1  m.8344A>G mutation and epilepsy experienced myoclonic seizures.

2 or EPM1 by displaying progressive ataxia and myoclonic seizures.

3 behind the role of LGI1 in susceptibility to myoclonic seizures.

4 tonic-clonic seizures (GTCS) and a second to myoclonic seizures.

5  age, the homozygous mutant mice all exhibit myoclonic seizures accompanied by rapid jumping and runn

6 e found that mice lacking cystatin B develop myoclonic seizures and ataxia, similar to symptoms seen

7 -THP) had a significantly lower incidence of myoclonic seizures and less EEG activity following penty

8                  Expanded phenotypes include myoclonic seizures, auditory or visual hallucinations, a

9  By 1 year most Spnb3(-/-) animals develop a myoclonic seizure disorder with significant reductions o

10 , but nearly all P20-22 and P30-46 mSMEI had myoclonic seizures followed by generalized seizures caus

11 e (PTZ)-induced generalized tonic-clonic and myoclonic seizure incidence and severity.

12 ents, and include tonic-clonic, absence, and myoclonic seizures, including status epilepticus.

13    All but one of these patients had similar myoclonic seizures induced by linguistic activities othe

14 pilepsy (PME) is a syndrome characterized by myoclonic seizures (lightning-like jerks), generalized c