ライフサイエンスコーパス: myocardial blood flow

1 y flow reserve equals stress divided by rest myocardial blood flow).

2 uring the absolute level of rest and maximal myocardial blood flow.

3 their WT is commensurate with the degree of myocardial blood flow.

4 h intervention for determination of regional myocardial blood flow.

5 he role played by chronic reduction of basal myocardial blood flow.

6 icrocirculation and affect the regulation of myocardial blood flow.

7 in regulation and/or spatial distribution of myocardial blood flow.

8 ive metabolism despite restoration of normal myocardial blood flow.

9 ng allografts may be reflective of decreased myocardial blood flow.

10 ent normalized LV stroke volume and improved myocardial blood flow.

11 ease, whether contractile reserve depends on myocardial blood flow.

12 signals play an important part in regulating myocardial blood flow.

13 ul primary PCI predict MVI and decreased PET myocardial blood flow.

14 mography permits noninvasive quantitation of myocardial blood flow.

15 eatment is associated with an improvement in myocardial blood flow.

16 onary perfusion pressure, cardiac index, and myocardial blood flow.

17 to assess myocardial perfusion and quantify myocardial blood flow.

18 mechanisms of uptake of markers for regional myocardial blood flows.

19 In normal segments, baseline myocardial blood flow (0.95+/-0.32) increased (P<0.001)

20 thin 20 min was greater in ischemic regions (myocardial blood flow, 0.28 +/- 0.26 mL.min(-1).g(-1)) t

21 .26 mL.min(-1).g(-1)) than in normal tissue (myocardial blood flow, 0.52 +/- 0.19 mL.min(-1).g(-1)) (

22 nal mechanical function and PET for regional myocardial blood flow ([(15)O]water) and oxygen consumpt

23 Mean +/- sem left ventricular myocardial blood flow 2 mins after each respective drug

24 Stress myocardial blood flow (2.25+/-0.46 versus 1.78+/-0.43 mL

25 g field by real-time MCE correlate well with myocardial blood flow and can identify coronary stenosis

26 Regional myocardial blood flow and contractile function in ischem

27 nia positron emission tomography to quantify myocardial blood flow and coronary flow reserve.

28 Myocardial blood flow and coronary reserve were normal i

29 Hyperglycemia is associated with altered myocardial blood flow and energetics and can lead to a p

30 Resting myocardial blood flow and EPI, HED and phenylephrine ret

31 PET with [13N]ammonia measured myocardial blood flow and flow reserve in 15 patients wi

32 Myocardial blood flow and flow reserve showed significan

33 this investigation was to determine whether myocardial blood flow and flow reserve, based on quantit

34 Myocardial blood flow and glucose utilization were asses

35 ubidium-82 allows quantification of absolute myocardial blood flow and may have utility for risk stra

36 Likewise, myocardial blood flow and microvascular function in the

37 Baseline positron emission tomography myocardial blood flow and myocardial flow reserve were r

38 maging are likely attributable to changes in myocardial blood flow and myocardial oxygen supply-deman

39 ptake of (99m)Tc-N-NOET reflects reperfusion myocardial blood flow and not viability in a canine mode

40 ium with MCE is best using quantification of myocardial blood flow and provides improved accuracy com

41 the effects of dobutamine stress on regional myocardial blood flow and relative myocardial 99mTc-sest

42 However, with restoration of myocardial blood flow and separation from cardiopulmonar

43 culated as the ratio of hyperemic to resting myocardial blood flow and subdivided according to the pr

44 Myocardial blood flow and substrate metabolism were meas

45 ynamic effects of transplant vasculopathy on myocardial blood flow and vasomotion.

46 itative assessment of coronary perfusion and myocardial blood flow, and discuss their application in

47 To determine whether regional differences in myocardial blood flow are associated with regional dysfu

48 ed techniques for noninvasive measurement of myocardial blood flow are now available.

49 onal recovery was Peak MCIxbeta, an index of myocardial blood flow (area under the curve, 0.83).

