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1 gan (thigh versus arm) would provide further myocardial protection.
2 ntifying a role for GSNO in angiogenesis and myocardial protection.
3 secondary stroke protection rather than for myocardial protection.
4 tivation is antiapoptotic and contributes to myocardial protection.
5 diomyocytes, which in turn leads to improved myocardial protection.
6 membrane current, which may be important in myocardial protection.
7 s that may benefit from better postoperative myocardial protection.
8 at-shock protein 70 (HSP 70) plays a role in myocardial protection.
9 ior to ischemia induces preconditioning-like myocardial protection.
10 radykinin B(2) receptor blocker, HOE 140, on myocardial protection.
11 esearch stimulated by interest in endogenous myocardial protection.
12 rane fluxes and metabolic demand and improve myocardial protection.
13 cardioplegia in increasing doses to enhance myocardial protection.
14 ium (GIK) is a potentially useful adjunct to myocardial protection.
15 cardiopulmonary bypass (CPB), 3) methods of myocardial protection, 4) techniques for aortic occlusio
17 ether these effects could be used to enhance myocardial protection afforded by crystalloid cardiopleg
18 uce hibernation in active animals and afford myocardial protection after ischemia-reperfusion injury.
19 route could be a promising new strategy for myocardial protection against ischemia-reperfusion injur
20 shock protein 72 (HSP72) is known to provide myocardial protection against ischemia-reperfusion injur
21 ls during early myocardial repair, providing myocardial protection against ischemic damage by improvi
22 cule-1 (ICAM-1) has been reported to provide myocardial protection against leukocyte-mediated reperfu
25 tant implications for the design of tailored myocardial protection and operative strategies for diabe
26 ding care to high-risk patients and improved myocardial protection and surgical and anesthetic techni
28 yrin-B expression as required for vertebrate myocardial protection as hearts deficient in ankyrin-B s
29 perdynamic contractile response and enhanced myocardial protection, as evidenced by a reduction in my
33 tissue in myocardial injury, contributing to myocardial protection by the mediation of the FGFR1/beta
34 -blood (66:1) cardioplegia provides superior myocardial protection compared with dilute (4:1) cardiop
36 MP and heme, has recently been implicated in myocardial protection during ischemia and reperfusion.
37 KC activity in adult mouse hearts results in myocardial protection during ischemia or reperfusion, he
40 , we investigated the potential mechanism of myocardial protection following delta-opioid receptor ac
41 cardioplegia has been the gold standard for myocardial protection for years, but patients undergoing
43 a cytoprotective gene that confers long-term myocardial protection from ischemia/reperfusion injury.
44 ore, we hypothesized that (1) TNFR2 mediates myocardial protection from ischemia/reperfusion through
46 suggesting the importance of GSHPx-1 gene in myocardial protection from ischemic reperfusion injury.
47 normalization of beta-receptor function and myocardial protection from the effects of catecholamines
48 technically challenging, but new methods of myocardial protection have markedly improved the surgica
51 rction angina is associated with significant myocardial protection in the setting of primary percutan
53 by adipocytes is adiponectin, which confers myocardial protection, insulin-sensitization, and antiat
56 The possibility that an innate mechanism of myocardial protection might be inducible in the human he
58 hocardiography, and improved intra-operative myocardial protection techniques have all contributed to
59 ce to presently known principles of surgical myocardial protection using advanced cardioplegic techno
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