ライフサイエンスコーパス: myocardial ischemia

1 nt, repetitive coronary artery occlusion and myocardial ischemia).

2 l new targets for diagnosis and treatment of myocardial ischemia.

3 al ischemia, and (3) no ECG signs indicating myocardial ischemia.

4 MI, or urgent coronary revascularization for myocardial ischemia.

5 tle is known about lncRNAs in the setting of myocardial ischemia.

6 ed cardiac functional recovery after chronic myocardial ischemia.

7 the presence or absence of stress-inducible myocardial ischemia.

8 stress, thereby providing information about myocardial ischemia.

9 d arrhythmia vulnerability in the setting of myocardial ischemia.

10 in myocyte death and cardiac function after myocardial ischemia.

11 oxygen consumption leading to arrhythmia and myocardial ischemia.

12 uced wall motion abnormalities suggestive of myocardial ischemia.

13 s in which angiogenesis is impaired, such as myocardial ischemia.

14 nce of cardiac function and remodeling after myocardial ischemia.

15 d arrhythmia vulnerability in the setting of myocardial ischemia.

16 ise treadmill tests were performed to detect myocardial ischemia.

17 ainly studied patients with exercise-induced myocardial ischemia.

18 cal characteristics of mental stress-induced myocardial ischemia.

19 the left circumflex artery to induce chronic myocardial ischemia.

20 ruction, but is limited for the detection of myocardial ischemia.

21 ging (CTP) to detect atherosclerosis causing myocardial ischemia.

22 thermia may decrease arrhythmogenesis during myocardial ischemia.

23 of young adults evaluated by angiography for myocardial ischemia.

24 ion as a potential new strategy for treating myocardial ischemia.

25 onary angiography for evaluation of possible myocardial ischemia.

26 etecting obstructive atherosclerosis causing myocardial ischemia.

27 DA-approved biomarker for early diagnosis of myocardial ischemia.

28 option for the treatment of acute or chronic myocardial ischemia.

29 , BOLD magnetic resonance imaging can detect myocardial ischemia.

30 ng LV preloads, increased contractility, and myocardial ischemia.

31 udden death, especially when associated with myocardial ischemia.

32 ly viable alternative treatment strategy for myocardial ischemia.

33 -inducible gene therapy for the treatment of myocardial ischemia.

34 xhibit the highest accuracy for diagnosis of myocardial ischemia.

35 s improved survival in response to permanent myocardial ischemia.

36 ll-cell coupling in instances of anticipated myocardial ischemia.

37 ion), making RAS a new therapeutic target in myocardial ischemia.

38 in regional coronary blood flow in areas of myocardial ischemia.

39 swine with hypercholesterolemia and chronic myocardial ischemia.

40 via an intravenous injection 24 hours before myocardial ischemia.

41 leading cause of death in patients suffering myocardial ischemia.

42 vel therapeutic avenue for the prevention of myocardial ischemia.

43 ng on 254 patients referred with symptoms of myocardial ischemia.

44 or renal disease might actually precipitate myocardial ischemia.

45 ve (FFR-CT) is a novel, noninvasive test for myocardial ischemia.

46 arget for secondary disease prevention after myocardial ischemia.

47 T-segment depression (P=0.003) and therefore myocardial ischemia.

48 show that H2S levels increase in vivo during myocardial ischemia.

49 f transient ischemic attack (TIA)/stroke and myocardial ischemia.

50 closure on coronary collateral function and myocardial ischemia.

51 outcome depends on the extent of reversible myocardial ischemia.

52 ery disease but often correlates poorly with myocardial ischemia.

53 role in treating heart failure, angina, and myocardial ischemia.

54 be the coronary perfusion territories during myocardial ischemia.

55 proves the understanding of the mechanics of myocardial ischemia.

56 relevant outcomes, such as the detection of myocardial ischemia.

57 d to profile transcriptome remodeling during myocardial ischemia.

58 stem cell therapy in large animal models of myocardial ischemia.

59 eads to heart failure and death during acute myocardial ischemia.

60 ng ectopies to be arrhythmia triggers during myocardial ischemia.

61 s the reference standard for the presence of myocardial ischemia.

62 ias, in the presence of pathologies, such as myocardial ischemia.

63 in vivo in a porcine model of stress-induced myocardial ischemia.