50 l dimensions may result in abnormal regional myocardial blood flow as assessed by stress-induced myoc

51 lation between (11)C-PIB retention index and myocardial blood flow as measured with (11)C-acetate was

52 urately using noninvasive CMR-based absolute myocardial blood flow assessment than with invasive coro

53 ardiovascular magnetic resonance and reduced myocardial blood flow at positron emission tomography (P

54 In abnormal segments, myocardial blood flow at rest (0.73+/-0.19) increased at

55 graphy [N(13)]-ammonia, with quantitation of myocardial blood flow at rest and after adenosine stress

56 nervation and nitrogen-13 ammonia to measure myocardial blood flow at rest and after intravenous admi

57 PET with [13N]ammonia was used to measure myocardial blood flow at rest and during adenosine and d

58 Myocardial blood flow at rest and during adenosine-induc

59 Degree of hypertrophy, myocardial blood flow at rest and during hyperemia (hMBF

60 le reserve depends, in part, on the level of myocardial blood flow at rest and during inotropic stimu

61 Myocardial blood flow at rest in abnormal segments (0.50

62 Myocardial blood flow at rest was lower in contractile r

63 Using [(13)N]ammonia, we measured myocardial blood flow at rest, during adenosine-induced

64 hydroxyephedrine ([(11)C]HED) and to measure myocardial blood flow at rest, during hyperemia, and in

65 as an index of the observed heterogeneity of myocardial blood flow averaged, for [13N]ammonia, 9 +/-

66 t echocardiography permits the evaluation of myocardial blood flow both at rest and during pharmacolo

67 ion tomography (PET) not only as a tracer of myocardial blood flow but also as a marker of cell membr

68 cintigraphy assesses disparities in regional myocardial blood flow but does not directly detect hypox

69 Estimates of myocardial blood flow by [13N]ammonia were highly linear

70 g therapy increases stenotic segment maximal myocardial blood flow by approximately 45%.

71 otocol was used, including quantification of myocardial blood flow by N-13 ammonia.

72 low reserve (CFR; CFR=stress divided by rest myocardial blood flow) by positron emission tomography a

73 ctors were analyzed per short axis slice and myocardial blood flow calculated with a two-compartment

74 dial perfusion and determination of absolute myocardial blood flow can be achieved noninvasively usin

75 atial resolution and the fact that it tracks myocardial blood flow changes, it seems to have higher s

76 In steal segments, myocardial blood flow declined versus baseline (P<.001)

77 In the 8 patients with coronary steal, myocardial blood flow decreased from 90 +/- 18 ml/100 g/

78 In patients, analysis of regional myocardial blood flow demonstrated decreased myocardial

79 Correlation of VI and myocardial blood flow (determined by use of fluorescent

80 Baseline and follow-up hyperemic myocardial blood flow did not differ (11.8% +/- 9.4%; p

81 Neither basal skeletal muscle nor basal myocardial blood flow differed across groups; both skele

82 accuracy in terms of location and extent of myocardial blood flow differences as well.

83 The model accounts for heterogeneity in myocardial blood flow, differences in transport rates of

84 Myocardial blood flow distribution is known to be hetero

85 Analysis of myocardial blood flow distribution revealed two distinct

86 Hypothyroidism resulted in impaired myocardial blood flow due to a dramatic loss of arteriol

87 erfusion imaging in assessing alterations in myocardial blood flow due to coronary artery disease (CA

88 entify regional reductions in full-thickness myocardial blood flow during global coronary vasodilatio

89 Lower regional myocardial blood flow during hyperemia was associated wi

90 d and was associated with a 40% reduction in myocardial blood flow during treadmill exercise, whereas

91 detect the effects of regional variations in myocardial blood flow during vasodilation.

92 amics, isolated myocyte length (KOH method), myocardial blood flow (fluorescent microspheres), arteri

93 Redistribution of myocardial blood flow, from a region supplied by a sever

94 In this context, measurement of myocardial blood flow gives unique diagnostic informatio

95 At follow-up, stress myocardial blood flow had increased (P</=0.01) and hiber

96 Dynamic changes of myocardial blood flow have been observed after reperfusi

97 els, the absolute levels of rest and maximal myocardial blood flow have yet to be incorporated into r

98 during ischemia without a change in regional myocardial blood flow, heart rate, or systolic blood pre

99 d residual scatter bias for accurate cardiac myocardial blood flow imaging were 3-14 MBq/kg, 1.5-4.0,

100 graphy (PET) 13N-ammonia was used to measure myocardial blood flow in combination with 18F-fluorodeox

101 two approaches yield comparable estimates of myocardial blood flow in humans, which supports the vali

