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1 with low event rates in patients with normal myocardial perfusion.
2 ng SPECT to precisely quantify segment-level myocardial perfusion.
3 content, and increased vessel densities and myocardial perfusion.
4 e injection did not impair coronary flow and myocardial perfusion.
5 ta can lead to misinterpretation of regional myocardial perfusion.
6 D than in those without PTSD, denoting worse myocardial perfusion.
7 aine challenge evokes a sizeable decrease in myocardial perfusion.
8 integration of data on coronary anatomy and myocardial perfusion.
9 onary arteries is the prerequisite of normal myocardial perfusion.
10 essure (DBP) to levels that could compromise myocardial perfusion.
11 opathy (HCM) and is associated with abnormal myocardial perfusion.
12 increased with the extent of abnormality of myocardial perfusion.
13 yocardial strain), coronary artery flow, and myocardial perfusion.
14 There was no change in myocardial perfusion.
15 [N]NH3 imaging was performed to evaluate myocardial perfusion.
16 y showed visually concordant DE and regional myocardial perfusion abnormalities in 31 patients and ab
19 ents (17 men; 69+/-9 years) who had improved myocardial perfusion after the first injection but had r
20 ons about vascular territory distribution in myocardial perfusion analysis are frequently inaccurate
21 n) stress, including 15 subjects with normal myocardial perfusion and 27 patients referred for corona
22 The imaging data of patients with normal myocardial perfusion and 30 patients with mid-sized to l
24 injection is associated with improvements in myocardial perfusion and anginal symptoms in patients wi
25 not been systemically validated for absolute myocardial perfusion and coronary flow reserve (CFR) by
28 rteriolar density and significantly improved myocardial perfusion and endothelium-dependent vasorelax
31 d MPS underwent rest and adenosine stress 3D myocardial perfusion and late gadolinium enhancement CMR
35 (CMR) and positron emission tomography (PET) myocardial perfusion and myocardial perfusion reserve (M
36 ess-induced and adenosine-induced changes in myocardial perfusion and neurohormonal activation in CHF
38 armacokinetic studies in mice by quantifying myocardial perfusion and oxygen consumption with (11)C-a
40 circulation may represent less well-balanced myocardial perfusion and thus confer worse prognosis in
42 sitive patients demonstrated normal regional myocardial perfusion, and 3 DE-negative patients had (ap
45 t of myocardial edema, myocardial siderosis, myocardial perfusion, and diffuse myocardial fibrosis.
46 scar, halting adverse remodeling, increasing myocardial perfusion, and improving hemodynamic status a
47 ance (cine, T2* iron, vasodilator first pass myocardial perfusion, and late gadolinium enhancement im
49 ry angina is associated with improvements in myocardial perfusion, anginal complaints, and quality of
54 f the physiology of coronary circulation and myocardial perfusion; (b) describe the technical prerequ
56 ascularization) and quantitative measures of myocardial perfusion by [(13)N] ammonia positron emissio
57 ocker therapy has been also shown to improve myocardial perfusion by enhancing neoangiogenesis in the
60 cy of dynamic 3-dimensional (3D) whole heart myocardial perfusion cardiovascular magnetic resonance (
61 om nonischemic cardiomyopathy; evaluation of myocardial perfusion; characterization of hypertrophic c
65 an estimation of ischemic burden by using 3D myocardial perfusion CMR holds promise for noninvasive g
69 s to assess the diagnostic performance of 3D myocardial perfusion CMR to detect functionally relevant
70 s study was to compare ischemic burden on 3D myocardial perfusion CMR with (99m)Tc-tetrofosmin MPS.
