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1 3 hepatitis, grade 3 pneumonia, and grade 4 myocarditis).
2 sed cardiomyopathy and clinical suspicion of myocarditis.
3 to unravel the role of NOD2 in CVB3-induced myocarditis.
4 agnostic accuracy in patients with suspected myocarditis.
5 bsets of the inflammatory response in murine myocarditis.
6 severe heart failure at HT in children with myocarditis.
7 g cause of death in humans, can emanate from myocarditis.
8 d, consecutive patient cohort with suspected myocarditis.
9 ed 299 patients (0.7%) with the diagnosis of myocarditis.
10 tly different from control animals in severe myocarditis.
11 ra may be a suitable treatment for fulminant myocarditis.
12 id cell compartment, are protective in viral myocarditis.
13 tly between patients with and without active myocarditis.
14 arameters do not sufficiently reflect LGE in myocarditis.
15 teria is used to make the diagnosis of acute myocarditis.
16 hanisms of this important cause of pediatric myocarditis.
17 denoviruses are frequent causes of pediatric myocarditis.
18 standard was the clinical evidence for acute myocarditis.
19 characterized by hypertrophy, fibrosis, and myocarditis.
20 hematical models as they apply to autoimmune myocarditis.
21 oaches in patients suspected of having acute myocarditis.
22 mation and outcome in a mouse model of viral myocarditis.
23 review how autoimmunity is induced in viral myocarditis.
24 and cardiac proteins causes autoimmunity in myocarditis.
25 are frequently needed to diagnose giant-cell myocarditis.
26 of the sarcolemma play a role in CV-mediated myocarditis.
27 1 in cardiomyocytes had reduced CVB3-induced myocarditis.
28 actosylceramide showed significantly reduced myocarditis.
29 tes, qualifying this disease as eosinophilic myocarditis.
30 obal myocardium edema in patients with acute myocarditis.
31 An infection with CVB3 leads to myocarditis.
32 n determine the sexual dimorphism in EAE and myocarditis.
33 on of inflammatory cells in a mouse model of myocarditis.
34 2 (PAR2) in coxsackievirus B3 (CVB3)-induced myocarditis.
35 ardiac inflammation and injury in infectious myocarditis.
36 toreactive T cell responses in two models of myocarditis.
37 ruzi control and protection from fatal acute myocarditis.
38 cruzi infection, a known inducer of lymphoid myocarditis.
39 observed in the subgroup (n=130) with viral myocarditis.
40 characteristic of both human and mouse viral myocarditis.
41 from chronic MI and identification of acute myocarditis.
42 icant therapeutic challenge in patients with myocarditis.
43 is a potential therapeutic target for viral myocarditis.
44 ic T cell responses during subclinical viral myocarditis.
45 unmet need to identify high-risk patients in myocarditis.
46 gnetic resonance to detect and monitor acute myocarditis.
47 nt LGE has been shown to be a risk marker in myocarditis.
48 eir role in Coxsackievirus B3 (CVB3)-induced myocarditis.
49 sk stratification in patients with suspected myocarditis.
50 ts increased susceptibility to virus-induced myocarditis.
51 ands, both TS1 TEM and TN induced late-onset myocarditis.
52 cell epitopes that induce varying degrees of myocarditis.
53 ory response during the pathogenesis of CVB3 myocarditis.
54 ting diagnostic tools for risk assessment in myocarditis.
55 ation (2000-2015) with a listed diagnosis of myocarditis.
56 yocarditis, moderate myocarditis, and severe myocarditis.
57 une responses could be helpful in diagnosing myocarditis.
58 ntly higher need for mechanical ventilation (myocarditis 11% versus NICM 2% versus ICM 4%; P<0.001),
59 tages were found in patients with HCM (16%), myocarditis (15%), and hypertension (14%) but without re
60 iventricular mechanical circulatory support (myocarditis 19% versus NICM 2%, versus ICM 2%; P<0.001),
61 en patients with clinical diagnosis of acute myocarditis (25 years [23-38 years]; 78% males) were pro
63 hy (ICM), myocarditis patients were younger (myocarditis 43.4+/-14.2 years, NICM 49.8+/-12.4 years, a
64 01) and more frequently listed as status 1A (myocarditis 44% versus NICM 21% versus ICM 21%; P<0.001)
65 ), and extracoroporeal membrane oxygenation (myocarditis 5% versus NICM 0.4% versus ICM 1%; P<0.001).
