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1 ting with acute rhabdomyolysis and recurrent myoglobinuria.
2 cause of exercise-induced rhabdomyolysis and myoglobinuria.
3 severe isolated COX deficiency and recurrent myoglobinuria.
4 limb weakness, and in some cases, attacks of myoglobinuria.
5 l tide cycle, and the relative lack of renal myoglobinuria (26/26) suggested MSE onset on a rising ti
7 dered in patients with recurrent episodes of myoglobinuria and peripheral neuropathy presenting in la
8 ction of D3 on in vivo ARF (glycerol-induced myoglobinuria) and isolated proximal tubule injury respo
9 e presenting clinical features are myalgias, myoglobinuria, and an elevated serum creatine kinase.
10 terized by exercise intolerance, cramps, and myoglobinuria, and the other dominated by fixed, often p
11 ng with progressive exercise intolerance and myoglobinuria associated with complex III deficiency in
12 , especially of the exercise intolerance and myoglobinuria; biochemical and molecular features--molec
13 yopathy with severe exercise intolerance and myoglobinuria has been described in patients from northe
14 pin-2 deficiency in humans, leading to acute myoglobinuria in childhood or the inflammatory disorder
15 rse forms of acute renal tubular injury (eg, myoglobinuria, ischemia/reperfusion, urinary tract obstr
16 sing exercise intolerance, cramps, recurrent myoglobinuria, or fixed weakness, which often affects ex
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