64 ction in patients with residual or recurrent myocardial ischemia.

65 of women undergoing evaluation for suspected myocardial ischemia: 1) What is the relationship between

66 f patients at highest risk for perioperative myocardial ischemia, 2) evaluation and development of ca

67 ofiled the plasma miRNAs 4 h after transient myocardial ischemia (45 min) or sham procedure.

68 Myocardial ischemia (50 minutes followed by reperfusion)

69 ogether, the current study demonstrates that myocardial ischemia activates an innate immune signaling

70 Herein we tested the hypothesis that myocardial ischemia activates interleukin receptor-assoc

71 arrhythmias during the early phase of acute myocardial ischemia after ligation of the left anterior

72 ily (intravenous) for the first 7 days after myocardial ischemia also protected against the structura

73 ven in the absence of ECG changes indicating myocardial ischemia, an acute culprit lesion may be pres

74 92%, and a specificity of 72% for detecting myocardial ischemia and 86%, 92%, and 72%, respectively,

75 Mental stress can induce myocardial ischemia and also has been implicated in trig

76 Coronary spasm can cause myocardial ischemia and angina in patients with and thos

77 t it did not show a significant reduction of myocardial ischemia and angina versus placebo.

78 Myocardial ischemia and cardioprotection by ischemic pre

79 he-art noninvasive modality for detection of myocardial ischemia and coronary artery disease.

80 d locations, deviations from which can cause myocardial ischemia and death.

81 del of chronic ischemic heart disease (IHD), myocardial ischemia and exertional angina are caused by

82 important in making an accurate diagnosis of myocardial ischemia and for choosing the best interventi

83 Glucose deprivation mimics myocardial ischemia and induces apoptosis in cardiomyocy

84 ion, impairs vascular function, and promotes myocardial ischemia and injury in healthy firefighters.

85 n a variety of different settings, including myocardial ischemia and its complications, high-risk per

86 ficult to distinguish between that caused by myocardial ischemia and may impact both the treatment st

87 condition; however, the association between myocardial ischemia and MBs has increased their clinical

88 women, which contrasts with greater rates of myocardial ischemia and near-term mortality compared wit

89 zed inflammatory response is triggered after myocardial ischemia and necrosis and involves all compon

90 nclusion, the cvrPhone can be used to detect myocardial ischemia and periods of respiratory apnea usi

91 lnerability to fatal arrhythmia during acute myocardial ischemia and provides a plausible mechanistic

92 Myocardial ischemia and reperfusion in cytomegalovirus-s

93 alpha-mediated activation of monocytes after myocardial ischemia and reperfusion using a mouse model

94 sized that mild hypothermia induced prior to myocardial ischemia and reperfusion will have a benefici

95 ith oxidative or nitrosative stress, such as myocardial ischemia and reperfusion, increase mitochondr

96 g 4 common forms of myocardial inflammation: myocardial ischemia and reperfusion, sepsis, viral myoca

97 sonance imaging in an in vivo mouse model of myocardial ischemia and reperfusion.

98 achycardia exacerbates injurious episodes of myocardial ischemia and significantly increases the prod

99 aboratory protocol provides a model to study myocardial ischemia and the actions of novel and establi

100 ardium as well as the presence and degree of myocardial ischemia and the extent of the scar.

101 ular territories exhibiting stress-inducible myocardial ischemia and those with normal perfusion.

102 cardioprotective actions in animal models of myocardial ischemia and ventricular dysfunction through

103 m, so-called Prinzmetal's angina, leading to myocardial ischemia and ventricular tachycardia, which w

104 derwent diagnostic angiography for suspected myocardial ischemia and were found to have unobstructed

105 nitrite in 2 clinical models: stress-induced myocardial ischemia and whole-arm ischemia-reperfusion.

106 eart failure, stroke, or hospitalization for myocardial ischemia) and of death or end-stage renal dis

107 on, or urgent coronary revascularization for myocardial ischemia) and total coronary events (death fr

108 branch block, (2) other ECG signs indicating myocardial ischemia, and (3) no ECG signs indicating myo

109 rise in fibrinolytic capacity, asymptomatic myocardial ischemia, and an increase in plasma cardiac t

110 schemia, and fasting-dependent hypoglycemia, myocardial ischemia, and AV block.

111 cesses such as left ventricular hypertrophy, myocardial ischemia, and diabetic cardiomyopathy.

