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1 4c was potent as the prototypical quinolone, nalidixic acid (1), with an IC50 value of 58.3 microgram
2 molar tooth appearance on anaerobic colistin nalidixic acid (CNA) agar which likely facilitated its d
3 iated to determine whether Columbia colistin-nalidixic acid (CNA) agar would be an equally sensitive,
4 nto blood (blood agar plate [BAP]), colistin-nalidixic acid (CNA), and MacConkey agars in 5% CO2 for
5 sed to rifampicin (transcription inhibitor), nalidixic acid (gyrase inhibitor), or A22 (MreB-cytoskel
6  culture on MacConkey agar supplemented with nalidixic acid (MACnal) and compared to overnight broth
7  bind not only quinolone antibiotics such as nalidixic acid (NA) and flumequine (FLU), but also salic
8 R), sarafloxacin (SAR), oxolinic acid (OXO), nalidixic acid (NAL) and flumequine (FLU) were separated
9 al distribution of the organism and apparent nalidixic acid (NAL) resistance.
10 ous Fenton-like reactions for the removal of nalidixic acid (NAL), a recalcitrant quinolone antibacte
11  susceptibility, the proportion resistant to nalidixic acid (NAL-R) increased from 2008 to 2012 (Typh
12  Typhi [MDRST]); 758 (38%) were resistant to nalidixic acid (nalidixic acid-resistant S. Typhi [NARST
13                  The combination of neomycin-nalidixic acid (NNA) agar and a selective broth medium (
14 on blood agar medium containing neomycin and nalidixic acid (NNA).
15  were 2.2 [ampicillin (AMP), p=0.017] to 23 [nalidixic acid (NX), p<0.001] times more likely to harbo
16 ty of Todd-Hewitt medium with gentamicin and nalidixic acid (SBM) with our current method of direct p
17 0 microg of amphotericin B, and 20 microg of nalidixic acid (VAN) per ml.
18 occus isolates grown on blood agar, colistin-nalidixic acid agar (CNA), and mannitol salt agar (MSA);
19 B streptococcus, with culture using neomycin-nalidixic acid agar (NNA) and LIM broth.
20 eep blood agar, chocolate agar, and colistin-nalidixic acid agar after 24 to 48 h of incubation at 35
21 d sharply increasing trends in resistance to nalidixic acid and ciprofloxacin for both ST and SPA.
22 n it to be more sensitive to CCCP, PMA, PCP, nalidixic acid and DOC than the parent strain.
23                     We identified two drugs, nalidixic acid and dorzolamide, that potently inhibit th
24 xacin), and three quinolones (oxolinic acid, nalidixic acid and flumequine) in eggs is presented.
25 e induced in a LexA-dependent manner by both nalidixic acid and mitomycin C, identifying these as mem
26 on and the addition of the gyrase inhibitors nalidixic acid and novobiocin.
27 emiselective blood agar medium incorporating nalidixic acid and sulfamethazine (NAS) is described.
28 ays a role in modulating the SOS response to nalidixic acid and that the response is more complex tha
29 ns known to be required for SOS induction by nalidixic acid are RecA and RecBC.
30 antially reduced for SOS induction following nalidixic acid but not UV treatment, and which were also
31 ot hydrolyze hippurate, and was sensitive to nalidixic acid but resistant to cephalothin.
32   Disk diffusion using these antibiotics and nalidixic acid failed to detect some low-level-resistant
33 els of the AcrAB-TolC pump, thereby removing nalidixic acid from the organism.
34 A subset of SOS genes lost their response to nalidixic acid in the dnaQ mutant strain, while two test
35 usceptible to ciprofloxacin but resistant to nalidixic acid in vitro, a pattern associated with fluor
36                                Resistance to nalidixic acid may be useful in the identification of E.
37 howed increased survival on media containing nalidixic acid or rifampicin, but did not have an increa
38 ere uncovered as being uninducible by either nalidixic acid or UV treatment.
39                                              Nalidixic acid passed undegraded through the MBR and was
40 itt broth supplemented with 10 micrograms of nalidixic acid per ml and 15 micrograms of colistin per
41                     The cellular response to nalidixic acid perturbation was analyzed using this form
42 s to rifampicin resistance (RifR) (rpoB) and nalidixic acid resistance (NalR) (gyrA).
43 es of spontaneous mutation to rifampicin and nalidixic acid resistance in one medium and one fast str
44 e efflux contributes to the overall level of nalidixic acid resistance.
45 d an identical pattern on PFGE, and all were nalidixic acid resistant.
46 e, such as qnr, is often not detected by the nalidixic acid screen test.
47        Quinolone antibacterial drugs such as nalidixic acid target DNA gyrase in Escherichia coli.
48  perturb a GyrA-GyrA dimer interface allowed nalidixic acid to fragment chromosomes and kill cells in
49  A disk diffusion breakpoint was derived for nalidixic acid to serve as a surrogate marker for gyrase
50                               Novobiocin and nalidixic acid treatment both resulted in rapid loss of
51 specifically deficient in SOS induction upon nalidixic acid treatment by using a dinD::lacZ reporter
52 of DNA fragments by GyrA antiserum following nalidixic acid treatment of cells.
