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1 PCI MI definition and preventing PCI-related myonecrosis.
2 , that cause extensive tissue destruction in myonecrosis.
3 s to reduce tissue damage during clostridial myonecrosis.
4 ction did not correlate with the quantity of myonecrosis.
5 ents strongly associated with periprocedural myonecrosis.
6 and burden of CAD and degree of subclinical myonecrosis.
7 GAS interaction may prevent or attenuate GAS myonecrosis.
8 onin T is a sensitive and specific marker of myonecrosis.
9 educe the extent, but not the occurrence, of myonecrosis.
10 MRI technique allows direct visualization of myonecrosis.
11 e of infection, e.g., skin epithelium during myonecrosis.
12 ence in the muscle, resulting in devastating myonecrosis.
13 for the treatment of C. perfringens-mediated myonecrosis.
14 ine and morphine do not die from clostridial myonecrosis.
18 lts in greater suppression of periprocedural myonecrosis, although a survival benefit has not been de
19 availability of more sensitive biomarkers of myonecrosis and a new classification system from the uni
20 s strains produce severe diseases, including myonecrosis and enteritis necroticans, in humans and ani
21 ronary arteries would reduce peri-procedural myonecrosis and subsequently enhance event-free survival
22 ally used hydroxyl radical trap, reduces the myonecrosis and the mortality caused by an experimental
23 nflammation affects LV function by extending myonecrosis and/or altering LV remodeling remains unknow
24 ease was specific to C. perfringens-mediated myonecrosis; buprenorphine did not protect against disea
25 atients with minimal amounts of recent onset myonecrosis but elevated risk as indicated by CK-MB and
28 = 10), acute myocardial infarction (n = 10), myonecrosis due either to polymyositis or crush injuries
30 ng food poisoning, gas gangrene (clostridial myonecrosis), enteritis necroticans, and non-foodborne g
31 iseases, including gas gangrene (clostridial myonecrosis), enteritis necroticans, antibiotic-associat
32 ormal at 14 days with evidence of persisting myonecrosis, even though gene expression of regulators o
34 only a minor role in preventing the onset of myonecrosis in a mouse animal model of the disease (unpu
39 A streptococcal (GAS) necrotizing fasciitis/myonecrosis often necessitates extensive debridement to
41 or coated) approaches may be associated with myonecrosis related to atherosclerotic debris plugging t
43 ptococci in the dorsal muscle, and extensive myonecrosis that occurred far in advance of any systemic
44 MI definition sensitive for small degrees of myonecrosis (the occurrence of which, based on contempor
45 egments with "distal-type" procedure-related myonecrosis using high-resolution quantitative cardiovas
52 cular lymphocytic infiltrates and rare focal myonecrosis were evident on biopsy at 2 weeks postoperat
53 eria only PLC was necessary for the onset of myonecrosis, while at sublethal doses (10(6)) both PFO a
54 ed in muscle tissues from a patient with GAS myonecrosis who died of streptococcal toxic shock syndro
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