71 More recently developed 3-dimensional (3D) myocardial perfusion CMR, however, provides whole-heart
73 ation of abnormalities including border zone myocardial perfusion, contractile dysfunction, and LV wa
74 blished the feasibility of performing stress myocardial perfusion CT imaging in small groups of patie
75 roups of patients and have shown that stress myocardial perfusion CT in combination with CT coronary
76 se determinants of myocardial oxygen demand, myocardial perfusion decreased by 30% (103.7+/-9.8 to 75
78 R) is typically based on induction of either myocardial perfusion defect or wall motion abnormality.
80 I and CTA data may facilitate correlation of myocardial perfusion defects and subtending coronary art
82 der confidence at cardiac CT angiography, (b)myocardial perfusion defects were identified and scored
83 ry artery luminal stenosis and corresponding myocardial perfusion deficits in patients with suspected
87 ents in CT technology allow CT evaluation of myocardial perfusion during vasodilator stress, thereby
89 nctional MR images and dynamic assessment of myocardial perfusion from transit of intravascular contr
90 magnetic resonance for routine assessment of myocardial perfusion, function, and late gadolinium enha
94 y artery disease, given concerns for reduced myocardial perfusion if diastolic blood pressure is too
98 cian offices; this proportion was higher for myocardial perfusion imaging (74.8%) and cardiac compute
99 fusion (CTP) with cardiac magnetic resonance myocardial perfusion imaging (CMR-Perf) for detection of
100 eys the extensive literature on preoperative myocardial perfusion imaging (MPI) and outlines key tren
101 value of positron emission tomography (PET) myocardial perfusion imaging (MPI) and the improved clas
103 ve patients undergoing adenosine stress-rest myocardial perfusion imaging (MPI) by (99m)Tc-tetrofosmi
104 ate use criteria recommend performing stress myocardial perfusion imaging (MPI) for intermediate- to
105 -photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) has changed over time
106 aluation for appropriate use of radionuclide myocardial perfusion imaging (MPI) in multiple clinical
107 y (CCTA) to a strategy employing rest-stress myocardial perfusion imaging (MPI) in the evaluation of
108 Cardiovascular magnetic resonance (CMR) myocardial perfusion imaging (MPI) is a state-of-the-art
109 -photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) is an effective metho
113 consecutive patients underwent (82)Rubidium myocardial perfusion imaging (MPI) positron emission tom
115 segmentation of the left ventricle for SPECT myocardial perfusion imaging (MPI) quantification often
116 lity of a new protocol, IQ SPECT, to acquire myocardial perfusion imaging (MPI) studies in a quarter
117 It remains unclear whether the addition of myocardial perfusion imaging (MPI) to the standard ECG e
118 photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) underwent a comprehen
121 easibility of attenuation correction (AC) of myocardial perfusion imaging (MPI) with a virtual unenha
123 artery calcium score (CACS) as an adjunct to myocardial perfusion imaging (MPI) with SPECT for cardia
125 impact of appropriate use criteria (AUC) for myocardial perfusion imaging (MPI) with SPECT on the est
126 mance for positron emission tomography (PET) myocardial perfusion imaging (MPI) with Tc-99m single-ph
128 -photon emission computed tomography (SPECT)-myocardial perfusion imaging (MPI), a technique that is
129 tunity to lower the injected doses for SPECT myocardial perfusion imaging (MPI), but the exact limits
130 cent advances in CT coronary angiography and myocardial perfusion imaging (MPI), including PET MPI, i
136 h single-photon emission computed tomography myocardial perfusion imaging (SPECT MPI) has improved th
137 t single-photon emission computed tomography-myocardial perfusion imaging (SPECT-MPI) has high predic
138 ween October 2004 and September 2011 who had myocardial perfusion imaging after negative troponin T t
139 own CAD underwent prospectively simultaneous myocardial perfusion imaging and CAC scoring on a hybrid
140 2051 patients who underwent exercise stress myocardial perfusion imaging and echo (5.5+/-7.9 days),
142 consecutive CRT recipients with radionuclide myocardial perfusion imaging before CRT between January
143 were Veteran patients who underwent nuclear myocardial perfusion imaging between December 2010 and J
144 +/- 11.8 years), patients were referred for myocardial perfusion imaging between May 2008 and Januar
145 computed tomographic angiography and stress myocardial perfusion imaging by single photon emission c
146 was to determine the diagnostic accuracy of myocardial perfusion imaging by single-photon emission c
148 trocardiography, stress echocardiography, or myocardial perfusion imaging can reveal findings associa
151 h-spatial-resolution cardiovascular MR (CMR) myocardial perfusion imaging has been shown to be clinic
154 single-photon emission computed tomographic myocardial perfusion imaging improved from a summed stre
155 high-resolution and standard-resolution CMR myocardial perfusion imaging in patients with suspected
162 is of myocardial dynamic computed tomography myocardial perfusion imaging lacks standardization.
