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1  miR-431 plays a key role in maintaining the myogenic ability of skeletal muscle with age.
2 enic regulatory factors in vitro, suggesting myogenic activation.
3 e MEF2Calpha2 isoform in RMS cells increased myogenic activity and promoted differentiation in RMS ce
4 uitously expressed isoform that exhibited no myogenic activity and that MEF2Calpha2, the muscle-speci
5 tch that controls mesenchymal cell fate into myogenic and osteogenic lineages.
6 scles express dystrophin in up to 70% of the myogenic area and increased force generation following i
7  AMPKalpha1 deficiency had severely impaired myogenic capacity in regenerating muscle fibers.
8 TR, inhibition of miR-431 also repressed the myogenic capacity of human skeletal myoblasts.
9                                          The myogenic capacity of myoblasts decreases in skeletal mus
10  Importantly, elevating miR-431 improved the myogenic capacity of old myoblasts, while inhibiting end
11 rcise along with a decline in SC density and myogenic capacity.
12 reprogramming of muscle stem cells modulates myogenic cell fate.
13 nopathy, we investigated in vitro macrophage-myogenic cell interactions and found that Dysf-deficient
14                                           In myogenic cell lines, phosphorylation of p38beta residue
15 ast some ARMSs and the PAX3-FOXO1-expressing myogenic cells and demonstrate that fusion RNA profiling
16 e partially reprogrammed from differentiated myogenic cells and display a pluripotent-like state.
17  is transcribed from an intergenic region of myogenic cells and its expression is upregulated during
18 s that upregulate expression of PTPRQ in non-myogenic cells and MYF5/MYF6 in myogenic cells.
19          Here we detail a protocol to derive myogenic cells from human embryonic stem (ES) and induce
20 sue; therefore, an unlimited availability of myogenic cells has applications in regenerative medicine
21 e long been recognized as the main source of myogenic cells in adult muscle, most of the knowledge ab
22 uble mutant, whereas excessive production of myogenic cells is observed in the trunk.
23       At the forelimb level, endothelial and myogenic cells migrate from adjacent somites into the li
24 minantly expressed by macrophages but not by myogenic cells or capillary endothelia cells in injured
25 thesis that TRIM32 is involved in control of myogenic cells proliferation and differentiation.
26                   shRNA knockdown of CD82 in myogenic cells reduces myoblast proliferation, suggestin
27   Constitutive PKD activation in mouse C2C12 myogenic cells regulated metabolic genes and glucose met
28 n response to muscle injury, and the derived myogenic cells then fuse to repair damaged muscle fibers
29 through embryoid body method and MYF5-GFP(+) myogenic cells were sorted and characterized.
30 ICAM-1-ICAM-1 interactions was restricted to myogenic cells, as forced expression of ICAM-1 by fibrob
31 ntiation and retrospective identification of myogenic cells.
32  muscle tissues ('myobundles') using primary myogenic cells.
33 PTPRQ in non-myogenic cells and MYF5/MYF6 in myogenic cells.
34  were also seen in PTPRQ mRNA-expressing non-myogenic cells.
35  and are critical for differentiation of the myogenic cells.
36 e correction via homologous recombination in myogenic cells.
37 nts the adhesive and fusogenic properties of myogenic cells.
38              Although MRFs are essential for myogenic commitment and differentiation, timely repressi
39 SCs led to increased propensity for terminal myogenic commitment connected to impaired proliferative
40 which encodes a transcriptional regulator of myogenic commitment, via binding to the MyoD mRNA 3' unt
41 nges in the transcriptional machinery toward myogenic commitment.
42 n receptor-like kinase receptors, to enhance myogenic commitment.
43 ts level of expression correlates with their myogenic competence.
44 determine if AT1 R mechanoactivation affects myogenic constriction in skeletal muscle arterioles and
45 of AT1 Ra increases TRPM4 currents to induce myogenic constriction of cerebral arteries.
46 ributes significantly to force generation in myogenic constriction of cerebral resistance arteries.
47  candesartan, indicating that AT1 R-mediated myogenic constriction relies on Ca(2+) -independent down
48 mechanosensor that contributes to arteriolar myogenic constriction.
49 d knockdown of AT1 Rb selectively diminished myogenic constriction.