112 t pain with a low burden of obstructive CAD, myocardial ischemia, and events.

113 se aortic constriction, in pigs with chronic myocardial ischemia, and in end-stage human heart failur

114 b signaling led to Per2 stabilization during myocardial ischemia, and in this setting, Per2(-/-) mice

115 action, revascularization and stress-induced myocardial ischemia, and revascularization and TID (all

116 entricular systolic function, stress-induced myocardial ischemia, and TID.

117 Both myocardial ischemia- and HSP60-induced IRAK-1 activation

118 in atherogenesis and its major complication, myocardial ischemia; and summarizes LOX-1 modulation by

119 ry bypasses and their quantitative effect on myocardial ischemia are unknown.

120 treat Kawasaki disease patients who develop myocardial ischemia as a consequence of coronary artery

121 Myocardial ischemia assessments by 3 independent, blinde

122 n the bone marrow resulted in aggravation of myocardial ischemia-associated damage, which could not b

123 nary angiography for evaluation of suspected myocardial ischemia at 4 San Diego hospitals from 2005 t

124 rdiovascular magnetic resonance in detecting myocardial ischemia at rest secondary to severe coronary

125 netic resonance could be useful in detecting myocardial ischemia at rest.

126 Efforts should now focus on detecting myocardial ischemia before adverse LV remodeling begins,

127 Report of myocardial ischemia before or after onset of ocular cond

128 vels of free fatty acids are elevated during myocardial ischemia but also in other conditions that ha

129 r+ cells increased significantly early after myocardial ischemia but not after hind limb ischemia, in

130 Mental stress can trigger myocardial ischemia, but the prevalence of mental stress

131 has shown significant promise in addressing myocardial ischemia by promoting angiogenesis that helps

132 Primary end point was absolute change in myocardial ischemia by SPECT.

133 ecular imaging approach for detecting recent myocardial ischemia by using recombinant P-selectin glyc

134 Myocardial ischemia can occur without overt symptoms.

135 s may remain silent until later in life when myocardial ischemia can occur.

136 Although reperfusion following myocardial ischemia can prevent death by restoring nutri

137 The risks of myocardial ischemia, cardiovascular instability and cere

138 Myocardial ischemia causes cardiomyocyte death, adverse

139 CTA alone was a limited predictor of myocardial ischemia compared with SPECT, with a sensitiv

140 g DSE are more likely to have stress-induced myocardial ischemia compared with those with normal or h

141 ejection fraction in large animal models of myocardial ischemia, compared with placebo.

142 total of 868 women presenting with suspected myocardial ischemia completed an extensive baseline exam

143 Whether the cardiac mortality was from myocardial ischemia, congestive heart failure or arrhyth

144 gement of depression in women with suspected myocardial ischemia could reduce medical costs.

145 The primary end point was a composite of myocardial ischemia detected by continuous ECG and/or tr

146 ques, cardiac embolic source, TIA/stroke and myocardial ischemia differ among various ocular arterial

147 In CAD with severe LV dysfunction, inducible myocardial ischemia does not identify patients with wors

148 a protective role in the pathophysiology of myocardial ischemia due to coronary artery disease, limb

149 sently at low risk for manifesting inducible myocardial ischemia during cardiac imaging procedures.

150 emporal change in the frequency of inducible myocardial ischemia during cardiac stress testing.

151 to assess whether the frequency of inducible myocardial ischemia during stress-rest single-photon emi

152 ties in fatty acid metabolism resulting from myocardial ischemia, even many hours after symptom cessa

153 After myocardial ischemia, extracellular matrix (ECM) depositi

154 Interestingly, during the acute phase of myocardial ischemia, focal ectopies arising from this lo

155 ed mice were then subjected to 45 minutes of myocardial ischemia followed by reperfusion for up to 24

156 CRC 1116 Masterswitches in Myocardial Ischemia, German Research Council DFG.

157 Following myocardial ischemia, Gna(i2)(fl/fl)/PF4-Cre mice showed

158 al fibrillation, cardiomyopathy, and chronic myocardial ischemia had consistent patterns but were not

159 Since its original description 50 years ago, myocardial ischemia has been a recognized but underappre

160 The arrhythmogenesis of ventricular myocardial ischemia has been extensively studied, but mo

161 bearing recombinant human PSGL-1 can detect myocardial ischemia hours after resolution.

