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1 to the inhibition of histone deacetylases by n-butyrate.
2  DNA replication and late-gene expression by n-butyrate.
3 phorbol 12-myristate 13-acetate (TPA) and/or n-butyrate.
4 ates (k(3)) of 1-[(11)C]methyl-4-piperidinyl n-butyrate ([(11)C]BMP) hydrolysis determined by positro
5                                              n-Butyrate, a short-chain aliphatic carboxylic acid with
6 plants grown on fibroblasts and laminin with n-butyrate added to the incubation medium.
7  match" strategy showed that different SCFA (n-butyrate and acetate) appended to the same core sugar
8  liver because of the high concentrations of n-butyrate and related compounds to which the organ is o
9 was not inducible by HDAC inhibitors such as n-butyrate and trichostatin A (TSA).
10 ects occurred at a concentration of 3 mmol/L n-butyrate and with a treatment period of 8 to 12 hours.
11 ), was induced within 4 h of the addition of n-butyrate, and its 3.6-kb mRNA was resistant to inhibit
12                  An avidly metabolized SCFA (N-butyrate) augments crypt luminal alkalinization only s
13 teady-state PAI-1 mRNA caused by exposure to n-butyrate can be blocked by cycloheximide.
14                          We report here that n-butyrate can increase substantially the level of plasm
15                                        Thus, n-butyrate can influence the expression of multiple gene
16             We have reported previously that n-butyrate can reduce the level of beta-galactoside alph
17 didate with activity manifest through intact n-butyrate-carbohydrate linkages, reduced the invasion o
18 ot appear altered by treatment with 3 mmol/L n-butyrate for 6 hours.
19 expression of gabA, encoding the gamma-amino-n-butyrate (GABA) permease of the fungus Aspergillus nid
20 Th1 cells exposed to Ag and the G(1) blocker n-butyrate in primary cultures lose their ability to pro
21                Consequently, it appears that n-butyrate-induced anergy in Th1 cells correlated with t
22 ature of the protein apparently required for n-butyrate-induced Th1 cell anergy focused on the role o
23                Treatment of macrophages with n-butyrate led to the down-regulation of lipopolysacchar
24  lytic cycle activation in HH514-16 cells by n-butyrate or TSA.
25 iod) affected total fermentation product and n-butyrate performance parameters with corn fiber as the
26 roduction rate of 0.74 g COD l(-1) d(-1) and n-butyrate production rate of 0.47 g COD l(-1) d(-1) in
27 ic cycle using tetradecanoyl phorbol acetate/n-butyrate resulted in no virus production and an aberra
28                               The ability of n-butyrate to induce anergy in Ag-stimulated, but not re
29 gnition of an antigenic polypeptide, p40, in n-butyrate-treated BC-1 cells and by the absence of p40
30 0 to 20 times more antigen-positive cells in n-butyrate-treated BC-1 cells than in untreated cells wa
31 ated KSHV genomes by BC-1 cells, even though n-butyrate-treated cells contained numerous intranuclear
32 transcript encoding vIL-8 was detected in an n-butyrate-treated, MDV-transformed T-lymphoblastoid cel
33         Induction of the KSHV lytic cycle by n-butyrate was accompanied by the disappearance of host-
34         Induction of the KSHV lytic cycle by n-butyrate was associated with the expression of several
35  demonstrate that the short-chain fatty acid n-butyrate, which is secreted in high amounts by commens
36 m total fermentation product yield of 39%, a n-butyrate yield of 23% (both on a COD basis), a maximum

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