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1 ckers (atenolol, metoprolol, propranolol, or nadolol).
2 receiving metoprolol compared to propranolol/nadolol.
3 contractility with and without atropine and nadolol.
4 are beta3-sparing, including propranolol or nadolol.
5 eatment with the beta-adrenergic antagonist, nadolol (0.5 mg/kg), but not by the alpha-antagonist phe
6 Propranolol (10 mg/kg, intraperitoneally) or nadolol (10 mg/kg, intraperitoneally) administered 30 mi
7 with metoprolol (a beta(1)-AR antagonist) or nadolol (a beta(1)- and beta(2)-AR antagonist), but were
8 ed group, acute responders were treated with nadolol and acute nonresponders with nadolol+nitrates.
10 rker and racemic mixtures of the antagonists nadolol and propranolol demonstrated that the immobilize
13 d trial of endoscopic variceal ligation plus nadolol in preventing recurrent variceal bleeding and se
14 ranolol (n = 134), metoprolol (n = 147), and nadolol (n = 101) were analyzed, excluding patients <1 y
18 e observed that: (i) The beta-AR antagonists nadolol or carvedilol, given as a single i.v. injection
20 renergic receptor antagonists propranolol or nadolol or the alpha1- + beta1,2,3-adrenergic receptor a
22 ilar among the 4 beta-blockers, but in LQT2, nadolol provided the only significant risk reduction (ha
23 n LQTS, their efficacy differed by genotype; nadolol was the only beta-blocker associated with a sign
24 s for atenolol, metoprolol, propranolol, and nadolol were 0.71 (0.50 to 1.01), 0.70 (0.43 to 1.15) 0.
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