myelopathy/tropical spastic paraparesis [HAM/TSP]) is su
128 adult T cell leukemia and HTLV-1-associated myelopathy/tropical spastic paraparesis after the initia
129 gically distinct diseases: HTLV-1-associated myelopathy/tropical spastic paraparesis and adult T cell
130 T-cell lymphotropic virus type I-associated myelopathy/tropical spastic paraparesis has been reporte
131 T cell lymphotropic virus type I-associated myelopathy/tropical spastic paraparesis is a chronic pro
132 T-cell lymphotropic virus type I-associated myelopathy/tropical spastic paraparesis patients using r
134 iral load in patients with HTLV-I-associated myelopathy/tropical spastic paraparesis suggest that HTL
135 ent in 12 individuals with HTLV-1-associated myelopathy/tropical spastic paraparesis than in 29 asymp
136 , the inflammatory disease HTLV-1-associated myelopathy/tropical spastic paraparesis was associated w
137 Our analysis showed that HTLV-1-associated myelopathy/tropical spastic paraparesis was associated w
138 l nervous system, HAM/TSP (HTLV-I-associated myelopathy/tropical spastic paraparesis), by reducing th
139 ase human T cell leukemia virus-1-associated myelopathy/tropical spastic paraparesis, A6 also recogni
140 tis sicca in patients with HTLV-1-associated myelopathy/tropical spastic paraparesis, and HTLV-1-asso
141 tral nervous system (CNS), HTLV-I-associated myelopathy/tropical spastic paraparesis, characterized p
142 human T cell lymphotropic virus I-associated myelopathy/tropical spastic paraparesis, rheumatoid arth
159 f whom 39 (83%) had hearing loss, ataxia, or myelopathy; type 2 (secondary) iSS included 17 patients
160 g T cells in patients with HTLV-I-associated myelopathy was determined using MHC class I tetramers lo
162 wing criteria were included: (1) spondylotic myelopathy was suspected, (2) gadolinium enhancement was
163 it to define "clinical symptoms of traumatic myelopathy with no radiographic or computed tomographic
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