50     Results also support both neurogenic and myogenic contributions of polyQ AR to several acute aspe
51 e also observed in a model of MYOD1-mediated myogenic conversion of human fibroblasts, and in primary
52                                              Myogenic cultures were isolated from the skeletal muscle
53 nto the myogenic program also depends on the myogenic determination factor (MyoD) family of genes, bu
54 of myogenesis that lead to the activation of myogenic determination genes and subsequent differentiat
55 use models, we showed that COUP-TFII hinders myogenic development by repressing myoblast fusion.
56  that quiescent MuSCs express high levels of Myogenic Differentiation 1 (MyoD) transcript in vivo, wh
57 omoting myogenic lineage progression through myogenic differentiation 1 (Myod1) regulation.
58 rom Deltex2 knockout mice exhibit precocious myogenic differentiation and accelerated regeneration in
59 Pax7(Lo) subpopulation is enriched, enhances myogenic differentiation and accelerates muscle regenera
60 ion indicates that Mesp-b acts by inhibiting myogenic differentiation and by inducing the dermomyotom
61 us, DUX4-mediated activation of Ret prevents myogenic differentiation and could contribute to FSHD pa
62 al lethal, we explored its potential role in myogenic differentiation and development by generating a
63 G translocates to the nucleus in response to myogenic differentiation and sublethal dose of cisplatin
64 ediating the repressive effect of hypoxia on myogenic differentiation and suggests that inhibition of
65 mely paired box 7 (satellite cell) and early myogenic differentiation and terminal differentiation (m
66 ified that LSD1 is the only KDM required for myogenic differentiation and that KDM3B, KDM6A, and KDM8
67 el can help better understand the process of myogenic differentiation and the effects of mechanical c
68 22 was physiologically induced during normal myogenic differentiation and was transcriptionally regul
69                                        Using myogenic differentiation as a model, we found that reduc
70 cularly given the lack of drugs that promote myogenic differentiation available for potential clinica
71   These data demonstrate that Ascl2 inhibits myogenic differentiation by targeting MRFs and facilitat
72 on of NKX2-5 or mutant DMPK 3'UTR results in myogenic differentiation defects, which can be rescued b
73 Accordingly, MyoD or Myog expression rescues myogenic differentiation despite Ascl2 overexpression.
74 satellite cells impairs their activation and myogenic differentiation during muscle regeneration.
75                           Here, we show that myogenic differentiation factors regulate miR-182 levels
76 we identified HDAC3 as a major suppressor of myogenic differentiation from a high-efficiency Clustere
77 e identified three compounds which inhibited myogenic differentiation in C2C12 myoblasts; (+)-JQ1, PF
78 w that myomiR release accompanies periods of myogenic differentiation in cell culture and in vivo.
79 show that cell migration, proliferation, and myogenic differentiation in pre-culture SBB-treated grou
80 signaling rescues CNC cell proliferation and myogenic differentiation in these mutant mice.
81               We found that Deltex2 inhibits myogenic differentiation in vitro, and that skeletal mus
82 transient Myod1 induction efficiently drives myogenic differentiation into multinucleated myotubes.
83                     The process of stem cell myogenic differentiation is interpreted as the interplay
84  Pak1 and Pak2 display delayed expression of myogenic differentiation markers and myotube formation.
85 s repression of the Runx2 P1 promoter during myogenic differentiation of mesenchymal cells.
86 ycling that plays a nonredundant role in the myogenic differentiation of muscle precursors, limiting
87 ted ERK nuclear translocation induced robust myogenic differentiation of muscle progenitor/stem cells
88 -culturing the dKO-nmMSCs with dKO-MPCs, the myogenic differentiation potential of the dKO-MPCs was r
89                                              Myogenic differentiation proceeds through a highly coord
90 rs for noninvasive in vivo monitoring of the myogenic differentiation process from muscle precursor c
91 leads to Pax7 cleavage and initiation of the myogenic differentiation program.
92 form essential for normal progression of the myogenic differentiation programme.
