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1 tr(-/-) mice the structural integrity of the myotendinous and neuromuscular junctions are maintained.
2                                   For the 59 myotendinous and the 48 osteochondral diagnoses, the sen
3 cle, beta 1D was concentrated in costameres, myotendinous, and neuromuscular junctions.
4 n skin deep; it extends to the depths of the myotendinous architecture and the mechanical basis for p
5 tilage disease, microinstability of the hip, myotendinous injuries, and athletic pubalgia.
6 colemma localization, SPN is enriched at the myotendinous junction (MTJ) and neuromuscular junction (
7                              Movement of the myotendinous junction (MTJ) of the gastrocnemius mediali
8 responding tendon cells to form a functional myotendinous junction (MTJ) that allows for the force ge
9 ular matrix (ECM) that anchors fibers to the myotendinous junction (MTJ).
10 red for normal Laminin polymerization at the myotendinous junction (MTJ).
11 toward and subsequent differentiation of the myotendinous junction by priming formation of a protein
12 of muscle projections during early stages of myotendinous junction development.
13 rin expression during muscle development and myotendinous junction formation.
14 le-specific protein that is localized to the myotendinous junction in skeletal muscle.
15 , thus suggesting the important influence of myotendinous junction integrity on the severity of muscu
16  mdx/utr-/- background is the restoration of myotendinous junction integrity.
17 njections of choleratoxin subunit B into the myotendinous junction of MR or IR in monkeys.
18               In three animals the IR and MR myotendinous junction of one eye was injected simultaneo
19  alpha7 integrin and utrophin in maintaining myotendinous junction structure and enabling force trans
20 d at the sarcolemma, neuromuscular junction, myotendinous junction, and in peripheral nerve, but not
21  central nuclei at the perimysium and at the myotendinous junction.
22 ired for proper localization of PINCH at the myotendinous junction.
23 ole for dystrophin in maintaining vertebrate myotendinous junctions (MTJs) has been postulated previo
24 l muscle, talin 1 regulates the stability of myotendinous junctions (MTJs), but the function of talin
25 e fiber cytoskeleton, and the maintenance of myotendinous junctions (MTJs).
26 unctions (NMJs) and, as shown here, abnormal myotendinous junctions (MTJs).
27 al for muscle attachment and ECM assembly at myotendinous junctions (MTJs).
28  of those that underlie synapse formation at myotendinous junctions and that the outgrowth of seconda
29 unctions, which supports the hypothesis that myotendinous junctions are distinct domains in which the
30  underlying muscle guidance and formation of myotendinous junctions are poorly understood both in ver
31 table increase in myostatin concentration at myotendinous junctions during muscle unloading.
32      As assessed by atomic force microscopy, myotendinous junctions from normal and delta sgc-null mi
33 accumulates at the neuromuscular synapse and myotendinous junctions in adult skeletal muscle, and is
34 ues and is confined to the neuromuscular and myotendinous junctions in mature muscle.
35 is enriched at intersomitic junctions and at myotendinous junctions in somites and the myotome, where
36                                 However, the myotendinous junctions linking the DVM to the dorsal epi
37                                              Myotendinous junctions show adaptations to modified musc
38 keletal proteins that are highly enriched at myotendinous junctions that we hypothesize to be subject
39  enrichment at force transmitting sites, the myotendinous junctions, and costameres.
40   Ultrastructural analysis revealed abnormal myotendinous junctions, and functional tests showed a ni
41  confined in skeletal muscle to synapses and myotendinous junctions, and in kidney to the glomerular
42 zed tendon cells, resembling tendon cells at myotendinous junctions, form at the ends of these muscle
43 erentially concentrated at neuromuscular and myotendinous junctions, suggesting a role at sarcolemmal
44 hows that the mRNA is highly concentrated at myotendinous junctions, which supports the hypothesis th
45 ncluding intercalated discs, costameres, and myotendinous junctions.
46 en muscle fibers and/or myofibrils or at the myotendinous junctions.
47 evelopment, repair, and at neuromuscular and myotendinous junctions.
48 ge from cell motility to formation of stable myotendinous junctions.
49 mplex to control the development of thoracic myotendinous junctions.
50 ping muscle and cartilage, and xirp2a at the myotendinous junctions.
51 We also found a positive correlation between myotendinous strain injury and ringed fibers in the HSA(
52                   We found that muscles with myotendinous strain injury had an increased expression o
53 olecular and cellular adaptations to chronic myotendinous strain injury in mdx mice expressing a micr
54  well characterized, the chronic response to myotendinous strain injury is less clear.
55                                              Myotendinous strain injury is the most common injury of
56 nd the formation of rings are adaptations to myotendinous strain injury that help to prevent muscle n

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