162 sitron-emission tomography imaging agent for myocardial ischemia, hypoxic tumors, and brain disorders

163 d measure of large- and small-vessel CAD and myocardial ischemia, identifies patients at risk for CVD

164 icular ejection fraction, and stress-induced myocardial ischemia, improved discrimination of cardiac

165 electrocardiogram interpretation to diagnose myocardial ischemia in critically ill patients is unclea

166 sevoflurane did not reduce the incidence of myocardial ischemia in high-risk patients undergoing maj

167 gated the relationship between DE on CMR and myocardial ischemia in hypertrophic cardiomyopathy.

168 pared with propofol reduces the incidence of myocardial ischemia in patients undergoing major noncard

169 ation between mood and anxiety disorders and myocardial ischemia in patients with and without a CAD h

170 dverse cardiac events such as arrhythmia and myocardial ischemia in rats undergoing a dobutamine chal

171 enzyme inhibitors have been shown to relieve myocardial ischemia in stable patients and to improve ep

172 ic changes found during an episode of severe myocardial ischemia in the canine heart is presented.

173 Myocardial ischemia in this context occurs as a result o

174 Mice were subjected to 45 min of myocardial ischemia in vivo followed by 24 h of reperfus

175 deprivation (GD) in vitro, and during acute myocardial ischemia in vivo.

176 Myocardial ischemia induces the ER stress response; howe

177 In myocardial ischemia, induction of autophagy via the AMP-

178 ascular complications such as heart failure, myocardial ischemia/infarction, hypertension, thromboemb

179 and MR contrast agents for the diagnosis of myocardial ischemia is a case in point, as the utility o

180 Myocardial ischemia is characterized by reduced blood fl

181 sses mitochondrial function, protection from myocardial ischemia is enhanced as a result of a coordin

182 Thus, myocardial ischemia is lack of coronary blood flow with

183 Mental stress-induced myocardial ischemia is prevalent and a risk factor for p

184 Nevertheless, the assessment of myocardial ischemia is still not part of routine clinica

185 Tissue ischemia, such as transient myocardial ischemia, leads to release of cellular RNA in

186 tients who already harbor silent cTOD (i.e., myocardial ischemia, left ventricular hypertrophy, systo

187 During myocardial ischemia, low pH activates NHE1 and causes in

188 y arteries, patients with similar amounts of myocardial ischemia may vary widely in their symptoms.

189 microvascular dysfunction is responsible for myocardial ischemia-mediated myocyte death, and ultimate

190 investigate the metabolomic profile of acute myocardial ischemia (MIS) using nuclear magnetic resonan

191 nally, we transplanted hESC-ECs into a mouse myocardial ischemia model.

192 ions aimed at reducing mental stress-induced myocardial ischemia (MSIMI) have not been well studied.

193 Mental stress-induced myocardial ischemia (MSIMI) was defined as the developme

194 METHODS AND In different murine models of myocardial ischemia, myeloperoxidase deficiency profound

195 Associations of cardiac arrhythmia, myocardial ischemia, myocardial infarction, stroke, and

196 ients in 2003 and 2007 were derived from the Myocardial Ischemia National Audit Project (n=51 755).

197 Myocardial ischemia occurred in 75 patients (40.8%) in t

198 rate, implying a greater risk, especially of myocardial ischemia, of lowering diastolic BP, which is

199 eptum-ratio and LAVi >/=32 ml/m(2); however, myocardial ischemia on MPS was not a predictor.

200 The discriminative value for myocardial ischemia on myocardial perfusion scintigraphy

201 P<0.001), and was an excellent predictor of myocardial ischemia on SPECT-MPI in the presence of sten

202 ardiovascular disease except a suggestion of myocardial ischemia on treadmill testing and mild athero

203 among patients without objective evidence of myocardial ischemia or obstructive CAD.

204 severely ill as a result of pulmonary edema, myocardial ischemia, or cardiogenic shock.

205 t significantly reduced by MPI screening for myocardial ischemia over 4.8 years.