93       Importantly, Bhlhe40 knockdown rescues myogenic differentiation under hypoxia.
94     We previously showed that RSPO2 promoted myogenic differentiation via activation of WNT/beta-cate
95                                      Induced myogenic differentiation was associated with a decrease
96 immortalized cells retained the capacity for myogenic differentiation when treated with the steroid h
97 urthermore, the model of bexarotene-enhanced myogenic differentiation will provide an important avenu
98 myogenin and a specific Mef2 isoform induced myogenic differentiation without activating endogenous M
99  considered to play an important role during myogenic differentiation, chronological alterations in D
100 he dynamic protein changes that occur during myogenic differentiation, demonstrating the feasibility
101 sults predict the kinetics of the process of myogenic differentiation, including the number of cells
102 rference revealed that BRD4 was required for myogenic differentiation, whereas BRD3 down-regulation r
103 eta superfamily signaling is an inhibitor of myogenic differentiation, with elevated activity in aged
104 ion protein 1 (MYOD1)-mediated activation of myogenic differentiation.
105  offer a useful resource for others studying myogenic differentiation.
106 a molecular mechanism by which 4.1R promotes myogenic differentiation.
107 o enhance cellular proliferation and inhibit myogenic differentiation.
108 oft tissue cancer characterized by disturbed myogenic differentiation.
109 critical in muscle development and regulates myogenic differentiation.
110  detrimental effects of cachectic factors on myogenic differentiation.
111 a nanoscale stiffness range known to support myogenic differentiation.
112 inately regulate muscle-related genes during myogenic differentiation.
113 al regulators of muscle-related genes during myogenic differentiation.
114 m-dependent signalling pathways that control myogenic differentiation.
115 re cultured in low-serum medium to stimulate myogenic differentiation.
116 y the role of Wnt-beta-catenin signalling in myogenic differentiation.
117 y, up-regulates Myog expression and promotes myogenic differentiation.
118 nesis, indicating that Mdm2 is necessary for myogenic differentiation.
119  propose that ZBED6 plays a critical role in myogenic differentiation.
120 ted posttranscriptional switch that controls myogenic differentiation.
121 xpressed at the sarcolemma of myotubes after myogenic differentiation.
122 as BRD3 down-regulation resulted in enhanced myogenic differentiation.
123 anscription factor myogenin, which regulates myogenic differentiation.
124 reas siRNA-mediated knockdown of Ret induced myogenic differentiation.
125 to multiple lineages, such as neurogenic and myogenic differentiations; they also display a superior
126 or the muscular dystrophies (MD's) and other myogenic disorders.
127                           BETi-mediated anti-myogenic effects were also observed in a model of MYOD1-
128                                          The myogenic electric organ has evolved six times in fishes
129                             In contrast, the myogenic enhancer chromatin regions distal to MYOD1 were
130 n Mt, like MYOG itself, and exhibited nearby myogenic enhancer chromatin.
131 y be enhancers acting in concert to regulate myogenic expression of multiple adjacent genes.
132  myogenic regulatory factors-muscle-specific myogenic factor 5, myoblast determination 1, and myogeni
133 e characteristic SS18-SSX fusion oncogene in myogenic factor 5-expressing (Myf5-expressing) cells dev
134 rk for studying interactions between general myogenic factors and iTFs in evolutionary diversificatio
135 by the expression of certain combinations of myogenic factors.
136              Cells differentiating towards a myogenic fate on aligned topographies adopt a characteri
137 n in response to injury, indicating that the myogenic function of MLX is manifested in vivo.
138 es a major contribution to the regulation of myogenic function, while E2f2 appears to be less importa
139 m-regulated phosphatase that is required for myogenic gene expression and skeletal muscle differentia
140 yme, which is required for the activation of myogenic gene expression, is a calcineurin substrate.
141 indirectly on multiple promoters to increase myogenic gene expression.
142 ed more strongly with and recruited HDAC5 to myogenic gene promoters to repress muscle-specific genes
143 gned CNTs, as confirmed by the expression of myogenic genes and proteins.
144 nistically, Linc-RAM regulates expression of myogenic genes by directly binding MyoD, which in turn p
145 regulates nuclear PI5P and the expression of myogenic genes during myoblast differentiation.
146 orm us that Tbx1 is required upstream of key myogenic genes needed for core mesoderm cell survival an
147 s, NET39, Tmem38A, and WFS1, direct specific myogenic genes to the nuclear periphery to facilitate th
148 f knock-in reporter human iPS cell lines for myogenic genes which can be used for disease modeling, d
149 criptional regulation of conserved and novel myogenic genes.
150 uscle repair via macrophage secretion of pro-myogenic growth factors.