163 atic analysis of dynamic computed tomography myocardial perfusion imaging may permit robust discrimin
164 racy of the 3 most commonly used noninvasive myocardial perfusion imaging modalities, single-photon e
165 h BMI >/= 40 kg/m(2) should be scheduled for myocardial perfusion imaging on a conventional SPECT cam
166 ial blood flow as assessed by stress-induced myocardial perfusion imaging or a significant fall in di
167 mage quality of torso PET and compare stress myocardial perfusion imaging patterns with myocardial (1
168 ought to determine whether changes in stress myocardial perfusion imaging protocols and camera techno
170 significantly higher in patients with normal myocardial perfusion imaging results (6.5% +/- 5.4%) tha
171 econdary outcome was a comparison of nuclear myocardial perfusion imaging results and frequency of is
173 amera system for high-speed SPECT (HS-SPECT) myocardial perfusion imaging shows excellent correlation
174 -photon emission computed tomography (SPECT) myocardial perfusion imaging studies among patients with
175 ce radiation exposure to patients undergoing myocardial perfusion imaging studies, especially when co
176 ests a reference range of TID for (82)Rb PET myocardial perfusion imaging that is in the range of pre
177 were followed for 6 months after their index myocardial perfusion imaging to determine subsequent rat
179 ombined Noninvasive Coronary Angiography and Myocardial Perfusion Imaging Using 320-Detector Row Comp
180 act of increased body mass on the quality of myocardial perfusion imaging using a latest-generation g
182 n spent nuclear fuel cycle as well as toward myocardial perfusion imaging utilizing (82)Sr/(82)Rb iso
186 ardiography, stress echocardiography, and/or myocardial perfusion imaging were performed to identify
188 adenosine-stress dynamic computed tomography myocardial perfusion imaging with a second-generation du
191 tery disease (CAD) is ambiguous, but nuclear myocardial perfusion imaging with single-photon emission
192 onary artery calcium (CAC) scoring on top of myocardial perfusion imaging with single-photon emission
195 se myocardial fibrosis imaging, and absolute myocardial perfusion imaging, are poised to further adva
198 nd software for positron emission tomography myocardial perfusion imaging, which has advanced it from
207 dial infarction showed high concordance with myocardial perfusion in matched territories as revealed
208 s of angina, relevant clinical outcomes, and myocardial perfusion in patients with refractory angina.
209 on, resulting in significant improvements in myocardial perfusion in the setting of chronic ischemia.
211 he early postinfarction period when regional myocardial perfusion is often severely compromised.
