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1 signal generated in the atria and ends with myocardial contraction.
2 propagates electrical impulses to coordinate myocardial contraction.
3 index (SWI) and MRI radial strain quantified myocardial contraction.
4 the regulation of basal and beta-adrenergic myocardial contraction.
5 ties in the heart that would directly affect myocardial contraction.
6 echanism of anesthetic-induced depression of myocardial contraction.
7 lial cells release substances that influence myocardial contraction.
8 es acutely, indicating diminished economy of myocardial contraction.
9 ssential chemical energy source that governs myocardial contraction.
10 dium to extract a metric that reflects local myocardial contraction.
12 the cushions expanding in a wave during peak myocardial contraction and becoming undetectable during
13 vely spliced cTnT variants may desynchronize myocardial contraction and contribute to the pathogenesi
14 myocardial perfusion are related to delayed myocardial contraction and greater extent of dyssynchron
15 e found that overexpressing SERCA2a enhances myocardial contraction and relaxation in normal mouse he
18 patients who died during SE had identifiable myocardial contraction bands compared with 5 of 22 contr
24 as despite significantly improved indexes of myocardial contraction (end-systolic pressure volume rel
27 itivities and high specificities (among them myocardial contraction fraction ratio had the highest ar
29 ression alters the energy cost of increasing myocardial contraction in mouse hearts with pressure-ove
32 of the myofibrillar proteins, activation of myocardial contraction is thought to involve switch-like
34 To quantify ventricular size/shape, regional myocardial contraction, lateral shortening of the IPMD,
35 However, the initial proposal that depressed myocardial contraction leads to a 'compensatory' hypertr
36 re is associated with decreased longitudinal myocardial contraction measured by pulsed tissue Doppler
37 erformance beginning shortly after the first myocardial contractions occur in developing vertebrate e
38 ene blue to examine cGMP-mediated effects on myocardial contraction-relaxation, while other experimen
40 implications for the role of this protein in myocardial contraction, with particular relevance to bet
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