ライフサイエンスコーパス: nasal lavage

1 hils, eosinophils, and interleukin (IL)-6 in nasal lavage.

2 ollowed by standard dose of virus and serial nasal lavages.

3 ined by quantification of S. pneumoniae from nasal lavage and analysis of sinus tissue, respectively.

4 merase chain reaction analysis on cells from nasal lavage and induced sputum samples from all subject

5 Rhinovirus RNA was detected in both nasal lavage and lower airway cells from all eight subje

6 ssociation between ChoP(+) expression in the nasal lavage and the development of OM with culture-posi

7 al loads, and cytokines were quantified from nasal lavages and blood, and correlated to clinical seve

8 t spirometry, methacholine challenge (PC20), nasal lavage, and sputum induction at baseline and on Da

9 Nasal lavages at 0, 15, 60, and 120 min after lysine-asp

10 In addition, one rhinovirus from a nasal lavage contained an identical nucleic acid sequenc

11 Nasal lavage cytokine levels of IL-4 (P < 0.01), IL-5, a

12 We analysed cytokine levels in nasal lavage fluid (NLF) in 59 subjects (46 with asthma)

13 IL-8 levels were measured in nasal lavage fluid and serum on randomization, day 8, an

14 ction through analysis of cytokine levels in nasal lavage fluid and stimulated peripheral blood monon

15 tion of macrophage inflammatory protein 2 in nasal lavage fluid and subsequent recruitment of neutrop

16 the mouse, human CD49d(+) PMNs isolated from nasal lavage fluid during a viral respiratory tract infe

17 +) PMN frequency was significantly higher in nasal lavage fluid during acute respiratory symptoms in

18 We collected nasal tissue and nasal lavage fluid from patients with CRS and control su

19 = .6) but resulted in a greater decrease in nasal lavage fluid IL-8 levels by day 15 (P = .03).

20 Nasal lavage fluid levels of interleukin (IL)-6, tumor n

21 and measured the total leukocyte content of nasal lavage fluid obtained from 10 min to 4 h after the

22 cell differential counts were evaluated from nasal lavage fluid obtained from wild-type and IL-17A-de

23 nts with acute bronchiolitis was elevated in nasal lavage fluid on both Days 1-2 (p = 0.014) and Days

24 ning cytokine and chemokine levels in serial nasal lavage fluid samples from 15 volunteers experiment

25 higher level of NTHI 2019 per milliliter of nasal lavage fluid than chinchillas colonized with predo

26 The percentage of leukocytes observed in nasal lavage fluid was significantly increased 12, 24, 4

27 antibodies was measured in tissue extracts, nasal lavage fluid, and sera by using multiplex bead arr

28 IL-8, IFN-alpha, TGF-beta, and TNF-alpha in nasal lavage fluid, plasma, and serum obtained serially

29 leukotriene (cysLT) levels were measured in nasal lavage fluid.

30 y response (measured by the albumin level in nasal-lavage fluid) to nasal provocation.

31 We sought to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients wi

32 e assessed, the inflammatory cell content in nasal lavage fluids estimated, and the activation patter

33 tase protein was also elevated in NPs and in nasal lavage fluids from patients with CRSwNP.

34 ergen significantly increased NGF protein in nasal lavage fluids of subjects with allergic rhinitis,

35 We found that IL-6 and IFN-alpha levels in nasal lavage fluids peaked early (day 2) and correlated

36 ores, and concentrations of IL-6 and IL-8 in nasal lavage fluids were compared between treatment grou

37 The concentrations of NGF protein in nasal lavage fluids were not affected by provocation wit

38 er baseline concentrations of NGF protein in nasal lavage fluids, compared with control subjects.

39 Nasal lavage for flow cytometry and nasal swabs for vira

40 ers kept a daily diary of symptoms and had a nasal lavage for polymerase chain reaction once each wee

41 We collected nasal lavages from ragweed sensitized subjects at differ

42 eron-gamma, and RANTES were detected only in nasal lavages from two asthmatic subjects, who had the m

43 t during RSV bronchiolitis reduces serum and nasal lavage IL-8 levels and the occurrence of postbronc

44 te immune profile characterized by increased nasal lavage monocyte chemotactic protein-3, IFN-alpha2,

45 Nasal lavage (NAL) fluid samples were obtained from five

46 Biomarkers in nasal lavage (NL) fluid may be useful in determining the

47 Nasal lavage (NL) samples from 20 subjects who were expo

48 d serum on randomization, day 8, and day 15 (nasal lavage only).

49 specific IgE, IgG, and IgG4 were measured in nasal lavage samples at the conclusion of the sensitizat

50 se in prion seeding activity was observed in nasal lavage samples following nasotoxic treatment.

51 ct illness at 3-month intervals and analyzed nasal lavage samples for respiratory tract viruses at th

52 Nasal lavage samples from building occupants with upper

53 s of 105 DNA samples extracted from archived nasal lavage samples from high-risk infants/young childr

54 2 x 10(3) PFU, genomic diversity present in nasal lavage samples increased from 1 to 3 days postinfe

55 ofiled global patterns of gene expression in nasal lavage samples obtained during an acute, moderate,

56 both OMP and prion proteins were present in nasal lavage samples.

57 ch is specific for ORNs, was not detected in nasal lavage samples.

58 Nasal lavage specimens were assayed for interferon-gamma

59 leukocyte or interleukin-8 concentrations in nasal-lavage specimens, or on quantitative-virus titer.

60 analyze the presence of virus in cells from nasal lavage, sputum, bronchoalveolar lavage, bronchial

61 V species and types were determined in 1,445 nasal lavages that were prospectively collected from 209

62 Nasal lavage was performed for moderate to severe respir

63 sal biopsies, and Th1 and Th2 cytokines from nasal lavage were assessed in subjects with severe PAR (

64 arated by at least 3 wk; bronchoalveolar and nasal lavages were performed on three occasions: immedia

65 of rats in which the ORNs were destroyed by nasal lavage with ZnSO(4), D2-like radioligand binding w