67 significantly higher in patients with active myocarditis (65 ms [Q1-Q3, 61-70 ms]) than in patients w
69 is one of the most prevalent causes of acute myocarditis, a disease that frequently is identified onl
73 cement (LGE) has not been clarified in acute myocarditis (AM) with preserved left ventricular (LV) ej
75 (1) our experience in diagnosing giant-cell myocarditis and (2) the outcome of patients on combined
80 as accompanied by a profound exacerbation of myocarditis and by a significant increase in mortality a
81 a profound acceleration and exacerbation of myocarditis and by a significant increase in mortality.
82 ropic strain of T. cruzi displayed increased myocarditis and cardiac fibrosis compared to WT controls
84 tion and prevents the progression to chronic myocarditis and dilated cardiomyopathy (DCM) by inhibiti
85 development of autoimmune coxsackievirus B3 myocarditis and dilated cardiomyopathy in male BALB/c mi
86 years) with a history of biopsy-proven viral myocarditis and drug-refractory VT; 5 patients presented
87 ponses are elevated during acute and chronic myocarditis and have been found to contribute to cardiac
88 to discriminate between patients with acute myocarditis and healthy controls was 86% for T2>52 ms, 7
91 c alpha myosin heavy chain (alphaMYHC) cause myocarditis and mice tolerized to alphaMYHC are protecte
92 S100A8 and S100A9 aggravate CVB3-induced myocarditis and might serve as therapeutic targets in in
95 te phase is associated with high parasitism, myocarditis and profound myocardial gene expression chan
97 ally for patchy diseases such as lymphocytic myocarditis and sarcoidosis) using the gold-standard Dal
98 ors promotes CD4+ T-cell-mediated autoimmune myocarditis and subsequent inflammatory cardiomyopathy.
99 ic processes of coxsackievirus-induced viral myocarditis and to screen antiviral therapeutics for eff
101 lysis in 130 adult patients with acute viral myocarditis and viral prodrome within 2 weeks from the o
103 om ITAMY (ITalian multicenter study on Acute MYocarditis) and evaluated CMR results from 386 patients
104 ed heart failure, biventricular failure, and myocarditis), and explore management considerations for
106 is groups, including dilated cardiomyopathy, myocarditis, and ischemic and nonischemic cardiomyopathi
107 MB versus MBIso both in moderate and severe myocarditis, and MBCD4 signal correlated with CD4+ T-lym
108 piral load for uncomplicated, renal failure, myocarditis, and multi-organ failure patients were 8616,
109 atients with cardiac sarcoidosis, giant cell myocarditis, and myocarditis associated with connective
110 estinal perforation), one patient because of myocarditis, and one patient because of multiorgan failu
111 of the right subclavian artery, one case of myocarditis, and one pulmonary malignancy with pulmonary
112 lammatory mediator during adenovirus-induced myocarditis, and persistent adenovirus infection may con
114 s known about the pathogenesis of adenovirus myocarditis, and the species specificity of human adenov
115 ally and robustly in experimental autoimmune myocarditis, and thus allowed for an unprecedented insig
116 e long-term mortality in patients with viral myocarditis, and to establish the prognostic value of va
117 cenarios, such as suspected hypersensitivity myocarditis, anthracycline cardiomyopathy, cardiac tumor
118 ) and SAPS II, whereas immunosuppression and myocarditis as the reason for ECMO support were associat
119 rent standard Lake Louise criteria (LLC) for myocarditis as well as native T1, calculation of extrace
120 diseases caused by picornaviruses, including myocarditis, aseptic meningitis, encephalitis, hepatitis
121 iac sarcoidosis, giant cell myocarditis, and myocarditis associated with connective tissue disorders
122 ldren and adults is often the consequence of myocarditis associated with Coxsackievirus (CV) infectio
123 was significantly elevated in patients with myocarditis at FU0 (2.2 [2.0-2.3] versus 1.6 [1.5-1.7];
124 rarely develop life-threatening acute viral myocarditis (AVM), given that the causal viral infection
126 fected C57Bl/6 male mice developed increased myocarditis but reduced hepatic injury compared with inf
127 ositive patients compared with patients with myocarditis but without evidence of persistent CVB3 infe
128 arrhythmias are characteristic of giant cell myocarditis, but their true incidence, predictors, and o
129 re studied after the induction of autoimmune myocarditis by immunization with alpha-myosin-peptide; 2
131 hat Th1-type immunity protects against acute myocarditis by reducing viral replication and prevents t