206 is may contribute to worsen the prognosis of myocardial ischemia, peripheral arterial disease, ischem

207 fect of immediate treatment of perioperative myocardial ischemia (PMI) because of early graft failure

208 ole in atherogenesis and thrombosis-mediated myocardial ischemia, processes that are accelerated in d

209 Myocardial ischemia produces diastolic dysfunction.

210 er patients with syndrome X show evidence of myocardial ischemia (reduced perfusion and oxygenation)

211 tivity were measured in 2587 patients in the Myocardial Ischemia Reduction With Acute Cholesterol Low

212 ES trial and atorvastatin arm of the MIRACL (Myocardial Ischemia Reduction with Acute Cholesterol Low

213 nary syndromes (ACS) enrolled in the MIRACL (Myocardial Ischemia Reduction With Aggressive Cholestero

214 observed that blood selenium decreases after myocardial ischemia reperfusion and displays an inverse

215 ide, would target to injured heart tissue in myocardial ischemia reperfusion and injured leg muscle i

216 Anesthetized mice were subjected to myocardial ischemia reperfusion injury (coronary artery

217 Recent efforts to identify treatments for myocardial ischemia reperfusion injury have resulted in

218 We also measured damage after myocardial ischemia reperfusion injury using morphometry

219 on injury, whereas neon was only explored in myocardial ischemia reperfusion injury.

220 ly implicating MerTK in cardiac repair after myocardial ischemia reperfusion.

221 e by resident and recruited phagocytes after myocardial ischemia reperfusion.

222 -specific GRK2 ablation-each initiated after myocardial ischemia-reperfusion (I/R) injury-was investi

223 or in skeletal muscles protects mice against myocardial ischemia-reperfusion (I/R) injury.

224 specific beta(3)-AR agonists would attenuate myocardial ischemia-reperfusion (MI/R) injury via eNOS a

225 ardioprotective effects in a murine model of myocardial ischemia-reperfusion (MI/R) injury with a bel

226 emia is prevalent in patients who experience myocardial ischemia-reperfusion injury (IR).

227 targeted microbubble destruction attenuates myocardial ischemia-reperfusion injury and could avoid u

228 asound targeted hs-MB destruction attenuates myocardial ischemia-reperfusion injury and may avoid unw

229 ate that exercise protects the heart against myocardial ischemia-reperfusion injury by stimulation of

230 gen sulfide (H2S) is an attractive agent for myocardial ischemia-reperfusion injury, however, systemi

231 ole, and crosstalk with innate leukocytes in myocardial ischemia-reperfusion injury, wound healing, a

232 importance was tested in an ex vivo model of myocardial ischemia-reperfusion injury.

233 and limits infarct size in animal models of myocardial ischemia-reperfusion injury.

234 uction was investigated in a rodent model of myocardial ischemia-reperfusion injury.

235 s a valid therapeutic strategy in mitigating myocardial ischemia-reperfusion injury.

236 Furthermore, myocardial ischemia/reperfusion (I/R) in CMiPLA2gammaKO

237 at PKG activation with tadalafil would limit myocardial ischemia/reperfusion (I/R) injury and dysfunc

238 sis that ADAMTS13 reduces VWF-mediated acute myocardial ischemia/reperfusion (I/R) injury in mice.

239 important pathophysiological process during myocardial ischemia/reperfusion (I/R) injury, and the tr

240 Endothelial damage is a critical mediator of myocardial ischemia/reperfusion (I/R) injury.

241 were used to determine the role of SIRT1 in myocardial ischemia/reperfusion (I/R) tolerance.

242 examined whether Sirt1 is protective against myocardial ischemia/reperfusion (I/R).

243 vestigated the functional role of PKN during myocardial ischemia/reperfusion (I/R).

244 e (WT) littermates were subjected to in vivo myocardial ischemia/reperfusion (MI/R) and treated with

245 s play a major role in the early response to myocardial ischemia/reperfusion (MI/R) injury.

246 have now in an in situ pig model of regional myocardial ischemia/reperfusion addressed the role of mi

247 Myocardial ischemia/reperfusion in MBL2 KI mice revealed

248 Dual molecular imaging of myocardial ischemia/reperfusion injury allows characteri

249 cles of limb ischemia/reperfusion may reduce myocardial ischemia/reperfusion injury and improve patie

250 ated a more robust functional recovery after myocardial ischemia/reperfusion injury and significantly

251 Our data show that ADAMTS13 reduces myocardial ischemia/reperfusion injury in mice and indic

252 In response to myocardial ischemia/reperfusion injury in the mouse, FGF

253 lterations in endothelial BH4 content impact myocardial ischemia/reperfusion injury remains elusive.

