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1 g MyoD, myogenin, serum response factor, and myocyte enhancer factor 2.
2 n II-dependent transcriptional activities of myocyte enhancer factor 2.
3 ic translocation, resulting in activation of myocyte enhancer factor 2.
4 the derepression of the transcription factor myocyte enhancer factor 2.
5 ess cardiac transcription factors GATA-4 and myocyte enhancer factor-2.
6 or-2, c-Fos, c-Jun, activator protein-1, and myocyte enhancer factor-2.
7 teraction with the beta-catenin promoter via myocyte enhancer factor-2.
8 transcription factors Sp1 and Sp3 and/or the myocyte enhancer factor 2D.
9 is and inhibit transcriptional activation by myocyte enhancer factors 2.
10 represses the transcriptional activities of myocyte enhancer factor 2 and cAMP response element-bind
13 associated with class II histone deacetylase/myocyte enhancer factor-2 and calcineurin signaling path
14 e known downstream effectors of calcineurin (myocyte enhancer factor-2 and NFAT) cannot completely ac
15 ted pathological gene expression directed by myocyte-enhancer factor-2 and nuclear factor of activate
16 s, we identify rearrangements between MEF2D (myocyte enhancer factor 2D) and five genes (BCL9, CSF1R,
17 onal factors, such as stimulating protein 1, myocyte enhancer factor 2, and nuclear factor of activat
18 resses the levels of Kruppel-like factor 15; myocyte enhancer factor 2, and peroxisome proliferator-a
20 KLF4 promoter, and competitive blocking of a myocyte enhancer factor-2 binding site in the KLF4 promo
22 the transcriptional activation of Nur77 via myocyte enhancer factor 2-binding sites in its promoter.
23 novel Scl target gene, transcription factor myocyte enhancer factor 2 C (Mef2C) from Scl(fl/fl) feta
24 es Cabin1, a transcriptional corepressor for myocyte enhancer factor 2, creating a docking site for 1
25 of HDAC-1/2/3 with Nrf2 and HDAC-3/5/7 with myocyte enhancer factor-2; deacetylation of these factor
26 y by the pattern of expression of Drosophila myocyte enhancer factor 2 (DMEF2)-like immunoreactivity.
30 ctionally interacts with TEF-1 and also with myocyte enhancer factor 2 in a mammalian two-hybrid assa
31 These data suggest that ERK5 activation of myocyte enhancer factor 2-induced gene expression may pl
33 lial KLF4 expression through DNMT enrichment/myocyte enhancer factor-2 inhibition mechanisms of KLF4
34 um response factor) box transcription factor myocyte enhancer factor-2D (MEF-2D) acts as the principa
35 n of muscle-specific genes by members of the myocyte enhancer factor 2 (MEF2) and MyoD families of tr
36 d by the ASD-associated transcription factor myocyte enhancer factor 2 (MEF2) and that Brg1 regulates
37 g pattern of the muscle transcription factor myocyte enhancer factor 2 (Mef2) by promoting exclusion
41 We studied the role of transcription factor myocyte enhancer factor 2 (MEF2) during HSC activation.
44 ein-protein interactions with members of the myocyte enhancer factor 2 (MEF2) family of MADS domain t
45 p-helix (bHLH) transcription factors and the myocyte enhancer factor 2 (MEF2) family of MADS-box tran
46 exists between PGC-1alpha and members of the myocyte enhancer factor 2 (MEF2) family of transcription
49 vely activates transcription mediated by the myocyte enhancer factor 2 (MEF2) family of transcription
50 ression of muscle-specific genes through the myocyte enhancer factor 2 (MEF2) family of transcription
54 ta(B) or delta(C) induced transactivation of myocyte enhancer factor 2 (MEF2) gene expression and up-
58 ted the importance of the myogenic regulator myocyte enhancer factor 2 (MEF2) in muscle differentiati
59 The activity-dependent transcription factor myocyte enhancer factor 2 (MEF2) induces excitatory syna
62 sphorylation of the neuronal survival factor myocyte enhancer factor 2 (MEF2) leading to its inactiva
67 his conserved region is a consensus site for myocyte enhancer factor 2 (MEF2) proteins that we show i
70 gate the contribution of ERK5 and its target myocyte enhancer factor 2 (MEF2) to neurotrophin-3/TrkC-
72 vious studies have shown that members of the myocyte enhancer factor 2 (MEF2) transcription factor fa
73 unction as transcriptional repressors of the myocyte enhancer factor 2 (MEF2) transcription factor, f
74 ifferentiation through associations with the myocyte enhancer factor 2 (MEF2) transcription factor.
86 vel MHCI signaling pathway that requires the myocyte enhancer factor 2 (MEF2) transcription factors.
