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1 the endoscopist with benzodiazepine and/or a narcotic.
2  effects normally associated with the opiate narcotics.
3 ing the BBB in the physiological response to narcotics.
4  because of the side effects associated with narcotics.
5 s and were not using hormonal medications or narcotics.
6 performed on patients receiving preoperative narcotics.
7 evere, ultimately necessitating control with narcotics.
8 e pain and perhaps obviate the need for oral narcotics.
9 efined narcotics versus the predefined polar narcotics.
10 fusion that resolved with discontinuation of narcotics), 3 (0.3%) cases of atelectasis, 2 (0.2%) corn
11 nesthesia, one patient required supplemental narcotics (5 mg of oxycodone) and sedatives (1 mg loreza
12 in smoking (n=6), marijuana use (n=79), oral narcotic abuse (n=20), and intravenous drug use (n=21).
13                          Papaverine is a non-narcotic alkaloid found endemically and uniquely in the
14                                   The use of narcotics among patients with Crohn disease (CD) is ende
15 prohibited substances, including stimulants, narcotics, anabolic agents, diuretics, peptides, and gly
16                    Use of patient-controlled narcotic analgesia and duration of use decreased (63.2%
17 omyalgia, approximately 50% of whom required narcotic analgesia and/or were disabled, treatment with
18                                  Intrathecal narcotic analgesia is used increasingly in fast-tracking
19                                              Narcotic analgesia was required in 98% of general anesth
20  have been shown to be effective adjuncts to narcotic analgesia.
21 n all cases of pain, patients were receiving narcotic analgesia.
22   Many changes relate to improvements in non-narcotic analgesic techniques and use of various suprala
23                           Morphine, a potent narcotic analgesic used for the treatment of acute and c
24 atients) and reduction or discontinuation of narcotic analgesics (55% of patients), as well as improv
25 f entry onto the study, 15 patients required narcotic analgesics for bone pain; after treatment, eigh
26 s, and conservative therapy with bracing and narcotic analgesics gave little improvement.
27                Use and abuse of prescription narcotic analgesics have increased dramatically in the U
28      Postoperative pain relief (freedom from narcotic analgesics) was achieved in 153 of 185 patients
29 ents, reduced dependence on narcotic and non-narcotic analgesics, improved performance status and qua
30 e less likely than whites to be treated with narcotic analgesics.
31 t alleles might display altered responses to narcotic analgesics.
32  bodies eliminated the pain and the need for narcotic analgesics.
33 lity improvement vs. control periods, use of narcotic and benzodiazepine infusions were substantially
34 sible), and performed chart review to assess narcotic and benzodiazepine use and time from ventilator
35 lity in many patients, reduced dependence on narcotic and non-narcotic analgesics, improved performan
36 ter extubation, and respiratory arrest after narcotic and sedative medication.
37                               Total doses of narcotics and benzodiazepines increased after implementa
38 (alcohols and anilines) with predefined MOA (narcotics and polar narcotics) were investigated at diff
39 ics, beta-2 agonists, diuretics, stimulants, narcotics, and beta-blockers) spiked in human urine and
40            Antibiotics, antimotility agents, narcotics, and non-steroidal anti-inflammatory drugs sho
41 nical management of postsurgical reversal of narcotic anesthesia and opioid side effects as well as t
42 d patients who lived in close proximity to a Narcotics Anonymous meeting location.
43          These results confirm that tertiary narcotic antagonist quaternization substantially reduces
44                                   Quaternary narcotic antagonists that are assumed not to penetrate t
45 gastrointestinal stimulants, antibacterials, narcotics, antipsychotics, inotropes, digoxin, anestheti
46                                 Opioid-based narcotics are the most widely prescribed therapeutic age
47                     While morphine and other narcotics are the most widely prescribed therapy for mod
48    Application of the system to detection of narcotics at airport security control points is discusse
49                 A newly described condition, Narcotic Bowel Syndrome (NBS)/Opioid-Induced GI Hyperalg
50 s warranted to determine the epidemiology of narcotic bowel syndrome and delineate the most efficacio
51  underlying pathophysiological mechanisms of narcotic bowel syndrome are incompletely understood; how
52                                              Narcotic bowel syndrome is characterised by worsening ab
53                                     Although narcotic bowel syndrome is rarely diagnosed, given the c
54  (DM) exposed to sublethal doses of presumed narcotic chemicals with log Kow >/= 1.8.
55 t may facilitate development of valuable non-narcotic clinical analgesics utilizing cotreatment with
56                                   Given that narcotics commonly are administered to patients after se
57 neous or individual detection of three major narcotic components, heroin, noscapine and morphine at m
58 en preparations simultaneously, and 63% used narcotic-containing compounds.