254 N would protect the heart against subsequent myocardial ischemia/reperfusion injury via direct effect

255 n (Epac1(-/-)) protects against experimental myocardial ischemia/reperfusion injury with reduced infa

256 nd chronic inflammatory processes, including myocardial ischemia/reperfusion injury, sepsis, and adul

257 f early- and late-stage ECM remodeling after myocardial ischemia/reperfusion injury, which may have c

258 myocytes and immune cells and contributes to myocardial ischemia/reperfusion injury.

259 cycles of limb ischemia/reperfusion reduces myocardial ischemia/reperfusion injury.

260 y reduces the infarct size in a rat model of myocardial ischemia/reperfusion injury.

261 MTS13 could be of therapeutic value to limit myocardial ischemia/reperfusion injury.

262 lack of gamma2-AMPK sensitizes the heart to myocardial ischemia/reperfusion injury.

263 ction of infarct size in an in vivo model of myocardial ischemia/reperfusion injury.

264 ey mechanism of mitochondrial dysfunction in myocardial ischemia/reperfusion injury.

265 is not only the culprit but also a victim of myocardial ischemia/reperfusion injury.

266 ed as an important pathological mechanism in myocardial ischemia/reperfusion injury.

267 ected to experimental models of cerebral and myocardial ischemia/reperfusion injury.

268 eoxygenation and in the heart after regional myocardial ischemia/reperfusion, in the presence or abse

269 organ remote from the heart before sustained myocardial ischemia/reperfusion, was confirmed in all sp

270 ving von Willebrand factor multimers, during myocardial ischemia/reperfusion, we used a mouse model o

271 of the edematous reaction after experimental myocardial ischemia/reperfusion.

272 ed left ventricular function at 7 days after myocardial ischemia/reperfusion.

273 sts in stable form for at least 1 week after myocardial ischemia/reperfusion.

274 In 10 patients with inducible myocardial ischemia, saline and low-dose sodium nitrite

275 Myocardial ischemia slows ventricular relaxation and can

276 Patients referred for myocardial ischemia testing at 34 centers underwent rest

277 ads, and after a more stressful condition of myocardial ischemia that is known to robustly activate A

278 a dynamic interaction with the mechanics of myocardial ischemia that may be amenable to treatment.

279 During myocardial ischemia, the activity of AR is increased due

280 use heart can also regenerate in response to myocardial ischemia, the most common antecedent of heart

281 e medications that have been shown to reduce myocardial ischemia through diverse mechanisms and have

282 The primary endpoint was Thrombolysis In Myocardial Ischemia (TIMI) scale 2 or 3 flow in all trea

283 (Responses of Myocardial Ischemia to Escitalopram Treatment [REMIT]; N

284 he REMIT (Responses of Mental Stress-Induced Myocardial Ischemia to Escitalopram) study underwent psy

285 at Sestrin2 plays an influential role during myocardial ischemia to promote AMPK activation.

286 examined the effect of insulin resistance on myocardial ischemia tolerance.

287 During both permanent and transient myocardial ischemia, Trem-1 blockade also ameliorates ca

288 n addition, betaCys93Ala mutation results in myocardial ischemia under basal normoxic conditions and

289 61.7+/-11.9 years; 37% women) with suspected myocardial ischemia underwent stress perfusion cardiac m

290 ment of electrocardiogram interpretation for myocardial ischemia was fair, and improved with troponin

291 Myocardial ischemia was induced during stress testing in

292 Prevalence of obstructive CAD and myocardial ischemia was low (11.9% versus 12.7%, respect

293 The presence and extent of myocardial ischemia was quantified using the conventiona

294 secutive patients referred for evaluation of myocardial ischemia, we determined the net reclassificat

295 Myocardial ischemia, while causing cardiomyocyte injury,

296 ns on physical activity owing to angina) and myocardial ischemia, who were not candidates for revascu

297 sound to detect the inflammatory response to myocardial ischemia will be discussed.

298 up to develop a consensus on the syndrome of myocardial ischemia with no obstructive coronary arterie

299 lowing (ischemic postconditioning) sustained myocardial ischemia with reperfusion reduce infarct size

300 CTP was a better predictor of myocardial ischemia, with a sensitivity, specificity, PP