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1 b')2 at different activities (close to acute myelotoxicity).
2 b')2 at different activities (close to acute myelotoxicity).
3 vity groups the reduction was close to acute myelotoxicity.
4 d to evaluate the radioimmunotherapy-related myelotoxicity.
5 plete remission rates of 35% to 42%, without myelotoxicity.
6 had limited success because of dose-limiting myelotoxicity.
7 ntial value of several factors in predicting myelotoxicity.
8 ology were used to assess hematotoxicity and myelotoxicity.
9 counts at nadir was used as an indicator of myelotoxicity.
10 utics for preventing chemotherapy-associated myelotoxicity.
11 time and absence of pathways contributing to myelotoxicity.
12 w progenitor cells may contribute to benzene myelotoxicity.
13 ionship between topotecan exposure (AUC) and myelotoxicity.
14 correlation between the red marrow dose and myelotoxicity.
15 ndertaken to determine the factors affecting myelotoxicity after radioimmunotherapy (RAIT) with 131I-
18 es < or = 250 cGy resulted in < or = grade 2 myelotoxicity and a red marrow dose of 450 cGy resulted
19 combining these drugs increases the risk of myelotoxicity and may require granulocyte colony-stimula
20 observed vulnerability to antibiotic-induced myelotoxicity and prolonged neutropenia after nonmyeloab
21 gamma-H2AX values are associated with acute myelotoxicity and secondary blood malignancy may be wort
24 kely to have marrow targeting, prediction of myelotoxicity by conventional body and blood contributio
25 m (a key determinant of azathioprine-induced myelotoxicity) by using TPMT enzyme activity to establis
27 clonal antibodies, it has been reported that myelotoxicity cannot be predicted on the basis of the am
28 e title compound (14) showed reduced in vivo myelotoxicity compared to linezolid in a 14-day safety s
29 very of an oxazolidinone possessing improved myelotoxicity compared to linezolid, linezolid-like effi
32 oactivity method was not a good predictor of myelotoxicity for non-marrow-targeting (90)Y-antibody th
38 d leukemic transformation, and assessment of myelotoxicity in preclinical drug development requires a
41 hat 4-OH-CPA Cmax may determine its level of myelotoxicity indicates that the therapeutic index could
42 of red marrow radiation is important because myelotoxicity is often dose limiting in radioimmunothera
43 filtration rate, and common toxicity such as myelotoxicity is prevented with the use of hematopoietic
44 cent chemotherapy as significant factors for myelotoxicity may be important in the future design of c
45 re resistant to antifolates, suggesting that myelotoxicity occurs late in hematopoietic development.
46 okines to circumvent the acute dose-limiting myelotoxicity of cancer treatment, little is known about
48 lysis of the toxicity experiments showed the myelotoxicity of CPA was found to be closely correlated
49 sed to accurately predict absorbed doses and myelotoxicity of radioimmunotherapy with (177)Lu-cG250.
52 explore a possible role for apoptosis in the myelotoxicity of the phenolic metabolites of benzene.
54 ps are active in this area either to improve myelotoxicity profile of linezolid or to expand the spec
57 statin with cyclophosphamide would have less myelotoxicity than combinations using other purine analo
63 study provides a simple model for predicting myelotoxicity with reasonable accuracy in most patients.
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