59 and toxicity tests using an assumed baseline narcotic critical body residue (CBR) and a range of orga
60                                              Narcotic dependence decreased and activity level increas
61                                  Severe pain/narcotic dependency, tumor size larger than 10 cm, and n
62                         Apomorphine is a non-narcotic derivative of morphine, which acts as a dopamin
63 eatments for alleviating incisional pain and narcotic drug withdrawal symptoms, which are now in clin
64 intravenous propofol (2,6-diisopropylphenol, narcotic drug) infusion.
65 edures usually involve the administration of narcotic drugs as anesthetics or adjuvants.
66 le in mediating acute and chronic effects of narcotic drugs.
67 to extrinsic contamination of the parenteral narcotic fentanyl by a health care worker.
68                         Intravenous infusion narcotics (fentanyl, morphine, or hydromorphone) were us
69 n modulation should be tried before starting narcotics for pain control.
70  a brief interval after receiving additional narcotics for pain during the procedure.
71 more immediate concerns when choosing opiate narcotics for pain therapy.
72 evious studies have used oral or intravenous narcotics for supplementation.
73 ly less likely to receive a prescription for narcotics for their symptoms than patients in the nonsta
74 block, preoperative carbohydrate loading and narcotic free pain regimens.
75 recorded as being at work or as retrieving a narcotic from an automated dispensing cabinet in an area
76  neural dysfunction, inflammatory mediators, narcotics, gastrointestinal hormone disruptions, and ana
77                          Preoperative use of narcotics in patients undergoing abdominal surgery for C
78                        Despite common use of narcotics in the clinical management of severe traumatic
79                                          The narcotic independence rate at 1 year was 55% and continu
80 ining physiological responsiveness to opioid narcotics is considered, micro opioids derived from salv
81       Since stress influences the potency of narcotics, it may be an important physiological componen
82                                              Narcotics may be needed to provide relief in some cases.
83  a standard scale ranging from 0 to 10), and narcotic medication use (intravenous morphine equivalent
84 ctive application of regional analgesia, non-narcotic medications, and complimentary alternative opti
85  the antinociceptive actions of the alkaloid narcotic, morphine, following either i.p. or intracerebr
86  has been shown to decrease operative times, narcotic need and permit quicker return of bowel functio
87  interval, 1.37-2.26), respectively, that of narcotic nonusers.
88 ive sleep apnea; b) to examine the effect of narcotics on postoperative obstructive sleep apnea.
89 g, sleeping) and required significantly less narcotic opioids (P < .001); improvement in the patient'
90 -field sample testing of toxins, explosives, narcotics or other hazardous chemicals.
91 time, and 30% had not received any sedation, narcotic, or psychotropic drug in the previous 24 hrs.
92 r rate of infection or the use of sedatives, narcotics, or antibiotics in the catheter group.
93   Patients were assigned to receive either a narcotic- or a halothane-based anesthetic for adenotonsi
94 among those who did not receive prescription narcotics (P<0.0001).
95 ter ketorolac introduction required 58% less narcotics (P<0.001), recalled having less postoperative
96 generation of vapor samplers for explosives, narcotics, pathogens, or even cancer, and could inform f
97         Patients were less likely to receive narcotics prescriptions from primary care providers when
98 1.22-1.43]; HR(high), 1.33 [1.17-1.53]) to a narcotics-related conviction (HR(moderate), 2.23 [2.14-2
99                              To date, opioid narcotics represent the largest and most potent class of
100 eroidal anti-inflammatory drugs can decrease narcotic requirement and recovery time.
101     Perioperative outcomes and postoperative narcotic requirement were compared among patient groups.
102 es produced an analgesic effect that reduced narcotic requirements compared with patients who receive
103                          End points included narcotic requirements, glycemic control, islet function,
104       Blood loss, length of stay, parenteral narcotic requirements, resumption of diet, and return to
105 d postoperative sensory block that minimizes narcotic requirements.
106 ministration, a significant reduction in the narcotic's ability to produce antinociception during str
107  thought to be the cellular target of opioid narcotics such as morphine and heroin, mediating their e
108                     Operative time, doses of narcotics, surgical difficultly and hospital charges wer
109 ion and assessments of strategies to prevent narcotics tampering in all health care settings are need
110 ressants, pain killers, anti-psychotics, and narcotics that are poor substrates for microsomal CYP1A1
111                For patients taking long-term narcotics, the mean use per week was 639 mg (95% CI, 220
112 eral nutrition (TPN), and days of injectable narcotic therapy (all over 28 days), days in hospital (o
113 0001), (4) 2.6 additional days of injectable narcotic therapy (P <.0001), (5) 2.6 additional days in
114 r antagonists may improve the reliability of narcotic therapy.
115 ion of potent short-acting beta-blockers and narcotics to control hemodynamic variables are examples
116  "a physician providing a sufficient dose of narcotics to enable a patient to kill himself." Responde
117 to minimize the contribution of prescription narcotics to the nationwide opioid epidemic, reductions
118 tanding the intrinsic mechanisms involved in narcotic tolerance and dependence.