135 pathic) and can be mechanistically caused by myocarditis, conduction abnormalities, focal direct inju
136 ory microRNA-155 is upregulated during acute myocarditis, contributes to the adverse inflammatory res
137 gic encephalomyelitis (EAE) and experimental myocarditis, correlates with the natural variation in co
139 of two patients with melanoma in whom fatal myocarditis developed after treatment with ipilimumab an
142 After adjustment for severity of illness, myocarditis does not confer additional risk for wait-lis
143 with metastatic melanoma who developed fatal myocarditis during ipilimumab and nivolumab combination
145 s study, we used the experimental autoimmune myocarditis (EAM) model to determine the role of eosinop
147 ice developed severe experimental autoimmune myocarditis (EAM), in which mice are immunized with card
148 model of TnI-induced experimental autoimmune myocarditis (EAM), we demonstrated that both local and s
152 amatic presentation, patients with fulminant myocarditis (FM) might have better outcome than those wi
160 pite higher allosensitization, patients with myocarditis had similar post-transplant rejection, retra
164 he human heart long after the signs of acute myocarditis have abated are still not completely underst
166 regarding athletic participation after acute myocarditis have heightened the importance of early diag
167 Among children listed for HT, those with myocarditis have more severe heart failure than children
168 d countries, viral infections commonly cause myocarditis; however, in the developing world, rheumatic
171 AM-guided biopsy diagnosed ARVC in 11 (46%), myocarditis in 8 (33%), and idiopathic RVOT-VAs in 5 (21
172 demonstrate that ANT1 can induce autoimmune myocarditis in A/J mice by generating autoreactive T cel
173 Necroscopic findings were compatible to myocarditis in both, and immunohistochemistry for dengue
176 raction values for assessing the activity of myocarditis in patients with recent-onset heart failure
177 esonance (CMR) techniques to identify active myocarditis in patients with recent-onset heart failure.
179 l inflammation and leukocyte infiltration in myocarditis in the absence of a detectable decline in le
180 n for 147 patients with a diagnosis of acute myocarditis in the Extracorporeal Life Support Organizat
183 del will enable fundamental studies of viral myocarditis, including IFN-gamma modulation as a therape
192 ge of clinical symptoms, biopsy-proven viral myocarditis is associated with a long-term mortality of
195 f chronic dilated cardiomyopathy after viral myocarditis is complex and determined by host and viral
196 HT and used Cox models to determine whether myocarditis is independently associated with wait-list m
201 rders in which inflammation of the heart (or myocarditis) is the proximate cause of myocardial dysfun
203 ell and lymphocytic infiltrates, lymphocytic myocarditis-like foci, cardiomyocyte necrosis, and cardi
204 rhythmias, ischemia, and pericarditis and/or myocarditis-like syndromes, or they can be chronic, such
206 sample of patients with biopsy-proven active myocarditis, looking for accessible and valid early pred
209 - 23 ms in HTN subjects; p < 0.05), areas of myocarditis (median 22% LV with T1 >990 ms, as compared
211 l outcomes, including hydrops fetalis, fetal myocarditis, meningoencephalitis, neurodevelopmental del
214 zed granzyme B activity in hearts with acute myocarditis monitored by fluorescent molecular tomograph
215 athies in developed countries is lymphocytic myocarditis most commonly caused by a viral pathogenesis
216 safety end point was incidence of infectious myocarditis, myocardial rupture, neoplasm, hypersensitiv
219 al diagnosis includes myocardial infarction, myocarditis, neurogenic pulmonary edema, and nonischemic
222 onance imaging in only 24% of patients, with myocarditis occurring in 33% and no significant abnormal
223 io 41.5, 95% confidence interval 35.4-48.8), myocarditis (odds ratio 36.6, 95% confidence interval 21
226 th T2 and T1 mapping reliably detected acute myocarditis, only T2 mapping discriminated between acute
227 and November 2016 with a diagnosis of acute myocarditis (onset of symptoms <1 month) of whom 55 requ
228 lar dysfunction resulting from KD-associated myocarditis or between patients with and without coronar
230 larly, myocardial fibrosis in the absence of myocarditis or left ventricular hypertrophy, or other kn
233 ts with giant-cell myocarditis, eosinophilic myocarditis, or cardiac sarcoidosis and those <15 years
234 irus B3 (CVB3) is a causative agent of viral myocarditis, pancreatitis, and meningitis in humans.