102 True myocardial blood flow in ischemic (IS) and nonischemic (

103 st and reproducible measurements of regional myocardial blood flow in milliliters per minute per gram

104 lation during adenosine infusion showed that myocardial blood flow in neuropathic subjects was virtua

105 Dobutamine significantly increased myocardial blood flow in regions with and without a dobu

106 myocardial blood flow demonstrated decreased myocardial blood flow in territories supplied by stenoti

107 Baseline regional myocardial blood flow in the area at risk (AAR), measure

108 Normalized myocardial blood flow in the central infarct zone fell f

109 positron emission tomography measurements of myocardial blood flow in the evaluation and management o

110 ncrease in vascularization more than doubled myocardial blood flow in the infarcted myocardium.

111 ary pressure, idazoxan had no effect on mean myocardial blood flow in the LAD region (0.86 +/- 0.17 m

112 Regional myocardial blood flow in the reperfused infarct was redu

113 Correction of rest myocardial blood flow in viable asynergic segments, only

114 Ischemic zone (left circumflex) myocardial blood flows (in ml/min/g) were: baseline, 0.9

115 In normal segments, myocardial blood flow increased (P<.001) with dobutamine

116 Myocardial blood flow increased during the first stage o

117 After simvastatin, myocardial blood flow increased more compared with pretr

118 tile dysfunction have true reduction in rest myocardial blood flow is controversial.

119 tion of blood flow demonstrated that resting myocardial blood flow is reduced in hibernating myocardi

120 n cardiac and systemic hemodynamic function, myocardial blood flow, left ventricular wall thickening

121 produces no changes in hemodynamic function, myocardial blood flow, left ventricular wall thickening

122 itron emission tomography studies to measure myocardial blood flow (MBF) (in ml/g/min) at rest (MBFr)

123 In the control LCx zone, maximal myocardial blood flow (MBF) (measured by radioactive mic

124 enables near-simultaneous quantification of myocardial blood flow (MBF) and anatomical evaluation of

125 m destruction/refilling curves with regional myocardial blood flow (MBF) and contractile function.

126 is suggested that the integration of maximal myocardial blood flow (MBF) and coronary flow reserve (C

127 lows accurate, noninvasive quantification of myocardial blood flow (MBF) and coronary flow reserve (C

128 and neuronal (nNOS) NO synthase isoforms on myocardial blood flow (MBF) and coronary flow reserve (C

129 There is evidence that the quantitation of myocardial blood flow (MBF) and coronary flow reserve (C

130 tomography to quantify resting and hyperemic myocardial blood flow (MBF) and CVR.

131 The purpose of this study was to assess myocardial blood flow (MBF) and flow reserve in systemic

132 (DSE) were performed to quantitate regional myocardial blood flow (MBF) and function.

133 ines the effects of inotropic stimulation on myocardial blood flow (MBF) and glucose metabolism (MRGl

134 However, the extent of recovery of myocardial blood flow (MBF) and its potential physiologi

135 acquisition of PET enabled quantification of myocardial blood flow (MBF) and MFR using a previously v

136 Myocardial blood flow (MBF) and MVO(2) were measured dur

137 Myocardial blood flow (MBF) and myocardial flow reserve

138 The quantification of myocardial blood flow (MBF) and myocardial flow reserve

139 Absolute myocardial blood flow (MBF) and myocardial flow reserve

140 ventricular systolic and diastolic function, myocardial blood flow (MBF) and myocardial water content

141 Mismatch between areas of reduced myocardial blood flow (MBF) and reduced myocardial inner

142 ever, evidence for regional abnormalities in myocardial blood flow (MBF) and the potential mechanisms

143 quantifying subendocardial and subepicardial myocardial blood flow (MBF) and the relative coronary fl

144 ility of these 2 methods to quantify altered myocardial blood flow (MBF) and transmural distribution

145 Myocardial blood flow (MBF) and vasodilator reserve were

146 ssion tomography imaging was used to measure myocardial blood flow (MBF) at rest, during adenosine-in

147 e reduces cardiac work and may thereby lower myocardial blood flow (MBF) at rest.