151 d increasing MT values during the 28 days of myogenic in vivo differentiation and did not reach the v
152 used to noninvasively monitor the process of myogenic in vivo differentiation of hMPCs as a biomarker
153 to a softer blend of PDMS muprinted with FN, myogenic index, myotube width, and myotube length on mum
154 -derived mesenchymal stem cells and promotes myogenic induction of C2C12 mouse myoblasts, whereas dep
155 actor Bhlhe40 as a candidate mediator of the myogenic inhibitory effect of hypoxia.
156 and by recombinant IGF2, which activates the myogenic kinase Akt.
157 in the limb is required for the migration of myogenic limb progenitors.
158 regulates satellite cell behavior, promoting myogenic lineage progression through myogenic differenti
159 ll muscle progenitor state - a distinct cell myogenic lineage responsible for postnatal growth and re
160 s study characterized a unique population of myogenic lineage stem cells that can be isolated from ad
161 iation of activated satellite cells into the myogenic lineage.
162 e and cell differentiation to osteogenic and myogenic lineages.
163                      An in silico screen for myogenic long noncoding RNAs (lncRNAs) revealed nine lnc
164 the expression of the cardiac progenitor and myogenic marker Nkx2.5, or that of the myocardial marker
165 tory markers and increased the expression of myogenic markers in the skeletal muscle.
166 Differentiated cells exhibited expression of myogenic markers such as MyoD and Myog.
167 es TrxR1 levels and delays the expression of myogenic markers, suggesting the involvement of miR-23 i
168  by terminating Tbx6-dependent inhibition of myogenic maturation.
169 f the disease can be caused by neurogenic or myogenic mechanisms, we made use of the tet-On and Cre-l
170                                  We recorded myogenic MEPs after transcranial motor cortex stimulatio
171 lls also demonstrated delayed induction of 3 myogenic microRNAs (miRNAs), miR-1, miR-206, and miR-486
172  was examined in two mouse models of SBMA, a myogenic model that overexpresses wildtype androgen rece
173 ment accumulations were observed only in the myogenic model, even though axonal transport dysfunction
174 g in DMD patients should not only target the myogenic MPCs but should also attempt to prevent the act
175 ence, we termed this population of cells non-myogenic MSCs (nmMSCs); and (ii) a slowly adhering cell
176 ral circuits show that neurons can control a myogenic muscle organ not only by changing the contracti
177 al circuits have long been known to modulate myogenic muscles such as the heart, yet a mechanistic un
178 ing of the Caenorhabditis elegans pharynx, a myogenic muscular pump for feeding, and found three neur
179 e from the STS-131 mission exhibited reduced myogenic (Myf5 and -6) and adipogenic (Pparg, Cebpa, and
180               Finally, the comparison of the myogenic network architecture among different animal gro
181 sal physiological mechanism that may involve myogenic, neural control as well as metabolic regulation
182            Remarkably, deletion of Tgfbr2 in myogenic or chondrogenic progenitor cells does not manif
183 pidly adhering cell population, which is non-myogenic, Pax7(-) and express the mesenchymal stem cell
184 selected alveolar rhabdomyosarcoma (ARMS), a myogenic pediatric cancer whose exact cell of origin is
185          Tm2 overexpression rescued the hoip myogenic phenotype by promoting F-actin assembly at the
186 and fluorescent microscopy showed that their myogenic phenotype was sustained.
187 ty of the transduced cells, as well as their myogenic phenotype, were determined by flow cytometry an
188 ) SCs are shifted toward more differentiated myogenic population.
189 lled labeling and monitoring of lipogenic or myogenic populations of lung fibroblasts during fibrosis
190 , we define miRNA cocktails that promote the myogenic potential of human MiPs.
191         Silencing PW1/Peg3 markedly inhibits myogenic potential of mesoangioblasts in vitro through M
192 compromising each other, which indicates the myogenic potential of this combination of small RNAs.
193 rmal and these cells had evidence of reduced myogenic potential.
194 d generated a pool of endogenously corrected myogenic precursors in mdx mouse muscle.
195 se model ectopically expressing COUP-TFII in myogenic precursors to maintain COUP-TFII activity durin
196 ssion of TrxR1 during differentiation delays myogenic process, by negatively affecting the expression
197 t of our model and appear to orchestrate the myogenic process.