235 important human causative pathogen for viral myocarditis, pancreatitis, and meningitis, has evolved d
236 hy (NICM) and ischemic cardiomyopathy (ICM), myocarditis patients were younger (myocarditis 43.4+/-14
239 and endomyocardial biopsy can help identify myocarditis, predict risk of cardiovascular events, and
243 Twenty-nine patients were examined with a myocarditis protocol (group A), 10 patients with a stres
244 establish a mouse model of human adenovirus myocarditis, providing the means to study host and patho
246 es (5, 14%), dilated cardiomyopathy (3, 8%), myocarditis related (3, 8%), aortic dissection (3, 8%),
253 tibody asialogangloside GM-1 did not augment myocarditis severity in eosinophil-deficient Deltadouble
255 -asialo GM1 antibody significantly increased myocarditis severity, and was accompanied by elevated fi
258 drial proteins can be target autoantigens in myocarditis, supporting the notion that the antigens rel
260 significantly longer in patients with acute myocarditis than in control subjects (1185.3 msec +/- 49
262 arkers are commonly interpreted as resolving myocarditis, this assumption has not been confirmed as o
263 ty signal transducer IRAK4 exacerbates viral myocarditis through inhibition of interferon production
264 comparable symptoms, including inflammatory myocarditis, through chronic activation of the stimulato
265 conclusion, eosinophils drive progression of myocarditis to DCMi, cause severe DCMi when present in l
266 ation can be used effectively in adults with myocarditis to support the circulation while awaiting my
268 Six hundred seventy patients with suspected myocarditis underwent CMR including late gadolinium enha
269 entricular function and clinically suspected myocarditis underwent endomyocardial biopsy and CMR at 1
270 imental cytotoxic CD8+ T lymphocyte-mediated myocarditis using fluorescence reflectance imaging, vali
275 nificantly contributes to the development of myocarditis was hypothesized by the first physician-scie
280 We found that microRNA-155 expression during myocarditis was localized primarily in infiltrating macr
282 portance in the pathogenesis of CVB3-induced myocarditis, we aimed to unravel the role of NOD2 in CVB
283 condition for the development of the chronic myocarditis, we hypothesized that CD73 activity may coun
284 ace, smaller left ventricular dimension, and myocarditis were associated with death after transplanta
286 2009, 82 patients with biopsy-proven active myocarditis were consecutively enrolled and followed-up
288 bacteremia/sepsis, acute renal failure, and myocarditis were rare (each </= 2% of children) but asso
289 his discrepancy was particularly evident for myocarditis, whereas in infiltrative and storage disease
290 s known about the pathogenesis of adenovirus myocarditis, which is a significant impediment to the de
291 Previous studies suggest that children with myocarditis who receive heart transplantation (HT) may b
293 fractory cardiac electrical instability, and myocarditis with a robust presence of T-cell and macroph
294 old woman who was hospitalized for fulminant myocarditis with biventricular failure and cardiogenic s
297 ophilic IL-5Tg mice resulted in eosinophilic myocarditis with severe ventricular and atrial inflammat
298 n, we treated a patient with fulminant viral myocarditis with the interleukin-1 receptor blocking age
300 late heart failure pathogenesis during viral myocarditis, yet their identities and functions remain p
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