148 eral models for the quantitative analysis of myocardial blood flow (MBF) at stress and rest and myoca

149 Absolute quantitation of myocardial blood flow (MBF) by PET is an established met

150 We measured pre- and post-treatment myocardial blood flow (MBF) by positron emission tomogra

151 Myocardial blood flow (MBF) can then be calculated from

152 riglyceride levels (r=0.84), while hyperemic myocardial blood flow (MBF) decreased (r=-0.64).

153 i-Ethnic Study of Atherosclerosis (MESA) had myocardial blood flow (MBF) determined using cardiac mag

154 its functional consequence-that is, blunted myocardial blood flow (MBF) during stress.

155 of reconstruction algorithms on quantitative myocardial blood flow (MBF) estimation.

156 of misregistration on the quantification of myocardial blood flow (MBF) has not been studied.

157 e (RFR) is defined as the ratio of hyperemic myocardial blood flow (MBF) in a stenotic area to hypere

158 ia permits the noninvasive quantification of myocardial blood flow (MBF) in humans.

159 entricular channels created by laser improve myocardial blood flow (MBF) in the treated zones.

160 The ability to noninvasively evaluate murine myocardial blood flow (MBF) in vivo would provide an imp

161 normal coronary angiograms but with impaired myocardial blood flow (MBF) increases to cold pressor te

162 Impaired hyperemic myocardial blood flow (MBF) is associated with increased

163 detecting coronary artery disease (CAD) when myocardial blood flow (MBF) is quantified in absolute te

164 ubstantial controversy as to whether resting myocardial blood flow (MBF) is reduced in such circumsta

165 Myocardial blood flow (MBF) is the critical determinant

166 r injection and radioactive microspheres for myocardial blood flow (MBF) measurement.

167 ns of the AIF were compared with microsphere myocardial blood flow (MBF) measurements at linear regre

168 ector material may pave the way for absolute myocardial blood flow (MBF) measurements by SPECT.

169 ritories, and then compared with microsphere myocardial blood flow (MBF) measurements.

170 alterations of cardiac sympathetic tone and myocardial blood flow (MBF) regulation in subjects with

171 Routine quantification of myocardial blood flow (MBF) requires robust and reproduc

172 tachycardia unmasking a reduced endocardial myocardial blood flow (MBF) reserve is the mechanism of

173 the same because both drugs unmask abnormal myocardial blood flow (MBF) reserve.

174 Myocardial blood flow (MBF) responses to cold pressor te

175 Within 20 days, myocardial blood flow (MBF) responses to CPT were measur

176 study were to determine whether responses in myocardial blood flow (MBF) to the cold pressor testing

177 We quantified absolute myocardial blood flow (MBF) using a spin-labeling MRI (S

178 In group I patients, MCE-derived myocardial blood flow (MBF) velocity reserve (2.4+/-0.08

179 d by the rate of intensity rise (b) by QMCE; myocardial blood flow (MBF) was assessed by fluorescent

180 Myocardial blood flow (MBF) was assessed by microspheres

181 Basal and adenosine-stimulated myocardial blood flow (MBF) was determined by PET: after

182 In addition, myocardial blood flow (MBF) was determined quantitativel

183 Myocardial blood flow (MBF) was determined using radiola

184 Quantitative myocardial blood flow (MBF) was estimated from rest and

185 Myocardial blood flow (MBF) was measured by imaging (15)

186 Myocardial blood flow (MBF) was measured using magnetic

187 Myocardial blood flow (MBF) was measured using radiolabe

188 Myocardial blood flow (MBF) was measured with positron e

189 erated from the normal and stenosed beds and myocardial blood flow (MBF) was measured with radiolabel

190 Myocardial blood flow (MBF) was quantified with [13N]amm

191 Basal and adenosine-stimulated myocardial blood flow (MBF) were determined by positron

192 ty (VI) and radiolabeled microsphere-derived myocardial blood flow (MBF) were measured serially after

193 efore recanalization, the risk area (RA) and myocardial blood flow (MBF) were measured, and in vivo t

194 We measured myocardial blood flow (MBF) with (13)N-ammonia and PET a

195 We measured myocardial blood flow (MBF) with a PET scan at rest, dur

196 PET absolute myocardial blood flow (MBF) with H(2)15O and 13NH3 are w

197 Noninvasive measurements of myocardial blood flow (MBF) with PET revealed an abnorma

198 of myocardial oxygen consumption (MVO2) and myocardial blood flow (MBF) with PET.

