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1 tr(-/-) mice the structural integrity of the myotendinous and neuromuscular junctions are maintained.
4 n skin deep; it extends to the depths of the myotendinous architecture and the mechanical basis for p
6 colemma localization, SPN is enriched at the myotendinous junction (MTJ) and neuromuscular junction (
8 responding tendon cells to form a functional myotendinous junction (MTJ) that allows for the force ge
11 toward and subsequent differentiation of the myotendinous junction by priming formation of a protein
15 , thus suggesting the important influence of myotendinous junction integrity on the severity of muscu
19 alpha7 integrin and utrophin in maintaining myotendinous junction structure and enabling force trans
20 d at the sarcolemma, neuromuscular junction, myotendinous junction, and in peripheral nerve, but not
23 ole for dystrophin in maintaining vertebrate myotendinous junctions (MTJs) has been postulated previo
24 l muscle, talin 1 regulates the stability of myotendinous junctions (MTJs), but the function of talin
28 of those that underlie synapse formation at myotendinous junctions and that the outgrowth of seconda
29 unctions, which supports the hypothesis that myotendinous junctions are distinct domains in which the
30 underlying muscle guidance and formation of myotendinous junctions are poorly understood both in ver
33 accumulates at the neuromuscular synapse and myotendinous junctions in adult skeletal muscle, and is
35 is enriched at intersomitic junctions and at myotendinous junctions in somites and the myotome, where
38 keletal proteins that are highly enriched at myotendinous junctions that we hypothesize to be subject
40 Ultrastructural analysis revealed abnormal myotendinous junctions, and functional tests showed a ni
41 confined in skeletal muscle to synapses and myotendinous junctions, and in kidney to the glomerular
42 zed tendon cells, resembling tendon cells at myotendinous junctions, form at the ends of these muscle
43 erentially concentrated at neuromuscular and myotendinous junctions, suggesting a role at sarcolemmal
44 hows that the mRNA is highly concentrated at myotendinous junctions, which supports the hypothesis th
51 We also found a positive correlation between myotendinous strain injury and ringed fibers in the HSA(
53 olecular and cellular adaptations to chronic myotendinous strain injury in mdx mice expressing a micr
56 nd the formation of rings are adaptations to myotendinous strain injury that help to prevent muscle n
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