ライフサイエンスコーパス: nasal cartilage

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1 prevention of p53 degradation in developing nasal cartilage.

2 in articular cartilage, unlike the case for nasal cartilage.

3 a decrease in tissue proteoglycan content in nasal cartilage.

4 urified from interleukin-1-stimulated bovine nasal cartilage.

5 naling induces p53-mediated apoptosis in the nasal cartilage.

6 Purification of YKL-40 from resorbing bovine nasal cartilage and chondrocyte monolayers demonstrated

7 d cleavage of type II collagen, partially in nasal cartilage and completely in articular cartilage.

8 l a role for both genes in the fusion of the nasal cartilages and proper patterning of the mandible,

9 gated cytokine-induced destruction of bovine nasal cartilage at both the proteoglycan and collagen br

10 ions from native and trypsin-digested bovine nasal cartilage (BNC) and from engineered cartilage, gen

11 A bovine nasal cartilage (BNC) explant assay was performed to mea

12 In stimulated bovine nasal cartilage (BNC), >90% of collagen is released by d

13 Using bovine nasal cartilage conditioned media as a source of the agg

14 d media from interleukin-1-stimulated bovine nasal cartilage cultures.

15 ial collagenase could be readily detected in nasal cartilage cultures.

16 Col1a1, was found in the locations where the nasal cartilages exist in WT embryos.

17 The bovine nasal cartilage explant model and the destabilization of

18 Bovine nasal cartilage explants were cultured with 1 microM Ret

19 Using interleukin-1-stimulated bovine nasal cartilage explants where aggrecan depletion occurs

20 severely affected mutants exhibit defective nasal cartilage, failure of fusion between the nasal sep

21 The destruction of nasal cartilage in GPA is mainly mediated by fibroblasts

22 tion in the aggrecanase neoepitope NITEGE in nasal cartilage in response to IL-1.

23 . co-implanted with healthy allogeneic human nasal cartilage into immunodeficient pfp/rag2(-/-) mice.

24 und uniquely in the tracheal, auricular, and nasal cartilage of adults.

25 ty while concomitantly reducing apoptosis in nasal cartilage primordia, suggesting that enhanced BMP

26 The loss of type II collagen from nasal cartilage stimulated with IL-1 alpha was inhibited

27 II is processed normally in dermatosparactic nasal cartilage, suggesting the existence of another N-p

28 Autologous nasal cartilage tissues can be engineered and clinically

29 N terminus ARGSVIL from freeze-thawed bovine nasal cartilage using the monoclonal antibody BC-3.

30 Bovine nasal cartilage was stimulated to resorb with the additi

31 Bovine nasal cartilage was treated with affinity-purified fibro

32 In cultures of nasal cartilage with IL-1 alpha, almost all the proteogl

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