198 rium between proliferation and quiescence of myogenic progenitor and stem cells is tightly regulated
199 F-kappaB) and Notch signalling necessary for myogenic progenitor cell proliferation.
200      Furthermore, these PA scaffolds support myogenic progenitor cell survival and proliferation and
201 nse to hypertrophic stimuli and give rise to myogenic progenitor cells (MPCs) within the extracellula
202  of Pask in promoting the differentiation of myogenic progenitor cells, embryonic stem cells and adip
203 that cytoglobin is up-regulated in activated myogenic progenitor cells, where it localizes to the nuc
204 ely via interactions between CNC-derived and myogenic progenitor cells.
205 hts into both Wnt signalling and adult human myogenic progenitor differentiation.
206 tem cells (satellite cells, SCs) and derived myogenic progenitors (MPs); however, the factors respons
207 n be subcultured to produce large amounts of myogenic progenitors amenable to numerous downstream app
208 n in the fusion and differentiation of human myogenic progenitors and that dominant negative inhibiti
209                                          The myogenic progenitors do not die, but differentiate prema
210          Little is known about regulation of myogenic progenitors during tongue development when comp
211                         However, strikingly, myogenic progenitors fail to enter the limbs, leading to
212 ospective identification and purification of myogenic progenitors from human iPS cells.
213                                              Myogenic progenitors lacking Minion differentiate normal
214 man primary CD56(Pos) satellite cell-derived myogenic progenitors obtained from healthy individuals t
215 their endothelial identity and do not become myogenic progenitors or cardiomyocytes.
216                         Somites give rise to myogenic progenitors that form all of the muscles of the
217                                     Emerging myogenic progenitors were specified to form multinucleat
218           Re-orienting ghost fibers impacted myogenic progenitors' migratory paths and division plane
219                      Pax3-positive migratory myogenic progenitors, marked by expression of Lbx1, are
220                       In the Myf5-expressing myogenic progenitors, there is a cell-autonomous require
221 of genes, but Pax3 is not expressed in these myogenic progenitors, where different gene regulatory ne
222                               Entry into the myogenic program also depends on the myogenic determinat
223  Instead, the later emergence of a competing myogenic program and variable transgene dynamics over ti
224 ifferentiation led to an anticipation of the myogenic program because of an alteration of PcG protein
225  also restore MYOD's ability to activate the myogenic program in human senescent fibroblasts.
226 cle fate both by promoting an MRF-associated myogenic program in myoblasts and by maintaining an undi
227 -1 inhibition activates an miR-206-dependent myogenic program in RMS, offering a novel therapeutic st
228 ated Pax3/7-FoxO1 target genes and induced a myogenic program in RMS.
229 cetylation and deregulated activation of the myogenic program in SCs.
230 vior of these cells and their entry into the myogenic program is controlled by gene regulatory networ
231 porting a pivotal role for Shox2 in the core myogenic program orchestrating venous pole and pacemaker
232 pression that promotes the activation of the myogenic program, and is therefore termed Linc-RAM (Linc
233 everse senescence-mediated inhibition of the myogenic program.
234  cycle progression for the activation of the myogenic program.
235 DR precluded MYOD-mediated activation of the myogenic program.
236 egulation allows the progression through the myogenic programme.
237          RET is dynamically expressed during myogenic progression in mouse and human myoblasts.
238 nduced pluripotent stem cells (iPSCs) with a myogenic propensity are able to engraft into both cardia
239                                 Differential myogenic propensity did not influence pluripotency, but
240        Here, we determined that differential myogenic propensity influences the commitment of isogeni
241                             Importantly, MiP myogenic propensity is influenced by somatic lineage ret
242 urthermore, methods to enhance the intrinsic myogenic properties of MiPs are likely needed, given the
243                             Thus, these anti-myogenic proteins act as important inhibitors of synapse
244         Using progenitor markers, 3 distinct myogenic PW1(+) cell populations were isolated from the
245 al but concentration-dependent inhibition of myogenic reactivity.
246                                              Myogenic regulatory factor (MRF) genes, MYOD1, MYOG, MYF
247                                          The myogenic regulatory factor (MRF) MYF5 is the earliest to
248                                          The myogenic regulatory factor MRF4 is highly expressed in a
249                                          The myogenic regulatory factor MyoD has been implicated as a
250                                              Myogenic regulatory factors (MRFs), including Myf5, MyoD
251 ignificantly improved the gene expression of myogenic regulatory factors in vitro, suggesting myogeni
252 r cells express in a coordinated fashion the myogenic regulatory factors, while down-regulating the s
253    Conversely, the levels of expression of 3 myogenic regulatory factors-muscle-specific myogenic fac
254  the expression and functional activation of myogenic regulatory transcription factors (MRFs) are wel
255 RMS) is a pediatric malignacy of muscle with myogenic regulatory transcription factors MYOD and MYF5
256  plays an active role in both the arteriolar myogenic response and during changes in vascular tone in
257 rotein as a critical event to accomplish the myogenic response.