87 t corepressor that inhibits transcription by myocyte enhancer factor 2 (MEF2) transcription factors.
88 r localization and reduced expression of the myocyte enhancer factor 2 (MEF2) transcriptional target
89 ent fashion, the transactivation activity of myocyte enhancer factor 2 (MEF2), a protein known to coo
90 on is controlled by the transcription factor myocyte enhancer factor 2 (MEF2), but how MEF2 is activa
91 lves SOST expression reduction by inhibiting myocyte enhancer factor 2 (MEF2), which activates a dist
92 on, we have identified a perfectly conserved myocyte enhancer factor 2 (MEF2)-binding domain (-CTAAAA
93 ctivation of both Tcon and Treg cells led to myocyte enhancer factor 2 (Mef2)-induced expression of g
94 the present study we have determined that a myocyte enhancer factor 2 (MEF2)-related nuclear factor,
101 , flow induced the dissociation of HDAC5 and myocyte enhancer factor-2 (MEF2) and enhanced MEF2 trans
102 iation is controlled by interactions between myocyte enhancer factor-2 (MEF2) and myogenic basic heli
103 Class II histone deacetylases (HDACs) bind myocyte enhancer factor-2 (MEF2) and repress specific ge
104 report tripartite co-operation between MyoD, myocyte enhancer factor-2 (MEF2) and the thyroid hormone
106 ription in collaboration with members of the myocyte enhancer factor-2 (MEF2) family of MCM1-agamous-
109 at mediates interactions with members of the myocyte enhancer factor-2 (MEF2) family of transcription
111 hat mediate interactions with members of the myocyte enhancer factor-2 (MEF2) family of transcription
112 tic enzymes that depends on interaction with Myocyte Enhancer Factor-2 (MEF2) for their recruitment t
114 xpression of the muscle differentiation gene Myocyte enhancer factor-2 (Mef2) is normally delayed in
115 nmuscle bHLH proteins E12 or E47 or with the myocyte enhancer factor-2 (MEF2) protein, which acts coo
116 arval muscle splitting is likely mediated by myocyte enhancer factor-2 (MEF2) since in Mef2 hypomorph
117 sitide dependent protein kinase-1 (Pdk1) and myocyte enhancer factor-2 (Mef2) to trigger neurodegener
121 ig MAPK-1 (BMK1), an MAPK that activates the myocyte enhancer factor-2 (MEF2) transcription factor.
125 on (CGN) survival depends on activity of the myocyte enhancer factor-2 (MEF2) transcription factors.
126 ses GnRH gene expression via homeodomain and myocyte enhancer factor-2 (MEF2) transcription factors.
127 MMP10 gene transcription by associating with myocyte enhancer factor-2 (MEF2), a direct activator of
128 ter containing an essential binding site for myocyte enhancer factor-2 (MEF2), a stress-responsive tr
129 Three sites within the proximal promoter (myocyte enhancer factor-2 (MEF2), E-box, and CACC box) w
130 determine if hypertrophic signals activated myocyte enhancer factor-2 (Mef2), we studied mice carryi
131 is a member of class IIa HDACs that regulate myocyte enhancer factor-2 (MEF2)-mediated transcription
135 between the p38 pathway and a member of the myocyte-enhancer factor 2 (MEF2) group of transcription
137 dzelm, Cherry et al. (2015) demonstrate that myocyte enhancer factor 2D (MEF2D) binds and activates r
139 mediated autophagy regulated the activity of myocyte enhancer factor 2D (MEF2D), a transcription fact
140 ngs on the dysregulation of survival factor, myocyte enhancer factor 2D (MEF2D), by extrasynaptic NMD
141 lates and activates the transcription factor myocyte enhancer factor 2D (MEF2D), which plays a critic
142 emi-restricted transcription factors such as myocyte enhancer factor 2, nuclear factor of activated T
143 peptide) and transcription factor activity [myocyte enhancer factor-2, nuclear factor kappa B (NF-ka
144 tor), SRF (serum response factor), and MEF2 (myocyte enhancer factor 2) play critical roles in the me
147 In contrast, HDAC4 is directly recruited to myocyte enhancer factor 2 sites within target promoters
151 ated reduction of shear stress-mediated ERK5/myocyte enhancer factor 2 transcriptional activity, as w
152 nucleus and acts as a dominant inhibitor for myocyte enhancer factor 2 transcriptional activity.
153 r showed that the PKD-HDAC5 pathway mediated myocyte enhancer factor-2 transcriptional activation and
154 ctors, the serum response factor, Oct-1, and myocyte enhancer factor-2, whereas the other two genes (
155 d the pleiotropic transcription factor Mef2 (myocyte enhancer factor 2), which profoundly influences
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