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1 ates, in particular via the second messenger myo-inositol 1,4,5-trisphosphate, and phosphoinositides
2 e of large cholangiocytes by activation of D-myo-inositol 1,4,5-trisphosphate/Ca(2+) /calmodulin-depe
5 ability of human Arf1.GDP (Arf1.GDP) to bind myo-inositol (1,4,5)-trisphosphate (I(1,4,5)P(3)), the s
6 itol 1,3,4-trisphosphate (Ins(1,3,4)P3) or D-myo-inositol 1,4,5-trisphosphate (Ins(1,4,5)P3) over 80-
7 ic activity partial agonists at the platelet myo-inositol 1,4, 5-trisphosphate [Ins(1,4,5)P(3)] recep
8 brevicompactum, are potent agonists at the D-myo-inositol-1,4,5-trisphosphate [Ins(1,4,5)P3] receptor
10 ine)-mediated enhancement of intracellular D-myo-inositol 1,4,5-trisphosphate (IP(3)) and Ca(2+) accu
11 t inhibition of endoplasmic reticulum (ER) d-myo-inositol 1,4,5-trisphosphate (IP(3))-gated calcium r
15 igh affinity to intestinal SMC; stimulated D-myo-inositol-1,4,5-trisphosphate (IP3) formation, Ca2+ r
16 llular activity of phospholipase C-gamma, by myo-inositol 1,4,5-trisphosphate radioreceptor assay, de
17 phostin A (AdA) is a potent agonist of the d-myo-inositol 1,4,5-trisphosphate receptor (Ins(1,4,5)P3R
18 siently transfected with type I and type III myo-inositol 1,4,5-trisphosphate receptor (IP(3)R) isofo
20 ate (8), two analogues of the superpotent 1D-myo-inositol 1,4,5-trisphosphate receptor agonist adenop
21 hostin A (AdA), the most potent agonist of d-myo-inositol 1,4,5-trisphosphate receptors (IP(3)R), is
23 henylborate, a cell-permeant antagonist of D-myo-inositol 1,4,5-trisphosphate receptors, abrogates an
25 pase C products, namely diacylglycerol and d-myo-inositol 1,4,5-trisphosphate, were identified as pro
26 A variety of inositol phosphates including myo-inositol 1,4,5-trisphosphate, which is a secondary m
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