258 sis, reduced KV current density and restored myogenic responses in PAs from TgNotch3(R169C) mice, whe
259 in TgNotch3(R169C) and TgBAC-TIMP3 mice, and myogenic responses of brain arteries were likewise atten
260                             KV1 currents and myogenic responses were similarly altered in pial arteri
261                                      Altered myogenic responsiveness compromises tissue perfusion, ag
262 etion of p66Shc from these rats restored the myogenic responsiveness of renal preglomerular arteriole
263 lts demonstrate that the pharynx generates a myogenic rhythm in the presence of tonically released ac
264 y reprograms gene expression in BAT toward a myogenic signature, including increased expression of my
265 er human iPS cell line for MYF5, as an early myogenic specification gene, to allow prospective identi
266       IMuSCs express several pluripotent and myogenic stem cell markers; have the capability to form
267 , self-renewal/differentiation and fusion of myogenic stem cells (satellite cells).
268                      Differentiation of both myogenic stem cells and neural crest cells requires capz
269                    Satellite cells (SCs) are myogenic stem cells required for regeneration of adult s
270                Satellite cells are the major myogenic stem cells residing inside skeletal muscle and
271                   Muscle satellite cells are myogenic stem cells whose quiescence, activation, self-r
272 cell cycle withdrawal in fetal and postnatal myogenic stem cells, and assign to Ptpn11 signaling a ke
273 tion was observed in postnatal but not fetal myogenic stem cells.
274 estigated the relationship between different myogenic subpopulations and the function of canonical Wn
275      We found that Myf5- and MyoD-expressing myogenic subpopulations exist during embryonic tongue my
276          Our data demonstrate a lipogenic-to-myogenic switch in fibroblastic phenotype during fibrosi
277  cell population which is Pax7(+) and highly myogenic, termed muscle progenitor cells (MPCs).
278 e that a combinatorial anti-fibrotic and pro-myogenic therapy could be the foundation of future thera
279  in RH30 cells could be detected at the same myogenic time point(s).
280                    Conversely, we observed a myogenic-to-lipogenic switch during fibrosis resolution.
281 he AT1 R blocker losartan (1 mum) diminished myogenic tone and blocked stretch-induced cation current
282      HFD/STZ treatment induced a progressive myogenic tone augmentation in mesenteric and olfactory c
283 n-dependent inhibition of pressure-dependent myogenic tone consistent with previous reports of mechan
284 ilatory responses to an NO donor and loss of myogenic tone in KW animals were also rescued with Nox1
285 ooth muscle cell TNF drives the elevation of myogenic tone via enhanced sphingosine-1-phosphate (S1P)
286        However, we found that development of myogenic tone was greater in arteries from AT1 Ra knocko
287                                              Myogenic tone was unchanged, but over a range of transmu
288 depolarization and constriction to pressure (myogenic tone) were blunted in PAs from TgNotch3(R169C)
289 d from patients with diabetes have augmented myogenic tone, despite reasonable blood glucose control.
290 effect on blood glucose or resistance artery myogenic tone.
291  numerous physiological processes, including myogenic tone.
292  genes contributes to temporal regulation of myogenic transcription by restricting late gene expressi
293 m of the transcription start site of MyoD, a myogenic transcription factor gene.
294 ancer factor 2c) mRNA, which encodes the key myogenic transcription factor MEF2C.
295 man skin fibroblasts by forced expression of myogenic transcription factor MyoD, we performed quantit
296  and fusion associated with dysregulation of myogenic transcription factors and disruption of the nes
297 R-182 and provide insight into the role that myogenic transcription factors have in sarcoma progressi
298  induced Smad2/3 signaling and repression of myogenic transcription factors.
299 h muscle cell TNF augments resistance artery myogenic vasoconstriction in a diabetes model that induc
300                                    ABSTRACT: Myogenic vasoconstriction, which reflects the intrinsic

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