119 avenues for the study and treatment of pain, narcotic tolerance, and dependence.
120 ve the first indications of the link between narcotic toxicity and the chemical activity of organic c
121  its use is restricted to federally licensed narcotic treatment programs (NTPs).
122 efore their TP-IAT was 7.1 +/- 0.3 years and narcotic usage of 3.3 +/- 0.2 years.
123  room (3.2 vs 4.7, P = .003), interval total narcotic use (6.7 vs 12.5 mg, P = .003 at <4 hours and 0
124  = .01 at 8-12 hours), and total intravenous narcotic use (9.2 vs 17.2 mg of morphine sulfate equival
125  of stay (4 versus 7 days), and decreased IV narcotic use (all P < 0.05.Postoperative morbidity was e
126 as associated with reduced LOS and decreased narcotic use after donor nephrectomy.
127 significantly reduces postoperative pain and narcotic use after LVHR.
128                                      Overall narcotic use also decreased by nearly 50% (45.6 vs. 21.3
129 rbidity, as well as significant decreases in narcotic use and cost.
130 months 9 and 12 after randomization, with no narcotic use and no further sphincter intervention.
131            Among adult patients, the odds of narcotic use at 1 year were increased by previous endosc
132  on the outcome implications of prescription narcotic use before kidney transplantation.
133  in patients that is associated with illicit narcotic use by health care workers.
134 reatic pain" similar to preoperative levels, narcotic use for any reason, and islet graft failure at
135                   Patients with preoperative narcotic use had a longer mean (SD) length of stay (11.2
136 7% of evaluable patients, with a decrease in narcotic use in 56%.
137          There was a significant increase in narcotic use in control patients in the first 24 hours (
138  measures were pain over 6 hours, parenteral narcotic use over 24 hours, duration of hospitalization,
139           Pain scores were lower and 24-hour narcotic use was less in patients who received lidocaine
140 ariable analysis indicated that preoperative narcotic use was the only independent risk factor associ
141 onor recipients with the highest quartile of narcotic use were 2.3 times (aHR, 2.27; 95% confidence i
142 y, wake-up times, emetic symptoms, pain, and narcotic use were compared.
143           Patients with a history of chronic narcotic use were excluded.
144 nalog pain scales) and subjective (mobility, narcotic use) scores were monitored before and after ver
145 ssociated with significant reduction in i.v. narcotic use, a rapid return to diet, and shorter hospit
146 were gathered regarding ethanol abuse, pain, narcotic use, and recurrent acute exacerbations requirin
147 omitant depression, chronic pain, alcohol or narcotic use, and/or take several preparations simultane
148 racture progression had significantly higher narcotic use, change in Karnofsky performance score, and
149                       Laparoscopic approach, narcotic use, length of stay, 30-day readmission, ileus
150                      Before starting regular narcotic use, patients with CD should be considered for
151 ience through improved pain control and less narcotic use, without increased length of stay or compli
152 ermine what effect this may have on pain and narcotic use.
153 d clinical variables, including preoperative narcotic use.
154 ductions in bone turnover markers, pain, and narcotic use.
155 rs, mechanical ventilation, benzodiazepines, narcotics, use of physical restraints, and exposure to v
156  Subgroup analysis indicated that outpatient narcotic users had increased incidence of adverse postop
157 rative outcomes compared with inpatient-only narcotic users.
158 up is designed for a quick identification of narcotics using automated sampling.
159 points were patient-reported outcomes, total narcotic utilization, and complications.
160 result in a classification of the predefined narcotics versus the predefined polar narcotics.
161 omated dispensing cabinet in an area where a narcotic was administered to each of the 3 case patients
162                              The relief from narcotics was sustained.
163 forcement Administration's list of scheduled narcotics was used to query opioid use.
164 Concomitant tricyclic antidepressants and/or narcotics were continued if therapy was stabilized prior
165 nal surgery, and chronic preoperative use of narcotics were independently correlated with POI on mult
166                           Patients receiving narcotics were more likely to have a current smoking hab
167 ial confidence interval: 63%, 100%) for whom narcotics were prescribed prior to the procedure reporte
168 es) with predefined MOA (narcotics and polar narcotics) were investigated at different levels of biol
169 ounds were significantly lower than those of narcotics, whereas no differences were found between end
170 nd some insight into the pharmacokinetics of narcotics while on uteroplacental support has been gaine
171          Serious adverse events included the narcotic-withdrawal syndrome and sirolimus-associated pn
172 tients who had at least 1 pharmacy claim for narcotics within 1 month before surgery was 41.5 years a
173 profound analgesic properties of intrathecal narcotics without motor blockade make them an excellent

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