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1 ng to its designation as mK17n (n stands for nail).
2 lipophilic 'active' in a microneedle-porated nail.
3 tation of solvent penetration into the human nail.
4 d as functions of time and of depth into the nail.
5 iosteum and stabilized via an intramedullary nail.
6 y is limited to the area associated with the nail.
7 dergo Wnt-dependent differentiation into the nail.
8 lial tissues such as: skin, cornea, hair and nail.
9 ation and later conversion to intermedullary nail.
10 on and later conversion to an intramedullary nail.
11 long fingers and an opposable hallux with a nail.
12 ast, mBMSCs and BCs formed abnormal bone and nail.
13 t model compound from nanoparticles into the nail.
14 drug delivery into microneedle-treated human nail.
15 rogressively undermined the integrity of the nail.
16 elopmental anomalies of the hair, teeth, and nails.
17 ysis and is localized in the matrix of human nails.
18 and contraindication for the use of flexible nails.
19 otpads and presence of supernumerary ventral nails.
20 reased number of mitotic cells in transgenic nails.
21 he nail keratogenous zone resulted in longer nails.
22 (n = 10); hair, 2.13 +/- 2.984 ng/g (n = 9); nails, 0.88 +/- 0.335 ng/g (n = 9); sweat, 1.90 +/- 1.69
23 isorder diagnosed by the triad of dysplastic nails, abnormal skin pigmentation, and oral leukoplakia;
25 milies characterized by a split-foot defect, nail abnormalities of the hands, and hearing loss, due t
27 earing loss (SNHL), amelogenesis imperfecta, nail abnormalities, and occasional or late-onset retinal
29 r with later conversion to an intermedullary nail and documented the postoperative clinical condition
31 ype in mouse, marked lesions are seen in the nail and nail bed and sebaceous glands of PC and SM pati
32 chia congenita (PC), a disorder in which the nail and other epithelial appendages are profoundly aber
34 Trochanteric entry-locked intramedullary nailing and submuscular bridge plating have also recentl
36 e 15 children (26.7%) including: hypoplastic nails and shortened fifth fingers (one), microtia with c
39 , enthesium, dactylitis, spine, and skin and nails), and coming to consensus on optimal treatment rec
41 palms, soles, body folds, genitals, face, or nails, and concomitant joint disease, are also important
42 characterized by dysplasia of the patellae, nails, and elbows and FSGS with specific ultrastructural
44 common and debilitating disease of the skin, nails, and joints, with an acknowledged but complex gene
45 linical and dermoscopic examination of skin, nails, and mucous membranes was performed, and skin biop
48 al SCs, located at the interface between the nail appendage organ and adjacent epidermis, which physi
49 other epidermal appendages: skin, teeth, and nails--as well as lacrimal, mammary, salivary, sebaceous
51 highly restricted and most prevalent in the nail bed and matrix, leading to its designation as mK17n
52 use, marked lesions are seen in the nail and nail bed and sebaceous glands of PC and SM patients, res
54 d K17 exhibit severe lysis restricted to the nail bed epithelium, where all three genes are robustly
55 Because Msx1 is strongly expressed in the nail bed mesenchyme, it has been proposed that the Msx1-
57 sal membranes, acral skin (soles, palms, and nail bed), and skin with chronic sun-induced damage have
61 m (P < 0.05), suggesting that human hair and nails can be used as biomarkers to assess human exposure
63 quences of Notch1 activation in keratinizing nail cells were investigated in a transgenic mouse model
69 presenting with FPPK alone, or with minimal nail changes, carry mutations in KRT16; however, most FP
79 ations for a better understanding of PsA and nail disease and for an improved understanding of the ps
82 s of PsA, psoriasis and psoriatic-associated nail disease to show how the prevailing autoimmunity con
83 isease, psoriatic arthritis, and severity of nail disease with concomitant impairment of quality of l
85 e physician's global assessment of psoriatic nail disease, and enthesitis (using the PsA-modified Maa
86 physician's global assessment of psoriatric nail disease, and the PsA-modified MASES index in each g
87 rthritis (PsA), and by implication psoriatic nail disease, have been considered as autoimmune disorde
88 with male gender, increased body mass index, nail disease, psoriatic arthritis, larger plaques, more
89 a history of atopy, autoimmune disease, and nail disease, thus deconstructing the clinical heterogen
98 that Lgr6-expressing cells give rise to the nail during homeostatic growth, demonstrating that Lgr6
103 (hazard ratio [HR] 3.89, 95% CI 2.18-6.94), nail dystrophy (HR 2.93, 95% CI 1.68-5.12), and interglu
104 tion is characterized by cutaneous erosions, nail dystrophy and exuberant vascular granulation tissue
105 oliative erythroderma, hypotrichosis, severe nail dystrophy and failure to thrive, two heterozygous m
106 oliative erythroderma, hypotrichosis, severe nail dystrophy and failure to thrive, two heterozygous m
107 later onset and less frequent occurrence of nail dystrophy and keratoderma in PC-K6b, PC-K6c, and PC
108 ed individuals is primarily characterized by nail dystrophy and late onset of mild skin fragility and
110 skin disorder characterized predominantly by nail dystrophy and painful palmoplantar keratoderma.
