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1 sample to develop new insights for achieving myocardial regeneration.
2 cover molecular mechanisms that will promote myocardial regeneration.
3 novel off-the-shelf microtissue product for myocardial regeneration.
4 d have been unthinkable only a few years ago-myocardial regeneration.
5 would represent the most powerful cells for myocardial regeneration.
6 ulate cardiomyocyte expansion in therapeutic myocardial regeneration.
7 rdium provides a path to realize sustainable myocardial regeneration.
8 ecovery was due to the cytokines rather than myocardial regeneration.
9 may provide a molecular strategy to promote myocardial regeneration.
10 cally to stimulate resident CPCs and promote myocardial regeneration.
11 protein appears to have no obvious effect on myocardial regeneration.
12 on combinatorial cell approaches to support myocardial regeneration.
13 Here we review efforts to induce myocardial regeneration.
14 from mechanisms that are distinct from true myocardial regeneration.
15 sses hold enormous potential for therapeutic myocardial regeneration.
16 he use of BM-derived cardiac progenitors for myocardial regeneration.
17 l non-coding RNAs called microRNAs (miRs) in myocardial regeneration.
18 is feasible, safe or can provide sufficient myocardial regeneration.
19 c growth factors has been shown to result in myocardial regeneration after acute myocardial infarctio
20 SF) and stem cell factor (SCF) could promote myocardial regeneration after coronary artery occlusion
24 nd 6) explore the potential of stem cells in myocardial regeneration and repair in hearts damaged by
25 dings shed new light on our understanding of myocardial regeneration and suggest potential novel ther
26 duce pathological cardiac remodeling, induce myocardial regeneration, and improve ventricular functio
27 lial cell transplantation into the heart for myocardial regeneration, and induction of angiogenesis f
30 recently shown to play an essential role in myocardial regeneration by regulating different processe
31 the lack of unequivocal evidence in favor of myocardial regeneration by the injection of BMCs in the
37 cardiac progenitor cells (hCPCs) may promote myocardial regeneration in adult ischemic myocardium.
39 ings define a mechanistic basis for enhanced myocardial regeneration in transgenic mice overexpressin
43 he mechanism of benefit is unclear, although myocardial regeneration is not a significant factor.
46 rdiomyocyte cell-cycle activity and promotes myocardial regeneration, leading to improved function af
48 ll-mediated regeneration to promote enhanced myocardial regeneration, persistence of donated cells, a
49 hanged since our survey of the landscape for myocardial regeneration powered by adult stem cells 4 ye
50 f cellular adoptive transfer for therapeutic myocardial regeneration, problems remain for donated cel
53 In seven acute infarcts, foci of spontaneous myocardial regeneration that did not involve cell fusion
54 rstand the role of fusion in stem cell-based myocardial regeneration, the present study was designed
58 peutic strategy, the induction of endogenous myocardial regeneration was investigated by initiating c
59 ion of miR-133 levels after injury inhibited myocardial regeneration, while transgenic miR-133 deplet
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