53 chromosome replication was blocked by either nalidixic acid treatment or thymine starvation, the tran
54 hed greater than 10-fold in the medium after nalidixic acid treatment, suggesting these were released
55 ically necessary for SOS induction following nalidixic acid treatment.
56  several additional SOS genes in response to nalidixic acid using real-time PCR.
57 ween high efflux and increased resistance to nalidixic acid was found.
58                    The deficient response to nalidixic acid was rescued by the presence of the wild-t
59  genes not previously known to be induced by nalidixic acid were also reproducibly upregulated.
60 rom the production of an antibacterial drug (nalidixic acid) was investigated employing a membrane bi
61 antibiotics (vancomycin, amphotericin B, and nalidixic acid), and the efficacy of solid (Herrold's eg
62 r characteristics, (ii) the concentration of nalidixic acid, (iii) the 48 organics identified in the
63                                         When nalidixic acid, a DNA synthesis inhibitor, was added to
64 on-inducible lexA mutant hypersusceptible to nalidixic acid, a property restricted to fluoroquinolone
65 azole, 312 (28%) to tetracycline, 19 (2%) to nalidixic acid, and 6 (0.5%) to ciprofloxacin.
66           In contrast, addition of rifampin, nalidixic acid, and chloramphenicol had little effect on
67 ant resistant to the prototype of quinolone, nalidixic acid, and created complexes on DNA detected by
68 eased spontaneous resistance to rifampin and nalidixic acid, and MMC/uvrD double mutants exhibited hi
69 g tetracycline, chloramphenicol, ampicillin, nalidixic acid, and rifampin.
70 ion elongation was blocked by hydroxyurea or nalidixic acid, arrested cells contained one partially r
71 DNA-damaging agents, such as mitomycin C and nalidixic acid, caused only limited elongation.
72 r alkaline phosphatase, and was resistant to nalidixic acid, cephalothin, and trimethoprim-sulfametho
73 , the MICs and inhibition zone diameters for nalidixic acid, ciprofloxacin, levofloxacin, and ofloxac
74                     The prototype quinolone, nalidixic acid, kills wild-type Escherichia coli only by
75 cs, gentamicin, ceftazidime, nitrofurantoin, nalidixic acid, ofloxacin.
76  were selected on the basis of resistance to nalidixic acid, representing a variety of the most preva
77 eptibility to various antibiotics, including nalidixic acid, rifampin, novobiocin, and chloramphenico
78 re resistant to ampicillin, chloramphenicol, nalidixic acid, streptomycin, sulfisoxazole, tetracyclin
79                                              Nalidixic acid, the prototype antibacterial quinolone, i
80 bial mixture of polymyxin B, amphotericin B, nalidixic acid, trimethoprim, and azlocillin (PANTA) was
81  PANTA reagent (polymyxin B, amphotericin B, nalidixic acid, trimethoprim, and azlocillin), reconstit
82 oteins involved in the cytotoxic response to nalidixic acid, we screened for E. coli mutants specific
83 o the selection of the first clinical agent, nalidixic acid, were ever published by the discoverers.
84 he wild, stranded dolphins were sensitive to nalidixic acid, whereas the isolates from the collection
85          Overall, these results suggest that nalidixic acid-induced DNA breaks are generated either b
86 repair DNA damage via UV-induced DNA damage, nalidixic acid-induced double-strand breaks, and methyl
87  fraction of kanamycin-resistant (Km(r)) and nalidixic acid-resistant (Nal(r)) isolates showed reduce
88                             About 10% of the nalidixic acid-resistant (Nal(r)) mutants in a transposi
89 ompared to 100% of those inoculated with the nalidixic acid-resistant (Nal(r)) parent and 100% of tho
90 triaxone was observed, 20 isolates (7%) were nalidixic acid-resistant (NARST).
91 ations in the gyrA gene were present in most nalidixic acid-resistant isolates.
92  758 (38%) were resistant to nalidixic acid (nalidixic acid-resistant S. Typhi [NARST]) and 734 NARST
93        We found a strong association between nalidixic acid-resistant Salmonella enterica serotype En
94  experiments, detection and enumeration of a nalidixic acid-resistant strain of E. coli O157 in bovin
95                                              Nalidixic acid-resistant strains harbored mutations in G
96 chloramphenicol, but all were susceptible to nalidixic acid.
97 and exhibited intermediate susceptibility to nalidixic acid.
98 ted for strongly by ciprofloxacin but not by nalidixic acid.
99 concentrations of rifampicin, kanamycin, and nalidixic acid.
100 m-sulfamethoxazole; 4 were also resistant to nalidixic acid.
101 pecifically deficient in the SOS response to nalidixic acid.
102 %) demonstrated a MIC > or = 16 microg/mL to nalidixic acid.
103 tment, and which were also hypersensitive to nalidixic acid.
104 om exogenous agents such as UV radiation and nalidixic acid.
105 ve and represent a more specific response to nalidixic acid.
106 e found to be upregulated in the presence of nalidixic acid.
107 lyze indoxyl acetate and their resistance to nalidixic acid.
108 ment of Health were tested for resistance to nalidixic acid.

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