199 ing regions with adequate collateral-derived myocardial blood flow (MBF) within the risk area (RA), w

200 all (PW) thickness, thickening, quantitative myocardial blood flow (MBF), and MBF reserve were measur

201 , left ventricular ejection fraction (LVEF), myocardial blood flow (MBF), and myocardial flow reserve

202 Myocardial blood flow (MBF), myocardial oxygen consumpti

203 tron emission tomography for measurements of myocardial blood flow (MBF), myocardial oxygen consumpti

204 d by increases in regional contractility and myocardial blood flow (MBF), particularly in the infarct

205 ssure of CO2 (PETco2) increases cerebral and myocardial blood flow (MBF), suggesting that it may be a

206 Systemic and coronary hemodynamics, myocardial blood flow (MBF), whole blood viscosity (WBet

207 ng intravenous adenosine (ADO) infusion, and myocardial blood flow (MBF).

208 The product A . beta represents myocardial blood flow (MBF).

209 w enable absolute quantification of regional myocardial blood flow (MBF).

210 d pool attenuation) with microsphere-derived myocardial blood flow (MBF).

211 emission tomography for the determination of myocardial blood flow (MBF); myocardial oxygen consumpti

212 ty study was undertaken to determine whether myocardial blood flow (MBF, mL/g/min) could be quantifie

213 al coronary pressure to 52 +/- 3 mm Hg, mean myocardial blood flow measured with microspheres was 0.8

214 dard liquid meal on whole heart and regional myocardial blood flow, measured by means of dynamic posi

215 (VEGF), endothelial progenitor cell assays, myocardial blood flow measurements, and histopathologic

216 positron emission tomographic metabolic and myocardial blood flow measurements, assessment of gene e

217 compared the effects of arbutamine stress on myocardial blood flow, myocardial MIBI uptake, and systo

218 annels), we measured mean arterial pressure, myocardial blood flow, myocardial tissue oxygen tension,

219 Myocardial blood flow of the anterior left ventricular w

220 High HMR was associated with decreased myocardial blood flow on PET (myocardial perfusion reser

221 There were no differences in myocardial blood flow or oxygen delivery among groups; h

222 y altering electrical stability or impairing myocardial blood flow, or both.

223 commonly used tool for the quantification of myocardial blood flow, other modalities, including singl

224 Hemodynamics, myocardial blood flow, oxygen, and lactate metabolism we

225 SPECT perfusion imaging delineates relative myocardial blood flow, patients with global left ventric

226 y makes robust, quantitative measurements of myocardial blood flow possible are highlighted.

227 al and global ventricular function, absolute myocardial blood flow quantification, and myocardial tis

228 ere euthanized after measurement of regional myocardial blood flow (radioactive microspheres) and in

229 cular pacing-induced heart failure, regional myocardial blood flow (radioactive microspheres) and reg

230 PET data can be used effectively to compare myocardial blood-flow rates at rest and stress levels.

231 Regional myocardial blood flow ratios between the injection site

232 We tested the hypothesis that the myocardial blood flow reserve (MBFR) is decreased during

233 Myocardial blood flow reserve may be impaired by cardiov

234 owever, little is known about the underlying myocardial blood flow response (MBF) in these patients.

235 However, myocardial blood flow response to cold pressor test incr

236 s in humans with ischemic heart disease that myocardial blood flow response to dobutamine is linearly

237 fasting plasma insulin levels decreased, and myocardial blood flow responses to cold pressor test nor

238 Myocardial blood flow responses to dipyridamole were sim

239 In humans with ischemic heart disease, myocardial blood flow responses to dobutamine and adenos

240 estigated the hemodynamic, neurohumoral, and myocardial blood flow responses to mental stress in 17 p

241 aim of this study was to correlate regional myocardial blood flow (RMBF) derived from [15O]H2O PET w

242 s in regional myocardial perfusion (regional myocardial blood flow [RMBF]), as measured by colored mi

243 nificantly higher levels of left ventricular myocardial blood flow than either vasopressin alone or e

244 nfarcted tissues demonstrated higher pre-CAO myocardial blood flow than salvaged tissues.

245 tamine resulted in a significant increase in myocardial blood flow that correlated significantly with

246 ography, based on a functional assessment of myocardial blood flow, thereby guiding antiischemic and

247 0.17 mL.min-1.g-1), but LNNA decreased mean myocardial blood flow to 0.49 +/- 0.09 (P < .01).