113 Subsequently, 2 other family members who had nail dystrophy were also correctly diagnosed as having d
114 disorder, characterized by oral leukoplakia, nail dystrophy, and abnormal skin pigmentation, as well
117 ers, epidermolytic palmoplantar keratoderma, nail dystrophy, enamel dysplasia, and sparse woolly hair
118 n abnormalities is typically associated with nail dystrophy, leucoplakia, bone marrow failure, cancer
119 bilitating plantar keratoderma, hypertrophic nail dystrophy, oral leukokeratosis, and a variety of ep
120 inant disorder characterized by hypertrophic nail dystrophy, oral leukokeratosis, and palmoplantar ke
121 ociated with a higher likelihood of PsA were nail dystrophy, scalp lesions, and intergluteal/perianal
128 o human genetic disorders: monilethrix, hair-nail ectodermal dysplasia, pseudofolliculitis barbae and
130 n, was lacking in the beta-catenin-deficient nail epithelium and that genetic deletion of Wntless (Wl
131 hat genetic deletion of Wntless (Wls) in the nail epithelium led to the lack of Wnt activation in ost
132 These results reveal a critical role for the nail epithelium on the digit bone during homeostatic reg
133 thin cells of the nail matrix portion of the nail epithelium, as well as in a subset of cells in the
134 nce in the DRI score at 3 months in favor of nail fixation (mean score, 44.2 in the nail group and 52
136 icular fracture of the distal tibia, neither nail fixation nor locking plate fixation resulted in sup
146 and typified by dystrophic lesions affecting nails, glands, oral mucosa, and palmar-plantar epidermis
147 ut not at 12 months (mean score, 23.1 in the nail group and 24.0 in the plate group; adjusted differe
148 or of nail fixation (mean score, 44.2 in the nail group and 52.6 in the plate group; adjusted differe
151 nths between groups (mean score, 29.8 in the nail group vs 33.8 in the plate group; adjusted differen
156 f topically applied chemicals into the human nail has been visualized and characterized using stimula
157 ngal agent used to treat mycoses of skin and nails, has recently been demonstrated to be a potential
160 actures are plate fixation or intramedullary nailing; however, despite recent evidence, the optimal m
163 d that each altered its own diffusion in the nail in an apparently concentration-dependent fashion.
166 erline patients who underwent intramedullary nailing in comparison with those who underwent external
167 al activity against dermatophytes that cause nail infection than conventional terbinafine preparation
168 nt episodes of shingles, a widespread fungal nail infection, fungal dermatitis, oral herpetic lesions
171 terms of age of onset of symptoms, extent of nail involvement, and impact on daily quality of life.
172 entation of the skin did not have mucosal or nail involvement, suggesting 2 distinct mechanisms.
176 ditary leukonychia (porcelain nails or white nails) is a rare nail disorder with an unknown genetic b
179 croscopy (AFM) and developed a novel "bed of nails"-like approach that uses quartz glass nanopillars
180 oped skin fragility, blisters, erosions, and nail loss on their paws - all features of EBA patients.
