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1 A similar pattern occurred following nasal administration.
2 vivo studies showed mucosal absorption after nasal administration.
3 ung epithelial cells in vivo following intra-nasal administration.
4 induced in DBA/1 mice was studied after the nasal administration (before disease induction) of 1 of
9 e showed that blocking IL-10 concurrent with nasal administration of Ag abolished the disease-suppres
13 t mucosal tolerance in EAG can be induced by nasal administration of an immunodominant peptide from t
22 Collectively, these data suggest that the nasal administration of GAD65 peptides induces a Th2 cel
25 t mucosal tolerance in EAG can be induced by nasal administration of recombinant rat alpha3(IV)NC1 an
26 tolerance in EAG by examining the effect of nasal administration of recombinant rat alpha3(IV)NC1.
27 Here, we demonstrate that subcutaneous or nasal administration of small-molecule MC activators wit
30 holine receptor (TAChR), to demonstrate that nasal administration of synthetic sequences of the TAChR
34 ificantly different from that seen following nasal administration of these vectors to mouse lung (rAA
35 rance in EAG by examining the effects of the nasal administration of this peptide after the onset of
37 hology studies in rats after 5 days repeated nasal administration showed that Solutol HS15 had no tox
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