248 LNNA administration, idazoxan increased mean myocardial blood flow to 0.62 +/- 0.13 mL.min-1.g-1 (P <

249 Cold increased myocardial blood flow to 0.79 +/- 0.18 mL.min-1.g-1 in c

250 ooth muscle play a critical role in coupling myocardial blood flow to cardiac metabolism.

251 d deposits and with (11)C-acetate to measure myocardial blood flow to study the impact of global and

252 LNNA also reduced myocardial blood flow to the normal region during exerci

253 o myocardial viability beyond its value as a myocardial blood flow tracer.

254 tion in diabetes is associated with abnormal myocardial blood flow under rest and adenosine-stimulate

255 n of 11C-acetate for absolute measurement of myocardial blood flow using a simple compartmental model

256 entricular MFR was calculated as stress/rest myocardial blood flow using Rb-82 positron emission tomo

257 Microsphere-measured myocardial blood flow validated the perfusion results.

258 Myocardial blood flow velocity reserve correlated signif

259 However, myocardial blood flow velocity reserve in patients with

260 demonstrated significantly (P<0.0001) lower myocardial blood flow velocity reserve in vascular terri

261 After the development of CHF, resting myocardial blood flow was 25% lower than normal (P<0.05)

262 ients (>1 year after surgery, n=10), resting myocardial blood flow was also homogenous.

263 global and regional uptake values, and then myocardial blood flow was derived using the Renkin-Crone

264 Myocardial blood flow was determined in regions correspo

265 g model-independent deconvolution, hyperemic myocardial blood flow was evaluated, and ischemic burden

266 Rest myocardial blood flow was higher in the neuropathic subj

267 Mean myocardial blood flow was higher in the transplant patie

268 t baseline and all doses of norepinephrine), myocardial blood flow was lower in Kv1.5(-/-) mice than

269 Hyperemic myocardial blood flow was lower in patients than in cont

270 Myocardial blood flow was measured (colored microspheres

271 Myocardial blood flow was measured by microspheres and t

272 Myocardial blood flow was measured by radioactive micros

273 Myocardial blood flow was measured by using positron emi

274 Myocardial blood flow was measured in absolute units wit

275 One week after permanent LAD occlusion, myocardial blood flow was measured with microspheres dur

276 MCE was performed and myocardial blood flow was measured with neutron-activate

277 Myocardial blood flow was quantified with the use of 13N

278 During ischemia, regional myocardial blood flow was reduced 84% at the endocardium

279 Mean absolute myocardial blood flow was significantly higher in region

280 After revascularization, hyperemic myocardial blood flow was significantly higher in segmen

281 adenosine infusion, global left ventricular myocardial blood flow was significantly less in the neur

282 Myocardial blood flow was similar in patients without di

283 r, LV angiograms, hemodynamics, and regional myocardial blood flow were assessed.

284 etermined cardiac index and left ventricular myocardial blood flow were lower with 10% and 20% leanin

285 Measurements of myocardial blood flow were made with PET [13N]ammonia in

286 Resting and maximum (adenosine) myocardial blood flow were reduced in both PTU-S and PTU

287 ean myocardial flow reserve, and mean stress myocardial blood flow were significant predictors of adv

288 Rest and stress myocardial blood flows were calculated with factor analy

289 Nonischemic zone myocardial blood flows were: baseline, 0.99 +/- 0.18; st

290 rast agent, it cannot provide information on myocardial blood flow when injected directly into a coro

291 tes did not appear to contribute to regional myocardial blood flow, which may be a limitation of gene

292 Myocardial blood flow with 11C-acetate was obtained by f

293 However, estimates of myocardial blood flow with [13N]ammonia in humans have n

294 Brain and myocardial blood flow with active compression-decompress

295 Steal was defined as a decline in myocardial blood flow with adenosine >/=0.15 mL.min-1.g-

296 eart failure and the potential for improving myocardial blood flow with associated enhancement of reg

297 Dynamic imaging for quantification of myocardial blood flow with short-lived tracers, such as

298 CFR was quantified from stress/rest myocardial blood flow with the use of positron emission

299 DAN is associated with altered myocardial blood flow, with regions of persistent sympat

300 e hypothesized that sympathetically mediated myocardial blood flow would be impaired in diabetics wit