212 e relationship between coronary stenosis and myocardial perfusion is well established, little is know
213 ysis was applied to helical multidetector CT myocardial perfusion measurements, the correlation betwe
214 y late gadolinium enhancement (LGE) MRI, and myocardial perfusion/metabolism was evaluated by (99m)Tc
215 onance (CMR) with conventional 2-dimensional myocardial perfusion methods is limited by incomplete ca
216 ], wall motion abnormalities [WMA], abnormal myocardial perfusion, microvascular obstruction, late ga
220 criteria for identifying areas of decreased myocardial perfusion or for assessing tissue viability f
224 ardial flow reserve (MFR) with (13)N-ammonia myocardial perfusion PET have been implemented for clini
225 diagnostic performance of regadenoson (82)Rb myocardial perfusion PET imaging to detect obstructive c
228 ronary angiography after stress testing with myocardial perfusion positron emission tomography and wi
229 years, 50.5% women) referred for rest/stress myocardial perfusion positron emission tomography scans
230 or suspected CAD with troponin before stress myocardial perfusion positron emission tomography were f
231 ronary angiography after stress testing with myocardial perfusion positron emission tomography were f
232 ield in sucrose (SUC) versus EtOH; P=0.004), myocardial perfusion (ratio of blood flow to the at-risk
234 l blood-derived mononuclear cells (PBMCs) on myocardial perfusion recovery by using cardiac magnetic
235 <2.0, 3.18+/-1.42 mm Hg/cm per second versus myocardial perfusion reserve >/=2.0, 2.24+/-1.19 mm Hg/c
236 with decreased myocardial blood flow on PET (myocardial perfusion reserve <2.0, 3.18+/-1.42 mm Hg/cm
239 on tomography (PET) myocardial perfusion and myocardial perfusion reserve (MPR) measurements in patie
240 nalysis for each method, with stress MBF and myocardial perfusion reserve (MPR) serving as continuous
242 magnitude of change was proportional to the myocardial perfusion reserve (rho = 0.53; p < 0.01).
246 nt) were evaluated for perfusion upslope and myocardial perfusion reserve index with Student t test a
248 ention index to describe global and regional myocardial perfusion reserve using a dedicated solid-sta
252 stress (peak) for the assessment of regional myocardial perfusion (rMP), left ventricular ejection fr
253 erences in the prognostic accuracy of stress myocardial perfusion rubidum-82 (Rb-82) positron emissio
256 derwent a comprehensive echocardiogram and a myocardial perfusion scintigraphy (MPS) at inclusion.
258 e is progressive and recurring; thus, stress myocardial perfusion scintigraphy (MPS) is widely used t
261 yed to determine appropriateness ratings for myocardial perfusion scintigraphy (MPS), stress echocard
262 mean stenosis diameter 55%+/-11%), underwent myocardial perfusion scintigraphy for documentation of r
263 criminative value for myocardial ischemia on myocardial perfusion scintigraphy of all parameters was
264 There was no reduction in ischemic burden on myocardial perfusion scintigraphy or in the safety endpo
267 atients with available rest and stress gated myocardial perfusion single-photon emission computed tom
269 ardiovascular magnetic resonance while using myocardial perfusion single-photon emission computed tom
271 ween CE-SSFP and T2-STIR from this study and myocardial perfusion single-photon emission computed tom
272 improve the diagnostic accuracy of automatic myocardial perfusion SPECT (MPS) interpretation analysis
273 myocardial perfusion stress-rest changes in myocardial perfusion SPECT (MPS) studies for the optimal
274 nuation-corrected (AC) and noncorrected (NC) myocardial perfusion SPECT (MPS) with the corresponding
276 myocardial wall motion and thickening during myocardial perfusion SPECT are typically assessed separa
281 f this study was to determine whether stress myocardial perfusion (SPECT) optimized with stress-only
282 rdized MD, 0.331; 95% CI, 0.08 to 0.55), and myocardial perfusion (standardized MD, -0.49; 95% CI, -0
284 We aimed to improve the quantification of myocardial perfusion stress-rest changes in myocardial p
286 history of cardiac disease and with a normal myocardial perfusion study had either a low or a very lo
287 photon emission computed tomography (SPECT) myocardial perfusion study underwent coronary CT angiogr
288 alignment of coronary arterial segments and myocardial perfusion territories, designed for the CORE3
290 ve vascular-ventricular coupling and enhance myocardial perfusion, thereby potentially contributing t
296 dioxide (PetCO2) increased by 10 mm Hg, and myocardial perfusion was monitored with myocardial blood
300 mL/min/g; p = 0.03), whereas differences in myocardial perfusion were not statistically significant
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