ライフサイエンスコーパス: nasal epithelia

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1 d NO-dependent chloride transport across the nasal epithelia.

2 anion secretory currents in wild-type and CF nasal epithelia.

3 ve cells or fibers in the olfactory nerve or nasal epithelia.

4 y in the olfactory bulb, olfactory nerve, or nasal epithelia.

5 based lentivirus vector (GP64-FIV) to murine nasal epithelia.

6 ut the airways, while CNT2 was restricted to nasal epithelia.

7 In nasal epithelia 1-EBIO produced only a small transient e

8 experiments using cultures of primary human nasal epithelia and human bronchial epithelia have estab

9 It is also expressed by nasal epithelia and neural crest-derived facial structur

10 fate of the ectoderm from which the lens and nasal epithelia are derived.

11 n, spinal cord, sensory ganglia, retina, and nasal epithelia, as well as in the pituitary, and is ref

12 The presence of increased CCL5 levels in nasal epithelia at the time of bronchiolitis or the deve

13 (apical, basolateral, paracellular) in human nasal epithelia cultures using experimental (Ussing Cham

14 1.2-43.3; P = .028), and CCL5 expression in nasal epithelia during acute RSV infection (OR, 3.8; 95%

15 ted across the various regions of the bovine nasal epithelia following conjugation with deslorelin an

16 analysis documented persistent expression in nasal epithelia for approximately 1 year without signifi

17 We conclude that: (1) normal, but not CF, nasal epithelia have a constitutively active DPC-sensiti

18 f HCO3- via CFTR; and (2) both normal and CF nasal epithelia have Na+-independent, H2DIDS-sensitive A

19 es and persistently expresses a transgene in nasal epithelia in the absence of agents that disrupt th

20 ant asthma-associated methylation changes in nasal epithelia of adult white asthmatic patients.

21 al hCFTR expression was also observed in the nasal epithelia of CF mutant mice.

22 t) measurements are routinely carried out on nasal epithelia of CF patients in the clinic.

23 h 4PBA therapy inducing CFTR function in the nasal epithelia of deltaF508-homozygous CF patients.

24 uction of influenza virus replication in the nasal epithelia of HA vector-immunized mice suggested an

25 lture and of the Cl- transport defect in the nasal epithelia of mice homozygous for the deltaF508 mut

26 In vivo studies of CF mouse nasal epithelia revealed an increase in goblet cell numb

27 In CF nasal epithelia the activation of calcium-activated chlo

28 both CF mouse models, Cl(-) permeability of nasal epithelia was restored in a sustained manner by zi

29 f IB3-1 cells with 0.1-1 mM 4PBA and primary nasal epithelia with 5 mM 4PBA also resulted in the appe

30 ulose-formulated AcGP64-FIV transduced mouse nasal epithelia with much greater efficiency than simila

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