181 linical features comprised nail dystrophy or nail loss, marginal palmoplantar keratoderma, hypodontia
182 nt experiments established that transplanted nail LRCs can actively participate in functional nail re
185 typical phenotype, including severe hair and nail manifestations, we scrutinized the exome sequencing
186 ores that extended to a range of depths; the nail material adjacent to the ablated area was rendered
187 ese SCs dominantly delivering progeny to the nail matrix and differentiated nail plate, demonstrating
188 ail stem cells (NSCs) reside in the proximal nail matrix and that the mechanisms governing NSC differ
189 d that Lgr6 is expressed within cells of the nail matrix portion of the nail epithelium, as well as i
190 n strictly to suprabasal cells in epidermis, nail matrix, and other skin appendages during normal dev
196 have indicated that flexible intramedullary nails may lead to a shorter time to union and a decrease
197 s specifically localized to developing mouse nail mesenchyme at embryonic day 15.5, suggesting a cruc
200 amino)-1-(3-pyridyl)-1-butanol, and hair and nail nicotine levels were measured in 60 subjects enroll
203 with specialized pedal grasping (including a nail on the hallux) and a petrosal bulla likely evolved
204 on of the femur shaft with an intramedullary nail or an external fixateur with later conversion to an
207 r initial (<24 hours) intramedullary femoral nailing or external fixation and later conversion to an
209 altering cysteine-rich proteins of hair and nails or by means of another mechanism remains unknown.
212 urrent or persistent infections of the skin, nail, oral, and genital mucosae with Candida species, ma
213 is reliant on the presence of the overlaying nail organ and is mediated by a proliferative blastema.
217 nita (PC), a disorder typified by dystrophic nails, painful hyperkeratotic calluses in glabrous skin,
221 f genetically distinct conditions, including nail-patella syndrome and collagen type III glomerulopat
225 ription factor 1-beta (LMX1B) are a cause of nail patellar syndrome, a condition characterized by ske
226 r, an array of 100 pores in 0.2cm(2) area of nail permitting a 10(3)-fold increase in initial drug up
227 ts that are consistent with abnormal toe and nail phenotypes in individuals with Van der Woude and po
228 ntal skin features included [corrected] hair/nail phenotypes, while [corrected] the most common syste
229 on the face and lips and was associated with nail pigmentation, blue pigmentation on the hard palate,
231 shown that PLCD1 is a component of the human nail plate by proteomic analysis and is localized in the
232 or abnormalities in skin appendages, such as nail plate dystrophy and structural defects in hair.
233 rogeny to the nail matrix and differentiated nail plate, demonstrating their plasticity to adapt to w
238 te the cylindrical portion of the electrode: nail polish, epoxy, polyimide, and polypropylene coating
243 uiescent cells within the basal layer of the nail proximal fold, organized in a ring-like configurati
248 Treatment recommendations for 4 clinical nail psoriasis scenarios were developed based on the evi
249 e Health Assessment Questionnaire (HAQ), the Nail Psoriasis Severity Index (NAPSI), the physician's g
255 utation, this Wnt activation is required for nail regeneration and also for attracting nerves that pr
258 The effective treatment of diseases of the nail remains an important unmet medical need, primarily
261 exemplars that metallic pipe leaks caused by nails, rocks, and erosion corrosion autogenously repaire
267 between quintile selenium levels measured in nail samples and cognitive test scores, with adjustment
271 rther identified in vivo in urine and finger nail samples, this suggests that in vitro assays can rel
272 partial and complete laser poration of human nail samples, with the energy per pore and the exposure
274 tration of a model drug across laser-treated nails showed that complete poration resulted in essentia
275 Finally, for a patient with significant nail, skin, and joint disease, adalimumab, etanercept, u
280 K15-derived cells can contribute to both the nail structure and peri-nail epidermis, and more toward
282 was identified for the first time in finger nails, suggesting that this matrix may also indicate pas
283 n as immobile reservoirs, sequestered on the nail surface and in the microneedle-generated pores, fro
284 ded the following: whole blood, urine, hair, nails, sweat, brain tissue, breast milk, and explants.
285 ovative, magnesium-containing intramedullary nail that facilitates femur fracture repair in rats with
286 tabolites was higher in urine than in finger nails; the opposite was observed for the primary metabol
287 ith a detectable mutation, PC manifests with nail thickening and plantar keratoderma before school ag
288 as used to image D2O, PG-d8/DMSO-d6, and the nail through the O-D, -CD2, and -CH2 bond stretching Ram
290 emia (65%), asthenia (55%), dry mouth (45%), nail toxicity (35%), constipation (34%), decreased appet
292 ion treated by wide surgical excision of the nail unit followed by full-thickness skin graft reconstr
294 has shown that wide surgical excision of the nail unit was associated with a low rate of recurrence.
295 efficiency of wide surgical excision of the nail unit with full-thickness skin graft reconstruction
296 coated wires were selectively insulated with nail varnish, electrophoretic paint, or fast-setting epo
298 the multiple keratins expressed in hair and nail, which can be related to the common ancestry, clust
299 d in urine (97% DF) and identified in finger nails, while no DPHP metabolites were detected in vivo.
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